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1 dependent stimulation of the MAPK pathway by protease activated receptor 1.
2 own for epidermal growth factor receptor and protease activated receptor-1.
3 elium in vivo and in vitro by acting through protease-activated receptor 1.
4 mplex in response to signals transduced from protease-activated receptor-1.
5 pression of Card10 mRNA and protein, but not protease-activated receptor-1.
6 ities via endothelial protein C receptor and protease-activated receptor-1.
7 endent on endothelial protein C receptor and protease-activated receptor-1.
8 or, endothelial cell protein C receptor, and protease-activated receptor-1.
9 duced by activation of the thrombin receptor protease-activated receptor-1.
10 nts of the transforming pathway initiated by protease-activated receptor-1.
13 ar cell recruitment in vivo was dependent on protease-activated receptor-1 activation of the MEK/ERK/
14 at endothelial cells with thrombin or murine protease-activated receptor 1 agonist peptide resulted i
15 o acetylcholine, 5-hydroxytryptamine, or the protease-activated receptor-1 agonist peptide TFFLR.
16 nery, because these responses are induced by protease-activated receptor-1 agonists and phospholipase
18 normal antiapoptotic activity that requires protease activated receptor-1 and endothelial cell prote
19 ated through G(i)-coupled receptors, such as protease-activated receptor 1 and CXC chemokine receptor
20 d receptor 4 in human podocytes, and between protease-activated receptor 1 and protease-activated rec
21 otease-activated receptor 4, and between rat protease-activated receptor 1 and protease-activated rec
22 larization and neurogenesis were mediated by protease-activated receptor 1 and were independent of th
23 apoptosis of human brain endothelial cells, protease-activated receptor-1 and endothelial protein C
24 n vivo evidence for the physiologic roles of protease-activated receptor-1 and endothelial protein C
25 of thrombin was attenuated by application of protease-activated receptor-1 and protein kinase C antag
30 an platelets express two thrombin receptors, protease-activated receptors 1 and 4 (PAR1 and PAR4) and
32 activates platelets by binding and cleaving protease-activated receptors 1 and 4 (PAR1 and PAR4).
33 acids for alpha-thrombin's interaction with protease-activated receptors 1 and 4 (PAR1 and PAR4, res
34 telets revealed that DXA3 formation requires protease-activated receptors 1 and 4, cytosolic phosphol
35 derived from the third intracellular loop of protease-activated receptors-1 and -2 and melanocortin-4
36 is externalized via calcium mobilization and protease-activated receptors-1 and -4, and 48% is contai
38 population with symptomatic PAD and whether protease-activated receptor 1 antagonism with vorapaxar
42 elets by alternate mechanisms, including the protease-activated receptor-1 antagonist vorapaxar, have
44 itor leupeptin or two structurally different protease-activated receptor-1 antagonists (SCH79797 and
45 llebrand factor aptamers, thrombin receptor (protease-activated receptor-1) antagonists, and thrombox
46 thrombo-resistance, excessive activation of protease-activated receptor-1 by thrombin, and insuffici
47 ver, the endothelial protein C receptor- and protease-activated receptor-1-dependent protective signa
48 ing activities mediated by the activation of protease-activated receptor 1 (F2R, also known as PAR1)
49 oreover, an antibody to the thrombin site on protease-activated receptor-1 failed to block factor XII
51 ing the thrombin-activatable receptor PAR-1 (protease-activated receptor-1), in Runx1/Cbfb-deleted ML
54 ion of tissue factor, von Willebrand factor, protease-activated receptor-1, intercellular adhesion mo
56 g to several Gq-coupled receptors, including protease activated receptor 1, muscarinic acetylcholine
58 or, low-density lipoprotein-related protein, protease-activated receptor-1, or matrix metalloproteina
59 Although recent studies established that protease activated receptor 1 (PAR-1) mediates some of t
60 SB2-flot-2 cells had increased expression of protease activated receptor 1 (PAR-1) mRNA, a transmembr
61 ial protein C receptor-dependent cleavage of protease activated receptor 1 (PAR-1) on endothelial cel
62 actor (TF)-dependent thrombin generation and protease activated receptor-1 (PAR-1) activation contrib
63 s of shear stress on the expression of human protease activated receptor-1 (PAR-1) and tissue plasmin
64 hrombin generation and the thrombin receptor protease activated receptor-1 (PAR-1) contribute to live
68 to generate sickle mice deficient in either protease activated receptor-1 (PAR-1) or protease activa
71 FLD, we tested whether the thrombin receptor protease-activated receptor 1 (PAR-1) and hematopoietic
72 previously reported that thrombin activates protease-activated receptor 1 (PAR-1) and induces a myof
73 ism of Xa in a HeLa cell line that expresses protease-activated receptor 1 (PAR-1) but not PAR-2, -3,
74 epidermal growth factor receptor (EGFR) and protease-activated receptor 1 (PAR-1) in endothelial cel
76 tein C receptor (EPCR)-dependent cleavage of protease-activated receptor 1 (PAR-1) on endothelial cel
77 tein C receptor (EPCR)-dependent cleavage of protease-activated receptor 1 (PAR-1) on vascular endoth
82 eruli, aPC preserves endothelial cells via a protease-activated receptor-1 (PAR-1) and endothelial pr
86 wo targets of thrombin-mediated proteolysis, protease-activated receptor-1 (PAR-1) expressed by strom
87 expression results in overexpression of the protease-activated receptor-1 (PAR-1) in human melanoma
88 anges in endothelial thrombomodulin (TM) and protease-activated receptor-1 (PAR-1) in irradiated inte
91 hrombin activation of the G-protein-coupled, protease-activated receptor-1 (PAR-1) on THBS1 gene expr
93 T were significantly blocked in vitro by the protease-activated receptor-1 (PAR-1) silencing RNA; by
94 ttenuates signaling by the thrombin receptor protease-activated receptor-1 (PAR-1) through direct act
96 ar protease signalling system, including the protease-activated receptor-1 (PAR-1) whose tethered lig
97 eceptor for thrombin on endothelial cells is protease-activated receptor-1 (PAR-1), a member of the G
98 ological response to thrombin is mediated by protease-activated receptor-1 (PAR-1), a seven-transmemb
99 ed endothelial protein C receptor (EPCR) and protease-activated receptor-1 (PAR-1), as did its in viv
102 phorylation on serine residues and prevented protease-activated receptor-1 (PAR-1)-induced Ca(2+) ent
109 ed the involvement of the thrombin receptor [protease-activated receptor-1 (PAR-1)] in astrogliosis,
113 EGF receptor (EGFR) and a thrombin receptor (protease-activated receptor-1, PAR-1) increases the expr
114 nce demonstrates that the thrombin receptor (protease-activated receptor-1, PAR-1) plays a major role
115 , neurogenesis and brain repair were lost in protease activated receptor 1 (PAR1) deficient mice.
116 and requires the APC active site, EPCR, and protease activated receptor 1 (PAR1) on endothelial cell
117 eptor (EPCR) as a coreceptor for cleavage of protease activated receptor 1 (PAR1) on endothelial cell
119 et age (IPF, FSC) and activation through the protease activated receptor 1 (PAR1) thrombin receptor (
120 us-encoded glycoprotein C (gC) can stimulate protease activated receptor 1 (PAR1)-enhanced infection
127 R on endothelial cells, activates endogenous protease activated receptor-1 (PAR1) and induces PAR1-me
128 al protein C receptor (EPCR) and cleavage of protease activated receptor-1 (PAR1) and that may play a
129 blood coagulation protease thrombin through protease activated receptor-1 (PAR1) can disrupt endothe
130 pose that APC-mediated signaling through the protease activated receptor-1 (PAR1) can favorably regul
131 signaling pathway for the thrombin receptor protease activated receptor-1 (PAR1) in human platelets.
132 enase and activator of the G protein-coupled protease activated receptor-1 (PAR1), is an emerging new
136 l cell protein C receptor (EPCR, PROCR), and protease activated receptor-1 (PAR1, F2R) on the growth
138 participation of its principal receptor, the protease-activated receptor 1 (PAR1) appears to be limit
142 -1 has been shown to cleave and activate the protease-activated receptor 1 (PAR1) in noncardiac cells
151 onstrate a novel signaling mechanism whereby protease-activated receptor 1 (PAR1) mediates expression
154 des based on the third intracellular loop of protease-activated receptor 1 (PAR1) or PAR4 (refs.
156 using variants of human APC with or without protease-activated receptor 1 (PAR1) signaling function
157 of thrombin, and is associated with altered protease-activated receptor 1 (PAR1) signaling, as PAR1
159 lso acts as a signaling molecule by cleaving protease-activated receptor 1 (PAR1) to cause breast can
160 t protease that initiates cell signaling via protease-activated receptor 1 (PAR1) to regulate vascula
167 We show that thrombin, and other agonists of protease-activated receptor 1 (PAR1), activate TGF-beta
169 The G protein-coupled thrombin receptor, protease-activated receptor 1 (PAR1), mediates many of t
170 Thrombin activates platelets mainly through protease-activated receptor 1 (PAR1), PAR4, and glycopro
171 by cleaving its G-protein-coupled receptors, protease-activated receptor 1 (PAR1), PAR4, or both.
172 of mice deficient in the thrombin receptor, protease-activated receptor 1 (PAR1), provided definitiv
173 CFs express 190 GPCRs and that activation of protease-activated receptor 1 (PAR1), the most highly ex
174 othelial cytoprotective actions that require protease-activated receptor 1 (PAR1), whereas thrombin a
175 coagulation cascade and a potent trigger of protease-activated receptor 1 (PAR1)-mediated platelet a
177 signaling function of aPC, mediated through protease-activated receptor-1 (PAR1) and endothelial pro
179 synthesis in neuronal cells by APC required protease-activated receptor-1 (PAR1) and PAR3, which inh
183 rect oncogenic activity by signaling through protease-activated receptor-1 (PAR1) in carcinoma cells;
184 lood, Aisiku et al describe a novel class of protease-activated receptor-1 (PAR1) inhibitors that blo
191 activation of the G protein-coupled receptor protease-activated receptor-1 (PAR1) is an important sti
194 and thermodynamic parameters for individual protease-activated receptor-1 (PAR1) molecules in the ab
197 llular (i3) loop agonist, pepducin, based on protease-activated receptor-1 (PAR1) was solved by NMR a
199 protease, can trigger cellular responses via protease-activated receptor-1 (PAR1), a G protein-couple
208 tein ALIX regulates lysosomal degradation of protease-activated receptor-1 (PAR1), a GPCR for thrombi
209 entified the prototypical thrombin receptor, protease-activated receptor-1 (PAR1), as part of a novel
210 ular protease signaling system including the protease-activated receptor-1 (PAR1), for which thrombin
211 rotein-coupled receptor (GPCR) for thrombin, protease-activated receptor-1 (PAR1), is activated when
212 d facilitated APC cleavage and activation of protease-activated receptor-1 (PAR1), leading to enhance
213 irreversible proteolytic mechanism by which protease-activated receptor-1 (PAR1), the G protein-coup
214 assess SNX1 function in endocytic sorting of protease-activated receptor-1 (PAR1), we used siRNA to d
218 rotein C-endothelial cell protein C receptor-protease activated receptor 1 pathway on endothelial cel
219 viously demonstrated that the stimulation of protease activated receptor 1 promotes alphavbeta6 integ
220 fferences at the transcript level, including protease activated receptor-1, protease activated recept
227 us-forming activity of one of its receptors, protease-activated receptor-1, to dissect how this recep
228 thermore, MMP-1 induced vasoconstriction via protease-activated receptor-1, whose expression was sign
229 tory of myocardial infarction, inhibition of protease-activated receptor 1 with vorapaxar reduces the
230 by proteolytic cleavage on activation of the protease activated receptor-1, with antiangiogenic prope
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