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1                     Bacterial communities in protected bronchial brushings from 42 atopic asthmatic s
2 ssociated proapoptotic transcription factor, protected fibroblasts from 5-aza only when the cytotoxic
3 on of silencing in multiple organs and (iii) protects siRNAs from 5-to-3 exonucleases.
4 oughness, or in adjacent micro-porosity, can protect the surface from a strong wettability alteration
5                            The cleavage of a protecting group from a protein or drug under bioorthogo
6 irus (VACV) infection were less effective at protecting mice from a lethal pulmonary challenge with V
7 ins (OMPs) have shown encouraging results in protecting mice from A. baumannii infection, but monoclo
8 ane, establishes an impermeable barrier that protects the cell from a number of stressors in the envi
9                                          Vif protects HIV-1 from A3G-mediated restriction by forming
10              This CXXC domain allows TET1 to protect CGIs from aberrant methylation, but it also limi
11 assical and lectin pathways of complement to protect human tissue from aberrant activation.
12 rtantly, silencing endogenous Trib3 strongly protected neurons from Abeta insult.
13 reatment with an inhibitor of MCJ expression protects liver from acetaminophen-induced liver injury a
14  of HIV, uncovering a potential strategy for protecting women from acquisition of the virus.
15                     Telomeres employ TRF2 to protect chromosome ends from activating the DNA damage s
16 rate that the biosafety level 4 (BSL-4) suit protects workers from aerosols in a BSL-4 environment us
17      A dozen of these bacterial mutants also protect the host from age-related progression of tumor g
18      The complexation of aptamers with AuNPs protects the nanoparticles from aggregating in a high-sa
19 ow flavonoid compounds have the potential to protect the brain from aging and neurodegenerative disea
20 whereas inhibition of GSK-3beta and caspases protected mice from AILI.
21                         MEDI8852 was able to protect naive ferrets from airborne transmission of H1N1
22 ubtilis has rendered the engineered bacteria protected against phages from all three major families o
23 ntranasal administration of exogenous Sema3E protects mice from allergic asthma by reducing eosinophi
24 though antigen application before conception protected WT progeny from allergy, it aggravated allergi
25 ) that modulate transcriptional networks and protect cells from alpha-synuclein (alphaSyn) toxicity.
26                   Here we report that NCEH-1 protects dopaminergic neurons from alpha-synuclein-depen
27 es the cationic exopolysaccharide Pel, which protects bacteria from aminoglycoside antibiotics and co
28  Tfh cells decreased IgE antibody levels and protected mice from anaphylaxis, without affecting Th2 c
29 sis (Mtb) to replicate intracellularly while protecting it from antibiotic treatment.
30  we hypothesized that IL-6 may also directly protect beta cells from apoptosis.
31                               Using drugs to protect HG MKs from apoptosis and CD42b shedding, we als
32 tial for these inhibitory TRAIL receptors to protect hHSC from apoptosis opens new avenues for progno
33 ts both its kinase end RNase activities, and protects cells from apoptosis both in vitro and in vivo.
34              Here Liu et al. show that Sirt6 protects podocytes from apoptosis and inflammation by in
35 cellular antiadrenergic therapeutic strategy protecting the heart from arrhythmia and contractile dys
36 nse, it provoked a genomic response that may protect skeletal muscle from atrophy.
37 ude that O-AS function as direct defenses to protect plants from attack by both native pathogenic fun
38 od-retina barrier and the ocular environment protects ocular tissues from autoimmune attack.
39 vade central nervous system (CNS) tissue and protected the mice from autoimmunity.
40 microbial layer around the colony that helps protect the brood from bacterial pathogen infection, res
41 acrophage-specific in vivo knockdown of Irf5 protected ZDF rats from beta-cell loss and hyperglycemia
42 sis, to enhance the enzymes stability and to protect the transducer from biofouling.
43 Besides enzyme entrapment, the obtained gels protected the transducer from biofouling, enabling the r
44 d and licensed dengue vaccine that failed to protect seronegative individuals from breakthrough or en
45 nvironmental conditions as an ion sensor and protects Caulobacter from calcium deficiency stress, a u
46 eradicated intestinal inflammation and fully protected KO mice from carcinogenesis.
47                                              Protecting neurons from causative factors of secondary i
48        Here, we show that both modifications protect P. aeruginosa from certain pilus-specific phages
49 a polymer known as peptidoglycan (PG), which protects the cell from changes in osmotic pressure and s
50 ouble-strand break (DSB) repair pathway that protects the genome from chromosomal instability.
51 s essential vitamins and phytonutrients that protect us from chronic diseases.
52 bit IRI in adoptive transfer experiments and protected mice from cisplatin- and doxorubicin-induced n
53  MjAgo degradation, and recombinant histones protect DNA from cleavage in vitro.
54 r show that the clinical isolates frequently protect each other from clinically relevant antibiotics.
55                     We demonstrated that TSV protects rice chloroplasts from cold stress by interacti
56  cells, but not intestinal epithelial cells, protects mice from colitis-induced intestinal cancer and
57 TOR or MAPK signaling cascades, which act to protect the parasite from complement-mediated and osmoti
58 zed that KSHV miRNAs target human GADD45B to protect cells from consequences of DNA damage, which can
59  that addition of a specific bpy-diol ligand protects unprotected peptides from Cu(II) -mediated oxid
60 at lacked Fabp4/Fabp5 were less effective at protecting mice from cutaneous viral infection, and lung
61 ressor PTEN into the nucleus and in so doing protects it from cytoplasmic proteins that cause PTEN de
62 hereas the X-ray beam is often attenuated to protect the detector from damage caused by intense Bragg
63      We conclude that horizontal acquisition protects offspring from damage caused by high light and
64 sults reveal a novel mechanism as to how WRN protects telomere integrity from damage and telomere ero
65 er cellular activities, and antioxidants can protect oocytes from damages caused by aging.
66                   Nociceptor sensory neurons protect organisms from danger by eliciting pain and driv
67 es are specialized chromatin structures that protect chromosome ends from dangerous processing events
68 izing properties, and possess the ability to protect nerve cells from death at the calcium overload c
69 how that IDGF2 is independent of insulin and protects cells from death caused by serum deprivation, t
70 oxp1 Elevating expression of isoform-A or -D protects cortical neurons from death caused by the expre
71  that in mice the transcription factor STAT3 protects these cells from death and contributes to maint
72             The polymer coat was designed to protect the particles from decomposition while in circul
73 these kinases individually, is sufficient to protect neurons potently from degeneration.
74 e-stranded DNA (ssDNA) with high affinity to protect it from degradation and prevent secondary struct
75 omic plants expressing proinsulin and GAD to protect the autoantigens from degradation in an oral vac
76 ressed arms of reversed replication forks to protect them from degradation.
77  RAD51 nucleofilaments on regressed arms, to protect them from degradation.
78 ttenuated FoxO-mediated murf1 expression and protected sarcomeres from degradation in ncx1-deficient
79 ies, but also creates a steric hindrance for protecting the glycoproteins from degradation by proteas
80 tablish that the PhoP-activated protein MgtC protects PhoP from degradation by outcompeting ClpS for
81 gic or genetic inhibition of Cdc25A activity protects neurons from delayed death in vitro and in vivo
82 ding to their depletion, while an H2 agonist protects MB-HSCs from depletion after sepsis.
83                                        CenpH protects cyclin B1 from destruction by competing with th
84 e role during nutrient starvation, autophagy protects cells from detachment-induced cell death, terme
85 d virus, we were able to vaccinate swine and protect them from developing ASF.
86 matic adolescent hAPP mice was sufficient to protect these mice from developing cognitive impairment.
87 ta7) just before and following SIV infection protected rhesus macaques from developing AIDS and parti
88  twice-weekly injections until age 25 weeks, protects the animals from diabetes.
89  the CB1 gene (Ati-CB1-KO) was sufficient to protect adult mice from diet-induced obesity and associa
90                               Hepatic DsbA-L protects mice from diet-induced hepatosteatosis and insu
91 bility in gastric conditions and effectively protected encapsulated proteins from digestion.
92 son for nondisclosure in both populations is protecting the child from distressing information.
93 monstrate that KSHV miRNAs are important for protecting infected cells from DNA damage responses.
94 X)-dependent, and apocynin, a NOX inhibitor, protected cells from double-strand breaks induced by HDM
95 ular ROS, and interference with either event protects bacteria from double-stranded DNA breakage and
96                    We conclude that Honokiol protects the heart from Dox-cardiotoxicity via improving
97 ion in mice afflicted with p53-mutant tumors protected them from doxorubicin-induced neutropenia and
98 ound BGP-15 improved mitochondrial function, protecting neurons from dying in vitro and in vivo, and
99 alling through the SMAD1 and SMAD3 pathways, protecting the endothelium from endothelial-to-mesenchym
100                            MIR122 appears to protect the liver from ethanol-induced damage by reducin
101  with HA35 normalized these changes and also protected mice from ethanol-induced liver and intestinal
102 xpression and in cholesterol uptake, further protecting macrophages from excessive lipid accumulation
103 complete dendritic stripping following rtACS protects neurons from excitotoxic cell death by silencin
104 l responsiveness, whereas blocking CP-AMPARs protects terminals from excitotoxic swelling.
105 tb) administration, and has shown ability to protect animals from exposure to a pesticide, paraoxon a
106 hly crosslinked ECM impeded drug permeation, protecting tumor cells from exposure to small-molecule d
107 ecombination factors BRCA1, BRCA2, and RAD51 protect these structures from extended nucleolytic degra
108  chromatin compaction by nucleosome stacking protects nucleosomal DNA from external forces up to 4 pi
109 ical memory following cell culture expansion protects MSCs from fibrogenesis in the host wound enviro
110         PAD4 deficiency or DNase 1 similarly protected hearts from fibrosis.
111 eutic targeting of PLD4 using specific siRNA protected mice from folic acid-induced kidney fibrosis a
112                                         They protect children from food insecurity by trimming down t
113 alize and function exclusively at telomeres, protecting them from fusion events.
114 hich suggests the PS1/gamma-secretase system protects neurons from GD-induced death.
115                            Elevated plant Se protects plants from generalist herbivores and pathogens
116                  Presenilin1/gamma-secretase protects neurons from glucose deprivation-induced death
117                        Its original goals of protecting the public from hazardous chemicals were hind
118 e transporter expressed highly in the liver, protects mice from high-fat diet-induced and aging-induc
119  evidence has revealed that SLC13A5 deletion protects mice from high-fat diet-induced hepatic steatos
120 patic gluconeogenesis in the mouse liver but protects mice from high-fat diet-induced hyperglycemia.
121 rom an evolutionarily ancient mechanism that protects photosynthetic bacteria from high light stress,
122 olysaccharides (CPSs) play multiple roles in protecting bacteria from host and environmental factors,
123  HVEM-specific HSV-1 neutralizing antibodies protect mice from HSV-1 eye disease, indicating the crit
124 combined with vascular sequestration of DSAs protects islet grafts from humoral rejection.
125 ditional analyses revealed that FGF2 priming protected cultured DPCs from hydrogen-peroxide-induced c
126             Our findings indicate that PAI-1 protects mice from hypertension-induced cardiac fibrosis
127 ese cells toward proinflammatory phenotypes, protecting the kidney from hypertensive injury and fibro
128     These studies suggest that while the ISR protects OPCs from hypoxia in vitro, it does not appear
129 teract with the environment, particularly to protect pathogens from immune surveillance and host defe
130 gly, we discovered that TrxR1/NADPH directly protects PTP1B from inactivation when present during the
131 etylation, and the presence of purified CobB protects TopA from inactivation by such non-enzymatic ac
132 tolerance-inducing TLR4 ligands are known to protect animals from infection.
133 asite traversal and development, and did not protect mice from infection with PfCSP transgenic Plasmo
134 generation of the skin's barrier in order to protect the body from infection and dehydration and to h
135 in astrocytes regulates BBB permeability and protects the cerebellum from infection and immunopatholo
136 EETs at sites of airway epithelial damage to protect the host from infections in patients with chroni
137 tributions to public health needs and better protect the public from infectious disease often motivat
138  Lactococcus, Lactobacillus, and Coprococcus protect the liver from inflammation.
139                The blood-brain barrier (BBB) protects the brain from inflammation but the mechanisms
140                                          FP7 protected mice from influenza virus-induced lethality an
141 anizes large-scale chromosome structures and protects the genome from instability.
142 tibody from a single injection of VRC01 mRNA protects humanized mice from intravenous HIV-1 challenge
143         The piRNAs play an important role in protecting the genome from invasive transposons in the g
144  kidneys, and adoptive transfer of ILC2 also protected mice from IRI.
145 ogenetic C1 neuron (C1) stimulation (10 min) protected mice from ischemia-reperfusion injury (IRI).
146 ., Mulholland, M., Zhang, W. mTOR activation protects liver from ischemia/reperfusion-induced injury
147  lead for the development of therapeutics to protect the brain from ischemic injury.
148 se (GOT) to maintain cellular energetics and protect the brain from ischemic stroke injury.
149 lacement of p120 from the cadherin, and p120 protects VE-cadherin from K5.
150                                     GJA1-20k protects actin filament from latrunculin A disruption, p
151 e the efficacy of the HMP Airdrive system to protect liver grafts from lethal ischemic damage before
152 m previously infected mice was sufficient to protect uninfected mice from lethal pathogen challenge.
153 s responsive to favipiravir treatment, which protected all animals from lethal disease and reduced se
154                               Finally, D-RR4 protected Caenorhabditis elegans from lethal infections
155 elicited neutralizing antibody responses and protected mice from lethal VEEV and EEEV challenges at 1
156                                      It also protected naive mice from lethal influenza infection.
157  inflammatory monocyte macrophages partially protected these mice from lethal SARS.
158             Here, we demonstrate that IL-17A protects mice from lethal WNV infection by promoting CD8
159 FA are dynamically regulated and function to protect EC from lipotoxic stress and provide FA for meta
160                       Moreover, LD formation protects EC from lipotoxic stress, regulates EC glycolys
161 es transient amplitude to control levels and protects them from loss after OSI.
162 und, surprisingly, that neutrophils serve to protect the host from LPS-induced lethal inflammation.
163 ctively, our results demonstrate that YW3-56 protects animals from LPSS, and CitH3 is a reliable biom
164 RNA nucleotides in the drug binding site can protect cells from macrolide-induced killing, even with
165 t, as well as wood preservatives are used to protect facade materials from microbial spoilage.
166 ordable and sustainable treatment options to protect the public from micropollutants.
167 nitor of lipoprotein trafficking tasked with protecting the cell from mislocalized lipoproteins.
168 ppressed viral loads by more than 5 logs and protected animals from mortality.
169 ibodies, including the broadly binding ones, protected mice from mortality despite lacking neutraliza
170 ocytosis by macrophages and neutrophils, and protected mice from MRSA infection in two model systems.
171 21(Waf1/Cip1) blocks the ability of Foxp1 to protect neurons from mut-Htt-induced neurotoxicity.
172 ogenomics platform to identify strategies to protect neuronal cells from mutant huntingtin induced de
173 bservation indicates that MMR preferentially protects genes from mutation and has important consequen
174 the AMPK-TORC1 metabolic checkpoint, thereby protecting tumour cells from MYC-driven cell death, and
175 er wild-type or a nonfunctional SpeA mutant, protects mice from nasopharyngeal infection; however, on
176 n protection mutualisms, defensive symbionts protect their hosts from natural enemies, including para
177 all molecule TNFR inhibitor R-7050 partially protected hyperoxaluric mice from nephrocalcinosis and C
178 esence of G70S protein was not sufficient to protect mice from neurodegeneration in G70S/- mice, show
179               These data suggest that FX may protect HAdV-5 from neutralization but has minimal contr
180 hat forms an efficient permeation barrier to protect the cell from noxious compounds (1)(,)(2) .
181 st, yKu70/80 rapidly localizes to the break, protecting DNA ends from nuclease accessibility, and rec
182 s support a novel nuclear role for SmgGDS in protecting malignant cells from nucleolar stress, thus p
183 R in the renal proximal tubule cells did not protect the mice from obesity, but markedly attenuated t
184                                Loss of Cadm1 protected mice from obesity, and tract-tracing analysis
185 expresses capsular polysaccharides (CPSs) to protect itself from opsonophagocytic killing.
186                                           To protect the body from osmotic stress, posterior pituitar
187                 Osmosensing transporter ProP protects bacteria from osmotically induced dehydration b
188 eract inhibition by unfair competition: MLCP protects pCPI-17 from other phosphatases, while pCPI-17
189                   The presence of brain also protects embryos from otherwise-teratogenic agents.
190 f Myc in osteoclasts increases bone mass and protects mice from ovariectomy-induced (OVX-induced) ost
191  how kissing-bugs Rhodnius prolixus actively protect themselves from overheating.
192                                           To protect bone marrow from overirradiation, the maximum pe
193 and its major oxidized derivative, carnosol, protect lipids from oxidation.
194                          Silverskin extracts protected erythrocytes from oxidative AAPH- and H2O2-ind
195 yrase ARC4/5 domains seems to be crucial for protecting tankyrase from oxidative inactivation.
196 stimulate endothelial cell proliferation but protects astrocytes from oxidative stress.
197  on Cu-Zn superoxide dismutase (SOD1), which protects cells from oxidative stress.
198 e TUNEL assay, we show that 3MST-derived H2S protects chromosomal DNA from oxidative damage.
199 plained by productive substrate binding that protects LPMOs from oxidative self-inactivation.
200                       Carbonic anhydrase III protects osteocytes from oxidative stress.
201 yroid hormone both in vitro and in vivo, and protects osteocytes from oxidative stress.
202 nregulates p53 transcription and selectively protects noncancerous cells from p53-dependent apoptosis
203 lead to the development of new strategies to protect cereal crops from pathogen infection.
204 d over many millennia to be best equipped to protect the host from pathogen infection.
205 atopoiesis, which forms the immune system to protect the host from pathogen invasion.
206 ion of Clostridiales, but not Bacteroidales, protected neonatal mice from pathogen infection and abro
207 release of antimicrobials into the tears and protected the eye from pathogenic Candida albicans or Ps
208 nt controlling water and solute movement and protecting seed from pathogenic infection.
209 ides secreted by the innate immune system to protect the host from pathogens.
210  can act as a promising biofilter capable of protecting natural aquafers from pathogens.
211 skin microbiome acts as an important barrier protecting our body from pathogens and other environment
212 s Akt activation and osteoclastogenesis, and protects mice from pathological bone loss in disease mod
213 tive, interleukin-10-positive T cells, which protect animals from pathology, increased after malaria
214 roglobulin (beta2M) by cancer cells directly protected them from phagocytosis.
215  stabilizes the 5 end of the guide strand by protecting it from phosphatases and 5-to-3 exonucleases.
216    5-Vinylphosphonate modification of siRNAs protects them from phosphatases, and improves silencing
217            LpxL2-dependent lipid A acylation protects Klebsiella from polymyxins, mediates resistance
218  of these transcripts at prophase appears to protect cyclins from precocious degradation.
219                                           To protect the fan from predation, many species have evolve
220 gdom, and they can act as key adaptations to protect animals from predators [1].
221 diet-induced obesity during pregnancy, could protect obese offspring from progression of NAFLD.
222 s the deubiquitinase activity of ataxin 3 to protect beclin 1 from proteasome-mediated degradation an
223 t tunnel to host the elongating new peptide, protect it from proteolytic digestion, and guide its eme
224 ting that the IPR is a distinct pathway that protects animals from proteotoxic stress.
225 more efficacious than Esc(1-21) and LL-37 in protecting host from pulmonary bacterial infection after
226  and ATP synthesis at a level high enough to protect M. tuberculosis from Q203-induced bacterial deat
227 nhibition of glutathione biosynthesis, which protects cells from reactive metabolites, increased the
228                  In extreme conditions, pb10 protects the phage from releasing its genome.
229 dern stormwater management practices are not protecting surface waters from road salt contamination a
230          Alternatively, such compounds could protect nectar from robbers [2], provided that they do n
231 o produce systemic and local immunity and to protect animals from RSV challenge.
232  The RSV vaccine was immunogenic but did not protect older adults from RSV illness.
233 ression of atherosclerosis and aneurysms and protecting them from rupture.
234       Functionally, we established that Trx1 protects GC1 from S-nitrosocysteine-induced desensitizat
235 otection.In meiosis I centromeric cohesin is protected by Sgo2 from Separase-mediated cleavage ensuri
236                       Centromeric cohesin is protected by Sgo2 from Separase-mediated cleavage, in or
237 novel glycosylation-dependent mechanism that protects tumor cells from serum growth factor withdrawal
238  malaria-endemic areas of Africa, where they protect children from severe and uncomplicated Plasmodiu
239 g bNAbs CR6261 and CR9114 have been shown to protect mice from severe disease following challenge wit
240  can antagonize TLR4 activation in vitro and protect mice from severe influenza infection, most likel
241 especially those with p53-mutant tumors, may protect them from severe chemotoxicity while allowing tr
242 laxis with the ABL kinase inhibitor imatinib protected the mice from severe IgE-mediated anaphylaxis.
243 py approach for IFNgammaR1 deficiency, which protects mice from severe mycobacterial infections, ther
244 ptosis in a mammalian expression system, and protected cortical neurons from slow excitotoxic injury
245 with RTX before radiation exposure partially protected podocytes from SMPDL3b loss, cytoskeletal remo
246 ne marrow mesenchymal stromal cells (BM-MSC) protect AML blasts from spontaneous and chemotherapy-ind
247 aluate the ability of SPC to encapsulate and protect bacteria from stress conditions.
248 chanical therapy have the clear potential to protect hindlimb function from such adverse influence.
249 the aforementioned conformational switch can protect nitrogenase from such damage, confirming that th
250 SAM growth in concert with floral transition protects it from such terminating effects.
251 at outside-xylem hydraulic vulnerability can protect the xylem from tensions that would induce emboli
252                                     S-layers protect cells from the outside, provide mechanical stabi
253 CNS we reasoned that brain aromatization may protect circuits from the threats of peripheral infectio
254 observations suggest that FGF2 priming might protect DPCs from the post-trauma microenvironment in wh
255 ctors present in the CNS may physiologically protect neurons from the deleterious impact of AbetaOs.
256 lts suggest that using money to buy time can protect people from the detrimental effects of time pres
257 the two-dimensional (2D) high-mobility plane protect the semiconductor from the effect of dynamic dis
258 ha and modified its ubiquitination status to protect Topo IIalpha from the proteasomal degradation in
259 facilitate nuclear import and simultaneously protect unassembled RpL4 from the cellular degradation m
260 le methods to test water supplies onsite can protect vulnerable communities from the impact of contam
261 ulating PGRP-SB2 selectively in the fat body protected animals from the deleterious effects of overnu
262 onally in proximal tubular epithelial cells, protected mice from the development of fibrosis.
263           alpha4beta2-nAChR stimulation also protected neuronal responding from the detrimental effec
264 ains of V. cholerae, which are thought to be protected only from the toxicity of their own effectors.
265 s thought to be a glutathione precursor, NAC protected primary astrocytes from the toxicity of the pr
266 es in vitro and in vivo KCNQ2/3 coexpression protected SMIT1 activity from the otherwise inhibitory e
267                                 Cfh deletion protected the mice from the pathogenic subretinal accumu
268 etry genetic pathway as a key determinant in protecting A. baumannii from the bactericidal activity o
269 oting gametocyte-to-ookinete development and protecting ookinetes from the mosquito complement-like r
270 different function in Plasmodium falciparum, protecting ookinetes from the mosquito immune response.
271 ial cells form a barrier that is critical in protecting the host from the hostile luminal environment
272 njugation, transport, and excretion, thereby protecting the liver from the harmful effects of bile ac
273 ogether up-regulate ACKR2 in remote tissues, protecting them from the spread of inflammation.
274 tial component of the shelterin complex that protects chromosome ends from the DNA damage response ma
275 ys demonstrated that the optimized inhibitor protects macrophages from the toxicity of lethal factor.
276                                      Lamin A protects nuclei from the impact of actomyosin activity.
277 am to the eC surface without breaking vacuum protects the surface from the environment after fabricat
278 ch, whereas stimulation of alpha4beta2-nAChR protected delay cells from these deleterious effects.
279 strating widespread potential application in protecting crop plants from these pathogens.
280 ensitive to DISE, suggesting cellular miRNAs protect cells from this form of cell death.
281 contribution of biallelic RELA expression in protecting stromal cells from TNF-mediated cell death, t
282 merous cancers, sialylates TNFR1 and thereby protects tumor cells from TNF-induced apoptosis.
283                        Moreover, Flunarizine protected ECs from TNFalpha-induced increase in Angpt-2
284 thesis that myeloperoxidase inhibition could protect hematopoietic cells from TOP2 poison-mediated ge
285 rated that keratinocyte-derived GC synthesis protected skin from topical phorbol 12-myristate 13-acet
286 hort duration, these efficient nanofactories protect human cells from toxic reactive oxygen species f
287 inhibitor of NF-kappaB-inducing kinase (NIK) protects liver from toxin-induced inflammation, oxidativ
288 utaneous immunization with neoglycoconjugate protected mice from transnasal challenge with the highly
289 effectively voltage-clamped during Ca waves, protecting the heart from triggered arrhythmias.
290 ether, we demonstrate that Dnmt3a and Dnmt3b protect the epidermis from tumorigenesis and that squamo
291 hil elastase or by degrading NETs with DNase protects mice from type-2 immunopathology.
292 idues on RNF157 disrupts binding to CDH1 and protects RNF157 from ubiquitination and degradation.
293 onfers resistance to high oxygen tension and protects cells from undergoing ferroptosis in response t
294 In conclusion, melatonin and its metabolites protect melanocytes from UVB-induced DNA damage and oxid
295 y therefore investigated whether fat feeding protected mice from ventilator-induced lung injury.
296               Consumption of a high-fat diet protects mice from ventilator-induced lung injury in a m
297                RNA-guided Cas9 endonucleases protect bacteria from viral infection and have been crea
298               Chlorination of drinking water protects humans from water-born pathogens, but it also p
299 particularly during pregnancy and lactation, protects offspring from WD-induced developmental program
300 nt IL6 to LysM-Cre; Egfr(f/f) mice given DSS protected them from weight loss and restored epithelial

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