ライフサイエンスコーパス: proliferate in response to

1 lls expressing GMRbeta-F8 were still able to proliferate in response to 10 ng/mL of human GM-CSF, alt

2 RB51-vaccinated cattle had lymphocytes which proliferated in response to 80- and 49-kDa proteins of S

3 al heterotrophs, we demonstrated that Vibrio proliferate in response to a broad range of dust-Fe addi

4 , based on interactions with a self-peptide, proliferated in response to a homologous viral Ag in the

5 nodes of C3H/HeN mice skin-painted with PDC proliferated in response to a peptide carrier:PDC conjug

6 secondary lymphoid tissues of immunized mice proliferated in response to a variety of C. neoformans a

7 speculation that cephalopod populations are proliferating in response to a changing environment, a p

8 CD4+ cells from all AChR-immunized mice proliferated in response to AChR and recognized similar

9 y of CCR6(+) memory T cells from MG patients proliferating in response to AChR-derived peptides was s

10 by reducing the capacity of CD8(+) cells to proliferate in response to activation.

11 tion was found, however, in CD8 T cells that proliferate in response to acute viral infections.

12 The cells failed to proliferate in response to Ag except in the presence of

13 abled resting primary T cells to survive and proliferate in response to Ag in the absence of any exog

14 lity to produce IFN-gamma, or the ability to proliferate in response to Ag in vitro.

15 cells primed in the absence of B cells could proliferate in response to Ag presented by B cells and c

16 but exhibit split anergy in that they cannot proliferate in response to Ag unless exogenous IL-2 is p

17 r function, are impaired in their ability to proliferate in response to Ag-specific stimulation, and

18 n response to IL-2, but were not anergic and proliferated in response to Ag.

19 inly T cells and others where mainly B cells proliferated in response to allergen stimulation.

20 depleted of basophils but not with basophils proliferated in response to allergen.

21 es-free recipients and their splenic T cells proliferated in response to alloantigen.

22 cells (a subset of memory T cells) failed to proliferate in response to alloantigens (which the mice

23 onor or the intrinsically reduced ability to proliferate in response to alloantigens.

24 Neither LT1 nor LT3 proliferated in response to alloantigens.

25 regulatory cells prevented CD8+ T cells from proliferating in response to alloantigens and from becom

26 stasis by eliminating activated T cells that proliferated in response to an infection.

27 As prostate cancer cells proliferate in response to androgen steroids, CYP17A1 in

28 us, unlike larynges in vivo, muscle will not proliferate in response to androgen, indicating the nece

29 o determine in vivo which cardiac cell types proliferate in response to Ang II, to evaluate whether p

30 tors in the presence of IL-2 and continue to proliferate in response to anti-CD3 stimulation, whereas

31 lenocytes lacking both ICAM and CD28 fail to proliferate in response to anti-CD3-induced T cell signa

32 t B-lymphocytes from cyclin D2(-/-) mice can proliferate in response to anti-IgM and anti-CD40, but t

33 ansion, since it induced neonatal B cells to proliferate in response to anti-IgM, which was further e

34 TCRxgag cells failed to proliferate in response to antigen but demonstrated cyto

35 This inability to proliferate in response to antigen cannot be overcome by

36 expression was associated with a failure to proliferate in response to antigen receptor crosslinking

37 aired capacity of activated T and B cells to proliferate in response to antigen receptor-mediated act

38 T cells fail to proliferate in response to antigen when any 1, or more,

39 nt self-antigen-specific CD8 T cells fail to proliferate in response to antigen, thereby preventing a

40 f the cells allowed the TGF-beta(-) cells to proliferate in response to antigen.

41 While CD8+ T cells that proliferated in response to antigen had a PD-1(high) phe

42 T cells that proliferated in response to antigen in vitro were found

43 Failure of CD4(+) T cells to proliferate in response to antigenic stimulation is a ch

44 Although both the VLRA and the VLRB cells proliferate in response to antigenic stimulation, only t

45 These cell lines proliferated in response to APC in the presence of recom

46 The T cells proliferated in response to APC sensitized in vivo, but

47 -overexpressing T cells are autoreactive and proliferate in response to APCs in an MHC class II-depen

48 to induce calcium flux, become activated, or proliferate in response to B-cell receptor and/or CD40 t

49 e B cells consistent with their inability to proliferate in response to BCR cross-linking.

50 cells expressing these stereotypic receptors proliferate in response to beta-(1,6)-glucan.

51 cells of mature and immature phenotypes, and proliferate in response to bile duct injury.

52 regulating large biliary functions) and (2) proliferate in response to bile duct ligation (BDL) by a

53 Human T cells proliferate in response to both human umbilical vein end

54 In vitro, the DO11.10 cells did not proliferate in response to C. parvum antigen, and a tBla

55 dergoing a primary response to C. neoformans proliferated in response to C. neoformans lysate.

56 t enhancing the ability of cardiomyocytes to proliferate in response to cardiac injury, as well as tr

57 These T cells proliferated in response to CD1+ presenter cells, lysed

58 ation in response to second party cells, but proliferated in response to CMV Ags, PHA, and third part

59 cell lines, but not normal lung lymphocytes, proliferated in response to col(V).

60 In vitro, STAT5a/b-/- T cells did not proliferate in response to Con A or alloantigens but ent

61 eural cells were examined for the ability to proliferate in response to concanavalin A and purified p

62 In contrast, memory Th17 cells proliferated in response to conserved outer membrane pro

63 As conventional memory cells, they proliferate in response to CpG DNA and fail to extrude r

64 They also proliferated in response to culture supernatants collect

65 become activated and acquire the ability to proliferate in response to cytokines.

66 is activation is required for SCs to rapidly proliferate in response to damage, repeated rounds of da

67 These cells proliferate in response to degeneration, therefore incre

68 other inhibitory receptors and were able to proliferate in response to DENV-specific stimulation.

69 d in number, and show a decreased ability to proliferate in response to different growth factors in v

70 Transfected myoblasts proliferated in response to dimerizer (comparable with b

71 oportion of Treg, CD4 Tconv, and CD8 T cells proliferating in response to donor-derived, stimulated B

72 Rather, Casp8-deficient B cells failed to proliferate in response to dsRNA and LPS, ligands for TL

73 E7-specific CD4(+) T cells proliferated in response to E7-pulsed LCL but not unpuls

74 riers have a large pool of CD4+ T cells that proliferated in response to EBNA1 and consisted of disti

75 CD8(+) SAP(+) T cells, but not SAP(-) cells, proliferated in response to EBV and killed EBV-infected

76 -dependent cell-signaling may prime cells to proliferate in response to EGF by promoting Tyr-845 phos

77 ort that murine embryonic fibroblasts (MEFs) proliferate in response to EGF only when these cells exp

78 Type-1 astrocytes are known to proliferate in response to EGF, and are immunohistochemi

79 l growth factor receptors (EGFRs) and do not proliferate in response to EGF, heregulin (HRG) or other

80 cells from rejected but not quiescent grafts proliferated in response to enteric bacterial and donor

81 an endogenous population of stem cells that proliferate in response to environmental and pharmacolog

82 owed that they contain progenitor cells that proliferate in response to epidermal growth factor (EGF)

83 nt with a Y-to-F alteration at position 343) proliferate in response to Epo in a manner comparable to

84 Human peripheral blood T cells proliferate in response to Escherichia coli and Pseudomo

85 s express CD127 and Ki67 in vivo and readily proliferate in response to Escherichia coli in vitro.

86 In contrast, TAF in MED12-LM proliferated in response to estradiol, whereas progester

87 l CD3(+) CD4(+) and CD3(+) CD4(-) cells that proliferated in response to ex vivo stimulation with ina

88 Senescent cells do not proliferate in response to exogenous growth factors, yet

89 AINR cells can still proliferate in response to exogenous IL-2, but can no lo

90 roliferating, but can still be stimulated to proliferate in response to exogenous IL-2.

91 mide, or when anergic T cells are allowed to proliferate in response to exogenous IL-2.

92 a key element for development of KS lesions, proliferates in response to external cytokines, such as

93 Moreover, decidual T cells proliferated in response to fetal tissue, and depletion

94 h that present on the surface of a cell that proliferates in response to FGF-2 (Swiss 3T3 fibroblasts

95 etroviral superantigen, become activated and proliferate in response to Friend virus (FV) infection.

96 ANS8 CAR T cells specific for the A2 domain proliferated in response to FVIII and ANS8 CAR Tregs wer

97 e 1 diabetes and healthy subjects vigorously proliferate in response to GAD65 stimulation ex vivo, le

98 igenic markers, interacted with neurons, and proliferated in response to glial growth factor, confirm

99 disease and the remaining 2 had T cells that proliferated in response to gluten antigen in vitro.

100 When tested for their ability to proliferate in response to GM-CSF, only chimeric transfe

101 C II(mid/low)) that were CCR5(+)/CCR7(-) and proliferated in response to GM-CSF, but, unlike immature

102 The cells' ability to proliferate in response to growth factor stimulation is

103 5beta1 integrin enables endothelial cells to proliferate in response to growth factors, whereas adhes

104 Each cell population was determined to proliferate in response to heat-killed virulent H37Rv, w

105 be relevant to the mechanisms by which ducts proliferate in response to hepatic injury and to the hyp

106 T cells from anti-ICOS-treated mice did not proliferate in response to hFVIII stimulation and produc

107 pancreatic cancer lines to select lines that proliferate in response to HGF.

108 ific CD8(+) T cells to secrete cytokines and proliferate in response to HIV antigen stimulation.

109 y of HIV-1-specific CD4(+) memory T cells to proliferate in response to HIV antigens rather than an a

110 ells of viremic patients could be induced to proliferate in response to HIV antigens when costimulati

111 ells secreted cytokines, were cytolytic, and proliferated in response to HMW-MAA-expressing cell line

112 3 cells transfected with IL-12R beta 2 alone proliferated in response to huIL-12 with an ED50 of abou

113 discovery of carotid body stem cells, which proliferate in response to hypoxia and generate neurons

114 The precursor frequency of T cells proliferating in response to IECs ranged from 0.3%-0.9%.

115 tures, both T(EM) and T(CM) produce IL-2 and proliferate in response to IFN-gamma-treated allogeneic

116 to WT cells, the Gal3-KO microglia failed to proliferate in response to IGF-1.

117 have shown that uterine leiomyoma (UL) cells proliferate in response to IGF-I and display increased I

118 ession of IGF-IR caused the transfectants to proliferate in response to IGF-I in the absence of insul

119 m containing normal levels of glucose do not proliferate in response to IGF-I, whereas cells maintain

120 mice activated in vitro with anti-IgM do not proliferate in response to IL-1, but do so in response t

121 elta T cells spontaneously secrete IL-10 and proliferate in response to IL-10, TGF-beta, and contact

122 essing the STAT4 N-terminal mutant failed to proliferate in response to IL-12 and had limited Th1 cel

123 T cells were unable to produce IFN-gamma or proliferate in response to IL-12 despite the expression

124 a 2(-/-) mice failed to produce IFN-gamma or proliferate in response to IL-12 stimulation.

125 However, these cells did not proliferate in response to IL-13, and the IL-4 dose resp

126 Synovial fluid T lymphocytes proliferate in response to IL-15, demonstrating that con

127 er coculture with regulatory cells failed to proliferate in response to IL-2, but were not anergic an

128 rfered with the acquisition of competence to proliferate in response to IL-4 as well as IL-2.

129 hocytes from Stat6-deficient animals fail to proliferate in response to IL-4.

130 TAIL7 cells are pre-T-ALL cells that proliferate in response to IL-7 and IL-4.

131 have considerably shorter telomeres, do not proliferate in response to IL-7, and are prone to cell d

132 Mesenchymal cells dose-dependently proliferated in response to IL-1 beta, IL-6, and TNF-alp

133 a2 microglobulin, naive CD8 T cells scarcely proliferated in response to IL-15/IL-15Ralpha complex, w

134 These T cells proliferated in response to IL-2 without additional mito

135 eptor BR3 and the tec family kinase Btk, and proliferated in response to IL-4 plus CD40 stimulation.

136 Cells that have proliferated in response to IL-4 plus IL-12 are fully ab

137 In MM cells that proliferated in response to IL-6, exogenous IL-6 downreg

138 of MAPK, only in MM cells and B9 cells that proliferated in response to IL-6.

139 id progenitor population expresses IL-3R and proliferates in response to IL-3 and that IL-3 activates

140 The DO.11 CD4+ memory cells are capable of proliferating in response to IL-2 and IL-4, while naive

141 ition, they respond vigorously to cytokines, proliferating in response to IL-2, and secreting IFN-gam

142 Pulmonary mast cell lines were capable of proliferating in response to IL-3, IL-4, IL-10, and SCF,

143 y expanded CD4+ T cells were not anergic but proliferated in response to immobilized anti-CD3 and cou

144 to show that despite anergy in vitro, Tregs proliferate in response to immunization in vivo.

145 verted transgenic CD4+CD25+ suppressor cells proliferated in response to immunization and inhibited a

146 Additionally, T cells can continue to proliferate in response to immunogenic proteins expresse

147 s from E1-rLm responder rabbits were able to proliferate in response to in vitro E1 stimulation.

148 yclin D2-deficient strains of mice failed to proliferate in response to infection with a retrovirus c

149 cterial antigens are selected to survive and proliferate in response to infection, whereas those that

150 e(-)Sca-1(+)c-Kit(-) (LSK(-)) phenotype that proliferates in response to infection with nonlethal Pla

151 present in the normal liver but activate and proliferate in response to injury and inflammation.

152 alian cardiomyocytes have little capacity to proliferate in response to injury, a deficiency that und

153 By contrast, sigma-neoblasts proliferate in response to injury, possess broad lineage

154 inability to maintain tissue homeostasis and proliferate in response to injury.

155 ular events that prevent adult myocytes from proliferating in response to injury or stress.

156 In addition, the restored cells could proliferate in response to interleukin-2 (IL-2).

157 cells from all EmuLMP2A animals were able to proliferate in response to interleukin-7-dependent devel

158 e their c-kitLo counterparts, expressed TdT, proliferated in response to interleukin (IL)-7, and gene

159 ANBL-6, a myeloma cell line, proliferates in response to interleukin 6 (IL-6) stimula

160 OCI/AML3, both of which produce IL-1beta and proliferate in response to it.

161 ll lines, both of which produce IL-1beta and proliferate in response to it.

162 cells (lin-) from me/me mice markedly hyper-proliferated in response to Kit ligand (KL). stimulation

163 mbers of the ErbB receptor family and do not proliferate in response to known ligands for these recep

164 d no difference in the percentage of T cells proliferating in response to L. mexicana and L. major.

165 ly to foreign antigens in that they strongly proliferate in response to Leishmania-infected dendritic

166 ated hepatic stellate cells, which initially proliferate in response to liver damage and produce the

167 5aR) abrogated the ability of hepatocytes to proliferate in response to liver injury, providing a mec

168 thelial cells and placental cytotrophoblasts proliferate in response to low O(2).

169 activation threshold is low, such that they proliferate in response to lower concentrations of agoni

170 assic work has suggested that memory T cells proliferate in response to lower doses of antigen than n

171 to the mitochondrial inhibitor rotenone and proliferate in response to lowered oxygen conditions.

172 TAF(II)105-null B cells can proliferate in response to LPS, produce relatively norma

173 rom normal human intrahepatic cholangiocytes proliferated in response to LPS.

174 Naive T cells proliferate in response to lymphopenia and acquire the p

175 Moreover, they do not proliferate in response to lymphopenia in the absence of

176 In MLR, T cells did not proliferate in response to M. leprae-stimulated DC.

177 , since fully rested T cell clones failed to proliferate in response to mAb A12 stimulation, despite

178 alloreactive CD4+ T cell populations as they proliferate in response to major or minor histocompatibi

179 of T cell clones, including HD4-1C2, cannot proliferate in response to MBP(111-129) (MBP122:Lys).

180 t immunity depends on naive CD4 T cells that proliferate in response to microbial antigens, different

181 the long form of the human PRLR were able to proliferate in response to mIL-3, ovine prolactin, or hu

182 mulation, with the frequency of CD4+ T cells proliferating in response to minor Ags in the absence of

183 ed when quiescent cultures are stimulated to proliferate in response to mitogen activation.

184 FIV(+) cats do not produce IL-2 and fail to proliferate in response to mitogen stimulation.

185 + T cells are classically anergic, unable to proliferate in response to mitogenic antibodies to the T

186 Semen leukocytes proliferated in response to mitogenic and antigenic chal

187 eneralized defect in the ability of PBMCs to proliferate in response to mitogens and recall antigens.

188 sociated with the failure of host T cells to proliferate in response to mitogens or to mount memory r

189 dy, we show that latently infected cells can proliferate in response to mitogens without producing vi

190 5(-) cells from IL-2-treated HIV(+) patients proliferated in response to mitogens, specific antigens,

191 ere primed so that their splenic lymphocytes proliferated in response to MOG 35-55 and the mice produ

192 e marrow culture system and "primes" them to proliferate in response to monomeric IL-7.

193 r infected with S2308 have lymphocytes which proliferate in response to most of the same S2308 protei

194 pecific T cell clones, including MS2-3C8 can proliferate in response to mouse MBP(111-129) (MBP122:Ly

195 the c-Kit-negative (c-Kit(neg)) PHSC do not proliferate in response to multiple hematopoietic growth

196 trophic mice, muscle stem cells activate and proliferate in response to muscle degeneration, resultin

197 early-acting hematopoietic cytokines further proliferate in response to nerve growth factor (NGF).

198 do not differentiate, but rather continue to proliferate in response to NGF.

199 rries a T-cell receptor encoding Vbeta14 and proliferates in response to NOD islets, islet supernatan

200 Cholangiocarcinomas also produce and proliferate in response to nonneoplastic biliary epithel

201 oxisomes in Saccharomyces cerevisiae fail to proliferate in response to oleic acid; instead, one or t

202 cytes from strain 16M- or 201-infected goats proliferated in response to one-dimensional polyacrylami

203 cells isolated from tolerant double Tg mice proliferated in response to OVA peptide and produced IFN

204 zed or deleted during thymic development and proliferated in response to P0 in vitro.

205 CD4+ T cells, in the absence of monocytes, proliferated in response to PAECs only after swine leuko

206 r peptides, p199-213 and p228-242, that also proliferated in response to PAP protein stimulation.

207 hepatocytes deficient in c-myc were able to proliferate in response to partial hepatectomy.

208 -specific enzymes, accumulated glycogen, and proliferated in response to partial hepatectomy, as neig

209 ng alpha beta T cells from these individuals proliferated in response to PBMC infected with T. gondii

210 to soluble anti-mu, or PC-dextran, but they proliferate in response to PC, anti-mu, or anti-id conju

211 Porcine Muller cells proliferate in response to PDGF, but not IGF-I or IGF-II

212 that Pdgfra(PI3K/PI3K)-derived MEPMs do not proliferate in response to PDGF-AA treatment.

213 healthy control fibroblasts, SSc fibroblasts proliferated in response to PDGFAA, whereas a combinatio

214 Beta cells failed to proliferate in response to PDL in beta-cell-specific SMA

215 Isolated CD4(+) CD27(-) CD28(null) cells proliferated in response to peripheral blood mononuclear

216 ) cells displayed low intrinsic turnover but proliferated in response to persisting antigen, giving r

217 s from three of six Sezary syndrome patients proliferated in response to PHA if an anti-CD28 mAb was

218 were washed free of imatinib mesylate, they proliferated in response to PHA, demonstrating that inhi

219 (89.6PD) infection have lost the capacity to proliferate in response to phytohemagglutinin (PHA).

220 urprisingly, IL-15Ralpha(-/-) CD8(+) T cells proliferate in response to poly I:C when adoptively tran

221 eta genes normally yet failed to survive and proliferate in response to pre-TCR signaling.

222 mitans Omp29-specific CD4(+) Th1 clone cells proliferated in response to pretreatment of GEC with fix

223 findings show that human beta-cells fail to proliferate in response to PRL for multiple reasons, one

224 Ba/F3 transfectants expressing the long PRLr proliferated in response to PRL, intermediate PRLr trans

225 that human brite/beige adipocyte progenitors proliferate in response to pro-angiogenic factors, in as

226 We show that cyclin D1(-/-) MECs fail to proliferate in response to prolactin and identify IGF-2

227 ells transfected with both receptor subunits proliferated in response to purified, recombinant human

228 However, while both cell types proliferated in response to PXR ligands, the FGF19 promo

229 T cells from infected mice also proliferated in response to re-exposure to Ag, and CD8(+

230 the persisting T cells lost the capacity to proliferate in response to repeated Ag stimulation.

231 Activated/memory MBP-specific T cells proliferating in response to resting B cells presenting

232 g memory phenotype (CD45RA-) CD4+ T cells to proliferate in response to rotavirus, a ubiquitous intes

233 ells (MIHCs) transduced with wild-type c-Kit proliferate in response to SCF, whereas cells expressing

234 le to rapidly generate effector cytokines or proliferate in response to secondary infection.

235 Only CD62L+ NKTs survived and proliferated in response to secondary stimulation.

236 Naive T cells proliferate in response to self MHC molecules after tran

237 ficient host, normal T cells 'spontaneously' proliferate in response to self-MHC-peptide complexes.

238 allogeneic specificity, i.e., they kill and proliferate in response to several different allogeneic

239 several EGFR ligands but were stimulated to proliferate in response to several other mitogens.

240 ls in the bulge and secondary hair germ that proliferate in response to skin injury.

241 sitized stromelysin-1-deficient mice did not proliferate in response to specific soluble antigen dini

242 mpair the ability of T cells to subsequently proliferate in response to stimulation by the original o

243 In addition, they proliferate in response to stimulation with autologous A

244 ine whether Ov39-derived T-cell lines, which proliferate in response to stimulation with hr44, can tr

245 ate in inflamed joints in Lyme arthritis and proliferate in response to stimulation with the causativ

246 CD4(+)/CD8(+) DP lymphocytes were able to proliferate in response to stimulation with viral antige

247 h the Fc epsilon RI gamma-expressing T cells proliferated in response to stimulation by TCR-specific

248 Peripheral blood mononuclear cells proliferated in response to stimulation with purified pp

249 om both types of mice produced cytokines and proliferated in response to stimulator cells lacking B7

250 e that VH3H9/Vlambda1 anti-chromatin B cells proliferate in response to stimulatory oligodeoxynucleot

251 They do not produce IL-2 or proliferate in response to subsequent stimulation.

252 f Ras, prolonged Ras and ERK activities, and proliferate in response to subthreshold levels of growth

253 dies, however, have indicated that HSCs also proliferate in response to systemic infection and replen

254 nd failed to flux calcium, secrete IL-2, and proliferate in response to T cell antigen receptor ligat

255 ficiency in which the host's T cells fail to proliferate in response to T-cell mitogens and fail to m

256 Importantly, ZAP-70-reconstituted T cells proliferated in response to T-cell receptor stimulation.

257 itro, these effector cells lysed T9.F cells, proliferated in response to T9.F stimulator cells, and p

258 ty of human T cells to produce IFN-gamma and proliferate in response to TCR activation.

259 Naive CD8 T cells proliferate in response to TCR and CD28 signals, but req

260 levels but made their own IL-2 were able to proliferate in response to TCR stimulation.

261 Although sap(-/-) tetramer-reactive cells proliferated in response to TCR activation, they did not

262 oire of rare Ag-specific CD4(+) T cells that proliferated in response to tetanus toxoid (TT) presente

263 studies show that whereas dermal fibroblasts proliferate in response to TGF-beta1, oral fibroblasts h

264 U9 cells expressing DN-N17ras neither proliferated in response to TGFbeta1 nor down-regulated

265 a large proportion of gamma delta cells that proliferate in response to the causative spirochete, Bor

266 ransit amplifying progeny were stimulated to proliferate in response to the chronic application of gl

267 T cells demonstrated a decreased ability to proliferate in response to the CII immunodominant peptid

268 termine the identity of the first cells that proliferate in response to the death of hair cells and t

269 circulating T lymphocytes that specifically proliferate in response to the extracellular domain of D

270 0.BR T cells showed a diminished capacity to proliferate in response to the MMTV Sag.

271 tive bronchiolalveolar stem cells (BASCs) to proliferate in response to the oncogenic stimulus.

272 Importantly, postrevision cells do not proliferate in response to the tolerizing superantigen,

273 lls were CD43(pos)CD24(pos) on isolation and proliferated in response to the cytokine combination of

274 They proliferated in response to the protein Ag KLH presented

275 deuterium labelling to mark CD8 T cells that proliferated in response to the virus and then assessed

276 these drugs inhibited endothelial cells from proliferating in response to the angiogenic factors prod

277 the ability for RV-A16-specific Th1 cells to proliferate in response to their RV-A39 peptide counterp

278 is known, to show that CD4(+)CD25(+) T cells proliferate in response to their selecting self-peptide

279 beta T cells but not CD8+ alpha beta T cells proliferated in response to these T. gondii preparations

280 oduced IL-10 but not IFN-gamma and failed to proliferate in response to this treatment.

281 e with an immature phenotype (CD4-CD8-) that proliferates in response to thymocytes or PMA when cultu

282 iescent cells to re-enter the cell cycle and proliferate in response to tissue damage.

283 While S-Myc cells proliferated in response to TNF treatment, AN-Myc cells

284 that c-MPL(+) polyclonal T cells expand and proliferate in response to TPO, and persist longer after

285 ere we found that FRCs specifically of TDLNs proliferated in response to tumor-derived cues and that

286 elial and sub-epithelial compartments, which proliferate in response to unique growth factor signals.

287 We found that TCDD-CD4(+) cells actively proliferate in response to various stimuli but suppress

288 s from T9/mM-CSF-immunized rats specifically proliferated in response to various syngeneic glioma sti

289 ssing VEGFR from tumor-bearing mice directly proliferated in response to VEGF-A.

290 both produced IFN-gamma but could minimally proliferate in response to viral challenge.

291 These T cells proliferated in response to viral Ags and showed normal

292 esent, irradiation-sensitive, and capable of proliferating in response to wounding; smedwi-2(RNAi) ne