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1 least in part, via ecdysone synthesis in the prothoracic gland.
2 activity of target of rapamycin (TOR) in the prothoracic gland.
3 ially express 3B11 represent the presumptive prothoracic glands.
4 hat a portion of the timing may occur in the prothoracic glands.
5 has multiple target tissues as follows: the prothoracic gland and aorta in the larva and the crop an
6 t tissues, dib expression is observed in the prothoracic gland and follicle cells of the ovaries resp
7 or in this tissue, Tsl also localizes to the prothoracic gland and influences developmental timing.
10 Instead, we find that innervation of the prothoracic gland by the PG neurons is absent in gt hypo
12 idogenesis (downregulation); and (c) how the prothoracic gland cells convert cholesterol to the precu
13 ression of both genes is concentrated in the prothoracic gland cells of the developing ring gland.
17 vity of the insulin-signaling pathway in the prothoracic gland of Drosophila modulates ecdysone relea
18 in vitro secretion of ecdysteroids from the prothoracic glands of last instar larvae of Spodoptera l
19 is and subsequent ecdysteroidogenesis in the prothoracic glands of the tobacco hornworm, Manduca sext
21 rvae, torso is expressed specifically in the prothoracic gland (PG), and its loss phenocopies the rem
22 osophila, a component of the ring gland, the prothoracic gland (PG), assesses growth to determine whe
26 )/Target of Rapamycin (TOR) signaling in the prothoracic glands (PGs) regulates ecdysone biosynthesis
27 ircadian culture system from Drosophila, the prothoracic gland provides unique advantages for investi
28 on of calcium signaling in the steroidogenic prothoracic gland results in the accumulation of unrelea
29 in signaling, specifically in the Drosophila prothoracic gland, results in developmental arrest prior
30 revealed that the phosphorylation of Manduca prothoracic gland S6 is limited exclusively to serine re
31 steroid hormone 20-hydroxyecdysone from the prothoracic gland, the primary endocrine organ of juveni
32 ons of 20E in the hemolymph feed back on the prothoracic gland to decrease rates of ecdysteroidogenes
33 ctivin signaling regulates competence of the prothoracic gland to receive PTTH and insulin signals, a
35 iates a transductory cascade in cells of the prothoracic gland, which results in an increased rate of
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