ライフサイエンスコーパス: programmed cell death

1 ase and nuclease activity, key components of programmed cell death.

2 energy production, metabolite syntheses, and programmed cell death.

3 tion of sphingolipid metabolites that induce programmed cell death.

4 h damages the cancer cell structure inducing programmed cell death.

5 ways unique to oxidative stress response and programmed cell death.

6 e Bmf and Bim, which are known regulators of programmed cell death.

7 control, biofilm formation, persistence, and programmed cell death.

8 hat AtPDCD5 also participates in age-induced programmed cell death.

9 in cancer progression through regulation of programmed cell death.

10 otein with dual roles in redox signaling and programmed cell death.

11 g to CD47, a cell surface receptor, triggers programmed cell death.

12 cells die or also controls other aspects of programmed cell death.

13 f proteins responsible for the regulation of programmed cell death.

14 including control of cell proliferation and programmed cell death.

15 induced STAT1 signaling but did not initiate programmed cell death.

16 e absence of pathogen infection and enhances programmed cell death.

17 cell escape via induction of an inflammatory programmed cell death.

18 it the repair of DNA damage or initiation of programmed cell death.

19 daptive immunity; and dormancy induction, or programmed cell death.

20 uires the assistance of an unlikely process: programmed cell death.

21 e virus-infected cell was thought to undergo programmed cell death.

22 ich govern the edibility of cells undergoing programmed cell death.

23 tional components in the cell, the latter by programmed cell death.

24 he phenylpropanoid pathway and regulation of programmed cell death.

25 hanisms regulating ZBP1 activation to induce programmed cell death.

26 ced protein misfolding and Caspase-8-induced programmed cell death.

27 ling between immunity and dormancy induction/programmed cell death.

28 ) is a nondescribed effect secondary to anti-programmed cell death 1 (anti-PD-1) and anti-programmed

29 nhibitory receptors on immune cells, such as programmed cell death 1 (PD-1) and cytotoxic T-lymphocyt

30 d to CD8(+) T cell exhaustion, manifested by programmed cell death 1 (PD-1) and lymphocyte activation

31 Importance: Antagonist antibodies to programmed cell death 1 (PD-1) and programmed cell death

32 ([Mg(2+)]i) leads to defective expression of programmed cell death 1 (PD-1) and the NK activating rec

33 n monotherapy or in combination with an anti-programmed cell death 1 (PD-1) antibody in mouse models

34 e after lung transplantation is dependent on programmed cell death 1 (PD-1) expression on CD8(+) T ce

35 Pembrolizumab inhibits the programmed cell death 1 (PD-1) immune checkpoint and has

36 uccess of monotherapies based on blockade of programmed cell death 1 (PD-1) in human melanoma, most p

37 Importance: Programmed cell death 1 (PD-1) inhibitor-related pneumon

38 on that is associated with expression of the programmed cell death 1 (PD-1) inhibitory receptor.

39 reported that intratumoral expression of the programmed cell death 1 (PD-1) receptor can guide the id

40 ells), regulatory T cells, and percentage of programmed cell death 1 (PD-1)-expressing cells among CD

41 pecific T cells resided predominantly in the programmed cell death 1 (PD-1)-expressing T cell compart

42 tained expression of the inhibitory receptor programmed cell death 1 (PD-1).

43 by upregulating inhibitory receptors such as programmed cell death 1 (PD-1).

44 The development of programmed cell death 1 (PD-1)/PD-1 ligand 1 (PD-L1) che

45 Blockade of the programmed cell death 1 (PD-1)/programmed death-ligand 1

46 phocytes of the same subjects expressed high programmed cell death 1 (PD1).

47 T lymphocyte-associated protein (CTLA)-4 or programmed cell death 1 (PDCD1; also known as PD-1) elic

48 demonstrate that LN CD4 T cells that express programmed cell death 1 (PDCD1; also known as PD-1), whi

49 hereas their inhibitory receptor expression (programmed cell death 1 [PD-1] and cytotoxic T-lymphocyt

50 -lymphocyte-associated antigen 4 [CTLA-4] or programmed cell death 1 [PD-1]) inhibitor.

51 D-L1) to enrich for patient response to anti-programmed cell death 1 and anti-PD-L1 therapies.

52 xpression of the inhibitory surface receptor programmed cell death 1 compared with controls.

53 Increasing fractions of programmed cell death 1 high/cytotoxic T lymphocyte-asso

54 T-cell expression of the inhibitory receptor programmed cell death 1 increased 25-fold compared with

55 Inhibitors of the Programmed Cell Death 1: Programmed Cell Death 1 ligand 1 (PD-1:PD-L1) pathway, a

56 ), the IL-2 receptor alpha chain (CD25), and programmed cell death 1 ligand 1 (PD-L1).

57 protein 4 (CTLA4) or programmed cell death 1/programmed cell death 1 ligand 1 (PD1/PD-L1) generates d

58 Assessment of PD-L1 (programmed cell death 1 ligand 1) expression by immunohi

59 Importance: Assessment of PD-L1 (programmed cell death 1 ligand 1) expression by immunohi

60 (programmed cell death protein 1) and PD-L1 (programmed cell death 1 ligand 1) have demonstrated dura

61 In this study, we demonstrate that programmed cell death 1 ligand 2 (PD-L2) regulates the p

62 ls of B lymphocyte stimulator receptor 3 and programmed cell death 1 on B cells increased in patients

63 expression of the immunosuppressive molecule programmed cell death 1 on T cells and of CD200 on B-CLL

64 robiome of melanoma patients undergoing anti-programmed cell death 1 protein (PD-1) immunotherapy (n

65 To determine the efficacy of programmed cell death 1 receptor (PD-1) inhibitors in lo

66 s and guide treatment in trials of the PD-1 (programmed cell death 1) axis inhibitors.

67 s and guide treatment in trials of the PD-1 (programmed cell death 1) axis inhibitors.

68 ntly improve the efficacy of alphaPD-1 (anti-programmed cell death 1) treatment using the B16F10 mela

69 molecules (cytotoxic T-lymphocyte antigen 4, programmed cell death 1, and indolamine 2,3-dioxygenase

70 reased inhibitory molecule expression (e.g., programmed cell death 1, lymphocyte-activation gene 3, a

71 274 molecule (programmed death-ligand 1) and programmed cell death 1, markers of cytolytic activity,

72 T cells, such as programmed death-ligand 1, programmed cell death 1, or transforming growth factor b

73 red CD20+ B cells, and relatively few PD-1+ (programmed cell death 1-positive) T cells, an immunophen

74 T-lymphocyte-associated protein 4 (CTLA4) or programmed cell death 1/programmed cell death 1 ligand 1

75 Inhibitors of the Programmed Cell Death 1: Programmed Cell Death 1 ligand

76 ls and expression of the inhibitory receptor programmed cell death -1 PD-1 on CD4(+) T cells were sig

77 ytotoxic T-lymphocyte antigen 4 (CTLA-4) and programmed cell death-1 (PD-1) has significantly improve

78 Despite the importance of programmed cell death-1 (PD-1) in inhibiting T cell effe

79 Ligation of programmed cell death-1 (PD-1) in the tumor microenviron

80 Programmed cell death-1 (PD-1) is a coinhibitory recepto

81 Programmed cell death-1 (PD-1) is a negative regulator o

82 Programmed cell death-1 (PD-1) is an essential inhibitor

83 Programmed cell death-1 (PD-1) is an inhibitory receptor

84 The inhibitory immune receptor programmed cell death-1 (PD-1) is intricately regulated.

85 Programmed cell death-1 (PD-1) signaling contributes to

86 Anti-programmed cell death-1 (PD-1) treatment reduced the tum

87 strong accumulation of terminally exhausted programmed cell death-1 (PD-1)(high) T cell Ig mucin-3(+

88 f GC TFH (CXCR5(high)PD-1(high)) and CXCR5(+)programmed cell death-1 (PD-1)(low) cells were GFP(+) th

89 l immunoglobulin and mucin-3 (TIM-3), and/or programmed cell death-1 (PD-1), and acquire an anergic p

90 Programmed cell death-1 (PD-1)-targeted therapies enhanc

91 P=0.008), correlating with the expression of programmed cell death-1 before primary percutaneous coro

92 Programmed cell death-1 mRNA expression was increased in

93 The programmed cell death-1(PD-1)/PD-ligand 1 (PD-L1) pathwa

94 re subsequently treated with anti-PD-1 (anti-programmed cell death-1) therapy and experienced complet

95 The anti-Programmed cells Death-1 was stopped and a topical corti

96 the onco-miR, miR-21 and its target protein, programmed cell death 4 (PDCD4) in arsenic induced malig

97 Programmed cell death 4 (PDCD4) is a tumor suppressor an

98 Programmed cell death 4 (Pdcd4), a tumor invasion suppre

99 miR-21 down-regulated programmed cell death 4 and programmed cell death 4 prot

100 1 down-regulated programmed cell death 4 and programmed cell death 4 protein was decreased in periton

101 through direct repression of its target gene programmed cell death 4 Thus, miR-21 functions downstrea

102 BMs and promoted the expression of Bax (the programmed cell death acceleration protein) and the indu

103 tochondria during leaf senescence, a type of programmed cell death aimed at the massive retrieval of

104 gation of peroxisomes during final stages of programmed cell death and can be used as a marker of thi

105 to understand the role of the apoptosome in programmed cell death and disease.

106 Bcl-2) family proteins are key regulators of programmed cell death and important targets for drug dis

107 biological pathways related to neurogenesis, programmed cell death and insulin signaling from the uni

108 lationship between amyloid-controlled fungal programmed cell death and mammalian necroptosis.

109 olipids are emerging as second messengers in programmed cell death and plant defense mechanisms.

110 opherol and upregulation of genes related to programmed cell death and protein folding.

111 Bacterial programmed cell death and quorum sensing are direct exam

112 of poly GA in a cell culture model activates programmed cell death and TDP-43 cleavage in a dose-depe

113 ty is mediated by altered signals related to programmed cell death and the activation of various sign

114 SnTox1 induces programmed cell death and the up-regulation of pathogene

115 In this review we explore different types of programmed cell death and their impact on innate immune

116 retrograde signaling, plant hormone action, programmed cell death, and defense against pathogens hav

117 ity, causing cellular stress, and increasing programmed cell death (apoptosis) in the tissues require

118 Programmed cell death (apoptosis) is an integral part of

119 species from cells or to selectively induce programmed cell death (apoptosis) or uncontrolled cell d

120 d play a central role in the early stages of programmed cell death (apoptosis).

121 ) is a crucial death regulator that triggers programmed cell death, apoptosis.

122 The best-known forms of programmed cell death are apoptosis and a recently recog

123 Multiple pathways of programmed cell death are important in liver homeostasis

124 changes are likely due to the activation of programmed cell death as assessed by Annexin V staining

125 teral organ induction is driven by recurrent programmed cell death at the most distal edge of the roo

126 'hypersensitive response' (HR) that involves programmed cell death at the site of pathogen recognitio

127 ses and subsequently undergoes developmental programmed cell death, breaking down as the embryo grows

128 phagy machinery can control the mechanism of programmed cell death by serving as a scaffold rather th

129 hrough a recently described distinct form of programmed cell death called NETosis.

130 Furthermore, programmed cell death can also represent a defense mecha

131 echanisms in the control of host defense and programmed cell death cascades.

132 ted decay pathway and a gene associated with programmed cell death could explain why this pathway is

133 Necroptosis is a form of programmed cell death defined by activation of the kinas

134 ominently in necroptosis, a specific form of programmed cell death dependent on RIPK-1, RIPK-3, and t

135 are generated, most of which will be lost by programmed cell death due to a limited supply of neurotr

136 lective functions of SR-B1 ultimately affect programmed cell death, female fertility, platelet functi

137 and lissencephaly associated with defective programmed cell death from loss of CRADD function in hum

138 (ECM) is required to combat the induction of programmed cell death in a variety of distinct cell type

139 ) family, which are emerging as mediators of programmed cell death in a variety of processes that reg

140 icum) Cipk6 regulates immune and susceptible Programmed cell death in immunity transforming Ca(2+) si

141 t that released, soluble M1 protein triggers programmed cell death in macrophages (Mvarphi).

142 Mother Cell (MMC) specification, and delayed programmed cell death in megaspores and the tapetum, fea

143 Drugs aimed at inducing programmed cell death in neoplastic cells by re-engaging

144 in phytoplankton, NO was reported to mediate programmed cell death in response to diatom-derived poly

145 This process involves the induction of programmed cell death in specific cells within the tissu

146 iated IAV sensing is critical for triggering programmed cell death in the infected lungs.

147 y, down-regulation of transcripts related to programmed cell death in the spinal cord, and normalizat

148 aviolet (UV)-B radiation and participates in programmed cell death in the UV-B DNA damage response.

149 s in the nuclear architecture, and activated programmed cell death, in D. citri midgut cells.

150 Programmed cell death, including apoptosis, mitochondria

151 lines displayed increased resistance to the programmed cell death-inducing mycotoxin fumonisin B1, w

152 To define how different forms of programmed cell death influence immunity, we established

153 plicated its role in signaling to SI-induced programmed cell death involving a DEVDase.

154 ulator for SI in pollen and acts upstream of programmed cell death involving actin and activation of

155 Programmed cell death is a crucial factor in the progres

156 Moreover, programmed cell death is differentially regulated in the

157 We propose DNA damage-induced programmed cell death is employed by plants as a develop

158 In Arabidopsis, DNA damage-induced programmed cell death is limited to the meristematic ste

159 Another type of programmed cell death is necroptosis (programmed necrosi

160 ion of death receptor-mediated activation of programmed cell death is the aspartate-specific cysteine

161 The significance of this cell-type-specific programmed cell death is unclear.

162 oma-targeting peptides, can induce a form of programmed cell death known as ferroptosis in starved ca

163 remain viable or following a unique form of programmed cell death known as NETosis, which is depende

164 programmed cell death 1 (anti-PD-1) and anti-programmed cell death ligand 1 (anti-PD-L1 ) therapy for

165 tibody) in advanced solid tumours expressing programmed cell death ligand 1 (PD-L1) and report here o

166 bodies to programmed cell death 1 (PD-1) and programmed cell death ligand 1 (PD-L1) have shown remark

167 Programmed cell death ligand 1 (PD-L1) is part of an imm

168 The programmed cell death ligand 1 (PD-L1) participates in a

169 The programmed cell death protein 1 (PD-1)/programmed cell death ligand 1 (PD-L1) pathway is a nega

170 US Food and Drug Administration (FDA) detect programmed cell death ligand 1 (PD-L1) to enrich for pat

171 frequently overexpress the ligand for PD-1, programmed cell death ligand 1 (PD-L1), facilitating the

172 erance mechanisms, such as the expression of programmed cell death ligand 1 (PD-L1), the inhibition o

173 ts of IFN-gamma and sensitize tumors to PD-1/programmed cell death ligand 1 blockade-induced rejectio

174 or growth that is largely unaffected by PD-1/programmed cell death ligand 1 blockade.

175 d C-MET (300 vs 100; P < .001) and increased programmed cell death ligand 1 expression (0 vs 1.5; P <

176 Programmed cell death ligand-1 (PD-L1) is typically prod

177 f biopsy and repeat biopsy for assessment of programmed cell death ligand-1 (PD-L1) status.

178 l proliferation, but they did express higher programmed cell death ligand-1 (PDL1) than other neutrop

179 ed non-small-cell lung cancer (NSCLC) with a programmed cell death-ligand 1 (PD-L1) tumour proportion

180 nst T cell surveillance via stabilization of programmed cell death-ligand 1 (PD-L1).

181 lants, which are defective in damage-induced programmed cell death, maintain the cell identities and

182 cells undergoing apoptosis, suggesting that programmed cell death may limit viral dissemination in t

183 clude genes associated with trophic support, programmed cell death, microtubule disassembly, synaptic

184 y, antibody-dependent cellular phagocytosis, programmed cell death, modulation of enzymatic activity,

185 ion-induced kidney injury, inflammation, and programmed cell death observed in wild-type mice and pro

186 Programmed cell death occurs in a highly reproducible ma

187 complex multicellular defense reaction where programmed cell death of cells surrounding the primary s

188 thelial cell (TEC) functions and induces the programmed cell death of immature thymocytes.

189 hat ZHOUPI does not induce the developmental programmed cell death of the endosperm directly.

190 agmentation, that ultimately resulted in the programmed cell death of the parasite.

191 Programmed cell death or apoptosis of infected host cell

192 iated parasite death, which we term 'microbe-programmed cell death' or 'microptosis', is caspase inde

193 epidermal patterning, vascular development, programmed cell death, organ abscission, senescence, and

194 o purportedly plays an important role in the programmed cell death pathway (PD-1/PD-L1).

195 Pyroptosis is a form of inflammatory programmed cell death pathway activated by human and mou

196 Necroptosis is a programmed cell death pathway that has been shown to be

197 Necroptosis is an alternate programmed cell death pathway that is unleashed by caspa

198 urveillance exploits a fungal apoptosis-like programmed cell death pathway to maintain sterilizing im

199 dies to examine additional components in the programmed cell death pathway to test the hypothesis tha

200 n, and vRNP sensing to trigger activation of programmed cell death pathways during IAV infection.

201 This study reveals the effect of antiviral programmed cell death pathways on inflammation, shows th

202 ous growth structures, and the activation of programmed cell death pathways.

203 luenza virus infection induces activation of programmed cell death pathways.

204 ltiple pathways involved in regulating novel programmed cell-death pathways and inflammation.

205 Programmed cell death (PCD) and associated pathway genes

206 decade, studies have shown how instrumental programmed cell death (PCD) can be in innate and adaptiv

207 During limb development, programmed cell death (PCD) contributes to separation of

208 Programmed cell death (PCD) functions in a variety of pr

209 ole in effector-triggered immunity (ETI) and programmed cell death (PCD) in plants, is a novel transm

210 ernata f.sp. Lycopersici (AAL) toxin induces programmed cell death (PCD) in susceptible tomato (Solan

211 istematic cells indicated the involvement of programmed cell death (PCD) in the process.

212 Programmed cell death (PCD) induced by endoplasmic retic

213 Programmed cell death (PCD) is a crucial process both fo

214 Programmed cell death (PCD) is central to organism devel

215 Programmed cell death (PCD) is critical for development

216 Programmed cell death (PCD) is usually considered a cell

217 Programmed cell death (PCD) occurs in several forms incl

218 ic agonist peptide able to trigger selective programmed cell death (PCD) of at least lung, breast, an

219 spectrum killing activity, is able to induce programmed cell death (PCD) of CD38(+) multiple myeloma

220 hanges in gene expression can lead to either programmed cell death (PCD) or acclimation.

221 Necroptosis is a RIP1-dependent programmed cell death (PCD) pathway that is distinct fro

222 Several indicators for programmed cell death (PCD) that are often observed in p

223 effector-triggered immunity (ETI), involving programmed cell death (PCD), as a major defence mechanis

224 cally programmed signalling pathway known as programmed cell death (PCD).

225 ed to severe or chronic stress, UPR promotes programmed cell death (PCD).

226 ion occurs through developmentally regulated programmed cell death (PCD).

227 C synapses develop while 7% of RBCs undergo programmed cell death (PCD).

228 hypersensitive response (HR) is a localized programmed cell death phenomenon that occurs in response

229 that most hypertrophic chondrocytes undergo programmed cell death prior to bone formation.

230 cer cell to develop resistance to anoikis, a programmed cell death process triggered by substratum de

231 tion in Arabidopsis and also participates in programmed cell death programs.

232 CD8(+) function and signaling through the programmed cell death protein (PD)-1 exhaustion pathway

233 Blocking programmed cell death protein (PD)-1 signaling, which me

234 une checkpoints, such as the one mediated by programmed cell death protein 1 (PD-1) and its ligand PD

235 The programmed cell death protein 1 (PD-1) and programmed de

236 Signaling between programmed cell death protein 1 (PD-1) and the PD-1 liga

237 as performed to assess CD8, FOXP3, CD56, and programmed cell death protein 1 (PD-1) expression on str

238 6, IL-21, inducible costimulator, CXCR5, and programmed cell death protein 1 (PD-1) expressions incre

239 is a humanized monoclonal antibody targeting programmed cell death protein 1 (PD-1) found on T and pr

240 s that target the immune checkpoint receptor programmed cell death protein 1 (PD-1) have resulted in

241 sed the safety and antitumor activity of the programmed cell death protein 1 (PD-1) inhibitor pembrol

242 Programmed cell death protein 1 (PD-1) is an immune chec

243 1 and associated increased expression of the programmed cell death protein 1 (PD-1) ligands, PD-L1 an

244 The programmed cell death protein 1 (PD-1) pathway-blocking

245 the expression of the coinhibitory molecules programmed cell death protein 1 (PD-1), T cell immunoglo

246 of genes, including sustained expression of programmed cell death protein 1 (PD-1).

247 The programmed cell death protein 1 (PD-1)/programmed cell d

248 expression of negative checkpoint regulators programmed cell death protein 1 (PD1), programmed death-

249 bitors (including antibodies that antagonize programmed cell death protein 1 [PD-1]) have both opened

250 Rgammat(+) T cells expressing a low level of programmed cell death protein 1 and a high level of OX40

251 ee of which were randomized trials comparing programmed cell death protein 1 and programmed death lig

252 DSCs expressed reduced surface expression of programmed cell death protein 1 compared to healthy cont

253 cally blocking the interaction of PD-L1 with programmed cell death protein 1 impaired the ability of

254 owed significantly improved survival for the programmed cell death protein 1 inhibitor nivolumab comp

255 c HLA-I genotype influences response to anti-programmed cell death protein 1 or anti-cytotoxic T lymp

256 hocyte-associated antigen 4 (CTLA-4) and the programmed cell death protein 1 pathways (PD-1/PD-L1) ha

257 ally for patients who do not respond to anti-programmed cell death protein 1 treatment.

258 s with monoclonal antibodies targeting PD-1 (programmed cell death protein 1) and PD-L1 (programmed c

259 t that tumors with a genetic basis for PD-1 (programmed cell death protein 1) ligand expression are h

260 Based on the expression of CD86 and programmed cell death protein 1, CD8(+) T cells were div

261 checkpoint-blocking (ICB) antibodies against programmed cell death protein 1/programmed death ligand

262 totoxic-T-lymphocyte-associated protein 4 or programmed cell death protein 1/programmed death-ligand

263 oteomics demonstrated that both proapoptotic programmed cell death protein 5 and antiapoptotic macrop

264 n recent years, it has been established that programmed cell death protein ligand 1 (PD-L1)-mediated

265 es the immune system by interacting with the programmed cell death protein receptor 1, found exclusiv

266 sess the safety and efficacy of nivolumab, a programmed cell death protein-1 (PD-1) immune checkpoint

267 etween programmed death ligand-1 (PD-L1) and programmed cell death protein-1 (PD-1) leads to tumour-a

268 The programmed cell death protein-1 (PD-1) signaling pathway

269 Programmed cell death protein-1 (PD-1)/programmed death

270 Combination with anti-programmed cell death protein-1 antibodies promoted comp

271 Programmed cell death protein-1 expression was significa

272 Programmed cell death protein-1- checkpoint blockers hav

273 onal N-terminal CaMBD constitutively induced programmed cell death, providing in planta evidence of a

274 afety and efficacy of pembrolizumab (an anti-programmed cell death receptor 1 [PD-1] antibody) in adv

275 ety and potential benefit of nivolumab (anti-programmed cell death receptor 1) monotherapy beyond Res

276 ovel mechanism in which immune signaling and programmed cell death require nuclear pore rearrangement

277 n during newt limb regeneration depends on a programmed cell death response by myofibres.

278 rst inducing and subsequently intercepting a programmed cell death response.

279 include genes associated with neurotrophins, programmed cell death, synaptic function, sirtuins and a

280 These data identify both a new programmed cell death system and a novel role for HQNO a

281 ion of cellular RNA and thereby heat-induced programmed cell death that in turn supports the formatio

282 Apoptosis is a form of programmed cell death that is critical for basic human d

283 Apoptosis is a form of programmed cell death that is essential for the efficien

284 Ferroptosis is a form of programmed cell death that is pathogenic to several acut

285 Heartwood formation is marked by programmed cell death that occurs during the summer mont

286 se 3 (RIPK3) mediates necroptosis, a form of programmed cell death that promotes inflammation in vari

287 Necroptosis is a highly inflammatory form of programmed cell death that results from MLKL-mediated di

288 in pyroptosis, a highly inflammatory form of programmed cell death, that potentially further perpetua

289 nhibitor 1 (BI1) modulates ER stress-induced programmed cell death through yet-unknown mechanisms.

290 studies suggested that pyroptosis, a form of programmed cell death triggered during abortive HIV infe

291 more efficient DNA repair and abrogation of programmed cell death under salinity and genotoxic stres

292 ulation of sulforaphane displayed attenuated programmed cell death upon bacterial and oomycete effect

293 fed CA-MRSA underwent a novel form of lytic programmed cell death via a mechanism that required RIPK

294 Until recently, programmed cell death was conceived of as a single set o

295 al for LCL defense against TNFalpha-mediated programmed cell death, whereas EBV-induced BATF/IRF4 wer

296 cently, apoptosis was the best known form of programmed cell death, whereas necrosis was for a long t

297 rigger proliferation but instead resulted in programmed cell death, which is likely mediated by the E

298 Programmed cell death widely but heterogeneously affects

299 we demonstrate that lung neutrophils trigger programmed cell death with apoptosis-like features in As

300 Thus, HQNO-driven autolysis links programmed cell death with quorum sensing and biofilm fo