ライフサイエンスコーパス: pro-

1 Habituation to LOW PRO (0.7 g . d(-1)) compared with HIGH PRO (1.5 g . d(-1

2 o LOW PRO (0.7 g . d(-1)) compared with HIGH PRO (1.5 g . d(-1)) augments the postprandial availabili

3 = 0.25). kg(-1) . d(-1)) compared with HIGH PRO (1.5 g . kg(-1) . d(-1)) augments the postprandial a

4 hese labeled substrates also showed that the pro-(3S) proton exchanges with protons from the bulk med

5 od was greater after LOW PRO than after HIGH PRO (61% +/- 1% compared with 56% +/- 2%, respectively;

6 cently, the tetrapetide N-acetyl-Ser-Asp-Lys-Pro (Ac-SDKP) has emerged as a potent antifibrotic agent

7 odilator bradykinin and N-acetyl-Ser-Asp-Lys-Pro (Ac-SDKP), a physiological modulator of hematopoiesi

8 iewed lectures through Adobe Acrobat Connect Pro (Adobe Systems, San Jose, CA), and completed online

9 urrence of a cardiac event compared with Pro/Pro + Ala/Pro genotypes in multivariate analysis (odds r

10 nges between assessments in the clinical and PRO "anchor" variables were classified as improved, stab

11 Functional assays suggest that both pro- and active MMP-9 trigger alpha-smooth muscle actin

12 mmune responses by differentially regulating pro- and anti- inflammatory cytokine production in innat

13 entially modulate the expression of selected pro- and anti- inflammatory mediators such as IL-6 and I

14 ivity via B face, proposing a model that the pro- and anti-amyloidogenic activities of SAP are not mu

15 previous in vitro studies have reported both pro- and anti-angiogenic effects of Slits.

16 lated to be regulated by the balance between pro- and anti-angiogenic factors.

17 vascular endothelial growth factor signals, pro- and anti-angiogenic inflammatory chemokine signals,

18 owever, SMAD1/5/8 signalling results in both pro- and anti-angiogenic outputs, highlighting a poor un

19 doglin modulates the crucial balance between pro- and anti-angiogenic signaling by activin receptor-l

20 regulated through complex crosstalk between pro- and anti-angiogenic signals.

21 al exon to produce two families of isoforms, pro- and anti-angiogenic, only the former of which is up

22 flow cytometry to analyze the expression of pro- and anti-apoptotic Bcl-2 family members in T cells

23 Furthermore, by combining different pro- and anti-apoptotic Bcl-2 pairings together with CRI

24 poptotic proteins, WM samples expressed both pro- and anti-apoptotic Bcl-2 proteins at low levels sim

25 , called mito-priming, uses co-expression of pro- and anti-apoptotic Bcl-2 proteins to engineer Bcl-2

26 The expression of the pro- and anti-apoptotic genes bcl-2 and bax, respectivel

27 GSPE also modulated pro- and anti-apoptotic markers in the pancreas of rats

28 Changes in the equilibrium of pro- and anti-apoptotic members of the B-cell lymphoma-2

29 Pro- and anti-apoptotic members of this family keep each

30 Analysis of the pro- and anti-apoptotic pathways indicated no significan

31 nd division-influenced expression of several pro- and anti-apoptotic proteins within CFSE-labeled cul

32 nderstand some contradictory results on both pro- and anti-arrhythmic effects of blocking IKur.

33 nfiltration and modulating the balance among pro- and anti-autoimmune CD4(+) T-cell subsets.

34 cess as it is at the cross-roads of both the pro- and anti-coagulant pathways.

35 rons including cidABC and alsSD that display pro- and anti-death functions, respectively.

36 Our method facilitates discovery of novel pro- and anti-fibrotic agents in 384-well plate format a

37 further characterize the balance of systemic pro- and anti-inflammation early after burn and inhalati

38 to a phenotype that was intermediate between pro- and anti-inflammatory activation.

39 We infer that pro- and anti-inflammatory activities of beta-arrestin2

40 In various hepatotoxic models, both pro- and anti-inflammatory activities of recruited monoc

41 leukotriene B4, thus LTA4H exhibits opposing pro- and anti-inflammatory activities.

42 ogenesis and function, as well as release of pro- and anti-inflammatory adipocytokines.

43 Diet is a major source of pro- and anti-inflammatory bioactive compounds.

44 immune system and affect the balance between pro- and anti-inflammatory cells.

45 y mechanisms leading to an imbalance between pro- and anti-inflammatory components of the immune syst

46 id PGE(2), suggesting that interplay between pro- and anti-inflammatory compounds may be important in

47 ammatory responses through the regulation of pro- and anti-inflammatory cytokine production, (ii) the

48 ppressive, they displayed unique patterns of pro- and anti-inflammatory cytokine production, differen

49 Immunization with Ldp27(-/-) induced both pro- and anti-inflammatory cytokine responses and activa

50 Expression of pro- and anti-inflammatory cytokines (interleukin [IL]-1

51 phology and function in hypertension, and on pro- and anti-inflammatory cytokines (PICs and AIC) and

52 rs in the untreated CLP group, comprising 14 pro- and anti-inflammatory cytokines and 8 chemokines, g

53 ing threshold determines whether PRR-induced pro- and anti-inflammatory cytokines are reciprocally re

54 diatric acute respiratory distress syndrome, pro- and anti-inflammatory cytokines are strongly associ

55 in behaviour, and the changes in circulating pro- and anti-inflammatory cytokines exhibited in this m

56 Pro- and anti-inflammatory cytokines gene expression was

57 potency with simultaneous production of both pro- and anti-inflammatory cytokines hampers its potenti

58 d the circulating levels of a large panel of pro- and anti-inflammatory cytokines in asymptomatic, in

59 on activation; additionally, B cells produce pro- and anti-inflammatory cytokines in response to cert

60 gether, our data suggest that the balance of pro- and anti-inflammatory cytokines that determines the

61 is threshold; consequently, both PRR-induced pro- and anti-inflammatory cytokines were decreased.

62 ormed by flow cytometry, and serum levels of pro- and anti-inflammatory cytokines were determined by

63 Bronchoalveolar lavage levels of pro- and anti-inflammatory cytokines were not elevated i

64 While different profiles of pro- and anti-inflammatory cytokines were observed betwe

65 we hypothesized that patients with elevated pro- and anti-inflammatory cytokines would have higher m

66 a comprehensive leukocytosis, elevated serum pro- and anti-inflammatory cytokines, and evidence of in

67 /-) mice produced significant levels of both pro- and anti-inflammatory cytokines, including IL-1beta

68 The release of pro- and anti-inflammatory cytokines, including interleu

69 nogen activator inhibitor) or the release of pro- and anti-inflammatory cytokines.

70 surface marker expression and production of pro- and anti-inflammatory cytokines.

71 od correlated not only with higher levels of pro- and anti-inflammatory cytokines/chemokines (CCL2, I

72 injury, expression of adhesion molecules and pro- and anti-inflammatory cytokines/chemokines, microgl

73 The balance between pro- and anti-inflammatory effects could determine the s

74 mitter known to trigger pathways involved in pro- and anti-inflammatory effects in a dose-dependent m

75 To characterize HSL-C12's pro- and anti-inflammatory effects in host cells, we mea

76 f much debate, presumably due to the complex pro- and anti-inflammatory effects of this cytokine.

77 The balance between the pro- and anti-inflammatory effects of VSMCs and their ex

78 The sympathetic nervous system also has both pro- and anti-inflammatory effects on immune tissues and

79 The SNS has both pro- and anti-inflammatory effects on immunity, confound

80 criptional programs leading alternatively to pro- and anti-inflammatory effects.

81 ious reports showed that HSL-C12 causes both pro- and anti-inflammatory effects.

82 rachidonoyl-ethanolamine mediate an array of pro- and anti-inflammatory effects.

83 L-33/ST2 is a unique cytokine with potential pro- and anti-inflammatory effects.

84 ceptor-specific and combinatorial control of pro- and anti-inflammatory eicosanoid biosynthesis, and

85 r the genetically determined balance between pro- and anti-inflammatory eicosanoids in regulating TNF

86 (LTA4H) locus, which controls the balance of pro- and anti-inflammatory eicosanoids, reveals two dist

87 erial keratitis and is critical to balancing pro- and anti-inflammatory events, resulting in better d

88 ogenic T cells, zymosan induced a mixture of pro- and anti-inflammatory factors and Tregs, both in vi

89 This study demonstrated that pro- and anti-inflammatory factors could be resolved and

90 within the epithelium and a balance between pro- and anti-inflammatory factors in the mucosa.

91 mmune responses, including IL-27 that exerts pro- and anti-inflammatory functions.

92 s mucosal barrier function and expression of pro- and anti-inflammatory genes by intestinal epithelia

93 ey play a key role in the regulation of both pro- and anti-inflammatory immune responses.

94 difference in the macropinocytic ability of pro- and anti-inflammatory macrophages that correlates w

95 detailed analysis of the effect of NFAT5 in pro- and anti-inflammatory macrophages uncovered its abi

96 is maintained through an adequate balance of pro- and anti-inflammatory macrophages, we assessed the

97 rrelated with the macropinocytic activity of pro- and anti-inflammatory macrophages.

98 We investigated region-specific changes in pro- and anti-inflammatory markers in the mesocorticolim

99 The delicate balance between pro- and anti-inflammatory mechanisms, essential for gut

100 immune system, affecting the balance between pro- and anti-inflammatory mechanisms.

101 but mechanistically distinct, regulation of pro- and anti-inflammatory mediators in TR APOE4/4 murin

102 ion of miR-155 in response to the respective pro- and anti-inflammatory mediators LPS and IL-10.

103 a plethora of bioactive molecules including pro- and anti-inflammatory mediators.

104 sion profile of several genes encoding major pro- and anti-inflammatory mediators.

105 homeostasis that can rapidly switch between pro- and anti-inflammatory or regulatory modes to respon

106 hree novel associations implicating genes in pro- and anti-inflammatory pathways in Crohn's disease;

107 xygenase (5-LO) metabolism can activate both pro- and anti-inflammatory pathways, but their role in w

108 signalosome level to Mal/MyD88 and TRAM/TRIF pro- and anti-inflammatory pathways.

109 a second-hit insult, or an imbalance between pro- and anti-inflammatory pathways.

110 ses to UV light, homeostasis associated with pro- and anti-inflammatory processes, and genomic stabil

111 IL-6 has context-dependent pro- and anti-inflammatory properties and is now regarde

112 IL6 is a pleiotropic cytokine with both pro- and anti-inflammatory properties, which acts direct

113 is a cytokine with dual function displaying pro- and anti-inflammatory properties.

114 lammatory and autoimmune disorders with both pro- and anti-inflammatory properties.

115 ine in immune regulation, orchestrating both pro- and anti-inflammatory reactions.

116 or H. pylori MV in the stimulation of innate pro- and anti-inflammatory responses and in the suppress

117 the human immune system requires controlled pro- and anti-inflammatory responses for host defence ag

118 tablishment of a precise balance between the pro- and anti-inflammatory responses is critical to guar

119 ir dysfunction can, thus, manifest in either pro- and anti-inflammatory responses.

120 al structures necessitates a fine balance of pro- and anti-inflammatory responses; well-timed, approp

121 One of the key findings includes both pro- and anti-inflammatory roles of GR, and a future cha

122 stinct immune cell populations that can play pro- and anti-inflammatory roles, and thus the compositi

123 disintegrin and metalloprotease 17) controls pro- and anti-inflammatory signaling events by promoting

124 ceptor clustering; it also provides clues to pro- and anti-inflammatory signaling pathways branching

125 Although the balance between systemic pro- and anti-inflammatory signals is crucial to TB dise

126 city strain PR8/H1N1 resulted in overlapping pro- and anti-inflammatory states.

127 tions, as well as regulatory cytokines, both pro- and anti-inflammatory, that are not typically produ

128 a micturition control center that integrates pro- and anti-micturition cues.

129 n areas that are capable of carrying diverse pro- and anti-micturition signals, and whose activity mo

130 r genome sequencing efforts, suggesting both pro- and anti-neoplastic roles.

131 These pro- and anti-nociceptive effects were blocked by co-inj

132 is evidence that endocannabinoids have both pro- and anti-nociceptive effects.

133 onditions, but preclinical studies show both pro- and anti-nociceptive effects.

134 nal disruption of regulatory DNA to decouple pro- and anti-oncogenic functions of a dominant transcri

135 Ras-family small GTPase RAB25 can exert both pro- and anti-oncogenic functions.

136 Our results indicate that the pro- and anti-proliferative effects of ROS can be indepe

137 This creates an imbalance between pro- and anti-proteolytic and apoptotic factors and may

138 ucial for successful deployment of synthetic pro- and anti-QS strategies.

139 Pro- and anti-quorum-sensing molecules can be covalently

140 During HR, BLM has both pro- and anti-recombinogenic activities, either of which

141 Sgs1 has both pro- and anti-recombinogenic roles, and therefore its ac

142 OPBP1(Dpb11) provides binding sites for both pro- and anti-resection factors at DSBs, providing insig

143 macaque monkeys performing memory-guided and pro- and anti-saccades.

144 sal frequency, affect selection pressures on pro- and anti-social punishment.

145 , and bacterial DNA, shifting the balance of pro- and anti-survival signals toward apoptosis via indu

146 or rejection, regulating the balance between pro- and anti-tumor immunity.

147 ble of differentiating macrophages with both pro- and anti-tumor properties.

148 ors (TLRs) cluster in lipid rafts and induce pro- and anti-tumor responses.

149 emains controversial, with evidence for both pro- and anti-tumor roles.

150 he individual immune components that possess pro- and anti-tumorigenic functions in individual cancer

151 kinase C alpha (PKCalpha) can activate both pro- and anti-tumorigenic signaling depending upon cellu

152 Here we review the pro- and anti-viral effects of methyl-6-adenosine in dis

153 geneity, specifically in the distribution of pro- and antiadhesive sialic acids that protect underlyi

154 le organism-based environmental screening of pro- and antiandrogens.

155 tatic and host defense reactions and deliver pro- and antiangiogenic factors throughout the vascular

156 Given that VEGF can elicit both pro- and antiangiogenic responses depending upon the bal

157 le cell-based assay that responds to complex pro- and antiangiogenic soluble factors with an in vitro

158 and have implications for the design of both pro- and antiangiogenic therapies.

159 e delicate balance in the relative levels of pro- and antiangiogenic VEGF isoforms can result in eith

160 ng, oligomerization, and permeabilization by pro- and antiapoptotic Bcl-2 members at the single-vesic

161 Several pro- and antiapoptotic factors were tested for different

162 Similarly, pro- and antiapoptotic mammalian regulatory elements are

163 y of IFNs partly by affecting the balance of pro- and antiapoptotic members of Bcl-2 family proteins,

164 cells (DCs) is determined by the balance of pro- and antiapoptotic proteins.

165 osis, including via its interaction with the pro- and antiapoptotic proteins.

166 NAF-1 binds to both the pro- and antiapoptotic regions (BH3 and BH4) of Bcl-2, a

167 ar that the immune system also has important pro- and antiatherogenic functions.

168 s and that disruption of the balance between pro- and antiatherogenic immune cell subsets may trigger

169 is review, we discuss the novel concept that pro- and antiatherogenic immune responses toward unknown

170 In this REVIEW, we discuss the pro- and anticarcinogenic role of the microbiota, as wel

171 athways are identified that mediate both the pro- and anticoagulant activities of thrombin.

172 Standard coagulation studies as well as pro- and anticoagulant clotting factors were measured.

173 rs of hemostasis by expressing and releasing pro- and anticoagulant mediators into the circulation.

174 plastin time (PTT), and lower levels of both pro- and anticoagulants up to 24 hours.

175 overall response depending on the balance of pro- and antifibrillatory contributions.

176 merase Pin1 modulates the production of many pro- and antifibrogenic cytokines and ECM.

177 WT mice have similar lung levels of several pro- and antifibrotic mediators (TGF-beta, IL-13, JE, an

178 Our research is focused on identifying pro- and antiflaviviral miRNAs as a means of characteriz

179 e-week mortality was associated with greater pro- and antiinflammatory alterations of the innate immu

180 showed that dairy food consumption improved pro- and antiinflammatory biomarker concentrations compa

181 imental properties appeared favorable in the pro- and antiinflammatory cytokine balance, 1,25-dihydro

182 Plasma pro- and antiinflammatory cytokine profiles were perform

183 IgG molecules exert both pro- and antiinflammatory effector functions based on th

184 provide strong evidence that, by activating pro- and antiinflammatory mediators, beta-catenin signal

185 initial stages of inflammation by balancing pro- and antiinflammatory signals.

186 edox state via transcriptional regulation of pro- and antioxidant enzymes.

187 culature is tightly regulated by a wealth of pro- and antioxidant systems that orchestrate region-spe

188 stance can be modulated by administration of pro- and antiquorum-sensing compounds.

189 liver regeneration, and they define specific pro- and antiregenerative molecular targets whose regene

190 ripheral nervous system (PNS) and CNS is the pro- and antiregenerative responses of their glial cell

191 Thus, pro- and antirelapse circuitry remodeling is induced in

192 rom an experiment with three mixed blocks of pro- and antisaccade trials.

193 ide a formal likelihood function of actions (pro- and antisaccades) and reaction times based on previ

194 biology, focusing on relevant TLR-dependent pro- and antitumor pathways, and discuss clinical applic

195 microenvironment is complex, containing both pro- and antitumorigenic elements, and remains to be ful

196 The reactions exhibited both pro- and antitumorigenic potential and primarily corresp

197 that several MMPs including MMP-9 exert both pro- and antitumorigenic properties.

198 synergistic regulation of the expression of pro- and antitumorigenic target genes.

199 thways downstream of TNFR1 and -2 with known pro- and antiviral effects.

200 P1 is efficiently secreted because of stable pro- and catalytic domain interactions.

201 resident and homing immune cells along with pro- and counter-inflammatory cytokines.

202 ith significant biological functions in both pro- and eukaryotes.

203 ttern of proteins, recombinantly produced in pro- and eukaryotic hosts.

204 are identified, distinct differences between pro- and eukaryotic phosphosignalling systems become app

205 ing has been demonstrated in a wide range of pro- and eukaryotic species, attesting to its universal

206 e novo protein synthesis, and the release of pro- and mature IL-1beta from infected primary monocytes

207 s and report that in Drosophila melanogaster pro- and mature neurotrophins are capable of inducing de

208 n of a full-length polypeptide into separate pro- and mature-domains.

209 ot prevent pro-domain processing between the pro- and metalloprotease domain, but nevertheless, cause

210 activate RAG1 and RAG2 gene transcription in pro- and pre-B cells.

211 und that mTOR is highly activated during the pro- and pre-B stages of mouse B cell development.

212 ven, Belgium, and GBG 29/BIG 02-03) compiled pro- and retrospectively between 2003 and 2011 was compa

213 oprotein convertase Furin (RXXR) between the pro- and the catalytic domain.

214 commonly activated after seizure and produce pro- and/or anti-inflammatory factors.

215 The Bax/Bcl-2 (pro-/anti-apoptotic) genes expression in the islets was

216 Pro-/anti-inflammatory (IL-6/IL-10) cytokine secretion r

217 epithelial/stem cells, the regulation of the pro-/anti-inflammatory cytokine balance.

218 by reappearance of the visual landmark or a pro-/anti-reach instruction), the parietofrontal network

219 ization of ataxin-1 functional complexes and pro-/antiapoptotic and inflammatory pathways.

220 The analysis of pro-/antioxidant balance in rat blood revealed a mild pr

221 IgH V(D)J assembly occurs in progenitor (pro-) B cells followed by that of IgL in precursor (pre-

222 poptosis and both differentiation), and both pro- (Bad, Bax) and anti-apoptotic (Bcl-2, Bcl-xL) facto

223 e Ile-Gln-Ala (beta-CN f187-189) and Val-Glu-Pro (beta-CN f116-118) having ACE IC50 values of 32.9+/-

224 es and proteases, abrogated the formation of pro- but also anti-inflammatory eicosanoids, and restore

225 e, through changes in expression of critical pro-, but not anti-, apoptotic genes.

226 sue biopsies to measure the total (CD68(+)), pro- (CD14(+) = M1), and anti- (CD206(+) = M2) inflammat

227 ding motifs containing the signature Asp-Phe-Pro (DFP) tripeptide.

228 bone marrow, all B cell progenitors-from pre-pro-/early pro-B cells to immature B cells-were dramatic

229 Ile-Pro-Pro (IPP), Leu-Pro-Pro (LPP) and Phe-Pro (FP) were separated on a C18-column coupled to UV/VI

230 etectable in the effluent unless Gly-Pro-Arg-Pro (GPRP) was added to block fibrin polymerization.

231 ihydroxyphenylalanine, Arg > Val > Lys, Tyr, Pro > hydroxyproline > alpha-aminobutyric acid > Gln, Th

232 asic residues or Trp in the X2 position, and Pro >> Ala > Trp in the X3 position.

233 ng additional hydrogen-bonding capacity, the Pro-->2-Hyp conversion alters the active site and enhanc

234 coding variant (rs2233290) at position 151 (Pro-->Ala) in the TNFAIP3-interacting protein 1, TNIP1,

235 FK binding to WT GST-TRAF6 compared with the Pro --> Ala-substituted peptide.

236 Icare PRO (ICP) is a new Rebound tonometer that is able to mea

237 nd depression surveys and provided blood for pro- (IL-1beta, IL-6, IL-8, TNF-a, IFN-gamma) and anti-i

238 ere compounds that react preferentially with pro- (inactive) caspases.

239 ls (ENaC) via oxidant signaling to promote a pro- injury environment.

240 bsylated peptides Val-Pro-Pro (VPP), Ile-Pro-Pro (IPP), Leu-Pro-Pro (LPP) and Phe-Pro (FP) were separ

241 al-Pro-Pro (VPP), Ile-Pro-Pro (IPP), Leu-Pro-Pro (LPP) and Phe-Pro (FP) were separated on a C18-colum

242 ence for the trans-conformation in the order Pro < Hyp < [alpha-(1,4)GlcNAc]Hyp.

243 This included the shift from a pro ("M1") to an anti-inflammatory ("M2") phenotypic sta

244 conclusions supporting Netrin-4 as either a pro- or an anti-angiogenic factor.

245 KKbeta have been reported to modulate either pro- or anti- inflammatory programs, which may be specif

246 er the question whether netrin-4 acts either pro- or anti-angiogenic, angiogenesis in the retina was

247 ite dysregulation, leading to the release of pro- or anti-apoptotic factors which mediate cell surviv

248 nalling pathways or of altered expression of pro- or anti-apoptotic proteins can thus be compared.

249 tein E receptor 2 (apoER2) results in either pro- or anti-atherogenic effects depending on the ligand

250 The role of IL-6 as a pro- or anti-inflammatory cytokine is still unclear.

251 GM-MO) or M-CSF (M-MO), which do not release pro- or anti-inflammatory cytokines unless subjected to

252 vo without altering the examined gallbladder pro- or anti-inflammatory cytokines.

253 ds to differentiation of CD4(+) T cells with pro- or anti-inflammatory effector cell functions.

254 erest based on its ability to possess either pro- or anti-inflammatory effects mediated through p35-p

255 tes to differentiate and to be primed toward pro- or anti-inflammatory Mvarphis upon culture with GM-

256 gm, in which MSCs can be polarized towards a pro- or anti-inflammatory phenotype depending on the Tol

257 ic nervous system may partake in bifurcating pro- or anti-inflammatory responses to microbes.

258 PGE(2) plays complex pro- or anti-inflammatory roles in facilitating mucosal

259 bolized to reactive products that can act as pro- or anti-inflammatory signaling mediators.

260 t only in a subset of patients, represents a pro- or anti-inflammatory state, its association with pr

261 c-Jun are increased in macrophages following pro- or anti-inflammatory stimulations.

262 First, by classifying aging-related genes as pro- or anti-longevity, we define distinct pathways and

263 and activate distinct pathways that produce pro- or anti-mitogenic actions.

264 s of human hippocampal biology and levels of pro- or anti-neurogenic stimuli, weigh whether these cor

265 gastric juice has the potential to act as a pro- or anti-oxidative medium.

266 employing lepb-linked sequences upstream of pro- or anti-regenerative factors controlled the efficac

267 on to endothelial cells and to induce either pro- or antiangiogenic signaling.

268 ure supports the idea that Akt can be either pro- or antiangiogenic, possibly due to compensation by

269 roenvironment seem to define whether PAF has pro- or anticarcinogenic effects.

270 ast, a high-dose of MAGL inhibitors produces pro- or antidepressant effects on acute stress- or chron

271 itivity of the peripheral immune system to a pro- or antidepressant state, bone marrow (BM) chimeras

272 Recent studies have shown that pro- or antiinflammatory effector functions of IgG Abs a

273 rate due to the probability of a trial type (pro- or antisaccade) are best explained by faster or slo

274 n, and angiogenesis, resulting in an overall pro- or antitumoral effect.

275 anisms may regulate its capacity to act as a pro- or antiviral effector targeting viral DNA.

276 ets, and subsequently differentiated into M1 pro- or M2 anti-inflammatory macrophages on stimulation.

277 itivity to TNFalpha-induced apoptosis favors pro- over antiapoptotic program in cancer cells, and CDI

278 This rise was +0.9 +/- 2.3 mm Hg for Icare PRO (P = 0.01) and +0.7 +/- 1.8 mm Hg for Tono-Pen (P =

279 ading the neutrophil chemoattractant Pro-Gly-Pro (PGP) and rationalized that the failure of conventio

280 report that the matrikine acetylated Pro-Gly-Pro (PGP) stimulates vascular inflammation through activ

281 ombination step were largely demethylated in pro-, pre-, and mature B cells.

282 Retroviral transduction of Pax5-deficient pro-/pre-B cell lines with a doxycycline-inducible (TetO

283 MCPyV infection and transformation of pro-/pre-B cells are likely to induce the expression of

284 presses IL-7R/JAK/STAT signaling to restrict pro-/pre-B progenitor expansion and leukemia development

285 nfection was detected in the Arg/Arg vs. Arg/Pro + Pro/Pro model.

286 Duplications in a Pro-Thr-Ala-Pro (PTAP) motif in the p6 domain of Gag are frequently

287 A conserved late domain motif, Pro-Thr-Ala-Pro (PTAP), located in the p6 region of Gag (p6(Gag)), p

288 share high sequence identity and catalyze 4-pro-(R)-hydride transfer from NADPH to an electrophilic

289 dipeptides: Ala-Arg (AR), Arg-Ala (RA), Arg-Pro (RP), Arg-Glu (RE), and Glu-Arg (ER); and two non-ar

290 ve abstraction of the hydrogen atom from the pro-(S)-position.

291 nding proteins contain a Ser-any residue-Ile-Pro (SxIP) motif.

292 control group to 1.69, 1.71 and 1.63), and a pro- to an anti-inflammatory cytokine shift.

293 by avoiding pathogenesis requires regulated pro- to anti-inflammatory responses and the development

294 NOS-induced nuclear CLIC4 coincides with the pro- to anti-inflammatory transition of the cells becaus

295 DR5 downregulation and an inverted ratio of pro- to antiapoptotic molecules, both of which were reve

296 an thirty fold increase in the ratio of BDNF pro- to mature-domains in the brains of individuals with

297 andomly assigned clusters (1:1) with MapInfo Pro (version 11.0) to either the control or intervention

298 sepsis patients perhaps because of a greater pro- versus anti-inflammatory response.

299 In contrast to a simple race between pro- versus anti-response, our model incorporates a sens

300 The dabsylated peptides Val-Pro-Pro (VPP), Ile-Pro-Pro (IPP), Leu-Pro-Pro (LPP) and Phe-