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1 develop allosteric potentiators of this key prostaglandin receptor.
2 cy by inactivating the gene encoding the EP2 prostaglandin receptor.
3 activate nociceptive neurons independent of prostaglandin receptors.
4 uced by PGF2alpha is mediated by FP or other prostaglandin receptors.
5 and antisense primers for each of the three prostaglandin receptors.
6 onsistent with the proposed phylogeny of the prostaglandin receptors.
7 ased COX-2 in H9c2 cells, which also express prostaglandin receptors.
8 demonstrating the first intersection between prostaglandin receptor activation and NFAT signaling.
10 we added recombinant GDF-9, prostaglandins, prostaglandin receptor agonists, or cyclooxygenase inhib
11 he enhancement is not prevented by AH6809, a prostaglandin receptor antagonist, but is blocked by cap
14 ronotropic mechanisms such as muscarinic and prostaglandin receptor binding, stretch, extracelluar Ca
16 e fashion by a pathway involving EP2 and EP4 prostaglandin receptors, cyclooxygenase-2-dependent reac
17 gs demonstrate that COX-2 and its downstream prostaglandin receptor EP(1) signaling pathway accelerat
18 ceptors have been described, termed E-series prostaglandin receptors (EP(1)-EP(4)), that can be furth
21 subjects, there is a surprising lack of the prostaglandin receptor EP2 on Th17 cells; therefore, we
22 PGE2, most likely by signaling through the E prostaglandin receptor EP2, reduces radiation-induced ap
23 human purified naive T cells, PGE2 acts via prostaglandin receptor EP2- and EP4-mediated signaling a
25 GE2) on the brain and appears to require EP3 prostaglandin receptors (EP3Rs), but the specific neuron
27 , these results demonstrate a novel role for prostaglandin receptor EP4 in the mediation of barrier-e
28 isome proliferator-activated receptor delta, prostaglandin receptor EP4, and c-myc), each containing
29 pha (FP), thromboxane A2 (TP)] suggests that prostaglandin receptors evolved functionally from an anc
30 on reflects involvement of G protein-coupled prostaglandin receptors expressed by mucosal epithelial
31 amily have been evaluated at the eight human prostaglandin receptors for potential use in the treatme
32 mmation and injury, although the responsible prostaglandin receptors have not been fully identified.
33 A activator 6-benzoyl-cAMP, and agonists for prostaglandin receptors IP, EP2, and EP4 in infected but
34 e the role of the extracellular sequences in prostaglandin receptor-ligand interaction, chimeras were
36 bitors of cyclooxygenase-2 or antagonists of prostaglandin receptors may have therapeutic potential f
37 o restore both of the high- and low-affinity prostaglandin receptor numbers to within normal ranges i
39 (COX-2) inhibitors for inflammatory disease, prostaglandin receptors provide alternative targets for
41 uggest that isoPGE2 effects were mediated by prostaglandin receptor subtypes EP2/DP on preosteoclasts
43 support the concept of targeting protective prostaglandin receptors therapeutically after stroke.
44 , acts in an autocrine/paracrine fashion via prostaglandin receptors to activate G alpha s and increa
45 ding evidence that these cells express EP(4) prostaglandin receptors, which are known to activate G(s
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