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1 chronic kidney disease (CKD) are at risk of protein-energy malnutrition.
2 ance dialysis suffer from varying degrees of protein-energy malnutrition.
3 intake and contribute to the development of protein-energy malnutrition.
4 A total of 21% had CPEM and 8.1% had acute protein-energy malnutrition.
5 deficiency per se from those of generalized protein-energy malnutrition.
6 he major cause is the confounding effects of protein-energy malnutrition and inflammatory disorders,
8 tentially serious problem because indexes of protein-energy malnutrition are powerful predictors of m
10 e into naive LP or AP mice demonstrated that protein-energy malnutrition caused profound defects in h
15 Screening tools for the early detection of protein-energy malnutrition in older persons have been d
19 well-proven techniques for the treatment of protein-energy malnutrition include intradialytic parent
22 rdised YLL rates for diarrhoeal diseases and protein-energy malnutrition markedly decreased, ranking
25 l pathway relating maternal through to fetal protein-energy malnutrition (PEM) and compromised fetal
26 ein turnover and net protein loss induced by protein-energy malnutrition (PEM) has been well document
27 d function are more compromised in edematous protein-energy malnutrition (PEM) than in nonedematous P
28 tion between plasma albumin and the edema of protein-energy malnutrition (PEM) were investigated by m
30 he first year of life for moderate to severe protein-energy malnutrition, then followed up to 48 year
31 dies in which participants had high rates of protein-energy malnutrition, there was a significantly h
32 protein-energy malnutrition (CPEM) and acute protein-energy malnutrition were defined by the Waterlow
33 ts older persons at high risk for developing protein-energy malnutrition when they develop either psy
34 supplementation on catch-up growth in severe protein-energy malnutrition, with particular reference t
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