ライフサイエンスコーパス: prolactin

1 wo independent BCL6 small hairpin RNAs or by prolactin.

2 the nursing mother is induced by the hormone prolactin.

3 turn, serotonin stimulates the secretion of prolactin.

4 also found to be differentially sensitive to prolactin.

5 on, we continuously infused female mice with prolactin.

6 ucleus (ARC) to maintain low levels of serum prolactin.

7 y by the coordinated actions of TGF-beta and prolactin.

8 only by a very high dose of unlabeled ovine prolactin.

9 rons that normally suppress the secretion of prolactin.

10 likely mediated by elevations of the hormone prolactin.

11 by investigating transport of (125)I-labeled prolactin ((125)I-prolactin) into the brain of female mi

12 [3.19] vs 4.56 [2.01] mIU/mL; P < .001), and prolactin (14.14 [9.48] vs 9.97 [3.12] ng/mL; P = .01) a

13 o model to characterize the effects of DHEA, prolactin, 17beta-estradiol on insulin-growth factor-1 a

14 phere), CA-125 (53 mum diameter sphere), and prolactin (63 mum diameter sphere) in a single PBS assay

15 Prolactin, a key regulator of 20alpha-HSD, was lower (P=

16 udy also demonstrated that AFAP1 responds to prolactin, a lactogenic hormone, by forming a complex wi

17 is circuit markedly reduced the capacity for prolactin action both in the MPOA and throughout the net

18 Here, we evaluated the role of prolactin action in the MPOA using complementary genetic

19 l mothers abandoned their pups, showing that prolactin action on MPOA neurons is necessary for the no

20 nduced phosphorylation of STAT5, a marker of prolactin action.

21 Compliant matrices promoted physiological prolactin actions and activation of STAT5, whereas stiff

22 We report here for the first time that prolactin activates extracellular signal-regulated kinas

23 er systemic or intracerebroventricular (icv) prolactin administration.

24 Prolactin also suppressed 3-ketosteroid induction of CK5

25 the functional epitopes for site 1 of human prolactin and also identifies a set of residues that sup

26 ignificantly reduced secretion of IGFBP1 and prolactin and altered the expression of endometrial rece

27 ed with nuclear pSTAT5 in response to 500 ng prolactin and appeared to be more sensitive than dopamin

28 pendent synthesis and secretion of autocrine prolactin and downstream activation of the prolactin rec

29 bulins and thus displayed signals from total prolactin and IgG-bounded prolactin (macroprolactin) cor

30 that enables inducible hepatic production of prolactin and its cleavage product.

31 y despite normal levels of circulating serum prolactin and pituitary prolactin production.

32 Prolactin and placental lactogen signal through the prol

33 tudies investigating the association between prolactin and type 2 diabetes beyond pregnancy are rare

34 ment of endometrial decidualization (IGFBP1, prolactin) and c) endometrial receptivity (SPP1, MAOA, E

35 tradiol, testosterone, progesterone, hGH and prolactin) and the obtained results demonstrated an exce

36 tive protein, interleukin-6, growth hormone, prolactin, and cortisol were analyzed.

37 Growth hormone (GH), prolactin, and insulin are involved in P450 regulation,

38 driver pathways, including Wnt/beta-catenin, Prolactin, and insulin-like growth factor (IGF)1 signali

39 correlates of severity, including JAK/STAT, prolactin, and interleukin 9 signaling.

40 We assessed plasma levels of progesterone, prolactin, and lipids and placental expression of genes

41 hetic effects, increased plasma cortisol and prolactin, and reduced resting electroencephalogram spec

42 estosterone, dehydroepiandrosterone sulfate, prolactin, and sex hormone-binding globulin (SHBG) impro

43 ted hormones: estrone sulfate, testosterone, prolactin, and SHBG; change in AUC, 8.8 [P < .001] for G

44 t inhibition of JAK2 significantly decreases prolactin- and interferon gamma (IFN-gamma)-induced tyro

45 ons of the ECM to the physiologic actions of prolactin are increasingly understood, little is known a

46 ptor, connexin 43, and cyclooxygenase-2) and prolactins are down-regulated in pregnant smtnl1(-/-) mi

47 Collectively, these data unveiled prolactin as a retinal trophic factor that may regulate

48 tiangiogenic fragment of the nursing hormone prolactin as playing an important role.

49 We aimed to examine whether prolactin associates with type 2 diabetes prospectively

50 Total prolactin bioluminescence immunoassay in standard, contr

51 androgen activity) correlated with serotonin/prolactin, but posited aromatase activity correlated sig

52 pike duration of TIDA cells, increased serum prolactin can promote dopamine release to limit its own

53 Prolactin caused no significant change in IGF-1 levels a

54 Because prolactin communicates with immune cells during pregnanc

55 Tg mice were fed chow containing IC3, plasma prolactin concentrations increased threefold, BP increas

56 Therefore, serum prolactin concentrations reflect endogenous serotonin.

57 he Tg mice were fed normal chow (NC), plasma prolactin concentrations were comparable to those in fem

58 Prolactin controls the development and function of milk-

59 nsulin-like growth factor binding protein-3, prolactin, cortisol, adrenocorticotropic hormone, thyroi

60 Here we report that prolactin counteracts induction of the CK5-positive popu

61 However, the mechanism by which prolactin crosses the blood-brain barrier and enters the

62 yroid stimulating hormone, gonadotropin, and prolactin deficiencies, whereas for ACTH, growth hormone

63 The lowest icv prolactin dose (10 ng) induced pSTAT5 in the ARC, but wi

64 ontribute the enhancer/promoter for decidual prolactin (dPRL), which is dramatically induced during p

65 e findings identify a function for autocrine prolactin during normal development and demonstrate its

66 activation, progesterone receptor activity, prolactin effects, and aspects of estrogen receptor beta

67 rrelated these properties with their EPS and prolactin-elevating liabilities at therapeutic doses.

68 tor, while dissociation rates correlate with prolactin elevation.

69 The anterior pituitary hormone prolactin exerts important physiologic actions in the br

70 h a strong transcriptional activation of the prolactin family 2 subfamily c of growth factors.

71 ranscription array screen, we identified the prolactin family member proliferin (PLF1 and PLF4) as a

72 s evidenced by significantly lower levels of prolactin, fasting glucose, total cholesterol, and trigl

73 ne (LH), follicle-stimulating hormone (FSH), prolactin, fasting plasma glucose, and insulin levels we

74 te-based immunoassay allows detection of two prolactin forms in crude serum without additional manipu

75 cl-2, HER2, p53, E-cadherin, Ki67, survivin, prolactin, FOXA1) for survival impact.

76 M is driven in part by the 16-kDa N-terminal prolactin fragment (16K PRL), the underlying molecular m

77 et al. explore signaling downstream of this prolactin fragment and demonstrate that miR-146a is a cr

78 at CSF is not the major route by which blood prolactin gains access to neurons in the brain.

79 ctions allowed local spatial coordination of prolactin gene expression.

80 ith an anti-Prl antibody, or deletion of the prolactin gene.

81 We have shown previously that the murine prolactin/growth hormone family member proliferin plays

82 nfluramine significantly increased serotonin/prolactin in all groups (p < .0001).

83 fferences in the accessibility of the ARC to prolactin in blood may influence the responsiveness of t

84 resents a novel prodifferentiation effect of prolactin in breast cancer.

85 s, which may be explained by lower levels of prolactin in males.

86 n hypothesized that the receptor may bind to prolactin in the blood and translocate it into the cereb

87 Fenfluramine-induced serotonin/prolactin in the T-treated group was significantly highe

88 for maternal behavior, the specific role of prolactin in this brain region has remained elusive.

89 In stiff matrices, prolactin increased SRC family kinase-dependent phosphor

90 Prolactin increases proliferation and cell motility, pro

91 In vivo mouse studies found that local prolactin induced the amplification of prostate epitheli

92 , downstream signaling mechanisms underlying prolactin-induced adult neurogenesis are completely unkn

93 and critical signaling mechanism underlying prolactin-induced adult neurogenesis.

94 Our data establish a critical role for prolactin-induced behavioral responses in the maternal b

95 The two components of the prolactin-induced current appear to be mediated through

96 The underlying prolactin-induced current is composed of separate low- a

97 tivator of Transcription (STAT) 5A/B mediate prolactin-induced mammary development during pregnancy.

98 r, non-redundant functions are restricted to prolactin-induced mammary gland development and function

99 dly activates brain neurons, as evidenced by prolactin-induced phosphorylation of signal transducer a

100 This was confirmed by a significant loss of prolactin-induced phosphorylation of STAT5, a marker of

101 Therefore, we compared prolactin-induced signaling in different hypothalamic ne

102 its neurogenesis in the SVZ and OB following prolactin infusion or mating/pregnancy.

103 s-of-function mutation in PRLR, resulting in prolactin insensitivity.

104 Reduction of prolactin, insulin-like growth factor binding protein-3,

105 rolactin signals and that stiff matrices and prolactin interact in a feed-forward loop in breast canc

106 s the first reported evidence of altered ECM-prolactin interactions in breast cancer, suggesting the

107 The transport of prolactin into the brain was saturable, with transport e

108 ransport of (125)I-labeled prolactin ((125)I-prolactin) into the brain of female mice in the presence

109 This corresponds with biologic data that prolactin is etiologically important in tumor promotion.

110 Prolactin is internalised into early endosomes, where th

111 In the presence of ECM, prolactin is internalised via a clathrin-dependent, but

112 We now report that autocrine prolactin is required for terminal mammary epithelial di

113 Thus, a threefold increase in plasma prolactin is sufficient to increase BP significantly and

114 trophic axis, in which the pituitary hormone prolactin is tonically inhibited by tuberoinfundibular d

115 By using human prolactin knock-in mice, we demonstrate that prolactin-S

116 e was an increased risk for higher proximate prolactin levels [RR, >15.7 vs. </=8.1 ng/mL (i.e., top

117 Clinically, circulating prolactin levels and density of the extracellular matrix

118 cid (5-HIAA) and fenfluramine-induced plasma prolactin levels are markedly diminished in the Tph2 kno

119 ive neural circuit, indicating that changing prolactin levels can act at multiple sites and thus, imp

120 Following lactation, prolactin levels decline and mammary-specific STAT5-depe

121 Elevated prolactin levels during pregnancy and lactation may medi

122 No significant change was observed in serum prolactin levels following Opra Kappa administration, bu

123 Serum prolactin levels increased significantly, and to a compa

124 est that pregnant women with abnormally high prolactin levels may need special attention.

125 Our data suggest that prolactin levels measured <10 years before diagnosis are

126 on of 20alpha-HSD, which may be due to lower prolactin levels observed in these women.

127 s (RR) and 95% confidence intervals (CI) for prolactin levels on samples collected <10 (proximate) ve

128 These drugs are well known to elevate serum prolactin levels to varying degrees.

129 d high aldosterone, growth hormone (GH), and prolactin levels, thereby presumably fostering efficient

130 output of the 5HT system was assessed using prolactin levels.

131 nd induces molting, with no change in plasma prolactin levels.

132 hile CORT-KOs displayed higher GH- and lower prolactin-levels than controls under both diets.

133 et conditions as SST-KOs presented higher GH/prolactin-levels, while CORT-KOs displayed higher GH- an

134 signals from total prolactin and IgG-bounded prolactin (macroprolactin) correspondingly.

135 ng a physiological requirement for autocrine prolactin, mammary glands from lactation-defective Akt1(

136 Prolactin may be a mediator in the pathogenesis of type

137 neurons express prolactin receptors (Prlr), prolactin may regulate GABA secretion from TIDA neurons,

138 Overall, 2,468 cases and 4,021 controls had prolactin measured <10 years and 953 cases and 1,339 con

139 ficantly higher maximum mean levels of serum prolactin (men, 34.56 microg/L [95% CI, 29.75-39.37] vs

140 Increased levels of a cleaved form of prolactin (molecular weight 16 kDa) have been associated

141 free triiodothyronine, parathyroid hormone, prolactin, N-terminal pro-brain natriuretic peptide, 25-

142 To study the effects of prolactin on blood pressure (BP), we generated male mice

143 DA neurons, potentially mediating actions of prolactin on hypothalamic function.

144 );Akt2(+/-) mice failed to express autocrine prolactin or activate Stat5 during late pregnancy despit

145 ences between the groups were seen regarding prolactin or cortisol.

146 cing the PrP(C) signal sequence with that of prolactin or osteopontin.

147 dy temperature, pupil size, plasma cortisol, prolactin, oxytocin, and epinephrine.

148 atase activity correlated significantly with prolactin (p < .0008; r(2) = 0.95).

149 Both TGF-beta and prolactin pathways are crucial regulators of this proces

150 Prolactin plays an important role in maintaining a norma

151 Most conspicuously, mutations in prolactin (PRL) and its receptor (PRLR) have an impact o

152 d mediated mainly by the lactogenic hormones prolactin (PRL) and placental lactogens.

153 We identified prolactin (PRL) as a candidate autocrine factor.

154 The hormone prolactin (PRL) contributes to breast cancer pathogenesi

155 The hormone prolactin (PRL) frequently increases in the circulation

156 The hormone prolactin (PRL) has long been debated as a potential imm

157 studies have revealed an important role for prolactin (PRL) in breast cancer.

158 Accumulating evidence supports a role for prolactin (PRL) in the development and progression of hu

159 estigated the role of the lactogenic hormone prolactin (PRL) in the regulation of ABCG2.

160 Extrapituitary prolactin (Prl) is produced in humans and rodents; howev

161 s during pregnancy and lactation, when serum prolactin (Prl) levels are highly elevated.

162 cohol consumption has been shown to increase prolactin (PRL) production and cell proliferation of pit

163 Prolactin (PRL) regulates activity of nociceptors and ca

164 nses: proliferation, caspase activation, and prolactin (PRL) release.

165 (TIDA) neurons are the central regulators of prolactin (PRL) secretion.

166 The polypeptide hormone prolactin (PRL) stimulates breast epithelial cell growth

167 The lactogenic hormone prolactin (PRL) transcriptionally increases ZnT2 express

168 The mammotrophic hormone, prolactin (PRL), and/or its receptor are also expressed

169 e distribution of pituitary hormones such as prolactin (PRL), growth hormone (GH), adrenocorticotropi

170 phosphorylated Ser305-ERalpha in response to prolactin (PRL), implying that maximal ERalpha phosphory

171 at the estrogen-responsive pituitary hormone prolactin (PRL), signaling through hepatocyte-predominan

172 lating concentrations of total testosterone, prolactin (PRL), thyroid stimulating hormone (TSH), free

173 We have previously shown that the prolactin (PRL)-activated tyrosine kinase JAK2 phosphory

174 tent by somatolactotropes, the precursors of prolactin (PRL)-producing lactotropes.

175 xpression pattern of growth hormone (Gh) and prolactin (Prl).

176 on VEGF receptor 2 (VEGFR2) expressed in PD prolactin-producing cells known to impair gonadotrophin

177 necessary and sufficient to induce autocrine prolactin production in the mammary gland, Stat5 activat

178 of circulating serum prolactin and pituitary prolactin production.

179 The hormone prolactin promotes lactational differentiation of mammar

180 In mouse models, prolactin promotes mammary carcinomas that resemble lumi

181 We explored whether the hormone prolactin provides trophic support to retinal cells, thu

182 In response to systemic prolactin, pSTAT5-labeled cells were widely observed in

183 Peripherally administered prolactin rapidly activates brain neurons, as evidenced

184 To determine why, we explored the human PRL-prolactin receptor (hPRLR)-Janus kinase 2 (JAK2)-signal

185 male mice in the presence and absence of the prolactin receptor (PRLR(-/-)).

186 in and placental lactogen signal through the prolactin receptor (PRLR) and contribute to adaptive bet

187 ), is significantly reduced due to decreased prolactin receptor (Prlr) and ErbB4 expression in Xbp1-d

188 show that CN/Nfatc1 regulates expression of prolactin receptor (Prlr) and that canonical activation

189 led that >98% of ovarian cancers express the prolactin receptor (PRLR), forming the basis of a new mo

190 e prolactin and downstream activation of the prolactin receptor (Prlr)-Jak-Stat5 pathway.

191 Many genes that are activated by the prolactin receptor are associated with tumorigenesis and

192 We have scrutinized the prolactin receptor as an archetype model of homodimeric

193 olecular architecture of the monomeric human prolactin receptor by combining experimental and computa

194 t were due to a heterozygous mutation in the prolactin receptor gene, PRLR, resulting in an amino aci

195 On the basis of high expression of the prolactin receptor in the choroid plexus, it has been hy

196 The prolactin receptor is an archetype member of the class I

197 Overexpression of the prolactin receptor is seen in more than 95% of human bre

198 tion was associated with reductions in islet prolactin receptor levels, STAT5 nuclear localization an

199 h impaired intercellular junction formation, prolactin receptor trafficking, and alveolar lumen devel

200 igh-affinity ligand-binding interface of the prolactin receptor, resulting in a loss of downstream si

201 Using a novel conditional deletion of the prolactin receptor, we have identified functional subpop

202 Moreover, retinas from prolactin receptor-deficient mice exhibited photorespons

203 st cancer progression and therapy as loss of prolactin receptor-Stat5 signaling occurs frequently and

204 , indicating transport is independent of the prolactin receptor.

205 We found that MPOA neurons expressing prolactin receptors (Prlr) form the nexus of a complex p

206 As these neurons express prolactin receptors (Prlr), prolactin may regulate GABA

207 ng through hepatocyte-predominant short-form prolactin receptors (PRLR-S), constrained TNF receptor-a

208 f matrices also increased co-localization of prolactin receptors and integrin-activated FAK, implicat

209 e brain in PRLR(-/-) mice lacking functional prolactin receptors compared to control mice, indicating

210 In this model, the expression levels of prolactin receptors in the retina were upregulated.

211 tinal polypeptide, purinergic, androgen, and prolactin receptors were also expressed in gland of Wolf

212 Kisspeptin neurons, which express prolactin receptors, were recently identified as major r

213 w CSF 5-HIAA and fenfluramine-induced plasma prolactin reflects chronic, endogenous central nervous s

214 Here, hormone release studies show that prolactin release from isolated rat lactotrophs stimulat

215 the frontal cortex following treatment while prolactin release was blunted, suggesting desensitizatio

216 and 2.5-5 mm dbcAMP) these agents stimulate prolactin release, an inhibition is measured at higher c

217 symptoms, weight gain, sedation, increase in prolactin release, overall functioning, and quality of l

218 ecstasy") and blunted d-fenfluramine-induced prolactin release, substantiating the importance of alph

219 his may serve as a feedforward inhibition of prolactin release.

220 acilitate homeostatic feedback regulation of prolactin release.

221 erior to risperidone in terms of increase in prolactin release.

222 e activation of GPR10 by its cognate ligand, prolactin releasing peptide, promotes PI3K-AKT-mTOR path

223 peptide-1 (GLP-1) neurons and noradrenergic prolactin-releasing peptide (PrRP) neurons participate i

224 n neurons that are immunoreactive for either prolactin-releasing peptide or glucagon-like peptide 1,

225 s obtained for the PICK1 interaction partner prolactin-releasing peptide receptor (GPR10).

226 The prolactin-releasing peptide receptor and its bioactive R

227 sufficient for full signal transduction for prolactin-releasing peptide, rather than a deep, membran

228 l, epidermal growth factor, myeloperoxidase, prolactin, resistin and soluble tumor necrosis factor al

229 Castration partially reduced the serotonin/prolactin response and Letrozole partially blocked the e

230 y reduced the fenfluramine-induced serotonin/prolactin response in the presence or absence of DHT.

231 SF 5-HIAA and a blunted fenfluramine-induced prolactin response, but also blunted 5-HT(1A) agonist-in

232 receptors (Prlr) form the nexus of a complex prolactin-responsive neural circuit, indicating that cha

233 We characterized prolactin-responsive neurons within the MPOA at differen

234 factor, STAT5 (pSTAT5), was used to identify prolactin-responsive neurons.

235 ion, including the effects of medications on prolactin secretion and the complexities of making contr

236 tor agonist prescribed for the inhibition of prolactin secretion and treatment of Parkinson disease,

237 findings suggest that oxytocin can modulate prolactin secretion by exciting TIDA neurons, and that t

238 contribute to the dopaminergic inhibition of prolactin secretion diurnally, as their neuromedin S(+)

239 urons, known as neuroendocrine regulators of prolactin secretion from the pituitary gland, also relea

240 serotonin (5-HT) and SSRIs cause changes in prolactin secretion is not known.

241 he dynamic dopaminergic control of pituitary prolactin secretion, a key reproductive hormone.

242 ons are involved in the control of pituitary prolactin secretion, and the GABAergic subpopulation may

243 endocrine neurons involved in the control of prolactin secretion, we have shown that approximately ha

244 not necessary for the feedback regulation of prolactin secretion.

245 because they are required to regulate basal prolactin secretion.

246 pamine (TIDA) neurons, the main inhibitor of prolactin secretion.

247 ctional role distinct from the regulation of prolactin secretion.

248 rons had no effect on feedback regulation of prolactin secretion.

249 that the SST/CORT role in the control of GH/prolactin secretions is maintained under LF- and HF-diet

250 neurons that were not previously known to be prolactin-sensitive, notably in the medial amygdala, wer

251 Pregnancy hormones, such as prolactin, sensitize neural circuits controlling parenta

252 adjustment, women in the highest quartile of prolactin showed the lowest risk for diabetes compared w

253 The endoplasmic reticulum-targeting prolactin signal sequence did not affect StAR associatio

254 Prolactin/signal transducer and activator of transcripti

255 Furthermore, enhanced prolactin signaling also led to amplification of a lumin

256 The transcription factor STAT5 mediates prolactin signaling and controls functional development

257 , thereby sharply increasing the activity of prolactin signaling at the onset of lactation.

258 functional relationship between the ECM and prolactin signaling in breast cancer.

259 filing further revealed a potential role for prolactin signaling in regulating BCR editing.

260 TGF-beta signaling, coordinates TGF-beta and prolactin signaling to control alveologenesis and lactog

261 in CKO mice without interrupting full-length prolactin signaling, as indicated by normal nursing acti

262 uit is robust with respect to alterations in prolactin signaling.

263 Prolactin signalling depends on a cross-talk with baseme

264 alling, although the role of endocytosis for prolactin signalling is not known.

265 e biological processes, mediated through the prolactin signalling pathway.

266 s is a powerful regulator of the spectrum of prolactin signals and that stiff matrices and prolactin

267 rix stiffness potently regulates a switch in prolactin signals from physiologic to protumorigenic out

268 d consequences of increased ECM stiffness on prolactin signals to luminal breast cancer cells in thre

269 In the presence of prolactin, spontaneously oscillating TIDA cells depolari

270 A principal signaling mediator of prolactin, Stat5, promotes cellular differentiation of b

271 prolactin knock-in mice, we demonstrate that prolactin-Stat5a/b signaling promoted metastases formati

272 Prolactin-stimulated adult neurogenesis in the subventri

273 by retroviral infection of shRNA attenuates prolactin-stimulated neurogenesis in SVZ-derived adult n

274 Prolactin stimulates dopamine release from neuroendocrin

275 Prolactin stimulates milk production, whereas oxytocin i

276 the central electrophysiological actions of prolactin suggests a novel feedback mechanism.

277 Prolactin suppressed Pg-induced BCL6 through Jak2-Stat5

278 were documented in the MCF-7 cell line, and prolactin synthesis was assessed in GH3 rat pituitary tu

279 ed that NEDA neurons would be more sensitive prolactin targets than neurons in other regions because

280 Vasoinhibins are N-terminal fragments of prolactin that prevent BRB breakdown during diabetes.

281 drugs to elevate blood levels of the hormone prolactin, the mechanism for this hormonal imbalance is

282 ing mice with chronically elevated levels of prolactin, the rate of (125)I-prolactin transport into t

283 y alveoli during pregnancy are controlled by prolactin through the transcription factors STAT5A and S

284 stages of prostate tumorigenesis induced by prolactin to help determine whether this hormone or its

285 the accessibility of the arcuate nucleus to prolactin, together with intrinsic differences in the NE

286 in prostate epithelium of prostate-specific prolactin-transgenic mice.

287 There was no change in the rate of (125)I-prolactin transport into the brain in PRLR(-/-) mice lac

288 These data suggest that prolactin transport into the brain involves another as y

289 ated levels of prolactin, the rate of (125)I-prolactin transport into the brain was significantly inc

290 exual stimulation and coitus in female mice; prolactin-triggered oviductal fluid secretion clears the

291 o be involved in mammalian placentation: the prolactins (two clusters), serpins, cathepsins, and the

292 We measured prolactin via immunoassay in cases diagnosed from 1990 t

293 Expression of the decidualisation marker prolactin was decreased in Ct-infected ESC at both mRNA

294 e immunoassay (BLIA) for total and IgG-bound prolactins was developed on the base of Ca(2+)-regulated

295 The effects of increased plasma prolactin were abolished by a genetic absence of eNOS.

296 Estrone sulfate, testosterone, and prolactin were selected by stepwise regression and incre

297 Because CSF levels of (125)I-prolactin were very low, even up to 90 min after adminis

298 systems (growth hormone, erythropoietin, and prolactin) were studied, and the focus was on the bindin

299 e weight gain and greater increases in serum prolactin, whereas haloperidol decanoate was associated

300 Levels of prolactin, which exerts down-regulating control on P450s