ライフサイエンスコーパス: proliferating-cell

1 tant functions in cytokinesis in mitotically proliferating cells.

2 nd contained a higher frequency of TNFR2(hi) proliferating cells.

3 o undergird future work on the metabolism of proliferating cells.

4 formation on metabolic flux distributions of proliferating cells.

5 uvate kinase (PK) in directing metabolism of proliferating cells.

6 limiting the spread of enveloped viruses in proliferating cells.

7 tablishing highly active metabolic states in proliferating cells.

8 activator MYB3R4 was in complex with E2FB in proliferating cells.

9 lization and availability of beta-catenin in proliferating cells.

10 he time window of mitotic gene expression in proliferating cells.

11 tivates the expression of IGFBP3 in actively proliferating cells.

12 n family, known to regulate cell division in proliferating cells.

13 ining cellular redox homoeostasis in rapidly proliferating cells.

14 ating cells and the latter being enhanced in proliferating cells.

15 ding cells were at least as high as those of proliferating cells.

16 s peroxide-dependent uptake and retention in proliferating cells.

17 mes are essential for protein degradation in proliferating cells.

18 KB1 induces a progrowth metabolic program in proliferating cells.

19 ed villus height and a compensatory shift in proliferating cells.

20 t that ORP4 promotes the survival of rapidly proliferating cells.

21 ts, primarily by inhibiting DNA synthesis in proliferating cells.

22 proportion of persisting proviruses were in proliferating cells.

23 l accumulation because of increased death of proliferating cells.

24 required to maintain metabolic stability in proliferating cells.

25 vity, demonstrated the endothelial nature of proliferating cells.

26 e normally activated during cell division in proliferating cells.

27 s the most robust transcription machinery in proliferating cells.

28 to duplicate and to segregate efficiently in proliferating cells.

29 be higher in spontaneously quiescent versus proliferating cells.

30 eased nuclear size and crowding from rapidly proliferating cells.

31 duced G2 checkpoint control and apoptosis in proliferating cells.

32 alterations and genetic diversity in normal proliferating cells.

33 ses the expression of some of these genes in proliferating cells.

34 ested cells, whereas the opposite is true in proliferating cells.

35 e, highly vascularized tumors that contained proliferating cells.

36 surement of deuterium enrichment into DNA of proliferating cells.

37 cells, including at genes also repressed in proliferating cells.

38 eneity, and spatial distribution of necrotic/proliferating cells.

39 herapeutic agents that indiscriminately kill proliferating cells.

40 olysis, and a Warburg metabolic phenotype in proliferating cells.

41 Tumors initiate when a population of proliferating cells accumulates a certain number and typ

42 Proliferating cells acquire genome alterations during th

43 Proliferating cells actively control their size by mecha

44 Proliferating cells adjust their cell size depending on

45 2 may neutralize and elevate p53 in actively proliferating cells and also provides a rationale for us

46 pression of LAT induced a reduction of brain proliferating cells and concomitant microcephaly.

47 he enamel organ, where it remained devoid of proliferating cells and contained apoptotic cells with i

48 nd MCM7 Y600 phosphorylation are enhanced in proliferating cells and correlated with poor survival of

49 oncepts regarding metabolic reprogramming in proliferating cells and discuss their potential impact o

50 anti-CD3 and anti-CD28 reduced the number of proliferating cells and entry of cells into the cell cyc

51 SL baboons had lower numbers of proliferating cells and immature neurons than socially d

52 ential for maintenance of DNA methylation in proliferating cells and is considered an important cance

53 perfamily; the gene is strongly expressed in proliferating cells and is rapidly and transiently up-re

54 differentiation can result in a depletion of proliferating cells and organ failure.

55 protein accumulation pattern correlated with proliferating cells and was sub-localized to the nucleus

56 ped in-house, allowing for quantification of proliferating cells, and BM blood volume was estimated b

57 l gap, number of macrophages, blood vessels, proliferating cells, and collagen content in the connect

58 fundamental biological need, especially for proliferating cells, and controlling nutrient supply is

59 icited a relative reduction in tumor growth, proliferating cells, and microvessel density.

60 two distinct subsets, one that is induced in proliferating cells, and repressed otherwise, and anothe

61 ssociated with aerobic glycolysis in tumors, proliferating cells, and some other cell types.

62 racteristic of differentiating cells than of proliferating cells, and we identified CpG clusters that

63 In proliferating cells, APOBEC3A modestly elicited ATR but

64 Highly proliferating cells are particularly dependent on glucos

65 Proliferating cells are preferentially susceptible to in

66 and slower growth due to reduced numbers of proliferating cells as the result of increased apoptosis

67 , protein, and fatty acid synthesis found in proliferating cells as well as for neutralizing high lev

68 2, IL-2, and IFN-gamma), myofibroblasts, and proliferating cells, as well as increased anti-inflammat

69 The proliferating cells attained a steady state, their slow

70 or CDK2 activity and unexpectedly found that proliferating cells bifurcate into two populations as th

71 ata indicate that La is required not only in proliferating cells but also in nondividing postmitotic

72 P-1) is overexpressed in various cancers and proliferating cells, but the functional significance rem

73 omal fragments are generally eliminated from proliferating cells, but we know little about how mammal

74 us RNA-Seq (sNuc-Seq) with pulse labeling of proliferating cells by 5-ethynyl-2'-deoxyuridine (EdU) t

75 s promote gap junction communication between proliferating cells by promoting dynamical microtubule p

76 sive crypt degeneration and lower numbers of proliferating cells compared with wild-type control mice

77 When proliferating cells complete mitosis, a fraction of newl

78 It is not clear how stem and proliferating cells cope with accumulating endogenous DN

79 lockade of WNT/beta-catenin signaling in the proliferating cells decreases proliferation activity, bu

80 However, proliferating cells depend on growth-factor-induced incr

81 It is unclear how proliferating cells elicit suppression on cell prolifera

82 tors involved in renal repair, the number of proliferating cells, especially at the injured edge, the

83 tive DNA damage response are well studied in proliferating cells, especially with regards to the deve

84 Cancer and proliferating cells exhibit an increased demand for glut

85 Normal proliferating cells express sense (SncmtRNA) and antisen

86 TCP (for Teosinte branched1, Cycloidia, and Proliferating cell factor) genes from petunia, and have

87 TEOSINTE BRANCHED1-CYCLOIDEA-PROLIFERATING CELL FACTOR1 (TCP) transcription factors p

88 we report that teosinte branched1/cycloidea/proliferating cell factor1-20 (TCP20) and NIN-like prote

89 ion factor gene TEOSINTE BRANCHED1/CYCLOIDEA/PROLIFERATING CELL FACTOR1-20 (TCP20) was identified.

90 egulated TCP (TEOSINTE BRANCHED 1, CYCLODEA, PROLIFERATING CELL FACTORS) transcription factors, notab

91 continually diluting out timer components in proliferating cells (Figure 1A, right).

92 In proliferating cells, formin inhibition abolishes nuclear

93 Our data suggest that transformed proliferating cells from HCC are more sensitive to knock

94 found a significantly greater percentage of proliferating cells from mice fed the high-fat/high-chol

95 hin the niche, but by prematurely displacing proliferating cells from their normal epithelial milieu.

96 cells; however, in senescent cells, but not proliferating cells, H4K20me3 enrichment at gene bodies

97 ough the importance of folate metabolism for proliferating cells has been long recognized and attribu

98 Proliferating cells have been found along the entire ven

99 ulture apparatus (Nutrostat) for maintaining proliferating cells in low-nutrient media for long perio

100 PD0325901 treatment reduced aberrantly proliferating cells in neurofibroma and MPNST, prolonged

101 The rapidly proliferating cells in plant meristems must be protected

102 d, where they were the predominant source of proliferating cells in the aortic wall.

103 During early gonadogenesis, proliferating cells in the coelomic epithelium (CE) give

104 Three different populations of proliferating cells in the dentate gyrus (DG) were label

105 Proliferating cells in the ventricular zone stem cell co

106 5-bromo-2'-deoxyuridine (BrdU; a marker for proliferating cells) in vivo, consequently interfering w

107 Key targets of Cdk4 in proliferating cells include members of the E2F transcrip

108 severe damage to tissues containing actively proliferating cells, including bone marrow and the gastr

109 Rapidly proliferating cells increase glycolysis at the expense o

110 ivity and prevents the outgrowth of cilia in proliferating cells, independent of Aurora A and HDAC6.

111 find that depleting either SAFA or PANDA in proliferating cells induces senescence.

112 phosphate (SAICAR), a metabolite abundant in proliferating cells, induces PKM2's protein kinase activ

113 s additional evidence that the metabolism of proliferating cells is adapted to facilitate producing n

114 s suggest that increased (18)F-FLT uptake by proliferating cells is due to a greater fraction of (18)

115 lls, suggesting that end-joining activity in proliferating cells is more likely to be mutagenic.

116 is modification controls SAMHD1 functions in proliferating cells is not known.

117 we show that a major role of respiration in proliferating cells is to provide electron acceptors for

118 ata argue a major function of respiration in proliferating cells is to support aspartate synthesis.

119 n also promote adaptability and diversity in proliferating cell lineages, although whether WGD is ben

120 rasting effect in postmitotic neurons versus proliferating cell lines.

121 capture the transcriptional dynamics within proliferating cells, methods to differentiate nascent tr

122 We observed that, in the limited subset of proliferating cells, most displayed fermentation of gluc

123 To maintain the integrity of the genome, proliferating cells must be able to block progression th

124 Proliferating cells must cross a point of no return befo

125 bacteria can promote sustained expression of proliferating cell nuclear Ag and that human PMN undergo

126 polarization, PMN-SA had sustained levels of proliferating cell nuclear Ag expression, absence of cas

127 interacting partners of EndoQ and identified Proliferating Cell Nuclear Angigen (PCNA).

128 wn as the PIP degron, which binds DNA-loaded proliferating cell nuclear antigen (PCNA(DNA)) and recru

129 During DNA replication, proliferating cell nuclear antigen (PCNA) adopts a ring-

130 Proliferating cell nuclear antigen (PCNA) and alpha-smoo

131 Overexpression of cyclins and proliferating cell nuclear antigen (PCNA) and evidence f

132 ociated with decreased platinum drug-induced proliferating cell nuclear antigen (PCNA) and FANCD2 mon

133 Mtp53 increased the level of proliferating cell nuclear antigen (PCNA) and minichromo

134 th muscle mass, paired box protein 7 (Pax7), proliferating cell nuclear antigen (PCNA) and nicotinami

135 fespan of replication factories by retaining proliferating cell nuclear antigen (PCNA) and other repl

136 omethylates non-histone substrates including proliferating cell nuclear antigen (PCNA) and promotes c

137 through its interactions with two proteins, Proliferating Cell Nuclear Antigen (PCNA) and Replicatio

138 the essential replication accessory protein proliferating cell nuclear antigen (PCNA) and the scaffo

139 e show that the ternary complexes containing proliferating cell nuclear antigen (PCNA) and two non-cl

140 In this study, we identified the proliferating cell nuclear antigen (PCNA) as a nIGF-1R-b

141 shown previously that the sliding DNA clamp proliferating cell nuclear antigen (PCNA) associates wit

142 es efficient mono- and polyubiquitination of proliferating cell nuclear antigen (PCNA) by regulating

143 n multiple transient events in the reaction: proliferating cell nuclear antigen (PCNA) clamp binding/

144 nd the homotrimeric Saccharomyces cerevisiae proliferating cell nuclear antigen (PCNA) clamps using s

145 of alpha-smooth muscle actin (alpha-SMA) and proliferating cell nuclear antigen (PCNA) compared with

146 inding protein 1 (Ebp1), a key in regulating proliferating cell nuclear antigen (PCNA) expression and

147 s a mismatch, MutSalpha/beta, and DNA-loaded proliferating cell nuclear antigen (PCNA) for activation

148 Proliferating cell nuclear antigen (PCNA) forms a trimer

149 Proliferating cell nuclear antigen (PCNA) forms a trimer

150 xyuridine (EdU) labeling as well as Ki67 and proliferating cell nuclear antigen (PCNA) immunofluoresc

151 Proliferating cell nuclear antigen (PCNA) is a highly co

152 Proliferating cell nuclear antigen (PCNA) is a protein w

153 The sliding clamp proliferating cell nuclear antigen (PCNA) is an indispen

154 Proliferating cell nuclear antigen (PCNA) lies at the ce

155 esion and a helix perturbation that supports proliferating cell nuclear antigen (PCNA) loading by rep

156 Proliferating cell nuclear antigen (PCNA) loading by rep

157 early and mid-S phase and during DNA repair, proliferating cell nuclear antigen (PCNA) loading onto D

158 The homotrimeric sliding clamp proliferating cell nuclear antigen (PCNA) mediates Okaza

159 c expression of USP7 promoted the UV-induced proliferating cell nuclear antigen (PCNA) monoubiquitina

160 The E3 ligase RAD18 is necessary for proliferating cell nuclear antigen (PCNA) monoubiquitina

161 loading of cyclin-dependent kinase (CDK) and proliferating cell nuclear antigen (PCNA) onto chromatin

162 The sliding clamp proliferating cell nuclear antigen (PCNA) plays a vital

163 nt studies have shown that monoubiquitinated proliferating cell nuclear antigen (PCNA) plays an impor

164 Proliferating cell nuclear antigen (PCNA) plays an impor

165 ns wherein the interaction between RECQ5 and proliferating cell nuclear antigen (PCNA) promotes RAD18

166 e lagging strand template and anchors to the proliferating cell nuclear antigen (PCNA) sliding clamp

167 ted, while raised levels of doublecortin and Proliferating Cell Nuclear Antigen (PCNA) suggested incr

168 in the recruitment of the DNA clamp protein proliferating cell nuclear antigen (PCNA) to DNA lesions

169 show that Bub1 constitutively interacts with proliferating cell nuclear antigen (PCNA) via a highly c

170 -shaped virus 2 (SIRV2) and the host-encoded proliferating cell nuclear antigen (PCNA), a key DNA rep

171 Here, we show that monoubiquitinated Proliferating Cell Nuclear Antigen (PCNA), a marker of s

172 from Thermoplasma acidophilum interact with proliferating cell nuclear antigen (PCNA), an essential

173 n of Kdm4d impairs the recruitment of Cdc45, proliferating cell nuclear antigen (PCNA), and polymeras

174 ion, as evidenced by stabilization of Mcl-1, proliferating cell nuclear antigen (PCNA), and pro-caspa

175 reaction requiring Msh2-Msh6 (or Msh2-Msh3), proliferating cell nuclear antigen (PCNA), and replicati

176 Proliferating cell nuclear antigen (PCNA), bone sialopro

177 hen the Rad6/Rad18 complex monoubiquitinates proliferating cell nuclear antigen (PCNA), but the basis

178 st time, the ATP-binding capability of human proliferating cell nuclear antigen (PCNA), identified th

179 oleta precludes its ability to interact with proliferating cell nuclear antigen (PCNA), it enhances i

180 ssive replication with the replication clamp proliferating cell nuclear antigen (PCNA), respectively.

181 A We show that Rad51 inhibits recruitment of proliferating cell nuclear antigen (PCNA), the platform

182 Proliferating cell nuclear antigen (PCNA), the processiv

183 Lack of SIM, but not proliferating cell nuclear antigen (PCNA)-interacting mo

184 action between the Enok complex and the Elg1 proliferating cell nuclear antigen (PCNA)-unloader compl

185 d together they enhance the transcription of proliferating cell nuclear antigen (PCNA).

186 strongly stimulated by its interaction with proliferating cell nuclear antigen (PCNA).

187 is dependent on the interaction of TDG with proliferating cell nuclear antigen (PCNA).

188 nse (p.Ser228Ile) sequence alteration of the proliferating cell nuclear antigen (PCNA).

189 o DNA is mediated by their interactions with proliferating cell nuclear antigen (PCNA).

190 e demonstrated interactions between TXR1 and proliferating cell nuclear antigen (PCNA).

191 increased expression of Nox4, TNF-alpha, and proliferating cell nuclear antigen (PCNA).

192 Small molecule inhibitors of proliferating cell nuclear antigen (PCNA)/PCNA interacti

193 be stimulated by processivity clamps such as proliferating cell nuclear antigen (PCNA); however, the

194 ient for loading the heterotrimeric PCNA123 [proliferating cell nuclear antigen (PCNA)] clamp onto DN

195 we explore the interactions of S. cerevisiae Proliferating Cell Nuclear Antigen (yPCNA) with modified

196 In the absence of NKT cells hepatic proliferating cell nuclear antigen and cyclin B1 decreas

197 ion protein A, replication factor C-Delta1N, proliferating cell nuclear antigen and DNA polymerase de

198 Whereas association with proliferating cell nuclear antigen and participation in

199 a, leukocyte infiltration, and expression of proliferating cell nuclear antigen and tenascin-C.

200 Proliferating cell nuclear antigen and the checkpoint cl

201 We further show that proliferating cell nuclear antigen and the nucleosome co

202 Immunostaining with proliferating cell nuclear antigen and Von Willebrand fa

203 show that NEIL1 binarily interacts with the proliferating cell nuclear antigen clamp loader replicat

204 Proliferating cell nuclear antigen content and signal tr

205 l neurogenesis, measured by doublecortin and proliferating cell nuclear antigen expression, was also

206 instability 1 are responsible for unloading proliferating cell nuclear antigen from newly synthesize

207 incorporation and proportionate increases in proliferating cell nuclear antigen gene expression.

208 ts stem from differences in stabilization of proliferating cell nuclear antigen in an open conformati

209 eased the levels of cyclin D1, E, and A, and proliferating cell nuclear antigen in meningeal cells wh

210 A conserved proliferating cell nuclear antigen interaction protein b

211 )O(2) treatments both induced Rad18-mediated proliferating cell nuclear antigen mono-ubiquitination d

212 ither translesion DNA synthesis initiated by proliferating cell nuclear antigen monoubiquitination or

213 localizing to sites of DNA damage, inducing proliferating cell nuclear antigen monoubiquitination, a

214 The eukaryotic DNA polymerase sliding clamp, proliferating cell nuclear antigen or PCNA, is a ring-sh

215 e pathway dependent on polyubiquitylation of proliferating cell nuclear antigen provides a backup mec

216 e upon chronic cholestasis, but unexpectedly proliferating cell nuclear antigen was down-regulated at

217 LR assessed by Ki-67 and proliferating cell nuclear antigen was markedly decrease

218 to promote senescence, whereas cyclin D1 and proliferating cell nuclear antigen were decreased to red

219 Expression of hepatic cyclin B1 and proliferating cell nuclear antigen were evaluated by Wes

220 Ki-67 and proliferating cell nuclear antigen were used to measure

221 tethers it to the leading strand, and PCNA (proliferating cell nuclear antigen) binds tightly to Pol

222 t impede replication by ubiquitinating PCNA (proliferating cell nuclear antigen) using the RAD6-RAD18

223 Proliferating cell nuclear antigen, a DNA sliding clamp,

224 In reconstituted human systems, MutSalpha, proliferating cell nuclear antigen, and replication fact

225 fects resulted in PASMC proliferation (Ki67, proliferating cell nuclear antigen, and WST1 assays) and

226 were determined by methyltetrazolium, Ki-67, proliferating cell nuclear antigen, bromodeoxyuridine, a

227 o examine the mRNA and protein expression of proliferating cell nuclear antigen, cyclin D1, E-cadheri

228 d DNA ligase I, which compete for binding to proliferating cell nuclear antigen, is critical to preve

229 Significant reduction in levels of proliferating cell nuclear antigen, NF-kappabeta/p50, cy

230 esis phase of the cell cycle (e.g. P. patens proliferating cell nuclear antigen, ribonucleotide reduc

231 resulted in accumulation of chromatin-bound proliferating cell nuclear antigen, slowed cell division

232 gy with Cavalieri estimation; apoptosis with proliferating cell nuclear antigen, TUNEL, and caspase a

233 ed arteries as indicated by co-staining with proliferating cell nuclear antigen, whereas Notch3 was e

234 or how nucleosome binding protects Set8 from proliferating cell nuclear antigen-dependent degradation

235 included demonstration that p12 possesses a proliferating cell nuclear antigen-interacting protein-d

236 cluding the catalytic polymerase domain, the proliferating cell nuclear antigen-interacting region, t

237 gnificantly reduced (P < 0.05) the number of proliferating cell nuclear antigen-positive tubular epit

238 h cyclin-dependent kinase 2 (CDK2), CDK4 and proliferating cell nuclear antigen.

239 polymerase-alpha and the replication clamp, proliferating cell nuclear antigen.

240 sion by the replicative DNA polymerase delta/proliferating cell nuclear antigen/replication factor C

241 D1, and the cell cycle genes CYCLIN A2;1 and PROLIFERATING CELL NUCLEAR ANTIGEN1 in buds was suppress

242 tion is essential for mono-ubiquitination of proliferating-cell nuclear antigen in response to oxidat

243 the first structural characterization of the proliferating-cell-nuclear-antigen-associated factor p15

244 eferences in distinct regions, including the proliferating-cell-nuclear-antigen-interacting protein m

245 lated kinase 1/2 (ERK1/2) and a reduction of proliferating cell numbers.

246 These non-proliferating cells occupy key cellular niches and elabo

247 to intrafiber NRL variations, as relevant to proliferating cells of interphase chromatin, fibers subj

248 e characterization of gene expression in the proliferating cells of M. lignano, represented by somati

249 YAP and WPB2 are upregulated in actively proliferating cells of mouse and human epidermis and cSC

250 M proteins limit Hippo regulation of YAP, in proliferating cells only, by sequestering a cytosolic Hi

251 Non-proliferating cells oxidize respiratory substrates in mi

252 iving IRL-1620 displayed increased number of proliferating cells (P<0.0001) and cells positively stai

253 nd PCL3 are E2F-regulated genes expressed in proliferating cells, PCL1 is a p53 target gene predomina

254 In proliferating cells, PhIP treatment increased the freque

255 diminished, correlating with the loss of the proliferating cell population of germinal zones.

256 etween these mechanisms can push an actively proliferating cell population to transition into a weakl

257 In senescent cells, but not proliferating cells, promoters of active genes are excep

258 In genome sequences from proliferating cells, read depth along chromosomes reflec

259 Together, our findings suggest that proliferating cells rely on both MDH1 and LDH to repleni

260 Lipids are important nutrients that proliferating cells require to maintain energy homeostas

261 tion of normal cells into malignant, rapidly proliferating cells requires major alterations in cell p

262 In contrast to the role of nuclear RNF8 in proliferating cells, RNF8 operates in the cytoplasm in n

263 In proliferating cells, SAFA and PANDA recruit PRC complexe

264 Bromodeoxyuridine (BrdU) labeling of proliferating cells showed that ASCs produced in the dra

265 Proliferating cells showed undifferentiated morphology a

266 our results suggest that Ki-67 expression in proliferating cells spatially organises heterochromatin,

267 biosynthetic and energetic demand of rapidly proliferating cells such as cancer cells is met by metab

268 ation, similar to that seen in other rapidly proliferating cells such as cancer cells.

269 ;2/TAM expression was found primarily in non-proliferating cells such as guard cells, trichomes, and

270 Rapidly proliferating cells switch from oxidative phosphorylatio

271 ling centres are specialized clusters of non-proliferating cells that direct the development of many

272 together with perforin and (2) induction of proliferating cells that had greater capacity to produce

273 The proportion of proliferating cells that were Olig2(+) was similar throu

274 In proliferating cells, the largest contributor to cytosoli

275 CYP2W1 was also shown to sensitize proliferating cells to several compounds, demonstrating

276 ence, but the contribution of these clonally proliferating cells to the rebounding virus is unknown.

277 s, and with KLF4 in differentiating, but not proliferating, cells to promote expression of specialize

278 As proliferating cells transit from interphase into M-phase

279 Lamin B1 reduction in proliferating cells triggers senescence and formation of

280 Although its role in proliferating cell types and tissues has been extensivel

281 Cdc6 resynthesis, our results indicate that proliferating cells use a window of time in mitosis, bef

282 istribution of the antigen, vasculature, and proliferating cells using immunohistochemistry.

283 RNA was extracted from proliferating cells versus differentiated neural cells a

284 insight about proteins apportioned for newly proliferating cells versus for somatic maintenance.

285 y all animals and only limited cell death of proliferating cells was found within the generative zone

286 In proliferating cells, we found that hnRNPA1 binds and des

287 Substantial percentages of proliferating cells were also found in intestinal tissue

288 the jejunum, ileum, and colon, but very few proliferating cells were detected in lymph nodes (axilla

289 center formation was examined and numbers of proliferating cells were evaluated by Ki-67 staining.

290 However, proliferating cells were found only in the CMZ of turtle

291 9-23 peptide in proliferation assays and the proliferating cells were highly enriched in certain T-ce

292 Confocal microscopy also demonstrated that proliferating cells were substantial viral target cells

293 ion of mTORC1 and likely most predominant in proliferating cells where mTORC1 is highly active.

294 assical signaling via RelA was essential for proliferating cells, whereas the alternative signaling p

295 ccumulation of SMN in Cajal bodies of intact proliferating cells, which actively assemble snRNPs, as

296 resses stem cell-promoting genes in actively proliferating cells, which is essential for the progress

297 small number of stem cells or is composed of proliferating cells with approximate phenotypic equivale

298 unohistochemistry and identify central canal proliferating cells with Ki67 and newly generated cells

299 EdU we show, as others have previously, that proliferating cells within the cornea epithelium do cont

300 Hdac7 was highly expressed in proliferating cells within the growth plate.