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1 o our attention was the sudden onset of left proptosis.
2 who presented with facial pain and right eye proptosis.
3 y (EOM) restriction (78.6% vs 38.8% P<0.01), proptosis (64.3% vs 21.2%, P<0.01), elevated intraocular
4 disease presenting with apparent unilateral proptosis and determine the occurrence of exophthalmos i
13 ment, minimal diplopia, the usual absence of proptosis, and general preservation of visual acuity.
15 atients in Group 1 had mild residual ptosis, proptosis, and movement restriction at 12 weeks, none of
16 pital stay, and sequelae of disease (ptosis, proptosis, and movement restriction) were evaluated and
19 the recurrence of strabismus, diplopia, and proptosis, but did not correct deficits in the best corr
21 ted between the ages of 29 and 76 years with proptosis, eyelid swelling or a mass (10/14 cases), and
22 frequent fractures, craniosynostosis, ocular proptosis, hydrocephalus, and distinctive facial feature
24 evated intraocular pressure occurring during proptosis induced choroidal ischemia and that acoustic r
25 arget organ in relapsing polychondritis, and proptosis is a well-recognized manifestation of eye invo
27 ternal examination revealed 3 cm of relative proptosis of the left eye and a palpable mass in the lef
34 ients with GO between the CA-to-OA ratio and proptosis (P<0.001), lid fissure (P = 0.004), and intrao
36 dation and death, coronal synostosis, ocular proptosis, precocious sternal fusion, and abnormalities
37 teration in physical appearance secondary to proptosis, ptosis, and facial disfigurement, leading to
39 ngle closure glaucoma presented with 4 mm of proptosis, resistance to retropulsion, tortuous corkscre
40 , measured as continuous variables, included proptosis, the Clinical Activity Score, and results on t
41 ions such as hypotony, diplopia, strabismus, proptosis, tube erosion, failure, corneal decompensation
42 phic lesions that resemble scars, thin hair, proptosis, underdeveloped cheekbones, and marked acro-os
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