ライフサイエンスコーパス: promonocyte

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1 rement for C/EBP activators by HIV-1 only in promonocytes.

2 diated silencing of TNF-alpha in THP-1 human promonocytes.

3 his concept using TLR4-stimulated THP1 human promonocytes, a model that mimics the initiation and ada

4 ells, hamster ovary CHO cells, and the human promonocyte cell line U937 cells were not susceptible to

5 In a human promonocyte cell line, MIS was able to bind Src homology

6 expression of COX-2 and sIL-1 RA in a human promonocyte cell line, THP-1.

7 U937 promonocyte cell populations demonstrated significant he

8 n occurred in Jurkat T cells but not in U937 promonocytes, demonstrating a requirement for C/EBP acti

9 illing by primary macrophages or human THP-1 promonocytes differentiated to a macrophage phenotype.

10 disrupted in endotoxin tolerant THP-1 human promonocyte due to changes in transcription factor bindi

11 SIRT6 occurs in THP1 cells and primary human promonocytes during inflammation and in splenocytes from

12 mid completely abolished the accumulation of promonocytes in the bone marrow.

13 RA pretreatment of latently infected U1 promonocytes inhibits HIV-1 expression in response to th

14 12-O-tetradecanoylphorbol 13-acetate (PMA), promonocyte-like U937 cells differentiate into macrophag

15 Using a THP-1 human promonocyte model of endotoxin tolerance that simulates

16 Accelerated phagocytosis is a hallmark of promonocyte, monocyte, and macrophage activation and its

17 ed mice and found that it was blocked at the promonocyte stage in the bone marrow.

18 We found in TLR4-stimulated THP-1 promonocytes that SirT1 and SirT 6 support a switch from

19 ed primary human macrophages and human THP-1 promonocytes to characterize the role of PLD in phagocyt

20 We utilized a cell-free system from U937 promonocytes to test the hypothesis that stimulation of

21 We used THP-1 human promonocytes, which mimic gene silencing when rendered e

22 s of NF-kappaB occur in LPS responsive THP-1 promonocytes with recruitment and binding of NF-kappaB p

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