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1                                              Promonocytic CD4+ cells (U937) were made resistant to HI
2 irion production from a chronically infected promonocytic cell line (U1) and in acutely infected mono
3 e activation of HIV in the latently infected promonocytic cell line (U1) and T-cell line (ACH-2) up-r
4 d induction of HIV expression in U1 cells, a promonocytic cell line chronically infected with HIV.
5 in monocytes, we transiently transfected the promonocytic cell line HL-60 with a chloramphenicol acet
6   Measurements of cyclin T1 mRNA levels in a promonocytic cell line suggested that regulation of cycl
7  for HIV-1 infectivity, clones from the U937 promonocytic cell line that express similar levels of CD
8                       Treatment of the human promonocytic cell line THP-1 with TNF-TFOs at a nontoxic
9                   In this study, we used the promonocytic cell line THP-1, an established model for m
10 eNC2 and GG2EE cells as well as in the human promonocytic cell line THP-1.
11 f calcium mobilization and chemotaxis of the promonocytic cell line THP-1.
12 ave been studied by using the HIV-1-infected promonocytic cell line U1.
13 by Salmonella in the latently infected human promonocytic cell line U1.
14 tor alpha (TNFalpha) production in the human promonocytic cell line U38.
15 ary human monocytes, as well as in the human promonocytic cell line U38.
16  to plasma from diabetic patients, the human promonocytic cell line U937 exhibits a significant eleva
17 acetate (PMA)-induced differentiation of the promonocytic cell line U937 leads to persistent NF-kappa
18 al experiments revealed that exposure of the promonocytic cell line U937 to hypoxia resulted in incre
19 can be mimicked in vitro by treatment of the promonocytic cell line U937 with PMA.
20 d from a patient with AML-M2 t(8;21) and the promonocytic cell line U937.
21 from human peripheral blood and for THP-1, a promonocytic cell line used as a model system.
22        In vitro studies using THP-1 cells (a promonocytic cell line) confirmed that macrophages expre
23 ivator of transcription-1 (Stat1) in a human promonocytic cell line, which was previously shown to be
24 onstrated by experiments with the human U937 promonocytic cell line.
25 e examined a number of subclones of the U937 promonocytic cell line.
26 st, phorbol ester-induced differentiation of promonocytic cell lines into macrophage-like cells produ
27                                     Although promonocytic cell lines such as U937 have been used as i
28 ytes and upon differentiation of U1 and U937 promonocytic cell lines to macrophages.
29 y based upon nonphysiological stimulation of promonocytic cell lines which may respond and process TA
30 lood mononuclear cells and transformed T and promonocytic cell lines.
31  and following macrophage differentiation of promonocytic cell lines.
32  in cellular extracts from Jurkat T and U937 promonocytic cell lines.
33 R) function in cells expressing HIV-1, human promonocytic cells (U937) acutely or chronically infecte
34 st-transcriptional mechanism when human U937 promonocytic cells are stimulated to differentiate into
35                                   U937 human promonocytic cells chronically infected with HIV-1 (U1 c
36                   Upon differentiation, U937 promonocytic cells gain the ability to release a large f
37 r restriction of M-tropic HIV-1 infection in promonocytic cells occurs at the fusion/entry level, tha
38                                              Promonocytic cells stably overexpressing an IkappaBalpha
39 f phosphorylation in receptor function, U937 promonocytic cells were stably transfected to express th
40 ippled in their ability to replicate in U937 promonocytic cells, indicating that these sites are requ
41 f cervicovaginal epithelial cells and U1/HIV promonocytic cells, showed that multiple N-9 use can pro
42 protein (Cdk9-dn) in Jurkat T cells and U937 promonocytic cells.
43 i-infected phorbol-differentiated U937 human promonocytic cells.
44 2 T-lymphocytic cells but not in HDAC4(-) U1 promonocytic cells.
45  of NF-IL6 mRNA following activation of U937 promonocytic cells.
46 activated by gamma interferon (IFN-gamma) in promonocytic cells.
47  in HIV-mediated cell-to-cell fusion in U937 promonocytic cells.
48 to a cellular factor induced by IFN-gamma in promonocytic cells.
49 r cells as well as in cells of H9 T and U937 promonocytic human cell lines.
50 P0 prevented HSV-1-induced disintegration of promonocytic leukemia (PML) nuclear bodies, an intracell
51 ze to punctate nuclear regions identified as promonocytic leukemia protein (PML) oncogenic domains (P
52 lar kinetics were observed with U-937 (human promonocytic leukemia) cells, used as a model for the bl
53                              Using the human promonocytic THP-1 cell line, we demonstrated that the e
54 ort, we demonstrate that LPS-tolerized human promonocytic THP-1 cells develop cross-tolerance and no
55 we focused on the dynamic responses of human promonocytic THP-1 cells to lipopolysaccharide (LPS).
56                        Transfection of human promonocytic U-937 cells with an HTLV-I Tax expression v
57 nomenon was observed in chronically infected promonocytic U1 cells differentiated to macrophage-like
58 ear pool of NF-kappaB (RelA and RelB) in the promonocytic U937 cell line using dominant-negative Ikap
59 omplement content, and incubated with either promonocytic U937 cells or normal human peripheral blood
60 lity of TNF-alpha to induce apoptosis in the promonocytic U937 cells, (ii) the discovery of a cross-t
61 increased expression of MHCII genes in human promonocytic U937 cells, which represent immature antige
62 ing both isoforms are also observed in human promonocytic U937 cells.
63 ed RNA-dependent protein kinase (PKR) in the promonocytic U937 cells.
64 erential TNF-alpha susceptibilities of human promonocytic U937 subclones, deficient in or overexpress
65 in-D(3)-induced differentiation of the human promonocytic U937cells, whereas ectopic SET expression i

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