ライフサイエンスコーパス: protein-losing enteropathy

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1 re two late deaths and no patients developed protein losing enteropathy.

2 ath of animals due to severe noninflammatory protein-losing enteropathy.

3 mia and were suspected to have an associated protein-losing enteropathy.

4 a thromboembolic event, and 1 (2%) developed protein-losing enteropathy.

5 atrial level shunting, thromboembolism, and protein-losing enteropathy.

6 thmia treatment (32%), thrombosis (12%), and protein-losing enteropathy (8%).

7 3 Fontan revisions, and 1 patient each with protein-losing enteropathy and ascites.

8 e infant who had allergic enterocolitis with protein-losing enteropathy and had low birth weight.

9 own to be associated with the development of protein-losing enteropathy and with decreased survival.

10 provided, and data supporting the notion of protein-losing enteropathy being a consequence of severe

11 n, drug-nutrient interactions, anorexia, and protein-losing enteropathy can all contribute to the pro

12 CHD, long-term results are encouraging, and protein-losing enteropathy can be expected to resolve.

13 imary intestinal lymphangiectasia (PIL) is a protein-losing enteropathy characterized by tortuous and

14 ndent predictors of heart failure death were protein-losing enteropathy (HR, 7.1; P=0.0043), single m

15 f FPE are nonbloody diarrhea, malabsorption, protein-losing enteropathy, hypoalbuminemia, and failure

16 oman with sarcoidosis presenting with severe protein-losing enteropathy, hypoalbuminemia, and proxima

17 The molecular basis of protein-losing enteropathy is unknown.

18 ficant clinical findings (eg, heart failure, protein-losing enteropathy, or new arrhythmias), and som

19 duits, New York Heart Association III/IV, or protein-losing enteropathy/plastic bronchitis) 20 years

20 old) exhibiting chronic effusions (n = 4) or protein-losing enteropathy (PLE) (n = 5) after lateral t

21 Fontan takedown was required in 3% and protein-losing enteropathy (PLE) developed in 6%.

22 Patients with protein-losing enteropathy (PLE) fail to maintain intest

23 Patients with protein-losing enteropathy (PLE) following the Fontan op

24 Protein-losing enteropathy (PLE) is a rare syndrome of g

25 Protein-losing enteropathy (PLE), characterized by loss

26 Protein-losing enteropathy (PLE), the loss of plasma pro

27 ias, renal insufficiency, and development of protein-losing enteropathy (PLE).

28 Protein-losing enteropathy (present in 34 patients) reso

29 usly healthy 7-year-old boy presented with a protein-losing enteropathy secondary to a hypertrophic g

30 , diminished exercise capacity, arrhythmias, protein-losing enteropathy, somatic growth retardation,

31 n of complement, angiopathic thrombosis, and protein-losing enteropathy (the CHAPLE syndrome) is caus

32 re, including ventricular failure, cyanosis, protein-losing enteropathy, thromboembolism, and dysrhyt

33 d CMR parameters, showed that in addition to protein-losing enteropathy, ventricular indexed end-dias

34 ression model with clinical parameters only, protein-losing enteropathy was associated with transplan

35 , significant cyanosis was present in 7, and protein-losing enteropathy was present in 4.

36 inal pain and diarrhea caused by early-onset protein-losing enteropathy with primary intestinal lymph

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