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1 L-6/STAT3 axis in PCa patients has failed to provide therapeutic benefit.
2 and, as a consequence, targeting M-ILK could provide therapeutic benefit.
3 via enhanced regulatory T cell activity may provide therapeutic benefit.
4 m with AURKA inhibitors has the potential to provide therapeutic benefit.
5 ers of gene-modified cells are sufficient to provide therapeutic benefit.
6 to hasten the resolution of inflammation and provide therapeutic benefit.
7 multiple tumor types and targeting Bcl-2 may provide therapeutic benefit.
8 rferon gamma (IFNgamma), a cytokine that can provide therapeutic benefit.
9 lear whether systemic inhibition of IKK will provide therapeutic benefit.
10 development and selection of agents that may provide therapeutic benefit.
11 al compounds that affect aggregation and may provide therapeutic benefit.
12 aling or excitation-contraction coupling can provide therapeutic benefit.
13 n, and strategies to activate expression may provide therapeutic benefit.
14 Replacing Nemo through gene therapy might provide therapeutic benefits.
15 sition in the skin by dietary fish oil could provide therapeutic benefits.
16 diabetic stem/progenitor cell functions may provide therapeutic benefits.
17 nally restoring the physiologic gradient may provide therapeutic benefits.
18 es geared toward inhibiting inflammation may provide therapeutic benefits.
19 kers prevent T-tubule remodeling and thereby provide therapeutic benefits.
20 s from their pathophysiological fate and may provide therapeutic benefits.
21 nd in some instances metabolic interventions provide therapeutic benefits.
22 nistration of low-dose PMO25 in SMA mice can provide therapeutic benefit and phenotypic rescue, presu
23 aled corticosteroids for control, colchicine provides therapeutic benefit as measured by maintenance
24 st, ENaC inhibition via exogenous agents may provide therapeutic benefit, as has long been proposed.
25 nt with the BRAF V600E inhibitor vemurafenib provides therapeutic benefits but the common emergence o
26 utant Huntingtin messenger RNA (mRNA) should provide therapeutic benefit, but normal Huntingtin likel
27 ANKL can alter thymic output and potentially provide therapeutic benefit by enhancing anti-tumor immu
29 NAc-Hep or similar heparin derivatives might provide therapeutic benefits by diminishing bleeding com
31 king memory and that targeting GABA(B)Rs may provide therapeutic benefit for age-related impairments
33 f the 12/15LO pathway in the vessel wall may provide therapeutic benefit for early vascular inflammat
35 leukemia and that antagonizing Survivin may provide therapeutic benefit for patients with acute leuk
36 PBN inhibition of RPE65 catalytic action may provide therapeutic benefit for such retinal diseases.
37 ition of InsP3-mediated Ca2+ signaling could provide therapeutic benefit for the patients afflicted w
39 itochondrial function in muscle, which might provide therapeutic benefits for diseases associated wit
40 cells (MSCs) are ongoing for the purpose of providing therapeutic benefit for a variety of human dis
41 or accelerating hematopoietic recovery, thus providing therapeutic benefits for patients after clinic
44 inhibitors such as palbociclib are likely to provide therapeutic benefit in combination with BRAF inh
45 orally administered PPARgamma agonists could provide therapeutic benefit in demyelinating disease.
46 biogenesis and fatty acidbeta-oxidation and provide therapeutic benefit in diseases associated with
48 GCase, can effectively reduce alpha-syn and provide therapeutic benefit in human midbrain neurons.
49 (LNA)-modified antimiR (LNA-antimiR-34) can provide therapeutic benefit in mice with preexisting pat
50 pected underlying infections and appeared to provide therapeutic benefit in our cohort of patients wi
54 ive, high-affinity A(2B) AdoR antagonist may provide therapeutic benefit in the treatment of asthma.
55 cal inhibition of the DOT1L/AF10 complex may provide therapeutic benefits in an array of malignancies
56 ugh direct targeting of the cancer cells may provide therapeutic benefits in CLL by encouraging a cyt
61 challenge in an infectious disease model and provided therapeutic benefit in a mouse cancer model.
63 ally, pharmacologic inhibition of ROR-gammat provided therapeutic benefit in mouse models of intestin
64 ing of differently folded states of SOD1 has provided therapeutic benefit in mutant SOD1 transgenic m
65 nterference mediated silencing of ATXN1 mRNA provides therapeutic benefit in mouse models of the dise
66 These results suggest that MC stabilization provides therapeutic benefits in sepsis by inhibiting ex
67 ideal drug for sickle cell anemia (SCA) and provides therapeutic benefit through multiple mechanisms
69 uggest that strategies to maintain HSP72 may provide therapeutic benefit to enhance mitochondrial qua
70 that small molecule inhibitors of CRTC2 may provide therapeutic benefit to individuals with autoimmu
71 es that attenuate the CREB-CRTC2 pathway may provide therapeutic benefit to individuals with type 2 d
72 ounds that enhance cryptochrome activity may provide therapeutic benefit to individuals with type 2 d
73 cological targeting of beta(1) integrins may provide therapeutic benefit to overcome tumor cell resis
74 alone or in combination with paclitaxel, may provide therapeutic benefit to patients diagnosed with A
75 reventing this myosin isoform "switch" could provide therapeutic benefit to patients with heart failu
76 h paradoxical MAPK pathway activation and to provide therapeutic benefit to patients with RAS mutant
77 ggest that use of ER-selective ligands could provide therapeutic benefit to reduce the risk of AD by
78 adiponectin (APN), a pleiotropic adipokine, provides therapeutic benefit to prevent AngII-induced ad
79 HGF to its active form has the potential to provide therapeutic benefit where HGF/Met activity is as
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