ライフサイエンスコーパス: protein kinase C-beta

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1 hondrial function and longevity, by reducing protein kinase C-beta.

2 is increased after PMA-evoked activation of protein kinase C-beta.

3 ding those for annexin VI, interleukin-4 and protein kinase C-beta.

4 between PKCI-1 and the regulatory domain of protein kinase C-beta.

5 able transfection of expression plasmids for protein kinases C beta 1 (PKC beta 1) or PKC beta 2 into

6 lted in a preferential decrease in epidermal protein kinase C-beta 2 (PKC-beta 2) compared with PKC-a

7 We further identify a BAFF-specific protein kinase C beta-Akt signaling axis, which provides

8 tagged lipid-binding proteins such as epsin, protein kinase C-beta, and synaptobrevin.

9 x, including synaptotagmin I, cAMP-dependent protein kinase C beta, apolipoprotein E, protein phospha

10 A specific inhibitor of protein kinase C-beta blocked the substrate-mediated inc

11 a mechanism dependent on SNARE proteins and protein kinase C-beta but independent of dopamine D(2) r

12 binding to C2 domains of synaptotagmin I and protein kinase C-beta by nuclear magnetic resonance spec

13 Among the protein knase C isoforms tested, protein kinase C beta, delta, and epsilon were most effe

14 Haploinsufficiency or complete deficiency of protein kinase C beta, Fyn, CD22, Galphaq, or Galpha11 h

15 the Src-family protein tyrosine kinase Lyn, protein kinase C beta I/II (PKCbetaI/II), and Erk2-speci

16 Elevated expression of protein kinase C beta II (PKC beta II) is an early promo

17 the unphosphorylated turn motif (Thr(641) in protein kinase C beta II), one of three priming sites ph

18 tion of the interaction with Hsp70 slows the protein kinase C beta II-mediated recovery of cells from

19 irect binding of this class of activators to protein kinase C beta II.

20 Using homozygous null mice for the protein kinase C beta-isoform gene (PKCbeta null), PKCbe

21 trast, treatment with D-alpha-tocopherol and protein kinase-C beta-isoform inhibition (LY333531) prev

22 ds induced by diabetes mellitus can activate protein kinase C-beta isoforms and selectively inhibit i

23 Abnormal activation of protein kinase C-beta isoforms in the diabetic state has

24 ling, but GC cells signaled less through the protein kinase C-beta-NF-kappaB pathway and produced str

25 Using the HL-60 cell variant deficient in protein kinase C-beta (PKC-beta) and several stable PKC-

26 The role of protein kinase C-beta (PKC-beta) in apoptosis induced by

27 AI-1 induction was suppressed by a selective protein kinase C-beta (PKC-beta) inhibitor.

28 Protein kinase C-beta (PKC-beta) is immediately downstre

29 We have previously shown that protein kinase C-beta (PKC-beta) is required for activat

30 osolic phospholipase A2-alpha (cPLA2-alpha), protein kinase C-beta (PKC-beta), and synaptotagmin-IA (

31 B cells from protein kinase C-beta (PKC-beta)-deficient mice failed t

32 We have identified protein kinase C beta (PKCbeta) as a critical mediator o

33 we identify the calcium-regulated classical protein kinase C beta (PKCbeta) as a repressor of myogen

34 the recommended dose of enzastaurin, an oral protein kinase C beta (PKCbeta) inhibitor, for phase II

35 Protein kinase C beta (PKCbeta) participates in antigen-

36 Protein kinase C beta (PKCbeta) regulated MIF-induced IL

37 Protein kinase C beta (PKCbeta) was identified by gene-e

38 Using mice deficient for protein kinase C beta (PKCbeta) we show an essential rol

39 One key mediator of BCR signaling is protein kinase C beta (PKCbeta), which regulates the act

40 d that combined small-molecule inhibition of protein kinase C-beta (PKCbeta) and glycogen synthase ki

41 l insulin signaling, through the activity of protein kinase C-beta (PKCbeta) and nuclear factor kappa

42 While targeted disruption of the protein kinase C-beta (PKCbeta) gene in mice results in

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