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1 spectrometry as 2,3-dinor-5, 6-dihydro-8-iso-prostaglandin F2alpha.
2                                              Prostaglandin F2alpha (0.01 micromol/L; n=6), which redu
3  to 10(-6) mol/L), KCl (5 to 50 mmol/L), and prostaglandin F2alpha (10(-9) to 10(-6) mol/L) was compa
4 retory phospholipase A2 (sPLA2 ), and 11beta prostaglandin F2alpha (11betaPGF2alpha ), and, in a subg
5 nary metabolite of 15-F2t-isoprostane (8-iso-prostaglandin-F2alpha), 2,3-dinor-5,6-dihydro-F2t-IsoP,
6 ntake of dietary antioxidants, urinary 8-iso prostaglandin F2alpha (8-iso PGF2alpha) as a marker of o
7                          In this work, 8-iso-prostaglandin F2alpha (8-iso-PGF2alpha) was analysed in
8 ore abundant F2-isoprostanes produced, 8-iso-prostaglandin F2alpha (8-iso-PGF2alpha).
9 ell as products of lipid peroxidation (8-iso-prostaglandin F2alpha (8-isoPF2alpha) and 4-hydroxy-2-no
10                   Urinary excretion of 8-iso-prostaglandin F2alpha, an isomer of the PGG/H synthase (
11  inflammation-initiating mediators including prostaglandin F2alpha and leukotriene B4 and pro-resolvi
12 dative stress was assessed by urinary 8-iso- prostaglandin F2alpha and serum soluble NOX2-derived pep
13 ssess the relationship between urinary 8-iso-prostaglandin F2alpha and serum soluble NOX2-derived pep
14 inflammation-initiating mediators (including prostaglandin F2alpha) and select proresolving pathways
15 NEAC); 2) markers of systemic (urinary 8-iso-prostaglandin-F2alpha) and muscle (carbonylated protein
16  D2 (DP)1, DP2, prostaglandin E2 (EP)1, EP4, prostaglandin F2alpha, and thromboxane A2 receptors but
17 The identification of 2, 3-dinor-5,6-dihydro-prostaglandin F2alpha as the major urinary metabolite of
18   Selective inhibition of PGF2alphaEA versus prostaglandin F2alpha biosynthesis accelerates adipogene
19 h as prostamide/prostaglandin F synthase, to prostaglandin F2alpha ethanolamide (PGF2alphaEA), of whi
20 e eye of each cat was treated topically with prostaglandin F2alpha, fluprostenol (FP receptor agonist
21 ntiated constriction of isolated arteries to prostaglandin F2alpha in proportion to the amount of PVA
22                                  Addition of prostaglandin F2alpha, known to limit PPARgamma activity
23 lmonary vasoconstriction (NPV) stimulated by prostaglandin F2alpha (PGF2alpha ) and by the drug LY835
24 r prostaglandins, thromboxane A2 (TXA2 ) and prostaglandin F2alpha (PGF2alpha ), on skin microcircula
25                                        Thus, prostaglandin F2alpha (PGF2alpha) (FP receptor agonist),
26                          We demonstrate that prostaglandin F2alpha (PGF2alpha) as well as two analogu
27                                 For example, prostaglandin F2alpha (PGF2alpha) causes hypertrophy of
28     The angiotensin II type I (AT1R) and the prostaglandin F2alpha (PGF2alpha) F prostanoid (FP) rece
29  the role of parasite LBs in biosynthesis of prostaglandin F2alpha (PGF2alpha) has not been investiga
30                                              Prostaglandin F2alpha (PGF2alpha) is a product of cycloo
31                                              Prostaglandin F2alpha (PGF2alpha) is an important mediat
32 alpha,20beta-dihydroxyprogesterone (DHP) and prostaglandin F2alpha (PGF2alpha) levels rise in teleost
33 hase (cyclooxygenase or COX) enzyme product, prostaglandin F2alpha (PGF2alpha), has exhibited promise
34                                              Prostaglandin F2alpha (PGF2alpha), which is released fro
35 licited endothelium-dependent relaxations in prostaglandin F2alpha (PGF2alpha)-contracted strips of p
36              To understand the mechanisms of prostaglandin F2alpha (PGF2alpha)-induced protein synthe
37 tion of inflammatory lipid mediators such as prostaglandin F2alpha (PGF2alpha).
38 ignaling pathways by their endogenous ligand prostaglandin F2alpha (PGF2alpha).
39 -iPs, consists of four classes of isomers of prostaglandin F2alpha (PGF2alpha).
40  to the follicular phase using progesterone, prostaglandin F2alpha(PGF2alpha) and pregnant mare serum
41  five primary prostanoids: prostaglandin E2, prostaglandin F2alpha, prostaglandin I2, thromboxane A2,
42 pha as the major urinary metabolite of 8-iso-prostaglandin F2alpha provides the basis for the develop
43 -2'-deoxyguanosine (r = 0.53, P < 0.001) and prostaglandin F2alpha (r = 0.26, P < 0.001), and the gre
44  E2 receptors (EPs) EP1 (266) and EP4 (117), prostaglandin F2alpha receptor (FP) (61), and thromboxan
45 s secreted inflammatory mediators, including prostaglandin F2alpha, the cytokines TNF-alpha and IL-6,
46  the oxidant stress marker isoprostane 8-epi-prostaglandin F2alpha) were identical in incubations wit

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