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1 rent encephalitogenic determinants of myelin proteolipid protein.
2 chromosome which codes for the major myelin proteolipid protein.
3 a mutant form of the DM20 isoform of myelin proteolipid protein.
4 yelin autoantigens, myelin basic protein and proteolipid protein.
5 anscription factor at the promoter region of proteolipid protein.
6 sion of myelin basic protein and promoter of proteolipid protein.
7 s association with loss of the myelin marker proteolipid protein.
8 ithin the lesion, despite the persistence of proteolipid protein.
10 s, and the myelin associated glycoprotein to proteolipid protein 1 (MAG:PLP1) ratio, which declines i
11 ecurrent duplication of the dosage-sensitive proteolipid protein 1 (PLP1) gene but also by nonrecurre
12 eds of mutations in the X-linked myelin gene proteolipid protein 1 (PLP1) have been identified in hum
14 e ratio of myelin-associated glycoprotein to proteolipid protein 1 and both endothelin 1 and vascular
15 e ratio of myelin-associated glycoprotein to proteolipid protein 1 and several other proteins involve
16 d ratio of myelin-associated glycoprotein to proteolipid protein 1 are likely to be protective physio
18 ng leukodystrophy caused by mutations of the proteolipid protein 1 gene (PLP1), which is located on t
21 e ratio of myelin-associated glycoprotein to proteolipid protein 1 in post-mortem human brain tissue
22 e ratio of myelin-associated glycoprotein to proteolipid protein 1, correlated positively with the co
23 ptide ligand (APL) variants derived from the proteolipid protein-1 (PLP1) epitope were expressed on i
25 with a retroviral vector designed to encode proteolipid protein (101-157) targeted for secretion.
26 of copolymers to I-A(s) in competition with proteolipid protein 139-151 (blocking), cytokine product
27 of Ag-specific tolerance using two regimens, proteolipid protein 139-151 (PLP139-151) peptide-coupled
28 and a DNA vaccine encoding the self-peptide proteolipid protein 139-151 (PLP139-151) provides protec
29 olecule VLA-4 antagonist, to regulate active proteolipid protein 139-151 (PLP139-151)-induced R-EAE.
30 ybridoma stimulated by a set of three myelin proteolipid protein 139-151 altered peptide ligands.
31 144) from an autoantigenic peptide of myelin proteolipid protein 139-151 by a single amino acid subst
33 ipid protein 139-151 in CFA, GM-CSF fused to proteolipid protein 139-151 peptide inhibited EAE in SJL
34 ke E2, drastically suppressed EAE induced by proteolipid protein 139-151 peptide when given at initia
35 alomyelitis (EAE) induced in SJL/J mice with proteolipid protein 139-151 was demonstrated by using th
37 ncephalomyelitis (EAE) in humanized mice and proteolipid protein 139-151-induced EAE in SJL/J mice.
38 hat anti-GITR mAb treatment of SJL mice with proteolipid protein 139-151-induced experimental autoimm
39 ively induced EAE were able to present Ag to proteolipid protein 139-151-specific T cell lines in vit
43 he frequency and effector function of myelin proteolipid proteins 139-151/I-A(s)-tetramer(+) cells in
44 tified a promoter variant (-113C>A) in PLP2 (proteolipid protein 2) that results in an approximately
45 n immunoglobulin gene superfamily member, or proteolipid proteins, 4-hydrophobic domain-motif protein
47 ice injected i.p. with a peptide fragment of proteolipid protein (a candidate autoantigen in multiple
48 ression is associated with downregulation of proteolipid protein, a highly abundant myelin sheath com
49 ignals may control the surface expression of proteolipid protein, a process that involves reduced end
50 indicate that the adhesive properties of the proteolipid protein, along with the reduction of sialic
51 pathology and to mediate neuroprotection in proteolipid protein alpha-syn (PLP-SYN) mice, a transgen
52 reflected in decreased expression of MBP and proteolipid protein and a reduction in the total number
57 a point mutation in the mouse gene encoding proteolipid protein and is characterized by severe dysmy
59 elitis, stimulation of lymph node cells with proteolipid protein and recombinant murine interleukin (
60 the amounts of myelin basic protein, myelin proteolipid protein, and 2',3'-cyclic nucleotide 3'-phos
61 teins including myelin basic protein, myelin proteolipid protein, and 2'3'-cyclic nucleotide 3'-phosp
62 sociates with myelin proteins such as myelin proteolipid protein, and assembles lipid-rich microdomai
63 teins, including myelin basic protein (MBP), proteolipid protein, and myelin oligodendrocyte glycopro
64 for reactivity against myelin basic protein, proteolipid protein, and myelin oligodendrocyte glycopro
65 ein and a decrease in neurofilament protein, proteolipid protein, and several pro-inflammatory marker
66 n oligodendrocyte glycoprotein (MOG)35-55 in proteolipid protein- and MOG-induced models of EAE, resp
69 The loss of Cx47 was associated with loss of proteolipid protein at the chronic stage of MHV-A59 infe
70 nic Ags (guinea pig myelin basic protein and proteolipid protein) by lymph node cells from animals im
72 ts and are specific for two different myelin proteolipid protein-derived peptides presented by HLA-A2
73 t immunization with myelin basic protein and proteolipid protein determinants results in clinical dis
74 -cell responses to the immunodominant myelin proteolipid protein epitope (PLP139-151) did not arise b
75 (HI574-586) of an immunodominant self-myelin proteolipid protein epitope (PLP139-151) induced a rapid
76 nization of SJL mice with the immunodominant proteolipid protein epitope, PLP139-151, surface express
78 taining human myelin basic protein and human proteolipid protein epitopes, prevented clinical symptom
81 Xenopus, DM gamma, a membrane protein of the proteolipid protein family, is expressed in a subset of
91 activation in the peripheral lymphocytes nor proteolipid protein gene coding alterations were identif
92 indicate that, occasionally, females with a proteolipid protein gene duplication can manifest an ear
93 uorescent in situ hybridization identified a proteolipid protein gene duplication in both patients.
94 oscopic duplication that contains the entire proteolipid protein gene is the major cause of Pelizaeus
96 zaeus-Merzbacher disease, duplication of the proteolipid protein gene PLP1 is responsible, whereas de
99 n by oligodendrocytes expressing one copy of proteolipid protein gene secondary to selection for a fa
100 axons, and subsequently expressed the myelin proteolipid protein gene, initiating remyelination.
101 fect disease induced after immunization with proteolipid protein immunodominant peptide plus MBP.
102 c F1 mice, immunized 12-15 days earlier with proteolipid protein in complete Freund's adjuvant, were
103 ulates cell surface expression of the myelin proteolipid protein in cultured oligodendrocytes in unex
104 y acidic protein in Alexander disease and of proteolipid protein in hypomyelinating disorders such as
106 ought to be completely replaced by the newer proteolipid proteins in the terrestrial vertebrate CNS.
107 course is well documented for both MBP- and proteolipid protein-induced EAE, and recently has been s
108 major intrinsic membrane protein of myelin, proteolipid protein, interacts with rafts in oligodendro
110 issue, Yin et al. study mutant mice in which proteolipid protein is replaced by the peripheral myelin
111 ling view has been that expression of P0 and proteolipid proteins is mutually exclusive; P0, which me
112 ons of myelin-specific proteins (MBP, myelin proteolipid protein, MAG, and 2',3'-cyclic nucleotide,3'
114 uced by myelin oligodendrocyte glycoprotein, proteolipid protein, myelin basic protein, and renal tub
115 expression, including myelin basic protein, proteolipid protein, myelin oligodendrocyte glycoprotein
116 erized myelin antigens myelin basic protein, proteolipid protein, or myelin oligodendrocyte glycoprot
117 roliferative response and IL-2 production of proteolipid protein p139-151-specific T cells were signi
118 ctive T cells was demonstrated by inhibiting proteolipid protein (p139-151)-induced EAE and finding t
119 JL/J mouse by adoptive transfer of activated proteolipid protein peptide (PLP) 139-151-specific Th1 c
120 on with Listeria monocytogenes (LM) encoding proteolipid protein peptide (PLP) amino acids 178-191 (L
121 latory signals, impaired memory responses to proteolipid protein peptide (PLP), and do not develop PL
123 zed for disease with encephalitogenic myelin proteolipid protein peptide 139 to 151, and analysis was
126 specific for MBP exon 2, MBP peptide 89-101, proteolipid protein peptide 139-151, and OVA gave stimul
127 of a CD4+, Th1, VB2+ encephalitogenic SJL/J proteolipid protein peptide 139-151-specific T cell clon
129 ecific for the relapse epitope consisting of proteolipid protein peptide amino acids 139-151 clustere
131 During acute relapsing EAE induced by a proteolipid protein peptide of amino acids 178-191, tran
133 AR inhibited their capability to present the proteolipid protein peptide to PLP(139-151)-specific T c
135 t detectable in the infiltrates of mice with proteolipid protein peptide-induced experimental autoimm
136 ectively, these results indicate that myelin proteolipid protein peptide-specific CD8+ CTL may be an
138 ntal autoimmune encephalomyelitis induced by proteolipid protein, peptide 139-151-specific T cell lin
139 In relapsing/remitting EAE induced with proteolipid protein peptide139-151, lithium administered
140 Our data showed that an extensive array of proteolipid protein peptides could elicit autoreactivity
141 encoded by alternative transcripts from the proteolipid protein ( PLP ) gene, are major components o
143 ospot analysis (ELISPOT) assays, we followed proteolipid protein (PLP) 139--151-specific T cells enga
144 study, we have identified an MHC variant of proteolipid protein (PLP) 139-151 (145D) that renders PL
145 encephalitogenic epitope of a myelin antigen proteolipid protein (PLP) 139-151 in the peripheral repe
147 ompassing the immunodominant myelin epitope, proteolipid protein (PLP) 139-151, into the coding regio
148 Female B10.S mice are highly resistant to proteolipid protein (PLP) 139-151-induced experimental a
149 mpared the ability of anti-VLA-4 to regulate proteolipid protein (PLP) 139-151-induced R-EAE when adm
150 tides, myelin basic protein (MBP) 87-106 and proteolipid protein (PLP) 175-192, that are considered t
153 ducts of the proteolipid protein gene (PLP), proteolipid protein (PLP) and DM20, are major components
154 matic epitope mapping of responses to myelin proteolipid protein (PLP) as well as assaying responses
155 mutation in exon 3B of the PLP altering the proteolipid protein (PLP) but not the alternatively spli
156 ping series of 265 12-mer peptides of myelin proteolipid protein (PLP) by patients with isolated mono
157 s of myelin, myelin basic protein (MBP), and proteolipid protein (PLP) during postnatal brain develop
158 ific for the immunodominant encephalitogenic proteolipid protein (PLP) epitope (PLP139-151) as assess
160 SJL mice initiated by immunization with the proteolipid protein (PLP) epitope PLP139-151 is associat
161 nti-IL-23p19 during active disease inhibited proteolipid protein (PLP) epitope spreading and prevente
162 states in different cell compartments of the proteolipid protein (PLP) expressed in COS-7 cells.
165 Overexpression or lack of expression of proteolipid protein (PLP) gene by oligodendrocytes cause
168 enes containing portions of the mouse myelin proteolipid protein (PLP) gene fused to the lacZ reporte
170 nt protein (EGFP) driven by the mouse myelin proteolipid protein (PLP) gene promoter have been develo
175 areas, an increased number of OGs expressing proteolipid protein (PLP) mRNA compared with those expre
176 rade astrocytomas contained a high number of proteolipid protein (PLP) mRNA-positive cells and that t
177 ic for an encephalitogenic peptide of myelin proteolipid protein (PLP) peptide 139-151 (HSLGKWLGHPDKF
180 B6) specific for the encephalitogenic myelin proteolipid protein (PLP) peptide 139-151, on the experi
181 encephalomyelitis (EAE) induced with myelin proteolipid protein (PLP) peptide 139-151, whereas H-2 c
183 ning of the BBB in EAE mice immunized to the proteolipid protein (PLP) peptide, PLP 139-151, with or
184 eptides derived from sequences of the myelin proteolipid protein (PLP) presented by HLA class I molec
185 nit of functional NMDARs, using an inducible proteolipid protein (Plp) promoter-driven Cre-loxP recom
187 encephalomyelitis (EAE) induced with myelin proteolipid protein (PLP) residues 139-151 (HSLGKWLGHPDK
189 1, a chimera expressing the encephalitogenic proteolipid protein (PLP) sequence 139-151, induced devi
190 c presentation system, we demonstrate that a proteolipid protein (PLP) TCR antagonist peptide (PLP-LR
192 ized with the p139-151 determinant of myelin proteolipid protein (PLP) were transfected with a DNA co
193 enic mice on the SJL background specific for proteolipid protein (PLP)(139-151) develop a high incide
195 Multiple Ag peptides (MAPs) containing eight proteolipid protein (PLP)(139-151) peptides arranged aro
196 sing the HLA-DR or -DQ gene), we showed that proteolipid protein (PLP)(91-110) peptide induced classi
197 -cyclic-nucleotide 3'-phosphodiesterase, and proteolipid protein (PLP)) in primary human oligodendroc
198 -expressing cells co-expressed mRNA encoding proteolipid protein (PLP), a mature oligodendrocyte mark
200 allergic encephalomyelitis in SJL mice with proteolipid protein (PLP), brain ICOS mRNA and protein w
201 intervene in autoimmune responses to myelin proteolipid protein (PLP), encephalitogenic epitopes mus
202 ntaining both myelin basic protein (MBP) and proteolipid protein (PLP), induced antigen specific MBP
203 e, we show that NG2+ cells express mRNAs for proteolipid protein (PLP), myelin basic protein, and 2',
204 th antibodies to myelin basic protein (MBP), proteolipid protein (PLP), myelin-associated glycoprotei
205 undrum with regard to the function of myelin proteolipid protein (PLP), one of the major proteins in
206 ge when a tetraspan membrane protein, myelin proteolipid protein (PLP), replaced the type I integral
207 hybridization with oligonucleotide probes to proteolipid protein (PLP), the major protein in central
210 ssociated glycoprotein (MAG) but not P(0) or proteolipid protein (PLP), the structural proteins of co
211 ant of a non- glycosylated membrane protein, proteolipid protein (PLP), to examine the quality contro
212 expression of myelin basic protein (MBP) and proteolipid protein (PLP), two major myelin-specific pro
215 were determined from animals immunized with proteolipid protein (PLP)-139-151 (disease agonist), PLP
217 ism of a rTCR ligand (RTL) construct (I-A(s)/proteolipid protein (PLP)-139-151 peptide = RTL401) for
218 P1 and Ig-PLP-LR are chimeric Igs expressing proteolipid protein (PLP)-derived T cell agonist (PLP1)
219 mice, which are highly susceptible to myelin proteolipid protein (PLP)-induced experimental autoimmun
220 the human HLA-DRB1*0301 gene predisposes to proteolipid protein (PLP)-induced experimental autoimmun
223 (ELISPOT) assays to study, directly ex vivo, proteolipid protein (PLP)-specific memory cell reactivit
224 ) selectively modifies cytokine secretion in proteolipid protein (PLP)-specific, CD4+ T cell clones i
229 r ribonucleoprotein (hnRNP) H and F regulate proteolipid protein (PLP)/DM20 alternative splicing.
231 n (Ig) chimera carrying the encephalitogenic proteolipid protein (PLP)1 peptide corresponding to amin
233 +CD25-Foxp3- T cells specific for the myelin proteolipid protein (PLP)139-151 peptide can be converte
234 DEC205-mediated delivery of the self-peptide proteolipid protein (PLP)139-151 to DCs ameliorated clin
235 w chimeras, we show that activation of naive proteolipid protein (PLP)139-151-specific T cells in SJL
236 d multitransmembrane domain proteins, myelin proteolipid protein (PLP, 30 kDa) and DM-20 (26 kDa), fr
238 ion of 19 base pairs (bp) in intron 3 of the proteolipid protein (PLP/DM20) gene causes a neurologica
239 ed with an autoantigenic peptide from myelin proteolipid protein (PLP; PLP(139-151)) and used it to a
243 editary spastic paraplegia (HSP) type 2 is a proteolipid protein (PLP1)-related genetic disorder that
247 ng a minigene for residues 139-151 of myelin proteolipid protein (PLP139-151), a pathogenic self-Ag.
251 oxed conditional allele was crossed with the proteolipid protein promoter-driven inducible Cre allele
252 Our data suggest that NG2(+)/PDGFRalpha(+) proteolipid protein promoter-expressing progenitors gene
260 our study, we used the previously described [proteolipid protein/suppressor of cytokine signaling 1 (
261 enerated a transgenic mouse line [PLP/SOCS1 (proteolipid protein/suppressor of cytokine signaling 1)]
262 hed axons expressed and selectively targeted proteolipid protein to compact myelin and did not degene
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