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1 s of estrogen receptors or estradiol-induced progestin receptors.
2 estrogen receptors and/or estradiol-induced progestin receptors.
3 sterone, it appears to be mediated by neural progestin receptors.
4 estrogen receptors and/or estradiol-induced progestin receptors.
5 radiol and progesterone involve estrogen and progestin receptors.
6 ve no detectible impact on the expression of progestin receptors.
7 -1 down-regulated the expression of membrane progestin receptor alpha (mPRalpha), the intermediary in
8 el immunofluorescent technique to label both progestin receptors and Fos protein following progestero
9 dy examined the effects of administering the progestin receptor antagonist RU486 for the first 10 day
10 brain, changes in the activational state of progestin receptors because of dopamine D1 receptor stim
12 ng the mating-induced release of dopamine in progestin receptor-containing areas of the brain, change
14 cant increase in Fos-immunoreactivity within progestin receptor-containing cells in the medial preopt
15 e progesterone-induced Fos expression within progestin receptor-containing neurons may or may not be
16 terone treatment increased Fos expression in progestin receptor-containing regions, such as the ventr
17 The finding that sex differences exist in progestin receptor expression in the perinatal rat brain
18 ls housed in SP showed significantly reduced progestin receptor immunoreactivity (PRIR) in the VMH, m
19 ning estrogen receptor- or estradiol-induced progestin receptor-immunoreactivity in the ventrolateral
22 stics suggest the fish protein is a membrane progestin receptor mediating a "nonclassical" action of
23 gene with the characteristics of a membrane progestin receptor (mPR) in a fish model, spotted seatro
24 alpha), estrogen receptor-beta (ERbeta), and progestin receptor (PR) immunoreactivities are localized
25 or instance, the sex difference in perinatal progestin receptor (PR) immunoreactivity in the medial p
27 us, vessel invasion, estrogen receptor (ER)/ progestin receptor (PR) status, c-erb B-2, or Ki-67 expr
28 of estrogen receptor (ER) alpha, ERbeta and progestin receptor (PR) with LENK-labeled MF pathway pro
29 Only C57 and C57x129 mice had increases in progestin receptor (PR)-immunoreactivity (PR-IR) in the
37 xual receptivity and given the importance of progestin receptors (PRs) in mediating the responses of
38 e of P's actions at E2-induced intracellular progestin receptors (PRs) to facilitate lordosis was inv
39 -32 activation is thus an obligatory step in progestin receptor regulation of sexual receptivity in r
40 post-synaptic GPCRs, including the membrane progestin receptor, the corticotropin releasing hormone
41 ne with increase Fos expression also contain progestin receptors, we used a double-label immunofluore
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