ライフサイエンスコーパス: proliferating

1 otein expression, exit quiescence, and begin proliferating.

2 % of cells in the CC lining in adulthood are proliferating.

3 shRNA-mediated depletion of Kdm5b in proliferating adult NSCs decreased proliferation rates a

4 PT-Cy selectively eliminates proliferating alloreactive T cells, but whether and how

5 TV, inflammation, apoptosis, and number of (proliferating) alpha smooth muscle actin (alphaSMA) neoi

6 luent MelJuSo clones DCT(high)/CAV1(low) are proliferating and acquire fibroblast-like morphology, fo

7 as required to define the transition between proliferating and arrested cells inside the LRP, coincid

8 ity to disintegrate internalized MNP in both proliferating and contact-inhibited states.

9 Further, the numbers of proliferating and cytokine-producing lung ILC2s were inc

10 latent infection with HIV-1, while actively proliferating and differentiating HSPCs obtain predomina

11 ing a common principle of H3 eviction in the proliferating and endocycling domains of the root apex.

12 0alpha1)-Cre-expressing cells, predominantly proliferating and hypertrophic chondrocytes, using "Cre-

13 AV challenge and assessed the percentages of proliferating and IAV-infected, alveolar type II (AECII)

14 coxib-treated lesions further contained less proliferating and more apoptotic cells and exhibited low

15 l matrix, causing them to remain in a poorly proliferating and non-differentiating state.

16 in proliferating granule cell precursors, in proliferating and post-mitotic granule cells, and in Pur

17 The existence and interaction of proliferating and quiescent intestinal stem cells have b

18 ide insight into proteasome dynamics between proliferating and quiescent yeast in response to cellula

19 ngly with changes in gene expression between proliferating and senescent cells; however, in senescent

20 ndances in all four cell strains and between proliferating and senescent cells; however, in the four

21 we determine the distribution of H4K20me3 in proliferating and senescent human cells.

22 herefore, we compared PEsen fibroblasts with proliferating and transiently growth arrested controls u

23 We show that CLEC3A is present in resting, proliferating, and hypertrophic growth-plate cartilage a

24 trast, equally capable of inducing diabetes, proliferating, and producing cytokines.

25 CBSCs were present in the MI border zone and proliferating at 72 hours post-MI but had no effect on i

26 e large B cell lymphomas (DLBCLs) arise from proliferating B cells transiting different stages of the

27 on in digested lymphoid tissues, clusters of proliferating B cells with a GC-like phenotype can be ge

28 significant difference in the percentages of proliferating basal keratinocytes or adipocytes, nor in

29 also displayed reduced beta cell mass, fewer proliferating beta cells and reduced islet-specific gene

30 ormation and was later strongly expressed in proliferating blastemal cells.

31 nireview, we discuss the mechanisms by which proliferating cancer and T cells maintain a carefully co

32 Proliferating cancer cells are characterized by high rat

33 duced tumor growth by specifically targeting proliferating cancer cells but did not affect hypoxic, n

34 The complex metabolic logic of the proliferating cancer cells' appetite for glutamine-which

35 m oxidative phosphorylation to glycolysis in proliferating cancer cells, even under aerobic condition

36 icancer therapies target well-oxygenated and proliferating cancer cells, whereas there are no approve

37 Fetal or neonatal mouse hearts containing proliferating cardiac myocytes regenerate even extensive

38 Proliferating casein/T-effector cell counts were measure

39 Significantly, proliferating CD B cells express high levels of MHC clas

40 and primitive CD34(+)38(-), as compared with proliferating CD34(+)Hoechst(+)Pyronin Y(+) and CD34(+)3

41 mph nodes and a reduction in the fraction of proliferating CD4(+) Ki-67(+) TSCM in blood (but not lym

42 Proliferating CD62L+ cells downregulated or maintained C

43 These proliferating CD8 T cells had an effector-like phenotype

44 l SD-101 promoted infiltration of activated, proliferating CD8(+) T cells and led to a synergistic in

45 rns for core cell-cycle proteins in actively proliferating (CDK2-increasing) versus spontaneously qui

46 we report that teosinte branched1/cycloidea/proliferating cell factor1-20 (TCP20) and NIN-like prote

47 egulated TCP (TEOSINTE BRANCHED 1, CYCLODEA, PROLIFERATING CELL FACTORS) transcription factors, notab

48 interacting partners of EndoQ and identified Proliferating Cell Nuclear Angigen (PCNA).

49 During DNA replication, proliferating cell nuclear antigen (PCNA) adopts a ring-

50 ociated with decreased platinum drug-induced proliferating cell nuclear antigen (PCNA) and FANCD2 mon

51 th muscle mass, paired box protein 7 (Pax7), proliferating cell nuclear antigen (PCNA) and nicotinami

52 through its interactions with two proteins, Proliferating Cell Nuclear Antigen (PCNA) and Replicatio

53 the essential replication accessory protein proliferating cell nuclear antigen (PCNA) and the scaffo

54 e show that the ternary complexes containing proliferating cell nuclear antigen (PCNA) and two non-cl

55 In this study, we identified the proliferating cell nuclear antigen (PCNA) as a nIGF-1R-b

56 n multiple transient events in the reaction: proliferating cell nuclear antigen (PCNA) clamp binding/

57 s a mismatch, MutSalpha/beta, and DNA-loaded proliferating cell nuclear antigen (PCNA) for activation

58 Proliferating cell nuclear antigen (PCNA) forms a trimer

59 xyuridine (EdU) labeling as well as Ki67 and proliferating cell nuclear antigen (PCNA) immunofluoresc

60 Proliferating cell nuclear antigen (PCNA) lies at the ce

61 Proliferating cell nuclear antigen (PCNA) loading by rep

62 loading of cyclin-dependent kinase (CDK) and proliferating cell nuclear antigen (PCNA) onto chromatin

63 ns wherein the interaction between RECQ5 and proliferating cell nuclear antigen (PCNA) promotes RAD18

64 e lagging strand template and anchors to the proliferating cell nuclear antigen (PCNA) sliding clamp

65 from Thermoplasma acidophilum interact with proliferating cell nuclear antigen (PCNA), an essential

66 n of Kdm4d impairs the recruitment of Cdc45, proliferating cell nuclear antigen (PCNA), and polymeras

67 ion, as evidenced by stabilization of Mcl-1, proliferating cell nuclear antigen (PCNA), and pro-caspa

68 ssive replication with the replication clamp proliferating cell nuclear antigen (PCNA), respectively.

69 A We show that Rad51 inhibits recruitment of proliferating cell nuclear antigen (PCNA), the platform

70 Proliferating cell nuclear antigen (PCNA), the processiv

71 Lack of SIM, but not proliferating cell nuclear antigen (PCNA)-interacting mo

72 action between the Enok complex and the Elg1 proliferating cell nuclear antigen (PCNA)-unloader compl

73 increased expression of Nox4, TNF-alpha, and proliferating cell nuclear antigen (PCNA).

74 we explore the interactions of S. cerevisiae Proliferating Cell Nuclear Antigen (yPCNA) with modified

75 In the absence of NKT cells hepatic proliferating cell nuclear antigen and cyclin B1 decreas

76 Whereas association with proliferating cell nuclear antigen and participation in

77 We further show that proliferating cell nuclear antigen and the nucleosome co

78 incorporation and proportionate increases in proliferating cell nuclear antigen gene expression.

79 ither translesion DNA synthesis initiated by proliferating cell nuclear antigen monoubiquitination or

80 The eukaryotic DNA polymerase sliding clamp, proliferating cell nuclear antigen or PCNA, is a ring-sh

81 LR assessed by Ki-67 and proliferating cell nuclear antigen was markedly decrease

82 to promote senescence, whereas cyclin D1 and proliferating cell nuclear antigen were decreased to red

83 Expression of hepatic cyclin B1 and proliferating cell nuclear antigen were evaluated by Wes

84 Ki-67 and proliferating cell nuclear antigen were used to measure

85 tethers it to the leading strand, and PCNA (proliferating cell nuclear antigen) binds tightly to Pol

86 Proliferating cell nuclear antigen, a DNA sliding clamp,

87 were determined by methyltetrazolium, Ki-67, proliferating cell nuclear antigen, bromodeoxyuridine, a

88 d DNA ligase I, which compete for binding to proliferating cell nuclear antigen, is critical to preve

89 gy with Cavalieri estimation; apoptosis with proliferating cell nuclear antigen, TUNEL, and caspase a

90 or how nucleosome binding protects Set8 from proliferating cell nuclear antigen-dependent degradation

91 diminished, correlating with the loss of the proliferating cell population of germinal zones.

92 Although its role in proliferating cell types and tissues has been extensivel

93 Proliferating cells acquire genome alterations during th

94 Proliferating cells actively control their size by mecha

95 pression of LAT induced a reduction of brain proliferating cells and concomitant microcephaly.

96 Highly proliferating cells are particularly dependent on glucos

97 us RNA-Seq (sNuc-Seq) with pulse labeling of proliferating cells by 5-ethynyl-2'-deoxyuridine (EdU) t

98 When proliferating cells complete mitosis, a fraction of newl

99 It is not clear how stem and proliferating cells cope with accumulating endogenous DN

100 However, proliferating cells depend on growth-factor-induced incr

101 The rapidly proliferating cells in plant meristems must be protected

102 During early gonadogenesis, proliferating cells in the coelomic epithelium (CE) give

103 Proliferating cells in the ventricular zone stem cell co

104 Rapidly proliferating cells increase glycolysis at the expense o

105 s additional evidence that the metabolism of proliferating cells is adapted to facilitate producing n

106 To maintain the integrity of the genome, proliferating cells must be able to block progression th

107 Proliferating cells must cross a point of no return befo

108 These non-proliferating cells occupy key cellular niches and elabo

109 e characterization of gene expression in the proliferating cells of M. lignano, represented by somati

110 YAP and WPB2 are upregulated in actively proliferating cells of mouse and human epidermis and cSC

111 Non-proliferating cells oxidize respiratory substrates in mi

112 Together, our findings suggest that proliferating cells rely on both MDH1 and LDH to repleni

113 Lipids are important nutrients that proliferating cells require to maintain energy homeostas

114 tion of normal cells into malignant, rapidly proliferating cells requires major alterations in cell p

115 Proliferating cells showed undifferentiated morphology a

116 our results suggest that Ki-67 expression in proliferating cells spatially organises heterochromatin,

117 Rapidly proliferating cells switch from oxidative phosphorylatio

118 ling centres are specialized clusters of non-proliferating cells that direct the development of many

119 RNA was extracted from proliferating cells versus differentiated neural cells a

120 insight about proteins apportioned for newly proliferating cells versus for somatic maintenance.

121 However, proliferating cells were found only in the CMZ of turtle

122 ion of mTORC1 and likely most predominant in proliferating cells where mTORC1 is highly active.

123 5-bromo-2'-deoxyuridine (BrdU; a marker for proliferating cells) in vivo, consequently interfering w

124 l gap, number of macrophages, blood vessels, proliferating cells, and collagen content in the connect

125 In proliferating cells, APOBEC3A modestly elicited ATR but

126 omal fragments are generally eliminated from proliferating cells, but we know little about how mammal

127 In proliferating cells, formin inhibition abolishes nuclear

128 cells; however, in senescent cells, but not proliferating cells, H4K20me3 enrichment at gene bodies

129 severe damage to tissues containing actively proliferating cells, including bone marrow and the gastr

130 capture the transcriptional dynamics within proliferating cells, methods to differentiate nascent tr

131 We observed that, in the limited subset of proliferating cells, most displayed fermentation of gluc

132 In proliferating cells, PhIP treatment increased the freque

133 In contrast to the role of nuclear RNF8 in proliferating cells, RNF8 operates in the cytoplasm in n

134 assical signaling via RelA was essential for proliferating cells, whereas the alternative signaling p

135 surement of deuterium enrichment into DNA of proliferating cells.

136 ested cells, whereas the opposite is true in proliferating cells.

137 e, highly vascularized tumors that contained proliferating cells.

138 cells, including at genes also repressed in proliferating cells.

139 eneity, and spatial distribution of necrotic/proliferating cells.

140 herapeutic agents that indiscriminately kill proliferating cells.

141 olysis, and a Warburg metabolic phenotype in proliferating cells.

142 tant functions in cytokinesis in mitotically proliferating cells.

143 mes are essential for protein degradation in proliferating cells.

144 ts, primarily by inhibiting DNA synthesis in proliferating cells.

145 required to maintain metabolic stability in proliferating cells.

146 e normally activated during cell division in proliferating cells.

147 be higher in spontaneously quiescent versus proliferating cells.

148 duced G2 checkpoint control and apoptosis in proliferating cells.

149 alterations and genetic diversity in normal proliferating cells.

150 ses the expression of some of these genes in proliferating cells.

151 s, and with KLF4 in differentiating, but not proliferating, cells to promote expression of specialize

152 roteins such as the ring-shaped homotrimeric proliferating cellular nuclear antigen (PCNA).

153 Proliferating cholangiocytes and activated hepatic stell

154 MCs are recruited to proliferating cholangiocytes and promote fibrosis.

155 cretin receptor (SR) axis is up-regulated by proliferating cholangiocytes during cholestasis.

156 Crosstalk between CD4(+) T cells and proliferating chronic lymphocytic leukemia (CLL) tumor B

157 he full transcriptional activities of p53 in proliferating CLL cells may offer a possible therapeutic

158 ificity, and detects extramedullary sites of proliferating clonal plasma cells while providing import

159 es and dissected mouse placentae resulted in proliferating colonies that expressed known markers of T

160 Total body irradiation reduced numbers of proliferating crypts in Ah(Cre)/Met(fl/fl)/LacZ mice.

161 yze metabolite acquisition and allocation in proliferating cultured mammalian cells.

162 e single-cell measurements of p21 protein in proliferating cultures, we show that naturally occurring

163 ct by promoting the switch between quiescent/proliferating/differentiating myoblasts and by maintaini

164 the expression patterns of circular RNAs in proliferating (early-passage) and senescent (late-passag

165 Overall, the two independent examples of proliferating elements illustrate a general DNA transpos

166 Pa ESP was expressed in the proliferating embryonal mass, while it was absent in the

167 the female gametophyte and in the zygote and proliferating endosperm of the Arabidopsis (Arabidopsis

168 Apoptosis of proliferating endothelial cells and inhibition of angiog

169 c studies revealed that multiple myeloma and proliferating endothelial cells can promote CgA C-termin

170 In proliferating enterospheres derived from ENS progenitor

171 After allostimulation, distinct proliferating Eomes(lo)CTLA4(hi) and Eomes(hi)CTLA4(lo)

172 In proliferating epithelia of mammalian skin, cells of irre

173 ion where it controls the renewal of rapidly proliferating epithelial (matrix) progenitors and hence

174 Apical surfaces of proliferating epithelial sheets have been particularly w

175 butyrate likely preventing it from reaching proliferating epithelial stem/progenitor cells within th

176 G challenges induced marked accumulations of proliferating fibroblasts and of myofibroblasts, which w

177 Reducing CircPVT1 levels in proliferating fibroblasts triggered senescence, as deter

178 ceptor (p75(NTR)) is highly expressed in the proliferating GCPs, but is downregulated once the cells

179 issue demonstrated ZNHIT3 to be expressed in proliferating granule cell precursors, in proliferating

180 ts the gene from being induced by Activin in proliferating hESCs.

181 scute family bHLH transcription factor 1) in proliferating hippocampal stem cells, which prevents acc

182 il domain of SF1 is highly phosphorylated in proliferating human cells and is required for cell proli

183 pathways interact to maintain homeostasis in proliferating human cells, using fluorescent reporters f

184 requirement for SF1 SPSP phosphorylation in proliferating human cells.

185 pressed on the surface of senescent, but not proliferating, human diploid fibroblasts.

186 tor activation thresholds in a population of proliferating IgM(+)Nfkappab1(-/-)Fo B cells.

187 and IHH and, thereby control the transit of proliferating immature chondrocytes into mature hypertro

188 ellular synthetic lethality in quiescent and proliferating immature leukemia cells, and is thus a pot

189 In addition, we found that CD8 T cells proliferating in blood after PD-1 therapy of lung cancer

190 Plasmodium species cause malaria by proliferating in human erythrocytes.

191 speculation that cephalopod populations are proliferating in response to a changing environment, a p

192 y of CCR6(+) memory T cells from MG patients proliferating in response to AChR-derived peptides was s

193 oportion of Treg, CD4 Tconv, and CD8 T cells proliferating in response to donor-derived, stimulated B

194 orylation machinery at the stage of the fast proliferating intermediate progenitor cell.

195 warm-blooded animals that causes disease by proliferating intracellularly in muscle and the central

196 females are resistant, associated with fewer proliferating ISCs, suggesting a trade-off between highl

197 pidermal differentiation, with low levels in proliferating KCs and high levels in differentiated cell

198 taining stemness and renewal capacity of the proliferating keratinocyte compartment.

199 monstrate that CYGB has a protective role in proliferating keratinocytes grown under normal condition

200 ted by ZM241385, by increasing the number of proliferating keratinocytes.

201 cells, (ii) an expansion of the fraction of proliferating Ki-67(+) cells, and (iii) high levels of S

202 s and a decrease in the spatial proximity of proliferating (Ki67(+)) cells to CAFs impacting therapeu

203 In proliferating leaf zones, chloroplasts contain much lowe

204 Quiescent and proliferating leukemia cells accumulate highly lethal DN

205 We found that actively proliferating leukemia cells inhibited normal hematopoie

206 ent leukemia cells and BRCA/DNA-PK-deficient proliferating leukemia cells were sensitive to PARP1 inh

207 Rapidly proliferating leukemic progenitor cells consume substant

208 vivo, they are transformed into continuously proliferating lymphoblastoid cell lines (LCLs) that carr

209 rus (EBV) transforms B cells to continuously proliferating lymphoblastoid cell lines (LCLs), which re

210 Interestingly, locally proliferating macrophages were not classically activated

211 ds exhibited low in vitro toxicities in four proliferating mammalian cell lines and in isolated prima

212 lutamine are the major nutrients consumed by proliferating mammalian cells, but the extent to which t

213 gest that the greater reliance on HDR in the proliferating mammary gland, rather than a specific depe

214 We further demonstrated that proliferating MCL harbored an imbalance in Bcl-2 family

215 Chromosomes in proliferating metazoan cells undergo marked structural m

216 , Jak2, or Stat3 suppressed the formation of proliferating MGPCs in NMDA-damaged and FGF2-treated ret

217 combined with FGF2 enhanced the formation of proliferating MGPCs in the absence of retinal damage.

218 of pS6 in Muller glia and reduced numbers of proliferating MGPCs.

219 he upregulation of Pax6 and the formation of proliferating MGPCs.

220 ork of pathways that drives the formation of proliferating MGPCs; however, this pathway inhibits the

221 type are also found to be SIRT1-dependent in proliferating mouse embryonic fibroblasts and differenti

222 Proliferating MYCN/ALK(F1174L) neuroblasts display a dif

223 steps in differentiation are suppressed in a proliferating myoblast is much less clear.

224 idated its role to impede differentiation in proliferating myoblasts and carried out mechanistic stud

225 e that may repress muscle differentiation in proliferating myoblasts in the presence or absence of a

226 EZH2 premature degradation in proliferating myoblasts is prevented by low levels of PJ

227 Silencing MYF5 expression in proliferating myoblasts revealed that MYF5 promoted CCND

228 inase 2 (CK2), a serine/threonine kinase, in proliferating myoblasts.

229 In assessment of neurogenesis, the number of proliferating neural progenitor cells (NPCs) and the num

230 Two distinct cell types were identified as proliferating neural progenitors and immature neurons, b

231 rat brains, we observe cell cycle arrest of proliferating neural progenitors at three distinct stage

232 develop from a partitioned, unidirectionally proliferating neurectodermal domain that combines slowly

233 pulation led to the accumulation of actively proliferating neuroblasts and a lethal brain tumor pheno

234 The number of proliferating neuronal precursor cells was increased wit

235 -null mice lacked Olig2(+) OPCs, and instead proliferating neuronal precursors and GABAergic interneu

236 thood was largely predicted by the number of proliferating neuroprogenitors at each age.

237 a novel transgenic mouse paradigm to ablate proliferating NG2(+) cells after SCI to better understan

238 ork reveals previously unknown ways in which proliferating NG2(+) cells contribute to endogenous repa

239 We show that proliferating NG2(+) pericytes and glia largely segregat

240 Results reveal that loss of proliferating NG2(+) pericytes in the lesion prevented i

241 periphery, new blood vessel growth requires proliferating NG2(+) pericytes; if this were also true i

242 Supporting this hypothesis, proliferating NK cells did not express aldehyde dehydrog

243 After infusion of Cy, a marked reduction of proliferating NK cells was evident, suggesting selective

244 n treatment reduced the density of total and proliferating Nkx2.1(+) and Dlx2(+) cells in the MGEs an

245 Knockout of Khdrbs1 constricted the pool of proliferating NPCs by accelerating their cell cycle exit

246 probe's ability to monitor progression of a proliferating optical heterogeneity.

247 ion in a cell-density dependent manner, with proliferating p38alpha(-/-) cultures showing increased d

248 During pancreatic development, proliferating pancreatic progenitors activate the proend

249 In proliferating parasites, PVM-associated LC3 becomes imme

250 e agent revealed that uptake was confined to proliferating, PARP1-expressing cells.

251 revealed a 97% match between the profiles of proliferating PDG cells and their overlying nonprolifera

252 e results show that therapeutic targeting of proliferating PDGFRbeta(+) cells potently inhibits the f

253 To test this hypothesis we targeted proliferating PDGFRbeta(+) cells through independent dis

254 ucible genetic model to inhibit specifically proliferating PDGFRbeta(+) cells, ii) by treating mice w

255 Cs), characterized by three distinct stages: proliferating phase, involuting phase, and involuted pha

256 ds to their progressive elimination from the proliferating population and might apply to any kind of

257 These reprogrammed MG divide to produce a proliferating population of retinal progenitors that mig

258 taneously impair bone structure and increase proliferating potential of leukemic clone.

259 hogenic cells were mitotically inactive, but proliferating precursors were detected in primary cultur

260 nal programs take neuroepithelial cells from proliferating progenitors to differentiated neurons with

261 nt, to stimulate Muller glia to give rise to proliferating progenitors, and the network of signaling

262 forebrain where Olig2 is mostly expressed in proliferating progenitors, Olig2(+) cells in the cerebel

263 ting small eyes with a reduced CMZ and fewer proliferating progenitors, similar to gdf6a mutants.

264 id not result in widespread apoptosis in the proliferating progenitors.

265 The number of proliferating pulmonary PW1(+) cells and the proportion

266 iggers formation of stress granules (SGs) in proliferating, quiescent, and differentiated muscle cell

267 morphology of the tumor spheroids (layers of proliferating, quiescent, and necrotic cells).

268 oids and metabolism of the drug in the outer proliferating region of the spheroid.

269 Single-cell analysis revealed that, in vivo, proliferating resident macrophages can access this netwo

270 In disc1 mutant embryos, proliferating rx3+ hypothalamic progenitors are not main

271 ensory neuron regeneration and the number of proliferating Schwann cells in the distal stump after th

272 orm in injured nerves, although they contain proliferating Schwann cells with strikingly elevated c-J

273 , both the flown and ground cells were still proliferating slowly, as measured by the percentage of K

274 gnaling induces TGFbeta signaling converting proliferating SMCs to the contractile phenotype, even in

275 that the cells be maintained in an actively proliferating state.

276 dividing differentiated and dedifferentiated proliferating states in vivo.

277 and WWE domain-containing 1) is required for proliferating stem cells of the adult mouse hippocampus

278 ve not established whether SPEM derives from proliferating stem cells or differentiated, post-mitotic

279 maintaining homeostasis, quiescent or slowly proliferating stem/progenitor cells can acquire high pro

280 Herein, we identified a rapidly proliferating subpopulation of cells from the corneal en

281 cially in cancer immunotherapy, where highly proliferating T cells will encounter potentially large a

282 ies of Th1, effector-memory T cells, in situ proliferating T cells, and inhibitory PD1-PDL1 cells, ha

283 Rare proliferating tetraploid cells can emerge from acute pol

284 infiltrating T cells and expanded numbers of proliferating thyrocytes that highly express CD40.

285 ay between cortical cues and cell shape in a proliferating tissue.

286 ibution of cellular polygons is conserved in proliferating tissues among metazoans.

287 tor cells of embryonic human brain and other proliferating tissues, is co-expressed with components o

288 3 nuclear translocation is a common event in proliferating tissues.

289 ong-lived proteins as they transition from a proliferating to a quiescent state.

290 r glia are capable of de-differentiating and proliferating to become Muller glia-derived progenitor c

291 in power grids triggered by local overloads proliferating to downstream nodes eventually leading to

292 In the late stage, HeLa cells change from proliferating to migratory.

293 minent epithelial-free regions, enriched for proliferating tumor cells and dendritic cells (DCs).

294 Rapidly proliferating tumor cells upregulate specific amino acid

295 normal biosynthetic requirements of actively proliferating tumor cells.

296 feration, and Ca(2+) signaling is altered in proliferating tumor cells.

297 yzed in sorted CD3(+) CD4(+) CFSE(low) cells proliferating upon stimulation of PBMC with Dau c 1 or B

298 ls became sensitive to treatment and started proliferating when dissociated from the in vivo environm

299 decrease the number of palindromes in cells proliferating without telomeres.

300 Within the proliferating zone of the Arabidopsis root, regular symm