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1 xonuclease to remove mispaired 3' bases in a primer extension assay.
2 rapid amplification of cDNA ends (RACE) and primer extension assay.
3 ficial array could be quantified by a simple primer extension assay.
4 avage and to highly visible stop points in a primer extension assay.
5 site was identified by RNase protection and primer extension assay.
6 cific ligation assay and a single nucleotide primer extension assay.
7 cleavage were detected and analyzed using a primer-extension assay.
8 at least in the context of reporter gene and primer extension assays.
9 of the fxbA promoter that was identified in primer extension assays.
10 substrate for nucleic acid hybridization and primer extension assays.
11 -dependent toxT transcription by time course primer extension assays.
12 sults were compared with those of individual primer extension assays.
15 iptional start point (tsp) was identified by primer extension assay and a rapid amplification of cDNA
16 t of rRNA depurination in vivo using a novel primer extension assay and show that the temporal patter
19 osition 249, exhibits a mutator phenotype in primer extension assays and in the herpes simplex virus-
20 eir interactions using primase, helicase and primer extension assays, and a 'stripped down' reconstit
21 te that the purified O-ribosomes are pure by primer extension assays, and structurally homogenous by
24 e structure and function of mt-tRNA(Asp) The primer extension assay demonstrated that the m.7551A > G
25 of plasmid pIJ101 and, employing an in vitro primer extension assay, determined that the modification
26 ive allele-specific RT-PCR single nucleotide primer extension assays developed for two imprinted gene
29 ition, promoter consensus binding search and primer extension assay helped us to identify a new sigma
35 itatively, a strand bias was observed in the primer extension assay, in that polymerase synthesis ter
39 The results of ribonuclease protection and primer extension assays indicated that Bmp2 transcriptio
41 prtF and rofA transcripts by S1 nuclease and primer extension assays indicated that the same promoter
43 ensitive and highly reproducible multiplexed primer extension assay is described for quantitative mut
46 cipitated with HBV core antibody; and (iv) a primer extension assay maps the 5' end of the minus stra
47 on of Western blotting and a novel multiplex primer extension assay (MPEA), we showed that, although
59 (selective 2'-hydroxyl acylation analysed by primer extension) assays show that part of the regulated
61 varying the order of reagent addition in the primer extension assay showed no distinct differences in
63 ng site was near dsbG, Northern blotting and primer extension assays showed that OxyR binding to the
65 on mutants of LAP1 by in vitro transcription-primer extension assays showed that upstream elements in
66 ata from mRNA decay studies and quantitative primer extension assays support a model in which bound C
67 nt inhibitors of human telomerase by using a primer extension assay that does not use PCR-based ampli
72 y (ZIBS assay); (ii) an S1 nuclease cleavage-primer extension assay to map B-Z junctions; and (iii) a
74 e seven nucleotides were analyzed by a novel primer extension assay using a mixture of one dNTP compl
77 native substrate and RNA-chain terminator in primer-extension assays using a surrogate respiratory sy
78 etous yeast species, and the allele-specific primer extension assay was designed to identify a total
86 omatin immunoprecipitation-single-nucleotide primer extension assays, we measured the chromatin compo
90 nged this notion by presenting evidence from primer extension assays which appeared to indicate that
92 eloped an SNP genotyping method based on the primer extension assay with fluorescence quenching as th
93 This procedure combines the versatility of a primer extension assay with nucleotide-level resolution,
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