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1 lation of their differentiated phenotype and proliferate.
2 r metabolism to enable tumors to survive and proliferate.
3 ated with the ability of cardiac myocytes to proliferate.
4 ed macrophages to overcome host defenses and proliferate.
5 Cs had not redifferentiated and continued to proliferate.
6 aryotic cells that allow them to survive and proliferate.
7  to T cells, which in turn are activated and proliferate.
8 Barr virus (EBV) become activated, grow, and proliferate.
9 ma cells to effectively invade the brain and proliferate.
10 epend on the nucleotide synthesis pathway to proliferate.
11 gressive and stochastic fashion as the cells proliferated.
12 % of cells in the CC lining in adulthood are proliferating.
13 otein expression, exit quiescence, and begin proliferating.
14 ntagonism was observed as T. halophilus only proliferated (3 log increase) in the presence of Z. roux
15                          For cancer cells to proliferate, a balance must be built between biomass-for
16 ls lacking this activation mechanism fail to proliferate after hyperosmotic stress.
17 reated cells maintained a higher capacity to proliferate after removal of rapamycin and expressed mor
18 drocyte progenitors or polydendrocytes) also proliferate after SCI and accumulate in large numbers am
19                 PT-Cy selectively eliminates proliferating alloreactive T cells, but whether and how
20 culture either in bulk or individually, they proliferate and differentiate into both AT2- and AT1-lik
21 ents that initiate mesenchymal stem cells to proliferate and differentiate into odontoblast-like cell
22 pment, pancreatic progenitors simultaneously proliferate and differentiate into the endocrine, ductal
23                The decision of stem cells to proliferate and differentiate is finely controlled.
24 and, when recovered, retain their ability to proliferate and differentiate.
25 ct in RB1-negative cells, which continued to proliferate and express Ki-67.
26 eptive to embryo attachment by continuing to proliferate and failing to differentiate.
27 ad, cardiac endothelial cells were likely to proliferate and generate new endothelium following injur
28 ulsion or surface adhesion, enabling them to proliferate and invade new territories.
29 cellular decisions such as whether or not to proliferate and migrate.
30 une cells and CpG-B to activating B cells to proliferate and produce Abs and inflammatory cytokines.
31       We confirmed that the CD4+ lymphocytes proliferate and produce IFN-gamma in response to stimula
32 dentify a new mechanism by which human ILC3s proliferate and produce IL-22, and identify NF-kappaB as
33 o the inability of cells in the inner ear to proliferate and replace lost sensory receptors.
34 ted antigen-specific naive CD4(+) T cells to proliferate and secrete Th1-like cytokines.
35                                   ATII cells proliferate and spread onto the denuded basement membran
36 diseases, mitotically dormant cholangiocytes proliferate and subsequently acquire a neuroendocrine ph
37        Pancreatic endocrine progenitor cells proliferate and transiently express the bHLH transcripti
38 resident fibro/adipogenic progenitors (FAPs) proliferated and gave rise to adipocytes.
39                           Newer methods have proliferated and include quantitative PCR for organism l
40 and less than 2%, if any, of the total newly proliferated and molecularly confirmed scar-forming astr
41            Strikingly, BCAP(-/-) progenitors proliferated and produced more myeloid cells of both neu
42               Circulating T lymphocytes that proliferated and produced tumour necrosis factor alpha u
43 lood of piperacillin-hypersensitive patients proliferated and secreted TH1/TH2 cytokines alongside IL
44 MCs cultured under normal glucose conditions proliferated and synthesized ECM proteins in response to
45 as required to define the transition between proliferating and arrested cells inside the LRP, coincid
46                      Further, the numbers of proliferating and cytokine-producing lung ILC2s were inc
47  latent infection with HIV-1, while actively proliferating and differentiating HSPCs obtain predomina
48 0alpha1)-Cre-expressing cells, predominantly proliferating and hypertrophic chondrocytes, using "Cre-
49 AV challenge and assessed the percentages of proliferating and IAV-infected, alveolar type II (AECII)
50 coxib-treated lesions further contained less proliferating and more apoptotic cells and exhibited low
51 in proliferating granule cell precursors, in proliferating and post-mitotic granule cells, and in Pur
52             The existence and interaction of proliferating and quiescent intestinal stem cells have b
53 ide insight into proteasome dynamics between proliferating and quiescent yeast in response to cellula
54 ndances in all four cell strains and between proliferating and senescent cells; however, in the four
55 ed basal cell population able to self-renew, proliferate, and generate differentiated cells, consiste
56 ct both demonstrated that the cells adhered, proliferated, and maintained their phenotype well on the
57             However, CD8(+) dT degranulated, proliferated, and produced IFN-gamma, TNF-alpha, perfori
58 a leukemic stem cell (LSC) that self-renews, proliferates, and differentiates to give rise to a myelo
59   We show that CLEC3A is present in resting, proliferating, and hypertrophic growth-plate cartilage a
60 trast, equally capable of inducing diabetes, proliferating, and producing cytokines.
61 c reporting of health care data continues to proliferate as consumers and other stakeholders seek inf
62           Even though a very few hepatocytes proliferate at any given time in normal liver, the mecha
63 CBSCs were present in the MI border zone and proliferating at 72 hours post-MI but had no effect on i
64 on in digested lymphoid tissues, clusters of proliferating B cells with a GC-like phenotype can be ge
65 endrocyte precursor cells (OPCs) migrate and proliferate before differentiating into myelinating cell
66                                 HD10.6 cells proliferate by virtue of a transduced tetracycline-regul
67 nireview, we discuss the mechanisms by which proliferating cancer and T cells maintain a carefully co
68           The complex metabolic logic of the proliferating cancer cells' appetite for glutamine-which
69    Fetal or neonatal mouse hearts containing proliferating cardiac myocytes regenerate even extensive
70                                              Proliferating casein/T-effector cell counts were measure
71                                        These proliferating CD8 T cells had an effector-like phenotype
72 rns for core cell-cycle proteins in actively proliferating (CDK2-increasing) versus spontaneously qui
73  we report that teosinte branched1/cycloidea/proliferating cell factor1-20 (TCP20) and NIN-like prote
74                      During DNA replication, proliferating cell nuclear antigen (PCNA) adopts a ring-
75 ociated with decreased platinum drug-induced proliferating cell nuclear antigen (PCNA) and FANCD2 mon
76 th muscle mass, paired box protein 7 (Pax7), proliferating cell nuclear antigen (PCNA) and nicotinami
77  through its interactions with two proteins, Proliferating Cell Nuclear Antigen (PCNA) and Replicatio
78             In this study, we identified the proliferating cell nuclear antigen (PCNA) as a nIGF-1R-b
79 n multiple transient events in the reaction: proliferating cell nuclear antigen (PCNA) clamp binding/
80 s a mismatch, MutSalpha/beta, and DNA-loaded proliferating cell nuclear antigen (PCNA) for activation
81                                              Proliferating cell nuclear antigen (PCNA) forms a trimer
82                                              Proliferating cell nuclear antigen (PCNA) lies at the ce
83 loading of cyclin-dependent kinase (CDK) and proliferating cell nuclear antigen (PCNA) onto chromatin
84 n of Kdm4d impairs the recruitment of Cdc45, proliferating cell nuclear antigen (PCNA), and polymeras
85 A We show that Rad51 inhibits recruitment of proliferating cell nuclear antigen (PCNA), the platform
86 increased expression of Nox4, TNF-alpha, and proliferating cell nuclear antigen (PCNA).
87 we explore the interactions of S. cerevisiae Proliferating Cell Nuclear Antigen (yPCNA) with modified
88          In the absence of NKT cells hepatic proliferating cell nuclear antigen and cyclin B1 decreas
89                     Whereas association with proliferating cell nuclear antigen and participation in
90 incorporation and proportionate increases in proliferating cell nuclear antigen gene expression.
91                     LR assessed by Ki-67 and proliferating cell nuclear antigen was markedly decrease
92          Expression of hepatic cyclin B1 and proliferating cell nuclear antigen were evaluated by Wes
93                                    Ki-67 and proliferating cell nuclear antigen were used to measure
94  tethers it to the leading strand, and PCNA (proliferating cell nuclear antigen) binds tightly to Pol
95 were determined by methyltetrazolium, Ki-67, proliferating cell nuclear antigen, bromodeoxyuridine, a
96 gy with Cavalieri estimation; apoptosis with proliferating cell nuclear antigen, TUNEL, and caspase a
97 t ablate its ability to activate Cdk2 and to proliferate cells.
98                                              Proliferating cells acquire genome alterations during th
99 pression of LAT induced a reduction of brain proliferating cells and concomitant microcephaly.
100                                         When proliferating cells complete mitosis, a fraction of newl
101                 It is not clear how stem and proliferating cells cope with accumulating endogenous DN
102                                     However, proliferating cells depend on growth-factor-induced incr
103                                  The rapidly proliferating cells in plant meristems must be protected
104                  During early gonadogenesis, proliferating cells in the coelomic epithelium (CE) give
105                                      Rapidly proliferating cells increase glycolysis at the expense o
106     To maintain the integrity of the genome, proliferating cells must be able to block progression th
107                                    These non-proliferating cells occupy key cellular niches and elabo
108     YAP and WPB2 are upregulated in actively proliferating cells of mouse and human epidermis and cSC
109          Together, our findings suggest that proliferating cells rely on both MDH1 and LDH to repleni
110          Lipids are important nutrients that proliferating cells require to maintain energy homeostas
111                                              Proliferating cells showed undifferentiated morphology a
112                                      Rapidly proliferating cells switch from oxidative phosphorylatio
113                       RNA was extracted from proliferating cells versus differentiated neural cells a
114 insight about proteins apportioned for newly proliferating cells versus for somatic maintenance.
115 ion of mTORC1 and likely most predominant in proliferating cells where mTORC1 is highly active.
116                                           In proliferating cells, APOBEC3A modestly elicited ATR but
117                                           In proliferating cells, formin inhibition abolishes nuclear
118  capture the transcriptional dynamics within proliferating cells, methods to differentiate nascent tr
119   We observed that, in the limited subset of proliferating cells, most displayed fermentation of gluc
120   In contrast to the role of nuclear RNF8 in proliferating cells, RNF8 operates in the cytoplasm in n
121 assical signaling via RelA was essential for proliferating cells, whereas the alternative signaling p
122 e normally activated during cell division in proliferating cells.
123  be higher in spontaneously quiescent versus proliferating cells.
124 duced G2 checkpoint control and apoptosis in proliferating cells.
125  alterations and genetic diversity in normal proliferating cells.
126 ses the expression of some of these genes in proliferating cells.
127 ested cells, whereas the opposite is true in proliferating cells.
128 e, highly vascularized tumors that contained proliferating cells.
129 surement of deuterium enrichment into DNA of proliferating cells.
130 mes are essential for protein degradation in proliferating cells.
131 ts, primarily by inhibiting DNA synthesis in proliferating cells.
132  required to maintain metabolic stability in proliferating cells.
133 s, and with KLF4 in differentiating, but not proliferating, cells to promote expression of specialize
134 roteins such as the ring-shaped homotrimeric proliferating cellular nuclear antigen (PCNA).
135                                              Proliferating cholangiocytes and activated hepatic stell
136 ificity, and detects extramedullary sites of proliferating clonal plasma cells while providing import
137 es and dissected mouse placentae resulted in proliferating colonies that expressed known markers of T
138 ion, these T cells showed reduced ability to proliferate, confirming a state of T cell anergy.
139    Total body irradiation reduced numbers of proliferating crypts in Ah(Cre)/Met(fl/fl)/LacZ mice.
140 e single-cell measurements of p21 protein in proliferating cultures, we show that naturally occurring
141                                           To proliferate, developing Schwann cells (SCs) require YAP/
142 nervous system (ENS) progenitors to migrate, proliferate, differentiate, or survive within the distal
143 ct by promoting the switch between quiescent/proliferating/differentiating myoblasts and by maintaini
144  work suggests that the wtf multigene family proliferated due to meiotic drive and highlights the pow
145  the expression patterns of circular RNAs in proliferating (early-passage) and senescent (late-passag
146 the female gametophyte and in the zygote and proliferating endosperm of the Arabidopsis (Arabidopsis
147                                           In proliferating enterospheres derived from ENS progenitor
148 ion where it controls the renewal of rapidly proliferating epithelial (matrix) progenitors and hence
149       Both Cul9(-/-) and Cul9(Deltap53) MEFs proliferate faster and undergo spontaneous immortalizati
150            In addition, HBx-expressing cells proliferated faster than control and mutant HBx-expressi
151 G challenges induced marked accumulations of proliferating fibroblasts and of myofibroblasts, which w
152                  Reducing CircPVT1 levels in proliferating fibroblasts triggered senescence, as deter
153                         Biliary ductal cells proliferate from the portal areas of chronically damaged
154 nadal precursor cells and germ cells fail to proliferate fully and complete their respective differen
155 issue demonstrated ZNHIT3 to be expressed in proliferating granule cell precursors, in proliferating
156 ts the gene from being induced by Activin in proliferating hESCs.
157 ges: when stem cells of a particular lineage proliferate, how do the other lineages respond to mainta
158 il domain of SF1 is highly phosphorylated in proliferating human cells and is required for cell proli
159 pathways interact to maintain homeostasis in proliferating human cells, using fluorescent reporters f
160 pressed on the surface of senescent, but not proliferating, human diploid fibroblasts.
161  and IHH and, thereby control the transit of proliferating immature chondrocytes into mature hypertro
162 ellular synthetic lethality in quiescent and proliferating immature leukemia cells, and is thus a pot
163                    Fungal cells colonize and proliferate in distinct niches, from soil and plants to
164 te for the first time that Ct can infect and proliferate in ESC.
165 al heterotrophs, we demonstrated that Vibrio proliferate in response to a broad range of dust-Fe addi
166 is activation is required for SCs to rapidly proliferate in response to damage, repeated rounds of da
167  other inhibitory receptors and were able to proliferate in response to DENV-specific stimulation.
168 present in the normal liver but activate and proliferate in response to injury and inflammation.
169 dy, we show that latently infected cells can proliferate in response to mitogens without producing vi
170  that c-MPL(+) polyclonal T cells expand and proliferate in response to TPO, and persist longer after
171 show that pituitary-infiltrating lymphocytes proliferate in situ during AH, providing a previously un
172 rrays to select pancreatic cancer cells that proliferate in spite of cytotoxic drug treatment.
173                         Because tumour cells proliferate in suboptimal environments, and frequently s
174 P1 altered cell metabolism to allow cells to proliferate in the absence of MAPK signaling.
175 GBC population includes the basal cells that proliferate in the uninjured OE and is heterogeneous wit
176                      However, how Treg cells proliferate in these patients is not clear.
177 e we identified ccRCC lines whose ability to proliferate in vitro and in vivo is sensitive to wild-ty
178 n L. donovani and evaluated their ability to proliferate in vitro and trigger infections in mice.
179 ed NK cells are able to survive and possibly proliferate in vivo in humanized mice without exogenous
180 r these expanded NK cells can persist and/or proliferate in vivo in the absence of exogenous human cy
181 aybe related to their ability to migrate and proliferate in vivo rather than subset development or cy
182 culture rely less on glutamine catabolism to proliferate in vivo.
183 ribution to immune function, and capacity to proliferate in vivo.
184  lymphocyte responses, as memory CD8 T cells proliferated in an IL-15-dependent manner.
185                 In contrast, TAF in MED12-LM proliferated in response to estradiol, whereas progester
186                   Moreover, decidual T cells proliferated in response to fetal tissue, and depletion
187  ANS8 CAR T cells specific for the A2 domain proliferated in response to FVIII and ANS8 CAR Tregs wer
188 disease and the remaining 2 had T cells that proliferated in response to gluten antigen in vitro.
189 healthy control fibroblasts, SSc fibroblasts proliferated in response to PDGFAA, whereas a combinatio
190     Isolated CD4(+) CD27(-) CD28(null) cells proliferated in response to peripheral blood mononuclear
191                Only CD62L+ NKTs survived and proliferated in response to secondary stimulation.
192 deuterium labelling to mark CD8 T cells that proliferated in response to the virus and then assessed
193 ogenic SpA-deficient S. aureus mutant, cells proliferated in the BM survival niches and sustained lon
194 ransferred naive HEL-specific CD4(+) T cells proliferated in the eye draining lymph nodes, but did no
195                                 CTL019 cells proliferated in vivo and were detectable in the blood an
196       In addition, we found that CD8 T cells proliferating in blood after PD-1 therapy of lung cancer
197          Plasmodium species cause malaria by proliferating in human erythrocytes.
198 ompromised the ability of M. tuberculosis to proliferate inside lipid-rich foamy macrophages but not
199 orylation machinery at the stage of the fast proliferating intermediate progenitor cell.
200              Reactivating quiescent cells to proliferate is critical to tissue repair and homoeostasi
201 culation, extravasate at distant sites, then proliferate; it involves contributions from both the tum
202 taining stemness and renewal capacity of the proliferating keratinocyte compartment.
203  secrete lower levels of effector cytokines, proliferate less in response to cognate antigen, and upr
204             AhR-deficient (Ahr(-/-)) B cells proliferate less than AhR-sufficient (Ahr(+/+)) cells fo
205 , reduced expression of chemokine receptors, proliferated less, and produced less IFN- gamma, compare
206 ed epicardium formation, and Cdc42 null PECs proliferated less, lost polarity and failed to form vill
207                                Quiescent and proliferating leukemia cells accumulate highly lethal DN
208 ent leukemia cells and BRCA/DNA-PK-deficient proliferating leukemia cells were sensitive to PARP1 inh
209               Although most responding cells proliferated, ligand density was associated with time of
210  Moreover, macrophages recruited into muscle proliferated locally and were skewed toward a cyto-destr
211 from female to male, while floral stem cells proliferate longer, allowing for the production of addit
212 vivo, they are transformed into continuously proliferating lymphoblastoid cell lines (LCLs) that carr
213 rus (EBV) transforms B cells to continuously proliferating lymphoblastoid cell lines (LCLs), which re
214                       Interestingly, locally proliferating macrophages were not classically activated
215                               Chromosomes in proliferating metazoan cells undergo marked structural m
216 of pS6 in Muller glia and reduced numbers of proliferating MGPCs.
217 ork of pathways that drives the formation of proliferating MGPCs; however, this pathway inhibits the
218  that naive Tg CD4 T cells become activated, proliferate, migrate to the infected tissue, and acquire
219 yte precursors from hair follicle bulge that proliferate, migrate, and differentiate into mature mela
220                                These T cells proliferated modestly after skin transplantation and und
221            In vitro, IL4I1-deficient B cells proliferate more efficiently than their wild-type counte
222 over, E2f7/8-deficient primary keratinocytes proliferate more efficiently under stress conditions, su
223                  However, CMV tet(high) CTLs proliferated more in response to low CMV peptide concent
224                           The aged CPCs also proliferated more slowly than their young counterparts a
225  appeared to be lifted because ex-Th17 cells proliferated more than Th17 cells after stimulation.
226                EZH2 premature degradation in proliferating myoblasts is prevented by low levels of PJ
227 inase 2 (CK2), a serine/threonine kinase, in proliferating myoblasts.
228   Two distinct cell types were identified as proliferating neural progenitors and immature neurons, b
229 pulation led to the accumulation of actively proliferating neuroblasts and a lethal brain tumor pheno
230                                The number of proliferating neuronal precursor cells was increased wit
231 -null mice lacked Olig2(+) OPCs, and instead proliferating neuronal precursors and GABAergic interneu
232 thood was largely predicted by the number of proliferating neuroprogenitors at each age.
233  a novel transgenic mouse paradigm to ablate proliferating NG2(+) cells after SCI to better understan
234 ork reveals previously unknown ways in which proliferating NG2(+) cells contribute to endogenous repa
235                                 We show that proliferating NG2(+) pericytes and glia largely segregat
236                  Results reveal that loss of proliferating NG2(+) pericytes in the lesion prevented i
237  periphery, new blood vessel growth requires proliferating NG2(+) pericytes; if this were also true i
238                  Supporting this hypothesis, proliferating NK cells did not express aldehyde dehydrog
239  After infusion of Cy, a marked reduction of proliferating NK cells was evident, suggesting selective
240 n treatment reduced the density of total and proliferating Nkx2.1(+) and Dlx2(+) cells in the MGEs an
241                   Cells not expressing Lin37 proliferate normally, but DREAM completely loses its abi
242 K cells from individuals with GATA2 mutation proliferated normally in vitro, whereas lineage-negative
243 -knockout mouse embryonic fibroblasts (MEFs) proliferated normally; however, Cdk10-knockout MEFs deve
244 clonal colony during exposure to phages that proliferate on its surface.
245 rtality and show that chytrid can infect and proliferate on zebrafish tissue.
246 ansgenic (Tg) T cells from chronic infection proliferated on reinfection and were highly sensitive to
247 ost mammalian cells in tissue culture cannot proliferate or even survive in an environment that does
248 , decreased stemness and reduced capacity to proliferate or to differentiate following dexamethasone
249 ion in a cell-density dependent manner, with proliferating p38alpha(-/-) cultures showing increased d
250               During pancreatic development, proliferating pancreatic progenitors activate the proend
251                                           In proliferating parasites, PVM-associated LC3 becomes imme
252 e agent revealed that uptake was confined to proliferating, PARP1-expressing cells.
253 Cs), characterized by three distinct stages: proliferating phase, involuting phase, and involuted pha
254 e to IL-7, whereas more differentiated cells proliferated poorly.
255 taneously impair bone structure and increase proliferating potential of leukemic clone.
256 hogenic cells were mitotically inactive, but proliferating precursors were detected in primary cultur
257 nal programs take neuroepithelial cells from proliferating progenitors to differentiated neurons with
258 morphology of the tumor spheroids (layers of proliferating, quiescent, and necrotic cells).
259 lungs during infection with B. pertussis and proliferated rapidly after rechallenge of convalescent m
260 oids and metabolism of the drug in the outer proliferating region of the spheroid.
261                     In disc1 mutant embryos, proliferating rx3+ hypothalamic progenitors are not main
262 tem cells that provide the majority of newly proliferated scar-forming astrocytes that protect tissue
263 orm in injured nerves, although they contain proliferating Schwann cells with strikingly elevated c-J
264 pG-B and CpG-A activated B cells in vitro to proliferate, secrete Abs and IL-6, and that neither CpG-
265 Mammalian cardiomyocytes lose the ability to proliferate shortly after birth, and further increase in
266  that ectopically express the SA-Brg1 mutant proliferated similarly to the parental cells or cells ec
267  that the cells be maintained in an actively proliferating state.
268 ve not established whether SPEM derives from proliferating stem cells or differentiated, post-mitotic
269 8 engineering enabled low-avidity T cells to proliferate stronger and prevented PD-L1 upregulation an
270 cially in cancer immunotherapy, where highly proliferating T cells will encounter potentially large a
271 ies of Th1, effector-memory T cells, in situ proliferating T cells, and inhibitory PD1-PDL1 cells, ha
272 cipitates and their ample contact with Li2 O proliferate the reversible Sn --> Li x Sn --> Sn --> SnO
273  to face stress allowing them to survive and proliferate, thus contributing to vascular remodeling in
274 ay between cortical cues and cell shape in a proliferating tissue.
275 tor cells of embryonic human brain and other proliferating tissues, is co-expressed with components o
276 onse to injury, epithelial cells migrate and proliferate to cover denuded mucosal surfaces and repair
277 maged mitochondrial genomes will selectively proliferate to facilitate the tumorigenic process.
278  the mechanisms by which they are formed and proliferate to generate cellular dysfunction.
279 tions such as obesity, pancreatic beta-cells proliferate to prevent blood glucose elevations.
280 tubular epithelial cells of the nephrons can proliferate to repair the damage after AKI.
281  trafficked to the pancreatic lymph node and proliferated to the same extent in NOD and NOD-Idd22 mic
282 ong-lived proteins as they transition from a proliferating to a quiescent state.
283    In the late stage, HeLa cells change from proliferating to migratory.
284 normal biosynthetic requirements of actively proliferating tumor cells.
285  within the printed structures were shown to proliferate under culture conditions.
286 fically at the leading tumor edge, ceased to proliferate, underwent epithelial-mesenchymal transition
287 crimination is weak, uncooperative symbionts proliferate until they reach the equilibrium proportion
288 ion, Pten-null activated SCs can transiently proliferate upon injury and regenerate injured muscles,
289 lt stem cells maintain a quiescent state and proliferate upon injury.
290 yzed in sorted CD3(+) CD4(+) CFSE(low) cells proliferating upon stimulation of PBMC with Dau c 1 or B
291          CD4(+)CD161(+)Rho-effluxing T cells proliferated vigorously in response to stimulation with
292 f cancer cells is the ability to survive and proliferate when challenged with stressors such as growt
293 stimulate CD4(+) T cells to express CD25 and proliferate when exposed to superantigen or to cytomegal
294 mportantly, CD27-deficient T cells failed to proliferate when stimulated with CD70-expressing B cells
295 scent HBCs are activated to multipotency and proliferate when sustentacular cells are killed, but not
296  In adult rats, 3.6% +/- 0.9% of cells still proliferate, whereas, in mice, 10.3% +/- 2.3% of cells a
297 recognizing intact acquired MHC alloantigens proliferated, whereas those responding to allopeptide+se
298                                   Cells that proliferate within a confined environment build up mecha
299 s from peripheral blood survive, mature, and proliferate without SCF and KIT signaling in vitro.
300                                   Within the proliferating zone of the Arabidopsis root, regular symm

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