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1 ie, atypical teratoid/rhabdoid tumor and CNS primitive neuroectodermal tumor).
2 in's tumor of the chest wall, and peripheral primitive neuroectodermal tumor.
3 imilar to the neuroblastic rosettes in human primitive neuroectodermal tumors.
4 ver, it is also thought to be etiological in primitive neuroectodermal tumors.
5 mosomal translocation in Ewing's sarcoma and primitive neuroectodermal tumors.
6 2 with pancreaticoblastomas and 1 of 10 with primitive neuroectodermal tumors.
7 ld be a common mechanism in the causation of primitive neuroectodermal tumors.
9 e beginning of treatment: medulloblastoma or primitive neuroectodermal tumors, 57.8; germ cell tumors
10 own to be involved in Ewing sarcoma, related primitive neuroectodermal tumors and desmoplastic small
11 ial response (PR) in two patients with Ewing/primitive neuroectodermal tumors and included one PR and
12 rs (16 osteosarcomas, one Ewing sarcoma, one primitive neuroectodermal tumor, and one desmoplastic sm
13 velopment of gliomas, central nervous system primitive neuroectodermal tumors, and atypical teratoid/
14 sk medulloblastomas (MBs) and supratentorial primitive neuroectodermal tumors, and we report the outc
15 hich comprise Ewing's sarcoma and peripheral primitive neuroectodermal tumors, are highly aggressive
16 umors with multilayered rosettes (ETMRs) are primitive neuroectodermal tumors arising in infants.
17 d unpublished GEM models of medulloblastoma, primitive neuroectodermal tumor, astrocytoma, oligodendr
18 ), undifferentiated sarcoma, and soft tissue primitive neuroectodermal tumor at all sites except para
21 ety of substrates, in contrast to cells from primitive neuroectodermal tumors cells (n=6), which only
22 had low-grade glioma, 32 had medulloblastoma/primitive neuroectodermal tumor/embryonal tumor, 17 had
24 ve outcomes of patients with Ewing's sarcoma/primitive neuroectodermal tumor (ES/PNET) metastatic to
27 I-1, the hallmark of the Ewing's sarcoma and primitive neuroectodermal tumor family, encodes a fusion
28 ted to a single cell type (two patients with primitive neuroectodermal tumors, five with undifferenti
30 LI1 fusion gene found in Ewing's sarcoma and primitive neuroectodermal tumor, is able to transform ce
31 roglial-containing tumors, brain lymphoma or primitive neuroectodermal tumor make use of systemic adm
33 ients had foci of yolk sac tumor (n = 21) or primitive neuroectodermal tumor (n = 2), whereas 50 had
34 (n = 6), ependymoma (n = 8), medulloblastoma/primitive neuroectodermal tumor (n = 4), glioblastoma mu
35 nd those without a bone sarcoma (ie, neither primitive neuroectodermal tumor nor osteosarcoma) (HR fo
37 ffective in patients with Ewing's sarcoma or primitive neuroectodermal tumor of bone who had a relaps
38 patients with nonmetastatic Ewing's sarcoma, primitive neuroectodermal tumor of bone, or primitive sa
39 0 years old or younger with Ewing's sarcoma, primitive neuroectodermal tumor of bone, or primitive sa
41 Patients < or = 30 years with Ewing sarcoma, primitive neuroectodermal tumor or primitive sarcoma of
42 ) in children treated for medulloblastoma or primitive neuroectodermal tumor (PNET) and document the
43 all cases of Ewing's Sarcoma and peripheral Primitive Neuroectodermal Tumor (PNET) are associated wi
45 of FDG PET imaging of brain involvement with primitive neuroectodermal tumor (PNET) demonstrated mild
46 a (NB) and the Ewing sarcoma (ES)/peripheral primitive neuroectodermal tumor (PNET) family are pediat
47 patients with metastatic Ewing's sarcoma or primitive neuroectodermal tumor (PNET) of bone were ente
48 s of medulloblastoma, central nervous system primitive neuroectodermal tumor (PNET), and astrocytoma
52 one [osteogenic (OS) and Ewing's (ES) and/or primitive neuroectodermal tumor (PNET)] sarcoma, treated
53 astrocytoma and 158 children diagnosed with primitive neuroectodermal tumors (PNET) in the United St
54 moter developed bilateral retinoblastoma and primitive neuroectodermal tumors (PNET) of the midbrain.
55 ly (ESFT), such as Ewing's sarcoma (EWS) and primitive neuroectodermal tumors (PNET), are highly aggr
56 transgenic mice to the human medulloblastoma/primitive neuroectodermal tumor (PNETs) in location, his
60 enetic lesions in medulloblastomas and other primitive neuroectodermal tumors (PNETs) of the childhoo
61 E SCUTE 1 (HASH1) in cerebellar and cerebral primitive neuroectodermal tumors (PNETs), gliomas, and c
64 ith high-grade glioma and in two of six with primitive neuroectodermal tumors (PNETs)/ medulloblastom
65 response (PR) in one patient with peripheral primitive neuroectodermal tumor (PPNET), and a minimal r
67 , classically Ewing's sarcoma and peripheral primitive neuroectodermal tumor, share a common class of
68 lastoma, whereas experimental supratentorial primitive neuroectodermal tumors (sPNET) correspond to a
69 valuation of a panel of human supratentorial primitive neuroectodermal tumors (sPNET) showed low acti
70 o develop a new approach to the treatment of primitive neuroectodermal tumors we evaluated the effect
71 nd brain stem NSCs generated medulloblastoma/primitive neuroectodermal tumors, whereas forebrain NSCs
72 d transgenic animals that develop cerebellar primitive neuroectodermal tumors which model human medul
73 Cs were transformed with N-Myc, we generated primitive neuroectodermal tumors with divergent differen
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