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1 ere found in Munich Wistar Fromter rats with proliferative glomerulonephritis.
2 lupus erythematosus with antinuclear Abs and proliferative glomerulonephritis.
3 y revealed enlarged glomeruli with mesangial-proliferative glomerulonephritis.
4 ontaneous and chronic serum sickness-induced proliferative glomerulonephritis.
5 fuse (53%), focal (28%), and mesangial (13%) proliferative glomerulonephritis.
6 smaller quantities in patients with non-IgA proliferative glomerulonephritis and in healthy controls
7 ria at flare and was higher in patients with proliferative glomerulonephritis and in patients with im
9 gration in the anti-Thy 1 model of mesangial proliferative glomerulonephritis and provide evidence fo
10 lomerulonephritis, and diffuse endocapillary proliferative glomerulonephritis associated with systemi
12 gic diagnoses on recurrence included diffuse proliferative glomerulonephritis, focal proliferative gl
15 anded (ss) DNA (11F8) induces severe diffuse proliferative glomerulonephritis in nonautoimmune mice w
18 black 76%, other 36%; P < 0.05) and diffuse proliferative glomerulonephritis +/- membranous glomerul
19 fuse proliferative glomerulonephritis, focal proliferative glomerulonephritis, membranous glomerulone
20 1) normal by light microscopy, (2) mesangial proliferative glomerulonephritis, or (3) focal segmental
21 develop antiphospholipid syndrome (APS) and proliferative glomerulonephritis that is markedly accele
23 enterocolitis with adherent bacterial rods, proliferative glomerulonephritis, tubular necrosis, and
25 teria for acute transplant glomerulopathy or proliferative glomerulonephritis were not satisfied.
26 autoantibody levels and developed mesangial proliferative glomerulonephritis, which resembled system
27 (hu)Tg/CFH(-/-) mice spontaneously developed proliferative glomerulonephritis, which was accelerated
28 triggered severe nephritis, characterized by proliferative glomerulonephritis with crescent formation
29 Renal biopsy demonstrated a florid, diffuse, proliferative glomerulonephritis with glomerular immune
32 Anti-PR3 treated mice had mild pauci-immune proliferative glomerulonephritis, with infiltration of h
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