ライフサイエンスコーパス: promote proliferation

1 ion that maintains cell health, but does not promote proliferation).

2 t ER-positive breast cancers rely on CDK4 to promote proliferation.

3 ng with nutrients, to preserve viability and promote proliferation.

4 interactions mediated by Nanog dimerization promote proliferation.

5 of KLF5 and enhances the ability of KLF5 to promote proliferation.

6 tical RNA binding proteins, FBF-1 and FBF-2, promote proliferation.

7 1 overexpression, however, was sufficient to promote proliferation.

8 , the resulting chimeric protein efficiently promoted proliferation.

9 maturation and cell cycle exit and, instead, promoted proliferation.

10 CMZ, whereas a glucagon-receptor antagonist promoted proliferation.

11 FOG-1 specifies the sperm fate and that FBF promotes proliferation.

12 Here, we report that FOG-1 also promotes proliferation.

13 its stimulation of the PI-3K/p70 S6K cascade promotes proliferation.

14 expression of cyclin D1, E2F1, and E2F2, and promotes proliferation.

15 nal granulocytic differentiation and instead promotes proliferation.

16 rotects beta cells against glucotoxicity and promotes proliferation.

17 levels directed antiapoptotic signals while promoting proliferation.

18 remodeling by inhibiting VSMC apoptosis and promoting proliferation.

19 n of apoptosis in the absence of EPO without promoting proliferation.

20 s, supporting their functional importance in promoting proliferation.

21 K transformation, with the loss of autophagy promoting proliferation.

22 s/astrocytes, expression of signal molecules promoting proliferation (activated Notch1 and its downst

23 en frequently expressed in carcinomas, which promotes proliferation after regulated intramembrane pro

24 is is beneficial for cancer cells because it promotes proliferation against normal cells.

25 xosomes released by injured epithelial cells promote proliferation, alpha-smooth muscle actin express

26 MUC4 promotes proliferation, anchorage-dependent and-independ

27 most notably the stem cell factor SOX9, that promote proliferation and a metastatic phenotype.

28 on renal tubular epithelial cells (TEC) will promote proliferation and antiapoptosis during regenerat

29 MYC-S was previously shown to promote proliferation and apoptosis of immortalized rode

30 or, Snail, in recipient epithelial cells and promote proliferation and drug resistance.

31 onal mechanisms to rapidly induce genes that promote proliferation and efficiently attenuate their ex

32 How caspases promote proliferation and how cells are protected from t

33 IMP2's ability to promote proliferation and IGF action requires IMP2 phosp

34 Whereas other Th2 effectors promote proliferation and IL-4 production by naive T cel

35 ny types of human cancer, and is believed to promote proliferation and increased cell survival thereb

36 We sought to identify cytokines that can promote proliferation and induce or maintain IL-22 produ

37 Tnrc6a was previously reported to promote proliferation and inhibit differentiation of myo

38 genic effects in part through its ability to promote proliferation and inhibit p53-dependent apoptosi

39 ATMs were sufficient to promote proliferation and interferon-gamma production fr

40 absence of stromal cells, was sufficient to promote proliferation and invasion characteristic of a m

41 roblasts can be induced by agonists known to promote proliferation and invasion in vivo.

42 and that basal JNK activity is necessary to promote proliferation and maintain diploidy in breast ca

43 s the Hippo pathway effector Yorkie (Yki) to promote proliferation and maintenance of FSCs, but Hh al

44 Whereas the GTPase RhoA has been shown to promote proliferation and malignant transformation, the

45 ells secrete BMP4 in response to hypoxia and promote proliferation and migration of vascular smooth m

46 mutations potentiate the ability of Ack1 to promote proliferation and migration, suggesting that poi

47 oncoproteins such as cyclin D1 or HPV E7 to promote proliferation and morphogenesis in the absence o

48 sult also implies that the ability of ras to promote proliferation and oncogenic transformation can b

49 c epithelial and stromal cells, where it can promote proliferation and play a role in tissue regenera

50 larval somatic gonad functionally overlap to promote proliferation and prevent early meiosis.

51 ates Nmyc1 and suppresses Tbx2 expression to promote proliferation and specification of the atrial an

52 cells, IL-2 triggers signaling pathways that promote proliferation and survival by activating the STA

53 tly through cell surface GRP78 (CS-GRP78) to promote proliferation and survival of cancer cells; howe

54 t signaling pathway to dictate cell fate and promote proliferation and survival, the role of Pg in th

55 esyl protein transferase (FTase) activity to promote proliferation and survival.

56 tracellular matrix, stimulate migration, and promote proliferation and survival.

57 ivate beta-catenin-mediated transcription to promote proliferation and tissue expansion.

58 The adenoviral E1A proteins are able to promote proliferation and transformation, inhibit differ

59 s suggested that apoptotic caspases can also promote proliferation and tumor growth under certain con

60 lates EGF signaling at low cell densities to promote proliferation and, therefore, may be beneficial

61 t disrupts adhesion and induces signals that promote proliferation and/or migration.

62 After 1 wk of culture, IL-18 promoted proliferation and accelerated differentiation o

63 xpression of mutant NRAS and EIF1AX proteins promoted proliferation and clonogenic survival in LGSC c

64 crucial role in LRC fate, loss of p27(kip1) promoted proliferation and differentiation of LRCs in vi

65 , overexpression of a relevant ErbB4 isoform promoted proliferation and disturbed polarization of kid

66 lecules in response to S. aureus and greatly promoted proliferation and gamma interferon (IFN-gamma)

67 exposed epithelial cells activated DC, which promoted proliferation and HIV-1 replication of co-cultu

68 FGF signaling promoted proliferation and induced smooth muscle differe

69 ies, which showed that miR-4707-5p and MYBL2 promoted proliferation and metastasis.

70 lyses showed that overexpression of AK017368 promoted proliferation and restrained differentiation of

71 d on a stiff matrix secreted prosaposin that promoted proliferation and survival of mammary carcinoma

72 ate (ATP) is released from damaged cells and promotes proliferation and activation of a variety of im

73 , regulates the expression of HOX genes, and promotes proliferation and aggressiveness of neoplastic

74 In vitro, loss of REDD1 signaling promotes proliferation and anchorage-independent growth

75 hift in pulmonary arterial hypertension that promotes proliferation and apoptosis resistance in the p

76 The c-Myc oncoprotein promotes proliferation and apoptosis, such that mutation

77 ions-p73 mediates chemosensitivity while p63 promotes proliferation and cell survival-and are both ov

78 tic genes in inductive UGS mesenchyme, which promotes proliferation and cytodifferentiation of the pr

79 mbinase-activating gene 2-deficient mice, it promotes proliferation and development to the DP stage f

80 s it favors ECM production and autophagy and promotes proliferation and differentiation by limiting F

81 detection of interleukin-4, a cytokine that promotes proliferation and differentiation of B cells, t

82 eveal an integrin-Wnt7a-Decorin pathway that promotes proliferation and differentiation of neuroepith

83 The cytokine interleukin-15 (IL-15) promotes proliferation and effector capacity of CD8(+) T

84 protein abundance, we suggest that low FOG-1 promotes proliferation and high FOG-1 specifies spermato

85 te that activation of Ras in adult epidermis promotes proliferation and inhibits differentiation and

86 Taken together, lncRNA AK017368 promotes proliferation and inhibits differentiation of m

87 These results suggest that IGF-I promotes proliferation and inhibits osteoblastic differe

88 We further showed that Xrn2 promotes proliferation and inhibits squamous differentia

89 E(2) and vasoactive intestinal polypeptide, promotes proliferation and ion secretion and suppresses

90 EGF promotes proliferation and migration of stem/progenitor

91 epidermis, and a loss of NF-kappaB function promotes proliferation and oncogenesis.

92 requires two classes of mutations, one that promotes proliferation and one that blocks differentiati

93 LMP2A promotes proliferation and protects B cells from MYC-ind

94 In contrast, NOTCH3 promotes proliferation and receptor expression is increa

95 CD40 signaling promotes proliferation and rescues B-cells from apoptosi

96 ctivated in ATII cells after lung injury and promotes proliferation and spreading during repair.

97 ed that hypoxia-inducible factor (HIF)1alpha promotes proliferation and spreading of ATII cells durin

98 P. lncRNA AK017368 promotes proliferation and suppresses differentiation of

99 y controlling a transcriptional program that promotes proliferation and suppresses differentiation, i

100 In T and B cells, CREB activation promotes proliferation and survival and differentially r

101 showed in WEHI-231 cells that M2 expression promotes proliferation and survival and is associated wi

102 titutive tyrosine kinase activity of Bcr-Abl promotes proliferation and survival of chronic myelogeno

103 oblast models demonstrated that the receptor promotes proliferation and survival of extravillous trop

104 elial carcinoma cell lines and tumors, which promotes proliferation and survival via activation of th

105 In these neoplasms, HH signaling promotes proliferation and survival, contributes to the

106 aling elicited by growth factors and thereby promotes proliferation and TKI evasion downstream of HER

107 The adenovirus E1A oncoprotein promotes proliferation and transformation by binding cel

108 emonstrate a mechanism by which loss of Par3 promotes proliferation and tumorigenesis, which supports

109 Functionally, each promotes proliferation and viability, and they cooperati

110 ntrast to the well known activity of Sox2 in promoting proliferation and cell fate determination, our

111 rx1 maintains cells in a stem cell state by promoting proliferation and delaying expression of neura

112 d dendritic cells in addition to its role in promoting proliferation and differentiation of several c

113 h3 interaction constitutes a juxtacrine loop promoting proliferation and dissemination of ovarian can

114 ecovery of the mouse hematopoietic system by promoting proliferation and facilitating mobilization of

115 rcinogenesis in a preclinical mouse model by promoting proliferation and increased beta-catenin accum

116 , we establish a novel function of Angpts in promoting proliferation and invasion and inducing Tie-2

117 and inhibits fibrotic remodeling in part by promoting proliferation and migration of endothelial and

118 tive action of osteoclasts are implicated in promoting proliferation and migration of osteoblasts dur

119 , strikingly increases Th2 cell expansion by promoting proliferation and preventing apoptosis.

120 RC2 maintains small intestinal stem cells by promoting proliferation and preventing differentiation i

121 ole in the response of SCs to limiting BL by promoting proliferation and preventing premature SC diff

122 d activation of TAMs have been implicated in promoting proliferation and survival of cancer cells, as

123 e growth factor stimulation, with the former promoting proliferation and the latter promoting cell cy

124 pproximately 92 may mediate these effects by promoting proliferation and through posttranscriptional

125 e growth of triple-negative breast cancer by promoting proliferation and vascular dissemination of ca

126 pression counteracted p21Cip1 up-regulation, promoted proliferation, and drove retinoblastoma formati

127 1 protein expression increased beta-catenin, promoted proliferation, and inhibited p53-dependent apop

128 AP also expands basal epidermal progenitors, promotes proliferation, and inhibits terminal differenti

129 Therefore, FOG-3 normally promotes proliferation, and two copies of the fog-3 gene

130 pression, thereby increasing AKT activation, promoting proliferation, and decreasing c-JUN N-terminal

131 ely regulated expression of several genes to promote proliferation, apoptosis, and morphogenesis in r

132 Moreover, exposure to 2 Gy IR promotes proliferation arrest and differentiation in the

133 he Rb tumor suppressor and, when active, can promote proliferation as well as apoptosis.

134 Activation of beta2-AR signaling promotes proliferation associated with increased AKT, ex

135 articular against intracellular signals that promote proliferation at the expense of differentiation.

136 Acvr1 signaling promoted proliferation at early stages of lens formation

137 E9.5 cortex, suggesting that PACAP normally promotes proliferation at this stage.

138 itors of the canonical BMP signaling pathway promote proliferation but do not affect lineage choice,

139 ervations underscore that st does not always promote proliferation but may, depending on conditions a

140 suggest that signaling through RANK not only promotes proliferation but also inhibits the terminal di

141 dogenous Kras(G12D)-a common AML lesion that promotes proliferation but not self-renewal.

142 Furthermore, FGF promotes proliferation but PDGF favors differentiation.

143 Oncogenes such as HRAS(V12) promote proliferation by upregulating general transcript

144 nases mediates phosphorylation of STAT3, and promotes proliferation by accelerating G(1) --> S progre

145 to modulates myogenic cell determination and promotes proliferation by antagonizing the TGF-beta liga

146 rted recently that this transcription factor promotes proliferation by directly transactivating c-myc

147 d that overexpression of Id2 in primary AECs promotes proliferation by inhibiting a retinoblastoma pr

148 lts suggest a novel mechanism by which c-Myc promotes proliferation by stabilizing the mitotic spindl

149 Extracellular nucleotides promote proliferation, CXCL12-driven migration, and BM e

150 T cell receptor engagement promotes proliferation, differentiation, survival, or de

151 BMP7 thus promotes proliferation directly in nephron progenitors b

152 We find that SREBP1 and 2 promote proliferation downstream of mTORC1, and the acti

153 s string, Cut, and Hedgehog signaling, which promote proliferation during early oogenesis.

154 factor, early growth response gene 3 (Egr3), promotes proliferation during the transition from double

155 NAs encoding growth regulatory proteins that promote proliferation in a variety of cell types.

156 nsfection of p27kip1 siRNA was sufficient to promote proliferation in confluent cultures of HCECs fro

157 Gastrin and its precursors promote proliferation in different gastrointestinal cell

158 Both can stimulate chemotaxis and promote proliferation in endothelial cells and fibroblas

159 Cyclin D proteins promote proliferation in G1 and typically are down-regul

160 Transforming growth factor (ss1TGFss1) can promote proliferation in late stage cancers but acts as

161 nce the dephosphorylated C/EBPalpha does not promote proliferation in Rb-negative cells.

162 These results show that ROS do not promote proliferation in the Nkx3.1-null prostate, but i

163 in ovarian and endometrial cancer cells and promote proliferation in these cells.

164 ould impair the survival of native cells and promote proliferation in transplanted cells.

165 Stimulation of LD density promoted proliferation in colon cancer cells, whereas si

166 regime reduces mitochondrial metabolism and promotes proliferation in adult mouse cardiomyocytes, re

167 y lesions shortly after HER2 activation, but promotes proliferation in advanced primary tumour cells.

168 ted vascular smooth muscle cell motility but promotes proliferation in association with persistent be

169 PMA alone promotes proliferation in B-1 cells, but not in splenic

170 wo recent papers now show that Wnt signaling promotes proliferation in both stem cell populations, re

171 wers endogenous p27(kip1) protein levels and promotes proliferation in confluent cultures of rat CECs

172 Our findings indicate that WNT10B promotes proliferation in human corneal endothelial cell

173 by which the NG2-beta1-integrin interaction promotes proliferation in one case and motility in the o

174 Here we show that the miRNA miR-22 promotes proliferation in primary human cells, and throu

175 KLF5 promotes proliferation in vitro and in vivo and is induc

176 ects on S phase: driving DNA replication and promoting proliferation in diploid cells, even when deve

177 asion in skin cultures without significantly promoting proliferation in vitro and in vivo.

178 erates retinoblastoma development in mice by promoting proliferation, in part by reducing expression

179 silencing by RNA interference significantly promoted proliferation, indicating an inhibitory effect

180 The capacity to promote proliferation, inhibit differentiation and induc

181 cogenic properties, including the ability to promote proliferation, inhibit senescence, and collabora

182 zed route for PKA to activate a pathway that promotes proliferation, inhibits apoptosis, enhances tra

183 The fusion gene transcripts promoted proliferation, invasion, and motility with vari

184 ctor 5 (KLF5) is a transcription factor that promotes proliferation, is highly expressed in dividing

185 Although pax6 also promotes proliferation, it differentially regulates neur

186 functions in a ligand-independent manner to promote proliferation, migration, and invasion.

187 We also found that FGF-1 and FGF-2 promote proliferation, migration, and survival of cultur

188 We report that RAs promote proliferation, migration, and tube formation of

189 also demonstrated that RGC32 overexpression promoted proliferation, migration and tumorigenic growth

190 Recombinant fibromodulin promoted proliferation, migration, and invasion of HSCs,

191 vesicle-free (S4) human EOC ascites potently promoted proliferation, migration, and invasion of human

192 types, and this aberrant expression strongly promotes proliferation, migration and invasion through m

193 th factor (PDGF)-B to its receptor PDGFRbeta promotes proliferation, migration, and recruitment of pe

194 t role in the bronchiolization of alveoli by promoting proliferation, migration, and attenuation of a

195 se oxidation, while increasing [Ca(2+)]cyto, promoting proliferation, migration, and fission.

196 monstrating that one dose of wild-type fog-1 promotes proliferation more effectively than two doses -

197 , we found CD8+ T cells have the capacity to promote proliferation of BECs in low androgen condition.

198 ltured monocytes had a diminished ability to promote proliferation of both CD4(+) and CD8(+) T cells

199 t of TCRs by self-peptide:MHC complexes does promote proliferation of CD4+ T cells under severe lymph

200 se disparate signaling pathways cooperate to promote proliferation of cerebellar granule neuron precu

201 we propose that TGF-beta1 dysregulation may promote proliferation of ER-alpha-positive cells associa

202 ion of p38alpha does not induce apoptosis or promote proliferation of erythroid progenitors.

203 HIV integration into specific genes may promote proliferation of HIV-infected cells, slowing vir

204 We investigated how inflammation might promote proliferation of LPCs.

205 ogical pathways of programmed cell death may promote proliferation of malignant cells, and correction

206 hibitor, p21(cip1), to inhibit apoptosis and promote proliferation of NCCs, thereby maintaining a mul

207 bundance of serotonin (5-HT), its ability to promote proliferation of neural precursors, and reports

208 ates that DE-cadherin acts in glial cells to promote proliferation of neuroblasts.

209 ture differentiation of corticotropes and to promote proliferation of pituitary progenitors.

210 vitro-generated B10 cells were also found to promote proliferation of regulatory T cells in coculture

211 lished role in extending telomeres, TERT can promote proliferation of resting stem cells through a no

212 Drugs activating noncanonical Shh promote proliferation of satellite cells, which is aboli

213 d to identify exogenous soluble factors that promote proliferation of SSCs.

214 hat steatosis alters the microenvironment to promote proliferation of tumor initiating cells (TICs) a

215 igh levels of proangiogenic cytokines, which promoted proliferation of both endothelial and epithelia

216 NGF promoted proliferation of CD34(+) cells but not HRECs.

217 hereas those that developed into macrophages promoted proliferation of CD8(+) T cells only.

218 IL-22 also activated STAT3 and promoted proliferation of cultured BMOL cells (a mouse L

219 Ras-mediated hyperactivation of LKB1 promoted proliferation of GNMT-deficient hepatoma cells

220 Reducing levels of alpha1-ACT promoted proliferation of HCC cells in vitro.

221 CAP37 promoted proliferation of HCEC in a time- and dose-depen

222 ived cells that developed into migratory DCs promoted proliferation of influenza A virus-specific CD4

223 Exogenous Igf2 protein promoted proliferation of MB precursor cells (GNP) and a

224 Increase of PRP concentration promoted proliferation of MSCs, and 2.5% to 10% of PRP g

225 ased the intraprostatic pHe by 0.2 units and promoted proliferation of noninvasive C3 cells, which re

226 expression of WT1 decreased CDC73 levels and promoted proliferation of OSCC cells.

227 m optogenetically stimulated cortical slices promoted proliferation of pediatric and adult patient-de

228 Furthermore, it strongly promoted proliferation of TF1 cells, above the levels ob

229 In addition, pim-1 promoted proliferation of the BCR/ABL-transformed cells.

230 d media from myc-overexpressing B cell lines promoted proliferation of vascular endothelium in vitro,

231 cer and activator of transcription 5 (STAT5) promotes proliferation of a wide range of cell types, th

232 reases the number of immature beta-cells but promotes proliferation of both mature and immature beta-

233 Estrogen receptor-alpha (ERalpha) promotes proliferation of breast cancer cells, whereas t

234 (2014) show that microbial-derived butyrate promotes proliferation of cancer-initiated intestinal ep

235 Sonic hedgehog promotes proliferation of developing cerebellar granule

236 a driver mutation, NTHi-induced inflammation promotes proliferation of early adenomatous lesions in a

237 nal cofactor in the Hippo signaling pathway, promotes proliferation of embryonic cardiomyocytes by ac

238 Human chorionic gonadotropin (hCG) promotes proliferation of endogenous neural stem cells,

239 Activation of IL-4/IL-13 signaling promotes proliferation of FAPs to support myogenesis whi

240 Drosophila melanogaster ovary, Wingless (Wg) promotes proliferation of follicle stem cells located ~5

241 aken together, our results indicate that FAK promotes proliferation of glioblastoma cells by enhancin

242 ic skin lesions, induces differentiation and promotes proliferation of human keratinocytes.

243 stromal-dependent paracrine VEGF-A signaling promotes proliferation of human primary multiple myeloma

244 liferation, and increased CTGF in beta-cells promotes proliferation of immature (MafA(-)) insulin-pos

245 uggest a model in which high mTORC1 activity promotes proliferation of immature SCs and antagonizes S

246 ion, inflammatory cells produce IL-22, which promotes proliferation of LPCs via STAT3.

247 Taken together, our data show that the AhR promotes proliferation of MB cells, suggesting that this

248 from our laboratory has shown that Nef also promotes proliferation of myeloid cells through a signal

249 The Wnt/beta-catenin pathway promotes proliferation of neural progenitor cells (NPCs)

250 Fibroblast growth factor-2 (FGF-2) promotes proliferation of neuroprogenitor cells in cultu

251 esses differentiation of endocrine cells and promotes proliferation of Nkx6-1(+)Ptf1a(+) multipotent

252 We show that WNT pathway activation in vitro promotes proliferation of NSCs but not GNPs.

253 that demineralization of the dentin surface promotes proliferation of osteoblasts and early differen

254 regulation of cyclin D1 by HIF-1alpha, which promotes proliferation of ovarian cancer cells.

255 Strikingly, Tip30 deletion promotes proliferation of primary MECs and results in ra

256 ehog signaling in the epidermis in which SHH promotes proliferation of progenitors of the hair lineag

257 creased phosphorylation of histone H3, which promotes proliferation of T lymphocytes.

258 TNF-alpha promotes proliferation of thyrocytes in vitro, and anti-

259 trophectoderm with the inner cell mass (ICM) promotes proliferation of undifferentiated diploid troph

260 Importantly, we show that E2F3 promotes proliferation of VSMCs leading to increased IH,

261 Ex vivo, BMP inhibits, and Gremlin 1 promotes, proliferation of cultured BCC cells.

262 l capillary endothelial cells (BRCECs) while promoting proliferation of bovine cornea epithelial cell

263 he pathogenesis of hepatocellular carcinoma, promoting proliferation of cancer cells, the inflammator

264 at Zic1 and Zic4 have Shh-dependent function promoting proliferation of granule cell progenitors.

265 identifies p18 and p21 as novel targets for promoting proliferation of human beta cells and demonstr

266 prominent effect on macrophages, while also promoting proliferation of Igm(+) B cells and memory T c

267 enic system increased islet mass at birth by promoting proliferation of immature beta cells, in the a

268 hase of acute tubular necrosis in animals by promoting proliferation of injured tubular cells and dec

269 ated signaling and suggest a role of KLF5 in promoting proliferation of intestinal epithelia in respo

270 FGFs are commonly implicated in promoting proliferation of neural precursor cells, but i

271 Here we identify a role for NPY in promoting proliferation of postnatal neuronal precursor

272 Runx1 may act as a skin oncogene by directly promoting proliferation of the epithelial cells.

273 supporting instead a crucial role for SHH in promoting proliferation of these RP progenitors and for

274 The E2F1 transcription factor can promote proliferation or apoptosis when activated, and i

275 moterol, a specific partial agonist, did not promote proliferation or migration.

276 These include: promoting proliferation, protecting cells from activatio

277 the immature follicle to inhibit apoptosis, promote proliferation, stimulate production of steroid a

278 ating from the pre-T-cell receptor (pre-TCR) promote proliferation, survival and differentiation of i

279 s at the conclusion of PGC reprograming I to promote proliferation, survival and expression of the go

280 , insulin can synergize with inflammation to promote proliferation, survival, and dissemination of ca

281 ruiting Wingless (Wnt) signaling pathways to promote proliferation, survival, and motility.

282 Erythropoietin promoted proliferation, survival, and wound recovery in

283 of transcription 3 (STAT3) signaling, which promotes proliferation, survival, and metastasis of canc

284 type and induces c-Rel target genes, thereby promoting proliferation, survival, and more invasive bre

285 (AML) involves the cooperation of mutations promoting proliferation/survival and those impairing dif

286 erable evidence supports a role for PRL-1 in promoting proliferation, the biological regulators and e

287 of the graft endothelium to anti-HLA Abs may promote proliferation through the mTOR pathway.

288 y stromal myofibroblasts of the lower crypt, promotes proliferation through canonical beta-catenin ac

289 hese data show for the first time that ACA11 promotes proliferation through inhibition of NRF2 functi

290 t whereas intestinal cells induce genes that promote proliferation thymocytes induce expression of ge

291 n (SPOP) stabilize the TRIM24 protein, which promotes proliferation under low androgen conditions.

292 Thus, EGF promotes proliferation via the MAPK in COLO-357 cells bu

293 ecific miR-290 and miR-302 microRNA families promote proliferation whereas let-7 microRNAs inhibit se

294 iomes: alpha-Proteobacteria-rich communities promote proliferation, whereas Bacteroidetes or pathogen

295 protected cells from induced cell death and promoted proliferation, whereas GM-CSF alone was complet

296 We first show that CAV1 expression promotes proliferation, whereas it suppresses migration

297 Wnt signals act together to synergistically promote proliferation while maintaining the cells in an

298 g apoptosis--decreasing cell number--or that promote proliferation while simultaneously inhibiting ap

299 Both ligands enhance viability, and Tgfbeta1 promotes proliferation while ActB supports maturation.

300 damage through inhibiting cell apoptosis and promoting proliferation with important implications for