ライフサイエンスコーパス: procarcinogen

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1 reserved tissues of rodents treated with the procarcinogens.

2 , is essential for the bioactivation of many procarcinogens.

3 ltered metabolic activation of tobacco smoke procarcinogens.

4 of androgenic steroids and certain aromatic procarcinogens.

5 A13 (CYP2A13) activates the nicotine-derived procarcinogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-buta

6 nt in tobacco, and activation of the tobacco procarcinogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-buta

7 0s participate in xenobiotic detoxification, procarcinogen activation, and steroid hormone synthesis.

8 ominent roles in xenobiotic detoxication and procarcinogen activation.

9 CYP1A1 bioactivates several procarcinogens and detoxifies several xenobiotic compoun

10 tivation of hexamethylphosphoramide, a nasal procarcinogen, and 2,6-dichlorobenzonitrile (DCBN), a he

11 ign of chemopreventive strategies is whether procarcinogen bioactivation in an extrahepatic target ti

12 The bioactivation of procarcinogens by cytochrome P450 2W1 may be of signific

13 Thus, activation of procarcinogens by P450 lB1 may contribute to human tumor

14 the enzyme in tumors, in that activation of procarcinogens could lead to the accumulation of mutatio

15 Using the procarcinogen diethylnitrosamine (DEN) to initiate tumor

16 been shown to be formed in DNA from various procarcinogens (e.g., acrolein, malonaldehyde, vinyl chl

17 none, is one of the most potent and abundant procarcinogens found in tobacco and tobacco smoke, and g

18 ic aromatic hydrocarbons (PAHs), is a potent procarcinogen generated during the combustion of fossil

19 the dispositions of reactive metabolites of procarcinogens in humans, provided that exposures are ad

20 ) are important enzymes that detoxicate many procarcinogens, including HAAs.

21 A2 can catalyze the bioactivation of several procarcinogens, indicating a potential role in chemical

22 In all nodules of procarcinogen-induced murine hepatocellular carcinomas (

23 P450 2W1 catalyzed the activation of several procarcinogens, particularly polycyclic hydrocarbon diol

24 Metabolism of food- and tobacco-borne procarcinogens results in the exposure of DNA to toxic a

25 mportant enzymes in the detoxication of many procarcinogens, serve as a mechanism of bioactivation of

26 of benzo[a]pyrene (B[a]P), an environmental procarcinogen that activates Cyp1a1 transcriptional resp

27 yclic amines (HCAs) found in cooked meat are procarcinogens that are metabolically activated by N-hyd

28 els should be applicable to studies on other procarcinogens that require P450-mediated metabolic acti

29 ertain hepatotoxic chemicals, including some procarcinogens, their ability to monooxygenate, and ther

30 mbers, which have the capacity to metabolize procarcinogens to genotoxic carcinogens.

31 he cytochrome P450 enzymes, which metabolize procarcinogens to their activated forms.

32 key to block the conversion of environmental procarcinogens to their carcinogenic metabolites in both

33 Two major groups of procarcinogens, tobacco-specific nitrosamines and polycy

34 These procarcinogens undergo metabolic activation by N-oxidati

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