ライフサイエンスコーパス: probing depth

1 hment level, changes in bone level/fill, and probing depth.

2 probing (BOP), oral hygiene, and periodontal probing depth.

3 sound for high-spatial resolution imaging of probing depths.

4 strating a change of >/=1 mm, and changes in probing depths.

5 rences of 0.28 mm (95% CI, 0.18 to 0.37) for probing depth, 0.25 mm (95% CI, 0.14 to 0.36) for clinic

6 Examinations included: 1) probing depth, 2) clinical attachment level, and 3) oral

7 before the surgeries and after 12 months: 1) probing depth; 2) relative clinical attachment level; 3)

8 ty (P = 0.001), number of teeth (P = 0.006), probing depth 4 to 6 mm (P = 0.016), bleeding on probing

9 rrelated positively with FGS and periodontal probing depth (all rho >0.33; P <0.005).

10 n = 720) and dental calculus and periodontal probing depth among young adults were assessed using ful

11 onal studies, investigating features such as probing depth and attachment loss, are needed for the ap

12 nation; and 5) clinical parameters including probing depth and bleeding on probing.

13 ials should be analyzed according to initial probing depth and characteristics of the treated sites,

14 h participant, clinical parameters including probing depth and clinical attachment level were measure

15 improvement in the parameters of periodontal probing depth and clinical attachment level, have been p

16 and 2-hour OGTT were positively related with probing depth and clinical attachment level; blood gluco

17 Probing depth and clinical attachment loss (AL) were rec

18 ectly correlated with clinical parameters of probing depth and clinical attachment loss (AL).

19 erformed on all dental elements to determine probing depth and gingival recession.

20 MD with CHX group showed higher reduction in probing depth and percentage of periodontal diseases sit

21 Probing depth and plaque, papilla bleeding, and hyperpla

22 ceded another measurement change, changes in probing depths and relative clinical attachment levels a

23 lasma glucose, plaque index, gingival index, probing depth, and attachment loss when compared with th

24 ated significantly with IFCC units, clinical probing depth, and attachment loss.

25 eters, such as bleeding index, plaque index, probing depth, and clinical attachment level (CAL), were

26 Plaque and gingival indices, probing depth, and clinical attachment level were measur

27 ingivitis, bleeding on probing, suppuration, probing depth, and clinical attachment level were measur

28 al parameters (plaque index, gingival index, probing depth, and clinical attachment level) were recor

29 al parameters, gingival index, plaque index, probing depth, and clinical attachment level, were recor

30 eters, such as gingival index, plaque index, probing depth, and clinical attachment level, were recor

31 Plaque index, gingival index, probing depth, and clinical attachment loss were measure

32 ated with the extent of bleeding on probing, probing depth, and clinical attachment loss.

33 giene index-simplified, gingival index, mean probing depth, and loss of attachment.

34 ing on probing, modified bleeding index, GI, probing depth, and radiographic bone loss) did not reach

35 oral hygiene and greater loss of attachment, probing depth, and recession compared with controls.

36 onstrate symptomatic inflammation, increased probing depth, and tooth loss likely attributable to the

37 aper points from multiple sites with >/=5 mm probing depth, and whole genomic DNA was extracted.

38 ing tooth loss, plaque and bleeding indexes, probing depths, and clinical attachment loss (AL).

39 ival index; 3) bleeding on probing (BOP); 4) probing depth; and 5) attachment loss (AL).

40 icantly associated with increased changes in probing depth at 21 d of biofilm overgrowth (P </= 0.05)

41 rameters (plaque index, bleeding on probing, probing depth, attachment loss, and marginal bone loss),

42 inations at baseline including assessment of probing depth, attachment loss, gingival index, and plaq

43 s of patients were included according to the probing depth, bleeding on probing, and clinical attachm

44 Probing depth, bleeding on probing, gingival index, and

45 n included periodontal attachment loss (AL), probing depth, bleeding on probing, plaque index (PI), a

46 mplete periodontal examination consisting of probing depth, bleeding on probing, tooth mobility, ging

47 significant correlations were found between probing depth, CAL measures, and indicators of OSAS seve

48 Trained examiners measured probing depth, clinical attachment level (CAL), and blee

49 Probing depth, clinical attachment level (CAL), and kera

50 REC, probing depth, clinical attachment level (CAL), and widt

51 Clinical parameters were analyzed, including probing depth, clinical attachment level (CAL), bleeding

52 Outcomes examined were recession depth, probing depth, clinical attachment level (CAL), height o

53 ex, gingival index, vertical recession (VR), probing depth, clinical attachment level (CAL), keratini

54 reased sites with bleeding on probing (BOP), probing depth, clinical attachment level (CAL), waist ci

55 shed after a full clinical examination using probing depth, clinical attachment level, and bleeding o

56 Clinical parameters including periodontal probing depth, clinical attachment level, and bleeding o

57 Parameters related to probing depth, clinical attachment level, and bone loss

58 dontal status was assessed by measurement of probing depth, clinical attachment level, and extent and

59 tal status was assessed by criteria based on probing depth, clinical attachment level, and extent and

60 tal status was assessed by criteria based on probing depth, clinical attachment level, and extent and

61 Probing depth, clinical attachment level, and gingival a

62 Recession height, probing depth, clinical attachment level, and keratinize

63 8-OHdG, MDA, probing depth, clinical attachment level, and percentage

64 ) samples and clinical parameters, including probing depth, clinical attachment level, bleeding on pr

65 Samples were obtained first, with probing depth, clinical attachment level, bleeding on pr

66 drimestral recalls, evaluating plaque index, probing depth, clinical attachment level, furcation invo

67 l-mouth periodontal status was determined by probing depth, clinical attachment level, gingival bleed

68 red at baseline, 3 months, and 6 months were probing depth, clinical attachment level, GR height, wid

69 elded significant positive correlations with probing depth, clinical attachment level, IL-1beta, and

70 Probing depth, clinical attachment level, plaque index,

71 dontal clinical parameters recorded included probing depth, clinical attachment level, plaque index,

72 hemoglobin (HbA1c) levels were measured, and probing depth, clinical attachment levels, and bleeding

73 rameters (plaque index, bleeding on probing, probing depth, clinical attachment loss, and marginal bo

74 periodontal parameters: bleeding on probing, probing depth, clinical attachment loss, and plaque inde

75 Periodontal examination findings included probing depth, clinical attachment loss, bleeding on pro

76 odontal data recorded at each visit included probing depth, clinical attachment loss, bleeding on pro

77 Clinical attachment loss (AL); probing depth; decayed, missing, and filled teeth (DMFT)

78 iting the ionization region suggest that the probing depth depends upon the desorption temperature, a

79 ever, the CLM group presented lower means of probing depth for pockets >/=7 mm at 6 months (4.0 +/- 1

80 ain in clinical attachment, decreased pocket probing depth, gain in radiographic bone height, and ove

81 valuated: plaque index, bleeding on probing, probing depth, gingival recession, and clinical attachme

82 d by calibrated investigators, included CAL, probing depth, gingival recession, bleeding on probing (

83 full-mouth periodontal examination including probing depth, gingival recession, plaque index, and ble

84 of tooth vitality, no changes in periodontal probing depth, good preservation of the papillae, and no

85 papillae (n = 241; with bleeding-on-probing, probing depth >/= 4 mm, and clinical attachment loss >/=

86 sent in teeth with bleeding on probing and a probing depth >/=3 mm at one or more sites.

87 increased GCF-IL-1beta levels, and extent of probing depth >/=4 mm (P </=0.05).

88 the presence of one or more sites exhibiting probing depth >/=4 mm or clinical attachment level >/=4

89 defined as the presence of >/=4 sites with a probing depth >/=5 mm and a clinical attachment loss >/=

90 ect >/=4 mm deep along with an interproximal probing depth >/=6 mm and clinical attachment level >/=4

91 ts >/=4 mm deep, along with an interproximal probing depth >/=6 mm and clinical attachment loss >/=4

92 nt patients with periodontitis, and 3) had a probing depth >4 mm or clinical attachment loss >2 mm fo

93 iodontitis and the persistence of sites with probing depths >4 mm and bleeding on probing.

94 l aspects of premolars and molars exhibiting probing depths in the 4- to 5-mm range and 1- to 2-mm at

95 inical measurements such as plaque index and probing depth, in patients with CP (P < 0.001).

96 ing technology due to a substantially larger probing depth into the medium and sensitivity, compared

97 nds upon the desorption temperature, and the probing depth is estimated to be on the order of 5 nm fo

98 arameters including vertical recession (VR), probing depth, keratinized tissue (KT), and attachment l

99 is commonly acknowledged as a method with a probing depth limited by the escape depth of the photoel

100 hy' papilla (n = 69; no bleeding-on-probing, probing depth </= 4 mm, and clinical attachment loss </=

101 pH, fewer remaining teeth, fewer sites with probing depth </=4 mm, and a lower radiographic alveolar

102 s the first to use photoacoustic imaging for probing depth measurements with potential implications t

103 tool for periodontal examinations, including probing depth measurements, but is limited by systematic

104 The six factors evaluated (age, probing depth, mobility, furcation involvement, smoking,

105 ng spectroscopies characterized by different probing depths, namely X-ray magnetic circular dichroism

106 The outcome measure was improvement in probing depth of >/=2 mm.

107 ze is comparable to the approximately 150 nm probing depth of SPR, and (iii) possible deformation of

108 receive SubGPAP in periodontal pockets with probing depths of 4 to 9 mm, SupraGPAP in all other shal

109 llele carriers had a higher mean periodontal probing depth (P <0.05), mean clinical attachment level

110 structure significantly associated with high probing depth (P = 0.02) and high bleeding on probing (P

111 Sixty patients with probing depth (PD) >/= 5 mm and clinical attachment leve

112 who had one or more periodontal sites with a probing depth (PD) >/= 5 mm with bleeding on probing (BO

113 ) 79 participants had at least 14 teeth with probing depth (PD) >/=4 mm (generalized periodontitis [G

114 ight individuals presenting >/=12 teeth with probing depth (PD) >/=4 mm were selected.

115 respectively (P = 0.05); for >/=1 site with probing depth (PD) >/=4 mm, 48.3% and 100% (P <0.001); f

116 laque index (PI), bleeding on probing (BOP), probing depth (PD) >/=4 mm, and clinical attachment loss

117 okers showed lower mean number of sites with probing depth (PD) >/=5 mm after therapy.

118 sease included patients with >1 tooth with a probing depth (PD) >/=5 mm and >2 teeth with a clinical

119 Periodontitis was defined as >/=1 site with probing depth (PD) >/=5 mm and clinical attachment level

120 defined as radiographic bone loss of > 2 mm, probing depth (PD) >/=5 mm with bleeding on probing (BOP

121 one infrabony defect >/=3 mm in depth with a probing depth (PD) >/=6 mm were randomly treated with EM

122 laque index (PI), bleeding on probing (BOP), probing depth (PD) >3 mm, clinical attachment loss (AL),

123 ome variable was persistence of sites with a probing depth (PD) >4 mm and bleeding on probing (BOP) a

124 tential predictors of at least one site with probing depth (PD) >4 mm and bleeding on probing (BOP) a

125 The number of sites with probing depth (PD) >4 mm and bleeding on probing (BOP) p

126 ace (BGI) periodontal groups: 1) health, all probing depth (PD) <3 mm and bleeding on probing (BOP) <

127 .418, P <0.005), and CD4+ nadir and moderate probing depth (PD) (rho = 0.424, P <0.05).

128 he oral examinations, along with periodontal probing depth (PD) and assessments of bleeding on probin

129 Periodontal probing depth (PD) and attachment loss (AL) were summari

130 Probing depth (PD) and bleeding on probing were measured

131 d of >/= 2 years and that reported change in probing depth (PD) and clinical attachment level (CAL) a

132 ll participants to determine the periodontal probing depth (PD) and clinical attachment level (CAL) a

133 vels of PGE2 were positively correlated with probing depth (PD) and clinical attachment level (CAL) a

134 ter 12 months, clinical parameters including probing depth (PD) and clinical attachment level (CAL) w

135 Probing depth (PD) and clinical attachment level (CAL) w

136 egistration at six sites per tooth including probing depth (PD) and clinical attachment level (CAL).

137 e undertaken to evaluate mean differences in probing depth (PD) and clinical attachment level (CAL).

138 6 months, baseline and follow-up measures of probing depth (PD) and clinical attachment levels (CAL)

139 evere periodontitis based on measurements of probing depth (PD) and clinical attachment loss (AL) at

140 cation as well as continuous measurements of probing depth (PD) and clinical attachment loss (AL).

141 cantly predicted by presurgery interproximal probing depth (PD) and depth of osseous dehiscence at th

142 as it has only a mild effect on reduction in probing depth (PD) and gain in clinical attachment level

143 therapy has been shown to reduce periodontal probing depth (PD) and local inflammatory mediators in g

144 ients without obesity presented a lower mean probing depth (PD) at 6 months after therapy and a great

145 linical attachment loss (AL) and periodontal probing depth (PD) from six sites per tooth on all teeth

146 clinical attachment level (relative CAL) and probing depth (PD) measurements were made.

147 elative clinical attachment level (RCAL) and probing depth (PD) measures were made.

148 >/=2 teeth each with approximal sites with a probing depth (PD) of 5 to 7 mm and gingival index (GI)

149 Fourteen patients having sites with residual probing depth (PD) of at least 5 mm and 2 mm loss of cli

150 % of participants had at least one site with probing depth (PD) or clinical AL of >/= 3 mm.

151 The included studies had to report probing depth (PD) reduction after the therapy.

152 Greater mean probing depth (PD) reduction and greater mean gain in re

153 Mean probing depth (PD) reduction and mean clinical attachmen

154 Mean probing depth (PD) reduction and mean relative vertical

155 eta-analyses were performed for defect fill, probing depth (PD) reduction, and clinical attachment le

156 Parameters evaluated included probing depth (PD) reduction, clinical attachment level

157 single-mask, randomized, controlled study if probing depth (PD) was </=3 mm and attachment loss was <

158 Attachment loss (AL) and probing depth (PD) were measured at 6 sites per tooth on

159 tial reductions in gingival inflammation and probing depth (PD) with a gain in clinical attachment le

160 er 6 months were: 1) gingival index (GI), 2) probing depth (PD), 3) clinical attachment level (CAL),

161 including plaque index, gingival index (GI), probing depth (PD), and bleeding on probing (BOP) were m

162 index (VPI), gingival bleeding index (GBI), probing depth (PD), and bleeding on probing (BOP) were m

163 io (WHR), plaque index, bleeding on probing, probing depth (PD), and clinical attachment level (CAL)

164 nd biologic data, bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL)

165 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical attachment level (CAL)

166 l examinations of bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL)

167 uding modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL),

168 , gingival index, bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL),

169 aque index (API), bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL),

170 odified sulcus bleeding index, plaque index, probing depth (PD), and clinical attachment level (CAL),

171 us bleeding index (mSBI), plaque index (PI), probing depth (PD), and clinical attachment level (CAL).

172 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL).

173 with a record of bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL).

174 luded modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL).

175 ) and thickness (TKT) of keratinized tissue, probing depth (PD), and clinical attachment level (CAL).

176 bleeding index (mSBI), gingival index (GI), probing depth (PD), and clinical attachment level (CAL).

177 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL).

178 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical attachment level were c

179 nation, including bleeding on probing (BOP), probing depth (PD), and clinical attachment level, was p

180 with gingival index (GI), plaque index (PI), probing depth (PD), and clinical attachment loss (AL) in

181 ion, plaque index (PI), gingival index (GI), probing depth (PD), and clinical attachment loss (AL) we

182 laque index (PI), bleeding on probing (BOP), probing depth (PD), and clinical attachment loss (AL) we

183 The clinical attachment level (CAL), probing depth (PD), and gingival recession (REC) were as

184 ers included bleeding on probing (BOP), mean probing depth (PD), and mean clinical attachment level (

185 ight (ACH), clinical attachment level (CAL), probing depth (PD), and percentage of gingival sites tha

186 re (FMPS), full-mouth bleeding score (FMBS), probing depth (PD), and recession depth (RD) were record

187 rs including modified sulcus bleeding index, probing depth (PD), and relative attachment level (RAL)

188 luded modified sulcus bleeding index (mSBI), probing depth (PD), and relative vertical (RVAL) and hor

189 The primary outcome was probing depth (PD), and secondary outcomes were measured

190 e), bleeding on probing, gingival recession, probing depth (PD), and vertical (VAL) and horizontal (H

191 nducted for clinical attachment level (CAL), probing depth (PD), bleeding on probing, and gingival in

192 Periodontal status was assessed by probing depth (PD), bleeding on probing, and radiographi

193 clinical periodontal measurements, including probing depth (PD), bleeding on probing, gingival index,

194 parameters that could predict postoperative probing depth (PD), clinical attachment level (CAL) gain

195 Periodontal disease was characterized using probing depth (PD), clinical attachment level (CAL), alv

196 The clinical variables evaluated were probing depth (PD), clinical attachment level (CAL), and

197 Probing depth (PD), clinical attachment level (CAL), and

198 Clinical measurements, including probing depth (PD), clinical attachment level (CAL), and

199 mplified (OHI-S) score, gingival index (GI), probing depth (PD), clinical attachment level (CAL), and

200 Measurements of probing depth (PD), clinical attachment level (CAL), and

201 ding plaque index (PI), gingival index (GI), probing depth (PD), clinical attachment level (CAL), and

202 Before SRP and after 3 and 12 months, probing depth (PD), clinical attachment level (CAL), and

203 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), clinical attachment level (CAL), and

204 laque index, modified sulcus bleeding index, probing depth (PD), clinical attachment level (CAL), and

205 eeding index, supragingival dental calculus, probing depth (PD), clinical attachment level (CAL), and

206 The measurements were probing depth (PD), clinical attachment level (CAL), ble

207 Probing depth (PD), clinical attachment level (CAL), ble

208 Probing depth (PD), clinical attachment level (CAL), ble

209 Periodontal parameters including probing depth (PD), clinical attachment level (CAL), ble

210 At baseline, probing depth (PD), clinical attachment level (CAL), ble

211 included width of keratinized tissue (KTw), probing depth (PD), clinical attachment level (CAL), cli

212 Plaque index, gingival index, probing depth (PD), clinical attachment level (CAL), def

213 Pre- and post-therapy probing depth (PD), clinical attachment level (CAL), gin

214 Clinical parameters (probing depth (PD), clinical attachment level (CAL), gin

215 linical and radiologic parameters, including probing depth (PD), clinical attachment level (CAL), IBD

216 Clinical and radiologic parameters such as probing depth (PD), clinical attachment level (CAL), IBD

217 Clinical and radiologic parameters, such as probing depth (PD), clinical attachment level (CAL), int

218 Clinical periodontal parameters, including probing depth (PD), clinical attachment level (CAL), pla

219 ured at baseline, 3 months and 6 months were probing depth (PD), clinical attachment level (CAL), rec

220 The clinical examination included probing depth (PD), clinical attachment level, bleeding

221 Probing depth (PD), clinical attachment level, dichotomo

222 thickness, recession depth, recession width, probing depth (PD), clinical attachment level, gingival

223 h as plaque index (PI), gingival index (GI), probing depth (PD), clinical attachment loss (AL), and p

224 Periodontal probing depth (PD), gingival bleeding on probing (BOP),

225 ight (ACH), clinical attachment level (CAL), probing depth (PD), gingival bleeding, and supragingival

226 The whole-mouth periodontal probing depth (PD), gingival index (GI), and plaque inde

227 re evaluated: gingival bleeding index (GBI), probing depth (PD), myeloperoxidase (MPO) activity, alve

228 uded plaque index (PI), gingival index (GI), probing depth (PD), periodontal index, body mass index,

229 clinical analyses were carried out including probing depth (PD), plaque index (PI), bleeding on probi

230 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), relative attachment level (RAL), and

231 re analyzed, and clinical parameters such as probing depth (PD), relative clinical attachment level (

232 aque and bleeding on probing indices, pocket probing depth (PD), relative gingival margin position (G

233 Bleeding on probing (BOP), probing depth (PD), relative GR, clinical attachment lev

234 laque index, modified sulcus bleeding index, probing depth (PD), relative vertical attachment level (

235 minus baseline) for bleeding on probing and probing depth (PD), the patients were separated into hig

236 riodontist assessed attachment loss (AL) and probing depth (PD).

237 EGF-A, and TNF-alpha levels with periodontal probing depth (PD).

238 ng measures of clinical attachment level and probing depth (PD).

239 y outcome was postoperative change in pocket probing depth (PD).

240 tal status was assessed with: 1) periodontal probing depth (PD); 2) bleeding on probing (BOP); and 3)

241 included the following: 1) mean periodontal probing depth (PD); 2) clinical attachment level (CAL);

242 on-surgical periodontal treatment (NSPT): 1) probing depth (PD); 2) clinical attachment level (CAL);

243 cal documentation included the following: 1) probing depth (PD); 2) keratinized tissue width (KT); 3)

244 2) modified sulcus bleeding index (mSBI); 3) probing depth (PD); 4) clinical attachment level (CAL);

245 index; 2) modified sulcus bleeding index; 3) probing depth (PD); 4) relative clinical attachment leve

246 index (PI); 2) bleeding on probing (BOP); 3) probing depth (PD); and 4) clinical attachment level (CA

247 aque index (PI); 2) bleeding scores (BS); 3) probing depth (PD); and 4) clinical attachment level (CA

248 ertical clinical attachment level (VCAL); 3) probing depth (PD); and 4) level of gingival margin (LGM

249 iene index (OHI); 3) gingival index (GI); 4) probing depth (PD); and 5) clinical attachment level (CA

250 achment loss, >/= 2 interproximal sites with probing depth [PD] >/= 4 mm not on the same tooth, or on

251 In deep periodontal pocket analysis (probing depth [PD] >/= 7 mm at baseline), the test group

252 e index [PI], bleeding on probing [BOP], and probing depth [PD] >/=4 mm) and crestal bone loss (CBL)

253 s diagnosed with GSCP (>/=8 teeth presenting probing depth [PD] >/=5 mm and bleeding on probing [BOP]

254 presenting at least three residual pockets (probing depth [PD] >/=5 mm with bleeding on probing [BOP

255 GCF was collected from one diseased site (probing depth [PD] >4 mm, bleeding on probing [BOP], and

256 at three representative sites, one healthy (probing depth [PD] </=3 mm) and two diseased (PD >/=6 mm

257 odified sulcus bleeding index, plaque index, probing depth [PD], and clinical attachment level [CAL])

258 mination (plaque index, gingival index [GI], probing depth [PD], and clinical attachment level [CAL])

259 odified sulcus bleeding index, plaque index, probing depth [PD], and clinical attachment level [CAL])

260 odified sulcus bleeding index, plaque index, probing depth [PD], and clinical attachment level [CAL])

261 frequency, days since professional cleaning, probing depth [PD], and plaque index) were also determin

262 ing index [PBI]); and 3) periodontal status (probing depth [PD], attachment loss [AL]).

263 Pre-therapy and post-therapy clinical (probing depth [PD], clinical attachment level [CAL], and

264 tal parameters (including plaque index [PI], probing depth [PD], clinical attachment level [CAL], and

265 moglobin levels, and periodontal parameters (probing depth [PD], clinical attachment level, gingival

266 laque index [PI], bleeding on probing [BOP], probing depth [PD], clinical attachment loss [AL], and m

267 laque index [PI], bleeding on probing [BOP], probing depth [PD], clinical attachment loss [AL], and m

268 laque index [PI], bleeding on probing [BOP], probing depth [PD], marginal bone loss [MBL]) and fastin

269 Clinical data (probing depth [PD], plaque index [PI], gingival index [G

270 Clinical and radiologic parameters (i.e., probing depth [PD], relative vertical and relative horiz

271 omplete examinations (plaque/gingival index, probing depth [PD], vertical clinical attachment level [

272 sites in the contralateral quadrants having probing depths (PDs) of >/=4 mm were selected.

273 okers with chronic periodontitis and matched probing depths (PDs) using real-time polymerase chain re

274 ed plaque accumulation, bleeding on probing, probing depths (PDs), and clinical attachment loss.

275 l attachment levels (CALs; primary outcome), probing depths (PDs), plaque, and BOP also were recorded

276 Probing depths (PDs), tooth loss, alveolar bone levels,

277 4 months after surgery, clinical parameters (probing depths [PDs] and vertical clinical attachment le

278 by Er:YAG application in sites with initial probing depths [PDs] of >/=4.5 mm) and a control group (

279 luded a full-mouth evaluation of periodontal probing depth, plaque score, bleeding on probing, and cl

280 bleeding index (r = 0.409) and interproximal probing depth (r = 0.307).

281 re clinical attachment level gain (CALg) and probing depth reduction (PDr) with a follow-up >/= 6 mon

282 Pocket Probing Depth reduction (PPD), Clinical Attachment Level

283 propolis group showed significantly greater probing depth reduction and clinical attachment level ga

284 95% confidence interval [CI]: 1.46-2.87 mm), probing depth reduction was 2.97 mm (95% CI: 2.38-3.56 m

285 Probing depth reduction was 4.0 +/- 0.5 mm in OF and 3.2

286 ddition to a significant attachment gain and probing depth reduction, adjunctive use of a CTG to a bu

287 ienced significant improvements, in terms of probing depth reduction, clinical attachment level (CAL)

288 nd at least one of the following parameters: probing depth reduction, clinical attachment level gain,

289 uitters were more likely to have periodontal probing depth reductions (P <0.05) than non-quitters/osc

290 n, NH3 being the dominant source of opacity) probing depths to over ~8 bar; these regions probably co

291 Mean periodontal probing depth was 2 mm (SE 0.02), and 17% had bleeding o

292 the BspA-specific IgG titers and periodontal probing depth was observed when healthy and disease grou

293 Among the parameters, probing depth was the most significant predictor variabl

294 Although the change in probing depth was the primary outcome, bleeding on probi

295 ncreased plaque and periodontitis (increased probing depths) was attenuated by high systemic oxidativ

296 GR depth, keratinized tissue (KT) width, and probing depth were measured at baseline (T0), 1 year aft

297 Plaque index, bleeding on probing, and probing depth were recorded at four sites per tooth.

298 th baseline ABH loss (p<0.0001) and baseline probing depths were associated with subsequent ABD and A

299 ere treated with the contrast agent, and the probing depths were measured with novel photoacoustic im

300 Probing depths were reduced for both groups at all time