ライフサイエンスコーパス: progressive aphasia

1 language network in each variant of primary progressive aphasia.

2 arietal regions in logopenic variant primary progressive aphasia.

3 epresent a fourth variant of 'mixed' primary progressive aphasia.

4 , making this syndrome distinct from primary progressive aphasia.

5 tests; hence, none met criteria for primary progressive aphasia.

6 hanges that occur in the variants of primary progressive aphasia.

7 multimodal diagnostic evaluation of primary progressive aphasia.

8 inguish this syndrome from other variants of progressive aphasia.

9 ationship between semantic dementia (SD) and progressive aphasia.

10 , and therapeutic potential in patients with progressive aphasia.

11 gh detailed studies of patients with primary progressive aphasia.

12 atrophy pattern in semantic variant primary progressive aphasia.

13 the non-fluent/agrammatic variant of primary progressive aphasia.

14 ophy and eight for logopenic variant primary progressive aphasia.

15 highly variable in individuals with primary progressive aphasia.

16 most patients with semantic variant primary progressive aphasia.

17 type C consistently led to semantic primary progressive aphasia.

18 logical and neuroimaging features of primary progressive aphasia.

19 al fluency and grammar impairment in primary progressive aphasia.

20 anguage network in logopenic variant primary progressive aphasia.

21 c memory deficit in semantic variant primary progressive aphasia.

22 s and patients with semantic variant primary progressive aphasia.

23 ocessing of non-verbal sounds in the primary progressive aphasias.

24 s the harbinger of degenerative disease: the progressive aphasias.

25 = 54) and semantic (n = 96) variant primary progressive aphasias.

26 nsecutive series of 20 patients with primary progressive aphasia [12 with progressive non-fluent apha

27 a, 14 patients with semantic variant primary progressive aphasia, 25 patients with Alzheimer's diseas

28 bjects with the logopenic variant of primary progressive aphasia, 6 age-matched patients with AD, and

29 This study used patients with primary progressive aphasia, a clinical dementia syndrome charac

30 gains of function in a patient with primary progressive aphasia, a degenerative disease of the human

31 speech samples for 50 patients with primary progressive aphasia, along with neurodegenerative and no

32 Patients with logopenic variant primary progressive aphasia also showed significant hypersynchro

33 mporal dementia and semantic variant primary progressive aphasia (also called semantic dementia) are

34 atients with the semantic variant of primary progressive aphasia, also known as semantic dementia, an

35 on derived from the semantic variant primary progressive aphasia analysis was strongly connected with

36 , 12 patients with logopenic variant primary progressive aphasia and 13 patients with posterior corti

37 Thirty-five patients with primary progressive aphasia and 29 control subjects were recruit

38 ed 15 patients with semantic variant primary progressive aphasia and 57 patients with Alzheimer's dis

39 conclude that both semantic variant primary progressive aphasia and Alzheimer's disease are signific

40 We found that both semantic variant primary progressive aphasia and Alzheimer's disease are signific

41 Both semantic variant primary progressive aphasia and Alzheimer's disease had atrophy

42 Logopenic variant primary progressive aphasia and developmental dyslexia both mani

43 underlies verbal fluency deficits in primary progressive aphasia and further confirm the role of the

44 However, recent primary progressive aphasia and normal neurophysiological studie

45 ndrome and that varieties such as logopaenic progressive aphasia and progressive apraxia of speech ma

46 sound perception and recognition in primary progressive aphasia and specific disorders at perceptual

47 ent presented at age 54 years with logopenic progressive aphasia and, at autopsy, showed both frontot

48 tical atrophy than logopenic variant primary progressive aphasia) and higher-order visual network (lo

49 89 patients with semantic variant of primary progressive aphasia, and 30 patients with Huntington dis

50 antic variant and non-fluent variant primary progressive aphasia, and 46 healthy controls) described

51 nts (27.0%) with semantic variant of primary progressive aphasia, and 6 of 30 patients (20%) with Hun

52 cortical atrophy, logopenic variant primary progressive aphasia, and corticobasal syndrome).

53 the French composer who also suffered from a progressive aphasia, and painted his best-known work, 'B

54 of these impacted neural networks in primary progressive aphasia are lacking.

55 ristics of early and mild disease in primary progressive aphasia are poorly understood.

56 Revisions of criteria for logopenic primary progressive aphasia are proposed to address these challe

57 fty-eight autopsies of patients with primary progressive aphasia are reported.

58 volution of the logopenic variant of primary progressive aphasia as a distinct clinical entity and to

59 er leftward asymmetry for tangles in primary progressive aphasia but not in the amnestic Alzheimer-ty

60 speech production in each variant of primary progressive aphasia, by quantifying speech output along

61 A review of published primary progressive aphasia cases with adequate clinical and his

62 re not universal and that individual primary progressive aphasia cases with Alzheimer pathology exist

63 Francisco Memory and Aging Center's primary progressive aphasia cohort (n = 198) for history of lang

64 comprehension in 51 individuals with primary progressive aphasia, composed of all clinical variants a

65 es included frontotemporal dementia, primary progressive aphasia, corticobasal syndrome, and Alzheime

66 atrophy and the logopenic variant of primary progressive aphasia, differ from amnestic AD in distribu

67 while patients with semantic variant primary progressive aphasia discounted delayed rewards more stee

68 The Alzheimer's disease pathology in primary progressive aphasia displayed multiple atypical features

69 nsecutive series of 18 patients with primary progressive aphasia (eight with semantic variant, six wi

70 rtical-to-entorhinal tangle ratio in primary progressive aphasia establishes clinical concordance of

71 asia (nonfluent PPA; n = 15), fluent primary progressive aphasia (fluent PPA; n = 7), and amyotrophic

72 entral auditory impairment exists in primary progressive aphasia for non-linguistic stimuli.

73 t to that seen in the fluent form of primary progressive aphasia (fPPA), a neurodegenerative disease

74 stinguishing the semantic variant of primary progressive aphasia from the partially overlapping group

75 over, patients with semantic variant primary progressive aphasia had a significantly more prominent d

76 Patients with primary progressive aphasia had deficits of non-verbal sound ana

77 up, all participants with non-fluent primary progressive aphasia had evolved either corticobasal dege

78 developmental learning disability in primary progressive aphasia has been reported.

79 he connected speech of patients with primary progressive aphasia has often been dichotomized simply a

80 ore making a definitive diagnosis of primary progressive aphasia has promoted diagnostic specificity,

81 Observations in semantic variant primary progressive aphasia have inspired an alternative model f

82 atients with logopenic and nonfluent primary progressive aphasia have some deficits recognizing emoti

83 ssociated with syntactic deficits in primary progressive aphasia in a number of structural and functi

84 logical relationships in subtypes of primary progressive aphasia in hopes of utilizing language pheno

85 We examined 39 patients with primary progressive aphasia including logopenic variant (n = 14,

86 nt presenting with logopenic variant primary progressive aphasia initially thought to be due to Alzhe

87 Primary progressive aphasia is a clinical syndrome defined by pr

88 Primary progressive aphasia is a clinical syndrome that encompas

89 Primary progressive aphasia is a neurodegenerative clinical synd

90 Primary progressive aphasia is a neurodegenerative syndrome char

91 Primary progressive aphasia is a syndrome characterized by progr

92 l categorization of patients presenting with progressive aphasia is an area of controversy.

93 Progressive aphasia is best seen as a composite of two c

94 y contribute to the phenomenology of primary progressive aphasia is not established.

95 or natural kinds in semantic variant primary progressive aphasia is related in part to disease in vis

96 ecutive deficits), logopenic variant primary progressive aphasia (language deficits), and posterior c

97 Patients with logopenic variant primary progressive aphasia ('language variant of Alzheimer's di

98 = 10), and a third variant termed logopenic progressive aphasia (LPA; n = 10).

99 t subjects with logopenic variant of primary progressive aphasia (lvPPA) have beta-amyloid (Abeta) de

100 ipants with the logopenic variant of primary progressive aphasia (lvPPA) performed a recognition task

101 cortical atrophy (PCA), 12 logopenic primary progressive aphasia (lvPPA), 20 behavioural variant FTD

102 cortical atrophy in semantic variant primary progressive aphasia may follow connectional pathways wit

103 progressive biparietal syndrome (n = 2) and progressive aphasia (n = 6).

104 se, semantic dementia and non-fluent primary progressive aphasia (n = 9 each) were contrasted with co

105 n = 13; behavioural variant, n = 14; primary progressive aphasias, n = 21) and 27 control subjects.

106 The non-fluent/agrammatic variant of primary progressive aphasia (naPPA) is a young-onset neurodegene

107 the non-fluent/agrammatic variant of primary progressive aphasia (naPPA), but well-controlled clinica

108 three different clinical variants: nonfluent progressive aphasia (NFPA; n = 11), semantic dementia (S

109 n patients with nonfluent/agrammatic primary progressive aphasia (nfvPPA) and progressive supranuclea

110 asia (svPPA), (4) non-fluent variant primary progressive aphasia (nfvPPA) or (5) early onset Alzheime

111 svPPA), five with non-fluent variant primary progressive aphasia (nfvPPA)) and 17 healthy control sub

112 fficulty, in patients with nonfluent primary progressive aphasia (nonfluent PPA; n = 15), fluent prim

113 This study selected 15 patients with progressive aphasia on broad criteria, excluding only th

114 arger groups of patients with either primary progressive aphasia or a typical amnestic dementia.

115 Cases with predominant primary progressive aphasia or extra-pyramidal syndromes were ex

116 mporal dementia and semantic variant primary progressive aphasia patients alone confirmed this result

117 to a lesser extent or not at all in primary progressive aphasia patients whose syntax was relatively

118 ic and symmetric pathology cause the primary progressive aphasia phenotype, characterized by relative

119 ant frontotemporal dementia (bvFTD), primary progressive aphasia (PPA) and corticobasal syndrome (CBS

120 The dementia syndrome of primary progressive aphasia (PPA) can be caused by 1 of several

121 Primary Progressive Aphasia (PPA) is a behaviorally focal dement

122 Primary progressive aphasia (PPA) is a clinical dementia syndrom

123 Primary progressive aphasia (PPA) is a clinical syndrome charact

124 Primary progressive aphasia (PPA) is a focal dementia characteri

125 Primary progressive aphasia (PPA) is a neurodegenerative dementi

126 Primary progressive aphasia (PPA) is a neurodegenerative syndrom

127 Primary progressive aphasia (PPA) is a progressive language diso

128 Noninvasive brain stimulation in primary progressive aphasia (PPA) is a promising approach.

129 Primary progressive aphasia (PPA) is an uncommon degenerative de

130 Primary progressive aphasia (PPA) is characterized by an isolate

131 The semantic variant of primary progressive aphasia (PPA) is characterized by the combin

132 Primary progressive aphasia (PPA) refers to a disorder of declin

133 s in a large cohort of patients with primary progressive aphasia (PPA) variants defined by current di

134 nd genetic study of 31 patients with primary progressive aphasia (PPA), a decline in language functio

135 Primary progressive aphasia (PPA), a selective neurodegeneration

136 in up to one third of patients with primary progressive aphasia (PPA), but clinical features that pr

137 In the nonfluent variant of primary progressive aphasia (PPA), degeneration of the posterior

138 trophic lateral sclerosis (ALS), and primary progressive aphasia (PPA), including 281 AD, 256 ALS, 39

139 In primary progressive aphasia (PPA), speech and language difficult

140 word retrieval is a main symptom of primary progressive aphasia (PPA).

141 re-dominant pattern of deposition in primary progressive aphasia (PPA).

142 syntactic comprehension deficits in primary progressive aphasia reflect not only structural and func

143 ge network for the logopenic variant primary progressive aphasia region of interest, and the higher v

144 the non-fluent/agrammatic variant of primary progressive aphasia relates to the strength of connectiv

145 al patients in both semantic variant primary progressive aphasia samples.

146 phy in non-fluent/agrammatic variant primary progressive aphasia spreads over time from a syndrome-sp

147 as matched in age and gender to each primary progressive aphasia subgroup (n = 20, age = 65 +/- 5 yea

148 white matter tracts in the different primary progressive aphasia subtypes.

149 predilection for one or more of the primary progressive aphasia subtypes.

150 The semantic variant of primary progressive aphasia (svPPA) is typically associated with

151 tia (rtFTD), (3) semantic variant of primary progressive aphasia (svPPA), (4) non-fluent variant prim

152 (bvFTD), eight with semantic variant primary progressive aphasia (svPPA), five with non-fluent varian

153 a, including the semantic variant of primary progressive aphasia (svPPA), is strongly associated with

154 g state neuronal synchronizations in primary progressive aphasia syndromes.

155 s with bvFTD and semantic variant of primary progressive aphasia than in those with AD and is more li

156 e logopenic variant is a distinct subtype of progressive aphasia that may hold value as a predictor o

157 Within semantic variant primary progressive aphasia the right-handed and non-right-hande

158 um and caudate nucleus in non-fluent primary progressive aphasia (the corticobasal degeneration/progr

159 findings expand the differential of primary progressive aphasia to include prion disease.

160 ated performance in semantic variant primary progressive aphasia to ventral and medial portions of th

161 egion of atrophy in semantic variant primary progressive aphasia using cortical thickness analysis in

162 syntactic deficits in patients with primary progressive aphasia, using multimodal neuroimaging and n

163 ional spectral power changes in each primary progressive aphasia variant, compared to age-matched con

164 Each of the primary progressive aphasia variants showed different patterns o

165 owed significant differences between primary progressive aphasia variants themselves.

166 ork-specific neuronal dysfunction in primary progressive aphasia variants.

167 the non-fluent/agrammatic variant of primary progressive aphasia was derived in a group of 10 mildly

168 No subject with a diagnosis of primary progressive aphasia was identified with this mutation.

169 for all pathologies associated with primary progressive aphasia was the asymmetric prominence of atr

170 mporal dementia and semantic variant primary progressive aphasia were most likely to exhibit disgusti

171 atients with the semantic subtype of primary progressive aphasia, which is associated with marked tem

172 y were addressed in 72 patients with primary progressive aphasia who collectively displayed a wide sp

173 The association of logopenic primary progressive aphasia with Alzheimer's disease pathology w

174 arch has associated semantic variant primary progressive aphasia with distributed cortical atrophy th

175 Behaviourally, patients with primary progressive aphasia with non-semantic subtypes were seve

176 ealthy controls and in patients with primary progressive aphasia with relatively spared syntax, but t

177 imer's disease and logopenic variant primary progressive aphasia), with a trend towards lower (18)F-l

178 displays an atypical distribution in primary progressive aphasia yielded inconclusive results.