ライフサイエンスコーパス: protamine sulfate

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1 e coagulation assays, and reversibility with protamine sulfate.

2 onic detergents or the antimicrobial peptide protamine sulfate.

3 ionic strength buffer and buffers containing protamine sulfate.

4 tivate PKC in cells by the same mechanism as protamine sulfate.

5 % to 0.2% Reh cells), and in suspension with protamine sulfate (0.7% to 3.1% for Nalm-6 cells and 0%

6 before study agent (sample 1), 10 mins after protamine sulfate administration after cardiopulmonary b

7 ailed antagonism of the activation of PKC by protamine sulfate and did not involve competition with e

8 In a typical case, coassembly of protamine sulfate and perylene dye via electrostatic att

9 The effects of protamine sulfate and vanadate on Nephrin phosphorylatio

10 sulated with pancreatic islet cells by using protamine sulfate as a clinical-grade alginate cross lin

11 Protamine sulfate can be administered at the conclusion

12 s and other mammalian cells with ferumoxides-protamine sulfate complexes (FE-Pro), cellular toxicity,

13 ated SPIO used as an MRI contrast agent, and protamine sulfate, conventionally used to reverse hepari

14 ion of cellular PKC substrates that resemble protamine sulfate in their interactions with PKC may con

15 and high affinity SAPD binding and inhibited protamine sulfate-induced activity.

16 d chelation of extracellular calcium reduced protamine sulfate-induced damage, suggesting that calciu

17 eficient mice display impaired recovery from protamine sulfate-induced foot process effacement and li

18 On the contrary, protamine sulfate-induced phosphorylation at Tyr-1176/11

19 npo(-/-) mice display impaired recovery from protamine sulfate-induced podocyte foot process (FP) eff

20 l facet cell QIRs with the cationic protein, protamine sulfate, led to epithelial exfoliation and era

21 the cathepsin L inhibitor E64 all inhibited protamine sulfate-mediated barrier changes, which sugges

22 was measured in the presence and absence of protamine sulfate on the cytoplasmic side of the channel

23 n subjected to podocyte injury in vivo using protamine sulfate or nephrotoxic serum (NTS).

24 ted in GECs from puromycin aminonucleoside-, protamine sulfate-, or sialidase-treated rats, which sho

25 In a mouse model of podocyte injury, protamine sulfate perfusion of the Cfl1 mutant mouse ind

26 e podocyte foot process effacement following protamine sulfate perfusion.

27 Resveratrol potently inhibited protamine sulfate phosphorylation (IC(50) = 10 microM) b

28 ralized by heparin-binding proteins, such as protamine sulfate, platelet factor-4, and beta-thrombogl

29 ytoadherence, whereas the positively charged protamine sulfate promoted cytoadherence.

30 Administration of protamine sulfate rapidly damaged the isolated glomeruli

31 ons in this model using high-dose heparin or protamine sulfate support the pathogenic role of surface

32 infusion of the positively charged protein, protamine sulfate, the reverse was observed with mPF4(+/

33 of heparin alone or in the administration of protamine sulfate to reverse heparin anticoagulation dur

34 medium supplemented with growth factors, and protamine sulfate was replaced 4 times over a 48-hour pe

35 AC133(+) cells labeled with ferumoxide-protamine sulfate were mixed with either rat glioma or h

36 ctions with native PKCalpha were enhanced by protamine sulfate, which activates the enzyme without re

37 Cationic protamine sulfate, which forms similar complexes with he

38 lation of the cofactor-independent substrate protamine sulfate, which is a polybasic protein that act

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