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1 andin H(2) (PGH(2)) in the committed step of prostaglandin biosynthesis.
2 -2 (PGHS-2), catalyzes the committed step in prostaglandin biosynthesis.
3 ooxygenase (COX)-1 and COX-2, which catalyze prostaglandin biosynthesis.
4 COX) isoforms catalyze the committed step in prostaglandin biosynthesis.
5 inary excretion of lipids was used to assess prostaglandin biosynthesis.
6 indicate linkage between LPA signalling and prostaglandin biosynthesis.
7 gous to the enzymatic mechanism proposed for prostaglandin biosynthesis.
8 nd -2 (PGHSs) catalyze the committed step in prostaglandin biosynthesis.
9 ved to function as an important regulator of prostaglandin biosynthesis.
10 ade linking members of the MAPK pathway with prostaglandin biosynthesis.
11 nsaturated fatty acids in a critical step in prostaglandin biosynthesis.
12 serves as a biochemical link between NO and prostaglandin biosynthesis.
13 fications that alter COX functionalities and prostaglandin biosynthesis.
14 Xs)-1 and -2) catalyze the committed step in prostaglandin biosynthesis.
15 2, are responsible for the committed step in prostaglandin biosynthesis and are the targets of the no
16 (2) may contribute to negative regulation of prostaglandin biosynthesis and inflammation, suggesting
17 in doing both: transcriptionally inhibiting prostaglandin biosynthesis and its reversibility by an e
18 TGS2, which codes for COX-2, a key enzyme in prostaglandin biosynthesis, and AREG, which codes for th
19 wn to acetylate an active site serine, block prostaglandin biosynthesis, and give 15R-hydroxyeicosate
21 ch are responsible for the committed step in prostaglandin biosynthesis, and this has been considered
22 nt in cyclooxygenase-1, a critical enzyme in prostaglandin biosynthesis, are resistant to these rapid
23 a mechanism that has also been proposed for prostaglandin biosynthesis as well as for the formation
26 COX-2), which is the rate-limiting enzyme in prostaglandin biosynthesis from arachidonic acid and is
27 o vertebrate enzymes known to play a role in prostaglandin biosynthesis have been identified in the o
28 rleukin(IL)-1beta treatment markedly induced prostaglandin biosynthesis in diseased compared to healt
33 COX-2), an inducible rate-limiting enzyme in prostaglandin biosynthesis, is implicated in various phy
34 ible cyclooxygenase-2 (COX-2) gene regulates prostaglandin biosynthesis,is up-regulated in colorectal
36 c profiling identified an estradiol-enhanced prostaglandin biosynthesis signature in Tsc2-deficient (
37 ctions are related to its ability to inhibit prostaglandin biosynthesis, some of its beneficial thera
38 of the different cyclooxygenase isoforms in prostaglandin biosynthesis, surface hydrophobicity, and
39 es are responsible for the committed step in prostaglandin biosynthesis, the generation of prostaglan
40 7.4 cells that produce NO. and PGHS inhibits prostaglandin biosynthesis to the same extent as NOS inh
41 ygenase-2 (COX-2), a rate-limiting enzyme in prostaglandin biosynthesis, which was also increased at
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