1 nd a nephron model revealed hot spots in the proximal renal tubules.

2 ake in opossum kidney (OK) cells, a model of proximal renal tubules.

3 allowing normal G alpha(s) expression in the proximal renal tubules.

4 the basolateral membranes of hepatocytes and proximal renal tubules.

5 t of at least 10 mutations that cause severe proximal renal tubule acidosis in humans.

6 tly localized to epithelial cells, including proximal renal tubules and biliary epithelial cells.

7 nges alone can lead to PTH resistance in the proximal renal tubules and thus lead to impaired regulat

8 biting reuptake of the fusion protein in the proximal renal tubules, and, therefore, could significan

9  pancreatic beta-cells, small intestine, and proximal renal tubule are encoded by the 12 exons of the

10 ium-dependent amino acid transporters in the proximal renal tubule, causing a reduction in amino acid

11 mone (PTH), a major physiologic regulator of proximal renal tubule cell sodium-phosphate cotransport,

12 one inhibits sodium-phosphate cotransport in proximal renal tubule cells through activation of severa

13 posited along the basolateral surface of the proximal renal tubule in association with L-fucose, the

14 ate specific expression of cadherin-6 in the proximal renal tubules in normal human kidney and sugges

15 s produce PTHrP, and PTHrP expression in rat proximal renal tubules is upregulated in response to isc

16 e conclude that parathyroid hormone inhibits proximal renal tubule sodium-phosphate cotransport throu