ライフサイエンスコーパス: provocation

1 CM (LVOTG < 30 at rest but >/= 30 mm Hg with provocation).

2 the brain in the resting condition (without provocation).

3 (pulmonary function, bronchial methacholine provocation).

4 is associated with higher experienced anger provocation.

5 cellular protection after UVB carcinogenesis provocation.

6 ivity was evaluated by means of cold dry air provocation.

7 r these cells in the lung following allergen provocation.

8 ial exhaled nitric oxide, and a methacholine provocation.

9 e in triggering T cell responses upon immune provocation.

10 f mice and people with asthma after allergen provocation.

11 he steady state and after local aeroallergen provocation.

12 e DC differed in response to allergic immune provocation.

13 len (n = 12) before and after birch allergen provocation.

14 ntal elicitation of eczematous lesions after provocation.

15 BALF exosomes from before and after allergen provocation.

16 ng, exercise tolerance testing, and ajmaline provocation.

17 s the host response following an immunologic provocation.

18 alated perpetrated aggression more following provocation.

19 nduced eosinophilia associated with allergen provocation.

20 mast cell-deficient mice after aeroallergen provocation.

21 breathing (n = 5) before a 2-minute carbogen provocation.

22 lbumin level in nasal-lavage fluid) to nasal provocation.

23 y to attack, particularly following physical provocation.

24 nt of an ongoing airway response to allergen provocation.

25 enously and retinal DeltaPo2 during carbogen provocation.

26 en autoimmune pathogenesis and environmental provocation.

27 ne tolerance in the face of constant antigen provocation.

28 also reported higher levels of anger-related provocation.

29 57BL/6J) mice displayed an EPR following OVA provocation.

30 ed mast cell-deficient mice following an OVA provocation.

31 ar G-stress, but without the motion sickness provocation.

32 ess, was observed 5-30 minutes after antigen provocation.

33 circumstances, even during maximal bronchial provocation.

34 n their wild-type littermates after allergen provocation.

35 tralateral histamine, hours after unilateral provocation.

36 saline or BDP for 3 d before albuterol nasal provocation.

37 e responses to both allergic and nonallergic provocation.

38 pontaneous panic during the year after panic provocation.

39 e indeed potentiates aggressive responses to provocation.

40 accumulated after several days of continued provocation.

41 th atopy and healthy subjects after allergen provocation.

42 l prefrontal cortex connectivity during high provocation.

43 timuli resulting in cessation of the symptom provocation.

44 es aggressive behavior in response to social provocation.

45 14.6 mug/mL Fel d1, and matched diluent-only provocation.

46 al and amygdala reactivity when triggered by provocation.

47 cellular activation following a local nasal provocation.

48 nce was observed in change in pain after ice provocation.

49 ic responses after laboratory-based allergen provocation.

50 s likely have in the lung following allergen provocation.

51 Exercise testing, drug provocation, advanced cardiac imaging, and genetic testi

52 Recommendations on oral provocation and desensitization procedures have been mad

53 Previous studies have used symptom provocation and positron emission tomography to delineat

54 y controls were subjected to topical cowhage provocation and skin prick testing with histamine and as

55 sion is increased in asthmatics following Ag provocation and suggest that modulation of TRAIL and TRA

56 Mouse models of allergen provocation and/or transgenic gene expression have provi

57 ructive (LVOTG >/= 30 mm Hg at rest and with provocation), and latent HCM (LVOTG < 30 at rest but >/=

58 ulted in the loss of a main source of immune provocation, and a consequent increase in pathogenic imm

59 Although calcium provocation-angiography is highly effective in the ident

60 challenged with OVA also received a late OVA provocation at a higher concentration and were then expo

61 dually tailored to achieve effective symptom provocation at neutral, intermediate and strong intensit

62 ts occurring in the lungs following allergen provocation at several key moments: (i) the initiating p

63 Bronchial allergen provocations (BAP) were repeated at weeks 1, 2, 4, and 8

64 During the symptom provocation block, the participant could choose to rejec

65 t aggressive behaviors, such as responses to provocation, but that it also promotes nonaggressive beh

66 nd sudden cardiac death on catecholaminergic provocation by caffeine/epinephrine or programmed electr

67 Provocation by chronic immunosuppression, histology, and

68 aintains immune homeostasis despite constant provocation by environmental Ags.

69 in the intestinal mucosa, despite continuous provocation by environmental antigens, are jeopardized i

70 Aeroallergen provocation caused a 3-fold increase in the frequency of

71 In the second part, cold dry air provocation (CDA) was performed as a hyper-reactivity me

72 The diagnostic properties of a graded provocation challenge (PC) among children presenting wit

73 20 kD fraction (CPE w/o 20 kD) for allergic provocation challenge and immunotherapy.

74 NERD using blinded, placebo-controlled oral provocation challenge tests (OPCTs) or by self-reported

75 We therefore performed spirometry, bronchial provocation challenge, and fiberoptic bronchoscopy with

76 The results indicate that a CO(2) provocation challenge, in combination with ASL-MRI, is a

77 nt egg allergy need not be subject to an egg provocation challenge, reducing both costs and risks to

78 The patient refused an oral provocation challenge.

79 ypersensitivity, whereas in other cases oral provocation challenges are necessary to confirm the diag

80 or pepsinized cashew proteins and subsequent provocation challenges.

81 cs were performed (skin biopsy, subcutaneous provocation, clinical/laboratory assessment for thrombos

82 dy was to evaluate the colonoscopic allergen provocation (COLAP) test using the recombinant birch pol

83 y analysis, we defined AHR by a methacholine provocation concentration of 4 mg/mL or less, which led

84 d with the occurrence of aggression in a low provocation condition, but significantly predicts such b

85 retinal oxygenation response to a hyperoxic provocation (DeltaPo2) is strongly associated with subse

86 el individualized ecologically valid symptom provocation design: a live provocation functional magnet

87 nges of R5, R20, R5-R20, X5, Fres, ALX after provocations for ACh or Hist in all patients with asthma

88 lly valid symptom provocation design: a live provocation functional magnetic resonance imaging paradi

89 HTS nasal provocations identified significant, unique patterns of

90 Mood provocation in both depressed groups resulted in regiona

91 and early asthmatic responses after allergen provocation in patients with allergic asthma.

92 +) waves evoked by high extracellular Ca(2+) provocation in S2814D(+/+) mice into non-propagated Ca(2

93 mation resolved 48 h after a single allergen provocation in sensitized control mice, but not in anima

94 ype synthesis and is unusual in the apparent provocation in the reaction of an increase in core size.

95 er, the rapid release of IL-33 upon allergen provocation in vivo supports its role as a mediator of i

96 ated whether IL-33 is released upon allergen provocation in vivo.

97 inhibitory control of emotional reactions to provocations in antisocial individuals.

98 Ten healthy subjects underwent provocations in which single doses of methacholine, prev

99 This increased generosity in the absence of provocation indicates that testosterone can also cause p

100 During arrhythmia provocation induced by catecholamine injections, TG anim

101 Aeroallergen provocation induces the rapid accumulation of CD11c(+)MH

102 25 mg/ml) underwent single-dose methacholine provocations inducing at least 20% reduction in FEV1 aft

103 ot reactive (provoked) aggression (alcohol x provocation interaction).

104 curs with greater intensity and frequency as provocation is experimentally manipulated upwards, espec

105 Implementing control over these provocations is essential to healthy functioning; howeve

106 d flow in psoriatic plaques before and after provocations known to alter cutaneous vascular resistanc

107 herein depressive episodes occur with little provocation, may be reached by two pathways: many previo

108 rms by visual analog scale scores before ice provocation (median, 0.00; range, -3.30 to 0.70 for botu

109 rovocation, median MRC, 5; range, 3-5; after provocation, median MRC, 3; range, 1-4) and increased SI

110 n reduced muscle strength in HyperPP (before provocation, median MRC, 5; range, 3-5; after provocatio

111 ir home cage, both with and without physical provocation (mild tail pinch).

112 In this study, we used a defined OVA provocation model and mice deficient for specific leukoc

113 rrect diagnosis is crucial, but no validated provocation model exists, and other diagnostic tests hav

114 l mechanisms were evaluated in a human nasal provocation model.

115 oth spontaneously and in a resident/intruder provocation model.

116 d not to the sensitization phase of allergen provocation models.

117 for associations with findings from ajmaline provocation (n=332), exercise ECG (n=304), and signal-av

118 ously shown to be critical for the bacterial provocation of a pathogenesis-promoting host innate immu

119 alone, are probably important factors in the provocation of a relapse of vivax malaria.

120 wn function of human sebaceous glands is the provocation of acne.

121 Allergen provocation of allergic asthma patients is often charact

122 Provocation of an angerlike state results in T-wave alte

123 Provocation of an autoimmune response against NaV1.5 ind

124 n confirmed and expanded upon using allergen provocation of an inducible eosinophil-deficient strain

125 only in apoptotic processes but also in the provocation of angiogenic and proinflammatory responses.

126 Despite the frequent provocation of chest pain and electrocardiographic abnor

127 olves direct tissue toxic effects as well as provocation of destructive bystander immune responses.

128 second primate species with and without the provocation of increased peripheral homeostatic T-cell a

129 brogated regulatory cytokine production with provocation of inflammatory T-cell responses to apoptoti

130 ickness' procedure immersion was preceded by provocation of motion sickness in a human centrifuge.

131 The experimental provocation of panic in nonclinical subjects appears to

132 were compared with those of dexamethasone in provocation of Pneumocystis carinii pneumonitis in virus

133 5)O]H(2)O positron emission tomography after provocation of sadness with autobiographical memory scri

134 Sensorineural deafness in MWS, and provocation of symptoms by cold in FCU, are distinctive

135 if CNS substance P concentrations change on provocation of symptoms in PTSD patients.

136 of concordant functional change accompanying provocation of transient sadness in healthy volunteers a

137 The authors examined the effect of panic provocation on the subsequent development of panic attac

138 NOS2 protein was noted within 6 hours of LPS provocation only in the hearts of wild-type mice.

139 n the rate of positive responses to mannitol provocation or Feno measurements.

140 and visual analog scale before and after ice provocation over a 4-week period.

141 Predosing FEV1 dropped 10% over 4 wk of provocation (p = 0.001) and 7 of 9 returned to prestudy

142 specific lung resistance 4 to 8 h following provocation, p < 0.05 versus vehicle treatment) and airw

143 rs (0.53+/-0.06 before and 0.54+/-0.06 after provocation, P=.3).

144 esponsive to a script-driven imagery symptom provocation paradigm, were exposed sequentially to audio

145 increased subjective anxiety during symptom provocation (patients significantly more so) and activat

146 In the three conditions with pollen provocation, patients were pretreated with either cetiri

147 Following provocation, PCPA and TP+PCPA significantly increased ag

148 t when JNJ28307474 was only given during the provocation phase of the allergic reaction.

149 The pathogenic mechanism of provocation poliomyelitis may differ from that of polio

150 This phenomenon, labeled "provocation poliomyelitis," continues to cause numerous

151 chanism linking intramuscular injections and provocation poliomyelitis.

152 minal hyperactivity during patients' symptom provocation preceded subsequent deactivation during avoi

153 In the remitted group, mood provocation produced a unique rCBF decrease in pregenual

154 Provocation reduced muscle strength in HyperPP (before p

155 rtex helps ensure that irrelevant affordance provocations remain below the threshold for actual movem

156 CDA provocation resulted in a significant increase in rhinit

157 At every step of the provocation, scanning was performed at approximately the

158 utflow tract (LVOT) gradient at rest or with provocation, septal thickness, and baseline exercise dur

159 ine from 16 atopic asthmatics under allergen provocation, showing increases in metabolites of prostag

160 nged subjects reported immediate itch at the provocation site (short lasting, for 2-6 min).

161 gnificantly predicts such behavior in a high provocation situation.

162 NSAIDs hypersensitivity (which include drug provocations, skin testing and in vitro testing) and pro

163 During methacholine provocation, small airway dysfunction was more severe in

164 onse of ER subtypes to exercise and ajmaline provocation suggests underlying mechanisms of both abnor

165 lcholine leading to a positive acetylcholine provocation test (ACH test).

166 Conjunctival allergen provocation test (CAPT) reproduces the events occurring

167 icacy was assessed by comparing conjunctival provocation test (CPT) reactions at baseline, after 4 we

168 Conjunctival provocation test (CPT) response thresholds improved from

169 e diagnostic value of a skin test (ST), drug provocation test (DPT) and basophil activation test (BAT

170 ants of PG (PPL/MDM), AX and CLV and by drug provocation test (DPT) if required.

171 ablish the role of skin testing and the drug provocation test (DPT) in the diagnosis of nonimmediate

172 Patients confirmed as NIUA by positive drug provocation test (DPT) with acetylsalicylic acid (ASA) d

173 Evidence regarding drug provocation test (DPT) with antineoplastic and biologica

174 and a positive response to a nasal allergen provocation test (NAPT) without evidence of systemic ato

175 serum specific IgE and IgG4, nasal allergen provocation test (NAPT), and adverse events.

176 of Life Questionnaire (RQLQ), nasal allergen provocation test (NAPT), skin testing, serum levels of s

177 in symptom scores following a titrated nasal provocation test (TNPT) at baseline and after 5 months o

178 oconjunctivitis (ARC) using a titrated nasal provocation test (TNPT).

179 he summer, and she reacted positively to the provocation test at the work place, but not in the house

180 with optical coherence tomography during the provocation test for coronary spasm.

181 t past medical history underwent an ajmaline provocation test for investigation of suspected Brugada

182 scores at baseline and during a methacholine provocation test in 15 subjects with asymptomatic BHR, 1

183 The intracoronary acetylcholine provocation test is a safe technique to assess coronary

184 owder mannitol for inhalation as a bronchial provocation test is FDA approved however not currently a

185 E antibodies is useful, and a nasal allergen provocation test is needed to identify the culprit aller

186 Drug provocation test is vital to validate diagnostic tools;

187 traction Aggression Paradigm (a computerized provocation test of aggression), the Aggression Question

188 aeroallergens, and a positive nasal allergen provocation test response with release of inflammatory m

189 A provocation test using jellyfish samples was not perform

190 osis, a single-blind placebo-controlled drug provocation test was performed with aspirin or diclofena

191 tructive (LVOTG < 30 mm Hg at rest and after provocation test with amyl nitrite), obstructive (LVOTG

192 od and pollen allergy symptoms, conjunctival provocation test with birch pollen and Bet v 1, and in v

193 A bronchial provocation test with dry air was conducted in 42 player

194 measures design, patients underwent a nasal provocation test with either pollen or inactive control

195 A nasal allergen provocation test with multiple aeroallergens (NAPT-M), i

196 and exercise electrocardiograms and ajmaline provocation test.

197 nea was rated with the Borg score during the provocation test.

198 d had a positive bronchodilator or bronchial provocation test.

199 nd, severe symptoms might be provoked by the provocation test.

200 re diagnosed by a wheat ingestion + exercise provocation test.

201 e a review of published data for the role of provocation testing in the diagnosis of coronary spasm.

202 s, and safety of intracoronary acetylcholine provocation testing in white patients with unobstructed

203 B, UV-A, and visible radiation and broadband provocation testing showing similar results.

204 The intracoronary acetylcholine provocation testing was performed directly after angiogr

205 Drug provocation testing was undertaken by 87% of respondents

206 es with airway hyperresponsiveness (mannitol provocation testing) or inflammation (Feno).

207 tional exhaled nitric oxide (Feno), mannitol provocation testing, and (3)He gas magnetic resonance im

208 Conjunctival provocation tests (CPT) were performed before, during an

209 Conjunctival provocation tests (CPT), skin prick tests (SPT), BAT, an

210 ST), 12 by positive IgE and 42 by controlled provocation tests (CPT)].

211 Drug provocation tests (DPT) are commonly performed as part o

212 Nasal allergen provocation tests (NAPT) with Dermatophagoides pteronyss

213 Immediate responses to nasal allergen provocation tests (NAPTs) with Art v 3, Pru p 3, and mug

214 Single-blind, placebo-controlled oral provocation tests (OPTs) with the PPIs other than the cu

215 with a confirmed NSAID-H were found by oral provocation tests (OPTs).

216 Single-blind, placebo-controlled provocation tests (SBPCPTs) with aspirin were performed

217 inical benefit of cowhage and histamine skin provocation tests should be investigated in further stud

218 Many patients therefore require provocation tests to make a definitive diagnosis; these

219 However, provocation tests using intracoronary acetylcholine admi

220 Aspirin-supplemented provocation tests were performed to 4 cases who had nega

221 Nasal allergen provocation tests with Dermatophagoides pteronyssinus, A

222 iable tools (eg, questionnaires or bronchial provocation tests with small-particle stimuli) to assess

223 least two different NSAIDs or positive drug provocation tests), (ii) patients with more than two epi

224 Among pain provocation tests, a positive painful arc test result wa

225 s available in only half of the cases, while provocation tests, allergen quantification in homes, and

226 relies-as in humans-on elimination diet and provocation tests.

227 index and normal growth-hormone responses to provocation tests.

228 oup by a combination of diurnal profiles and provocation tests.

229 nsive and potentially risky, especially drug provocation tests.

230 lls) and (vii) In vivo biomarkers (including provocation tests?).

231 I intend this article to be a provocation that can help clarify our disciplinary inten

232 ly analysed the neural correlates of symptom provocation, the urge to avoid, rejection and relief.

233 llergen challenge and endobronchial allergen provocation through a bronchoscope.

234 ergen challenges, and endobronchial allergen provocations through a bronchoscope.

235 itching and whealing after standardized skin provocation to elicit Darier's sign.

236 lprit lesion (n = 76) but with acetylcholine provocation (total n = 346).

237 s (mean age, 38 years+/-12) before and after provocation (unilateral cooling, one calf).

238 ficient recipient mice subjected to allergen provocation using established models of respiratory infl

239 small airway dysfunction during methacholine provocation was associated with a higher increase in Bor

240 lesion, in whom intracoronary acetylcholine provocation was performed, had coronary spasm.

241 increase in dyspnea during the methacholine provocation was strongly and independently correlated wi

242 VA-specific IgG, but not IgE, before the OVA provocation, was capable of inducing the EPR in both str

243 interleukin (IL)-6 cytokine responses to LPS provocation were determined.

244 rways for several weeks in response to brief provocation with A. alternata in wild-type, Gm-csf- and

245 alculated the percent decrease in FEV1 after provocation with ACh or Hist.

246 methacholine chloride and after reversal of provocation with albuterol.

247 Nasal provocation with allergen significantly increased NGF pr

248 Before provocation with allergen, administration of anti-IL-5 a

249 ) and markedly longer itch persistence after provocation with cowhage (P < 0.01).

250 Provocation with des-Arg10-lysylbradykinin did not cause

251 d 1), and cockroach (Bla g 2) allergens; and provocation with histamine to test for bronchial hyperre

252 Noncarina PV triggers more commonly require provocation with isoproterenol infusion.

253 cumented with ictal video-EEG studies during provocation with language related tasks.

254 control subjects before and after bronchial provocation with methacholine chloride and after reversa

255 Bronchial provocation with methacholine was also performed in five

256 OVA-induced airway hyperresponsiveness after provocation with methacholine.

257 d to an increase in dyspnea during bronchial provocation with methacholine.

258 By pairing symptom provocation with online avoidance responses on a trial-b

259 cases developed positive responses after re-provocation with PG/PV.

260 in nasal lavage fluids were not affected by provocation with the vehicle for allergen or with histam

261 es with >2 different NSAIDs or positive drug provocation) with either cutaneous (MNSAID-UA, n = 25) o