ライフサイエンスコーパス: pseudotuberculosis

1 (Yersinia pestis, Y. enterocolitica, and Y. pseudotuberculosis).

2 ze modified peptide-cytidylate from Yersinia pseudotuberculosis.

3 nic Escherichia coli strains and by Yersinia pseudotuberculosis.

4 the virulence of the enteropathogen Yersinia pseudotuberculosis.

5 tosis (YopJ) in macrophages infected with Y. pseudotuberculosis.

6 global set of sRNAs expressed in vitro by Y. pseudotuberculosis.

7 n at 37 degrees C in Y. pestis but not in Y. pseudotuberculosis.

8 s isolates and its evolutionary ancestor, Y. pseudotuberculosis.

9 gue, has only recently evolved from Yersinia pseudotuberculosis.

10 in serum samples of mice vaccinated with Y. pseudotuberculosis.

11 es virulence in Yersinia pestis and Yersinia pseudotuberculosis.

12 acrophage response to internalized pYV(+) Y. pseudotuberculosis.

13 la enterica serovar Typhimurium and Yersinia pseudotuberculosis.

14 hms genes; identical genes are present in Y. pseudotuberculosis.

15 ion of inv in Yersinia enterocolitica and Y. pseudotuberculosis.

16 in the intestine delayed dissemination of Y. pseudotuberculosis.

17 t beta1 integrin-promoted uptake of Yersinia pseudotuberculosis.

18 regulation between Y. enterocolitica and Y. pseudotuberculosis.

19 s and the corresponding features in Yersinia pseudotuberculosis.

20 by YmoA in Y. enterocolitica and H-NS in Y. pseudotuberculosis.

21 n T cells exposed to low numbers of Yersinia pseudotuberculosis.

22 osynthesis of 3,6-dideoxyhexoses in Yersinia pseudotuberculosis.

23 recently from the enteric pathogen Yersinia pseudotuberculosis.

24 ctor in Yersinia enterocolitica and Yersinia pseudotuberculosis.

25 is acquired since the the divergence from Y. pseudotuberculosis.

26 specific manner and only in Y. pestis and Y. pseudotuberculosis.

27 ent or highly divergent in all strains of Y. pseudotuberculosis.

28 macrophages is conserved in Y. pestis and Y. pseudotuberculosis.

29 pneumoniae, Yersinia enterocolitica, and Y. pseudotuberculosis.

30 ogen that has recently emerged from Yersinia pseudotuberculosis.

31 plague, from the closely related species Y. pseudotuberculosis.

32 clinical equine isolates of Corynebacterium pseudotuberculosis.

33 mary infection of C57BL/6 mice with Yersinia pseudotuberculosis.

34 SS in the gastrointestinal pathogen Yersinia pseudotuberculosis.

35 from the gastrointestinal pathogen Yersinia pseudotuberculosis.

36 sera of C57BL/6J mice infected with Yersinia pseudotuberculosis.

37 od- and waterborne enteric pathogen Yersinia pseudotuberculosis A combination of population genetics,

38 Phagosomes containing phoP mutant Y. pseudotuberculosis acquired cathepsin D at a higher rate

39 In this article, we demonstrate that Y. pseudotuberculosis Ail from strains PB1, 2812/79, and YP

40 These results indicate that Y. pseudotuberculosis Ail recruits C4BP in a functional man

41 Y. pestis and the closely related Y. pseudotuberculosis also block the feeding of Caenorhabdi

42 Y. pestis and the closely related Yersinia pseudotuberculosis also make biofilms on the cuticle of

43 pathogen that evolved recently from Yersinia pseudotuberculosis, an enteric pathogen transmitted via

44 tive agent of plague, diverged from Yersinia pseudotuberculosis, an enteric pathogen, an estimated 15

45 in a type III-dependent manner from Yersinia pseudotuberculosis and also secreted from C. trachomatis

46 the macrophage cytokine response to live Y. pseudotuberculosis and analyzed the susceptibility of TL

47 Enteric pathogens such as Yersinia pseudotuberculosis and enteropathogenic Escherichia coli

48 if YopE is a protective antigen for Yersinia pseudotuberculosis and if primary infection with this en

49 ing to increased systemic colonization by Y. pseudotuberculosis and potentially enhancing adaptive im

50 ession of virulence-relevant functions in Y. pseudotuberculosis and reprogramming of its metabolism i

51 vity of Cif from the human pathogen Yersinia pseudotuberculosis and selected variants, and the positi

52 esent on the chromosomes of Y. pestis and Y. pseudotuberculosis and that this secretion system is not

53 receptors and for phages infecting Yersinia pseudotuberculosis and Vibrio cholerae.

54 n recognition of the enteropathogen Yersinia pseudotuberculosis and whether this results in an immune

55 m (TTSS) found in Y. pestis is present in Y. pseudotuberculosis and whether this system is important

56 which of these phenotypes descended from Y. pseudotuberculosis and which were acquired independently

57 ecretion system effector known as YopJ in Y. pseudotuberculosis and Y. pestis and YopP in Y. enteroco

58 xpression of many sRNAs conserved between Y. pseudotuberculosis and Y. pestis differs in both timing

59 This suggests that both Y. pseudotuberculosis and Y. pestis produce an oligosacchar

60 e and absence of biofilm and on wild-type Y. pseudotuberculosis and Y. pseudotuberculosis QS mutants.

61 Previous study in Yersinia pseudotuberculosis and Yersinia enterocolitica prompted

62 pestis and pYV in enteropathogenic Yersinia pseudotuberculosis and Yersinia enterocolitica) that med

63 In Yersinia pseudotuberculosis and Yersinia enterocolitica, RovA reg

64 s and two enteropathogenic species, Yersinia pseudotuberculosis and Yersinia enterocolitica.

65 PNPase also enhances the ability of Yersinia pseudotuberculosis and Yersinia pestis to withstand the

66 red for optimal T3SS functioning in Yersinia pseudotuberculosis and Yersinia pestis.

67 binding of the biofilm produced by Yersinia pseudotuberculosis and Yersinia pestis.

68 1 during the divergence of Y. pestis from Y. pseudotuberculosis, and are the first evidence of a nove

69 herichia coli, Salmonella enterica, Yersinia pseudotuberculosis, and Vibrio cholerae, among others.

70 enic Yersinia species: Y. enterocolitica, Y. pseudotuberculosis, and Y. pestis.

71 fully conserved between Y. pestis, Yersinia pseudotuberculosis, and Yersinia enterocolitica.

72 produced similar levels of antibodies to Y. pseudotuberculosis antigens and were equally resistant t

73 Yersinia pestis and Yersinia pseudotuberculosis are closely related facultative intra

74 thogenic Listeria monocytogenes and Yersinia pseudotuberculosis as well as commensal bacteria includi

75 e response) during primary infection with Y. pseudotuberculosis, as shown by flow cytometry tetramer

76 s, we mapped the RNA structurome of Yersinia pseudotuberculosis at three different temperatures.

77 Y. pseudotuberculosis binding to cells caused robust recrui

78 n allowing adhesion of M. nematophilum or Y. pseudotuberculosis biofilm to wild type C. elegans.

79 ne CCL28 was increased by loss of rfaH in Y. pseudotuberculosis but not in Y. pestis.

80 metabolism in the enteric pathogen Yersinia pseudotuberculosis by integrated transcriptome and [(13)

81 fficient invasin-promoted uptake of Yersinia pseudotuberculosis by nonphagocytic cells.

82 plays a critical role in the virulence of Y. pseudotuberculosis by participating in the regulation of

83 immune response against enteropathogenic Y. pseudotuberculosis by promoting Th17-like responses in m

84 isen from a less virulent pathogen, Yersinia pseudotuberculosis, by a rapid evolutionary process.

85 rthermore, this lung infection model with Y. pseudotuberculosis can be used to test potential therape

86 our results suggest that the Ysc T3SS of Y. pseudotuberculosis can function within macrophage phagos

87 do not completely block phagocytosis and Y. pseudotuberculosis can replicate in macrophages, it is i

88 asal inoculation with as few as 18 CFU of Y. pseudotuberculosis caused a lethal lung infection in som

89 eens showed that infection with wild-type Y. pseudotuberculosis caused an influx of neutrophils, whil

90 Infection with wild-type Y. pseudotuberculosis caused significantly more inflammatio

91 The enteric pathogen Yersinia pseudotuberculosis causes the disease yersiniosis.

92 and PNPase can be readily copurified from Y. pseudotuberculosis cell extracts, suggests that these tw

93 S expression levels among a population of Y. pseudotuberculosis cells following the removal of Ca(2+)

94 Here we report that Y. pseudotuberculosis cells with reduced RNase E activity a

95 as protective against lethal intragastric Y. pseudotuberculosis challenge.

96 potential association site for the Yersinia pseudotuberculosis chaperone-effector pair SycE-YopE.

97 genes found in Yersinia pestis and Yersinia pseudotuberculosis compared to other Yersinia species, a

98 pestis, unlike the closely related Yersinia pseudotuberculosis, constitutively produces isocitrate l

99 ith a different bacterial pathogen, Yersinia pseudotuberculosis, contain damaged DNA.

100 (A) modified with C10 or C12 acyl groups, Y. pseudotuberculosis contained the same forms as part of a

101 ssay (type III secretion system dependent Y. pseudotuberculosis cytotoxicity assay).

102 Yersinia pseudotuberculosis delivers several Yop effectors (e.g.

103 The virulence of a Yersinia pseudotuberculosis Delta yopM mutant in mice via an intr

104 sensitivity to host defense peptides, the Y. pseudotuberculosis DeltarfaH strain was not attenuated i

105 either the IP32953 or the 32777 strain of Y. pseudotuberculosis, demonstrating that the phenotype is

106 Last, Y. pseudotuberculosis did not require Ivy to counter lysozy

107 acrophages infected with wild-type pYV(+) Y. pseudotuberculosis died of apoptosis after 20 h.

108 , the deletion of the homologous genes in Y. pseudotuberculosis does not seem to impact the virulence

109 The gram-negative enteric pathogen Yersinia pseudotuberculosis employs a type III secretion system a

110 ops have some redundant functions or that Y. pseudotuberculosis employs multiple strategies for colon

111 Results of infections with Y. pseudotuberculosis expressing catalytically inactive Yop

112 Y. pseudotuberculosis expressing wild-type YpkA, but not a

113 To determine the Y. pseudotuberculosis factors important for growth during l

114 However, both Y. pestis and Y. pseudotuberculosis form biofilms that adhere to the exte

115 ts, suggesting that YopE and YopH protect Y. pseudotuberculosis from these defenses in BALB/c mice.

116 d water-borne transmission route of Yersinia pseudotuberculosis, from which Y. pestis diverged only w

117 The closely related Yersinia pseudotuberculosis, from which Y. pestis recently evolve

118 The enteropathogen Yersinia pseudotuberculosis, from which Y. pestis recently evolve

119 tected, indicating that as many as 13% of Y. pseudotuberculosis genes no longer function in Y. pestis

120 Studies in Y. pestis and Y. pseudotuberculosis have shown that YopM suppresses infec

121 To determine how wild-type Y. pseudotuberculosis hinders yop mutant survival, yop muta

122 volved from the gastrointestinal pathogen Y. pseudotuberculosis; however, it is not known at what poi

123 In summary, infection with Y. pseudotuberculosis in intestinal and systemic sites indu

124 be correlated with the presence of viable Y. pseudotuberculosis in macrophages.

125 CDP-D-glucose 4,6-dehydratase from Yersinia pseudotuberculosis in the resting state.

126 to persist in competition with wild-type Y. pseudotuberculosis, indicating that these two infection

127 Cell extracts from Yersinia pseudotuberculosis induced multinucleation in HEp-2 cell

128 Using this tool for Yersinia pseudotuberculosis-infected lymphatic tissues, we reveal

129 enzymatic activities of YopE and YopT, in Y. pseudotuberculosis-infected macrophages.

130 hagocyte in this disease model but not in Y. pseudotuberculosis-infected mice.

131 Upon TLR-2 stimulation, Y. pseudotuberculosis-infected monocytes activated caspase-

132 h these results, IL-10 is undetectable in Y. pseudotuberculosis-infected mouse tissues until advanced

133 des and associated lymphatics after Yersinia pseudotuberculosis infection and clearance.

134 Decreases in NK cells after Y. pseudotuberculosis infection did not correlate with YopM

135 otic response of TLR2(-/-) macrophages to Y. pseudotuberculosis infection is equivalent to that of wi

136 within SLOs during gastrointestinal Yersinia pseudotuberculosis infection to limit pathogen spread wa

137 In an orogastric model of Y. pseudotuberculosis infection, a Delta yopM mutant was de

138 h lipopolysaccharide (LPS) prior to Yersinia pseudotuberculosis infection, caspase-1 is activated by

139 In a mouse model of Yersinia pseudotuberculosis infection, we show that at least thre

140 en demonstrated in a mouse model of Yersinia pseudotuberculosis infection.

141 t isolated specific features of wild-type Y. pseudotuberculosis infection.

142 robiota or animal susceptibility to Yersinia pseudotuberculosis infection.

143 Yersinia pseudotuberculosis infects many mammals and birds includ

144 nstrate that, in addition to MyD88, Yersinia pseudotuberculosis inhibits TRIF signaling through the t

145 The sterol structure dependence of Y. pseudotuberculosis internalization differed from that of

146 Because a key step in Y. pseudotuberculosis internalization is interaction of the

147 ompared the sterol dependence of wildtype Y. pseudotuberculosis internalization with that of Deltainv

148 Efficient uptake of Yersinia pseudotuberculosis into cultured mammalian cells is the

149 Efficient entry of the bacterium Yersinia pseudotuberculosis into mammalian cells requires the bin

150 lin-like (Big) domains, such as the Yersinia pseudotuberculosis invasin and Escherichia coli intimin,

151 Transfer of the Yersinia pseudotuberculosis invasin gene into E.coli DH10B asd(-)

152 Quorum sensing (QS) in Yersinia pseudotuberculosis involves two pairs of LuxRI orthologu

153 pair formation proteins (Trb) from Yersinia pseudotuberculosis IP31758 are the mostly closely relate

154 apK and yapJ to three homologous genes in Y. pseudotuberculosis IP32953 (YPTB0365, YPTB3285, and YPTB

155 e report the complete genomic sequence of Y. pseudotuberculosis IP32953 and its use for detailed geno

156 inia, we constructed DeltarfaH mutants of Y. pseudotuberculosis IP32953 and Y. pestis KIM6+.

157 Yersinia pseudotuberculosis is a foodborne pathogenic bacterium t

158 Yersinia pseudotuberculosis is a Gram-negative bacterial pathogen

159 Furthermore, the Deltahfq mutant of Y. pseudotuberculosis is hypermotile and displays increased

160 , when antiphagocytosis is incomplete and Y. pseudotuberculosis is internalized by macrophages, the b

161 gulator of biofilms that is functional in Y. pseudotuberculosis, is a pseudogene in Y. pestis.

162 [2Fe-2S] protein, E 1, cloned from Yersinia pseudotuberculosis, is the only known enzyme whose catal

163 Yersinia pseudotuberculosis lcrF-null mutants showed attenuated v

164 Deletion of multiple sRNAs in Y. pseudotuberculosis leads to attenuation of the pathogen

165 d A, and an attenuated strain producing a Y. pseudotuberculosis-like hexa-acylated lipid A.

166 Yersinia pseudotuberculosis localizes to the distal ileum, cecum,

167 Although Y. pseudotuberculosis makes biofilms in other environments,

168 t three virulence determinants encoded by Y. pseudotuberculosis manipulate the Rho GTPase RhoG.

169 test these models, we constructed a Yersinia pseudotuberculosis mutant expressing YopD devoid of its

170 As the YopD(DeltaTM) Y. pseudotuberculosis mutant formed somewhat smaller pores

171 to promote cytoskeletal disruption, and a Y. pseudotuberculosis mutant lacking YpkA GDI activity show

172 Here, we constructed a Yersinia pseudotuberculosis mutant strain with arabinose-dependen

173 Yersinia pseudotuberculosis mutants deficient for the adhesins in

174 In addition, Y. pseudotuberculosis mutants expressing YopM proteins unab

175 nature-tagged mutagenesis, we isolated 13 Y. pseudotuberculosis mutants that failed to survive in the

176 Yersinia pseudotuberculosis mutants that overproduce the DNA aden

177 Yersinia pseudotuberculosis mutants that overproduce the DNA aden

178 Finally, competition infections with Y. pseudotuberculosis mutants with various abilities to ind

179 22 strains of Y. pestis and 10 strains of Y. pseudotuberculosis of diverse origin.

180 also indicates that biofilm formation by Y. pseudotuberculosis on C. elegans is an interactive proce

181 regulator, FlhDC, in biofilm formation by Y. pseudotuberculosis on C. elegans.

182 fects of spleen and liver colonization by Y. pseudotuberculosis on yop mutants in the intestines.

183 in macrophages is common to Y. pestis and Y. pseudotuberculosis or is a unique phenotype of Y. pestis

184 hat only a small fraction of either Yersinia pseudotuberculosis or Yersinia pestis bacteria express t

185 city induced by Yersinia pestis and Yersinia pseudotuberculosis paradoxically leads to decreased viru

186 Here the importance of PhoP for Y. pseudotuberculosis pathogenesis was investigated.

187 lication in macrophages are important for Y. pseudotuberculosis pathogenesis.

188 A Y. pseudotuberculosis phoP mutant was 100-fold less virulen

189 Y. pseudotuberculosis phoP mutants died at a low rate in ma

190 Y. pseudotuberculosis phoP mutants were unable to replicate

191 In a murine model of Y. pseudotuberculosis pneumonia, two compounds significantl

192 These changes from the Y. pseudotuberculosis progenitor included loss of insectici

193 a model in which a genetic change in the Y. pseudotuberculosis progenitor of Y. pestis extended its

194 ing those phylogenetically closest to the Y. pseudotuberculosis progenitor, contain a mutated ureD al

195 (rovA(Yent)) promoter is weaker than the Y. pseudotuberculosis promoter.

196 nd on wild-type Y. pseudotuberculosis and Y. pseudotuberculosis QS mutants.

197 ent of the pseudogene with the functional Y. pseudotuberculosis rcsA allele strongly represses biofil

198 versely, deletion of the urease operon in Y. pseudotuberculosis rendered it nontoxic.

199 Over the course of 7 days, wild-type Y. pseudotuberculosis replicated to nearly 1 x 10(8) CFU/g

200 iofilm on Caenorhabditis elegans by Yersinia pseudotuberculosis represents a tractable model for inve

201 Virulence in Y. pseudotuberculosis requires the plasmid-encoded Ysc type

202 ested that rfaH may be required for Yersinia pseudotuberculosis resistance to antimicrobial chemokine

203 uring C. trachomatis infections, in Yersinia pseudotuberculosis resulted in its secretion via the Yer

204 Disruption of the ail gene in Y. pseudotuberculosis resulted in loss of C4BP binding.

205 nfection of C. elegans with the wild-type Y. pseudotuberculosis resulted in the differential regulati

206 We report that oral infection with Yersinia pseudotuberculosis results in the development of two dis

207 Transcriptomic analysis of Y. pseudotuberculosis revealed that genes located in both o

208 s similar to recently published data from Y. pseudotuberculosis, revealing a potentially conserved me

209 During aerobic growth on glucose, Y. pseudotuberculosis reveals an unusual flux distribution

210 In Y. pseudotuberculosis, rovA transcription is controlled pri

211 he gram-negative bacterial pathogen Yersinia pseudotuberculosis secretes into macrophages a protease

212 no consensus in the literature on whether Y. pseudotuberculosis shares this property.

213 nism(s) of complement resistance in Yersinia pseudotuberculosis showed that the outer membrane protei

214 Six sRNAs are Y. pseudotuberculosis specific and are absent from the geno

215 ed from an ancestral enteroinvasive Yersinia pseudotuberculosis strain by gene loss and acquisition o

216 that it is possible to use an attenuated Y. pseudotuberculosis strain delivering the LcrV antigen vi

217 is strains (EV766 and KIM10(+)) and three Y. pseudotuberculosis strains (IP2790c, IP2515c, and IP2666

218 Y. pseudotuberculosis strains also varied greatly in their

219 nterocolitica strains and 2 (of 10) Yersinia pseudotuberculosis strains at the restrictive temperatur

220 Yersinia pseudotuberculosis strains expressing the mutant YopB pr

221 phoP(+) Y. pseudotuberculosis strains initiated replication in macr

222 A number of Y. pestis and Y. pseudotuberculosis strains of different biovars and sero

223 crophages compared to other Y. pestis and Y. pseudotuberculosis strains tested.

224 s possibility was investigated here using Y. pseudotuberculosis strains that express YopJ or YopH und

225 g these proteins were infected with Yersinia pseudotuberculosis strains that secrete functionally act

226 el of different Yersinia pestis and Yersinia pseudotuberculosis strains to determine whether Yops of

227 V-cured, pYV(+) wild-type, and yop mutant Y. pseudotuberculosis strains were allowed to infect bone m

228 HeLa cells infected with wild-type or Yop-Y. pseudotuberculosis strains were assayed for interleukin

229 Five of 18 Y. pseudotuberculosis strains, encompassing seven serotypes

230 ions were specific to a limited number of Y. pseudotuberculosis strains, including the high pathogeni

231 ous sequences from numerous Y. pestis and Y. pseudotuberculosis strains, we determined that these aut

232 ophages infected with wild-type or mutant Y. pseudotuberculosis strains.

233 ons had high levels of divergence between Y. pseudotuberculosis strains.

234 travenously with wild-type or yopM mutant Y. pseudotuberculosis strains.

235 Therefore, Y. pseudotuberculosis subverts intestinal barrier function

236 ling of macrophages infected ex vivo with Y. pseudotuberculosis, suggesting a mechanism by which this

237 ery of IcsA in Escherichia coli and Yersinia pseudotuberculosis, suggesting that the mechanism for po

238 terium and host following phagocytosis of Y. pseudotuberculosis suggests new roles for the T3SS in Ye

239 The Yersinia pseudotuberculosis surface protein invasin binds to mult

240 Following clearance of Yersinia pseudotuberculosis, sustained inflammation and associate

241 aling potential virulence determinants in Y. pseudotuberculosis that are regulated in a posttranscrip

242 racterized a glycosyl hydrolase (NghA) of Y. pseudotuberculosis that cleaved beta-linked N-acetylgluc

243 , serotypes, and strains of Y. pestis and Y. pseudotuberculosis that may relate to the evolution of t

244 xpressing invA, a gene product from Yersinia pseudotuberculosis that mediates cellular invasion, also

245 Yersinia pseudotuberculosis, the closely related food-and water-b

246 In competition infections with wild-type Y. pseudotuberculosis, the presence of wild-type bacteria s

247 estis, the agent of plague in humans, and Y. pseudotuberculosis, the related enteric pathogen, delive

248 Yersinia pseudotuberculosis, the relatively recent progenitor of

249 Yersinia pestis evolved from Y. pseudotuberculosis to become the causative agent of bubo

250 Here we show that Hfq is required by Y. pseudotuberculosis to cause mortality in an intragastric

251 ematic deletion analysis of YopM in Yersinia pseudotuberculosis to determine which regions are requir

252 nthesizing the invasin protein from Yersinia pseudotuberculosis to enhance cellular entry were able t

253 We compared the ability of Y. pestis and Y. pseudotuberculosis to infect the rat flea Xenopsylla che

254 intimately connected with the ability of Y. pseudotuberculosis to successfully establish replication

255 the T3SS can be utilized by intracellular Y. pseudotuberculosis to translocate Yops.

256 inhibited T3SS-dependent cytotoxicity of Y. pseudotuberculosis toward macrophages in vitro.

257 Y. pseudotuberculosis translocated to organs such as the li

258 Random screening of Y. pseudotuberculosis transposon insertion mutants with a C

259 YscU is a Yersinia pseudotuberculosis type III secretion system protein cru

260 how host cell sterol composition affected Y. pseudotuberculosis uptake.

261 Yersinia pseudotuberculosis uses a plasmid (pYV)-encoded type III

262 Yersinia pseudotuberculosis uses a type III secretion system (T3S

263 ough the closely related enteric pathogen Y. pseudotuberculosis uses quorum sensing system to regulat

264 tified by in vitro screening, using Yersinia pseudotuberculosis Using a mouse model of P. aeruginosa

265 tose biosynthetic gene cluster from Yersinia pseudotuberculosis VI.

266 s aeruginosa, Erwinia chrysanthemi, Yersinia pseudotuberculosis, Vibrio cholerae (30-70% sequence ide

267 ate the misregulation of RhoG by multiple Y. pseudotuberculosis virulence determinants.

268 nt mechanism by which YopM contributes to Y. pseudotuberculosis virulence.

269 ion of Y. pestis from the ancestral Yersinia pseudotuberculosis was a significant reduction in the co

270 lytically inactive yopJ mutant strains of Y. pseudotuberculosis was developed to further investigate

271 gosomes containing phoP(+) or phoP mutant Y. pseudotuberculosis was studied by using immunofluorescen

272 In mice infected with Yersinia pseudotuberculosis, we found that PP barrier dysfunction

273 Yop- mutants of Y.pseudotuberculosis were assayed for pore-forming activit

274 B, YopD, and YopE (BDE) secreted by Yersinia pseudotuberculosis were purified by affinity chromatogra

275 2001, 89 culture-confirmed cases of Yersinia pseudotuberculosis were reported in Finland; 55 (62%) we

276 plague bacillus) and its ancestor, Yersinia pseudotuberculosis (which causes self-limited bowel dise

277 biofilms produced by Yersinia pestis and Y. pseudotuberculosis, which bind the C. elegans surface pr

278 RovA in both Y. enterocolitica and Yersinia pseudotuberculosis while negatively regulated by YmoA in

279 ld in TLR4(-/-) macrophages infected with Y. pseudotuberculosis, while the apoptotic response of TLR2

280 B3286), we show that yapK is conserved in Y. pseudotuberculosis, while yapJ is unique to Y. pestis.

281 acrophages harboring intracellular pYV(+) Y. pseudotuberculosis with chloramphenicol reduced apoptosi

282 ent of both fH and C4BP by Ail may confer Y. pseudotuberculosis with the ability to resist all pathwa

283 Dissemination of Yersinia pseudotuberculosis within mice after oral inoculation wa

284 of pYV(+) but not pYV-cured or DeltayopB Y. pseudotuberculosis within phagosomes: only a small fract

285 , evolved from the enteric pathogen Yersinia pseudotuberculosis within the past 20,000 years.

286 inia, divergence of Y. enterocolitica and Y. pseudotuberculosis/Y. pestis during evolution has result

287 d RovA bind with a higher affinity to the Y. pseudotuberculosis/Y. pestis rovA (rovA(Ypstb/Ypestis))

288 leavage is specific for the Y. pestis and Y. pseudotuberculosis YapE orthologues but is not conserved

289 rsinia pestis, which causes plague, Yersinia pseudotuberculosis, Yersinia enterocolitica.

290 contribution of this region by generating Y. pseudotuberculosis yopD(Delta150-170) and yopD(Delta207-

291 After oral inoculation of mice, Yersinia pseudotuberculosis yopE and yopH mutants colonize the in

292 from the cell by the introduction of the Y. pseudotuberculosis YopE RhoGAP protein could be bypassed

293 ed to show efficient RhoG inactivation by Y. pseudotuberculosis YopE, a potent Rho GTPase activating

294 al domain (residues 1-129) from the Yersinia pseudotuberculosis YopH (YopH-NT) in complex with N-acet

295 pseudotuberculosis yopJ mutant was as virulent as the wi

296 pseudotuberculosis yopM mutant was compared to that of a

297 xes from Escherichia coli EC869 and Yersinia pseudotuberculosis YPIII to explore the evolution of CDI

298 Only one of six strains tested, the Y. pseudotuberculosis YPIII(p(-)) strain, was defective for

299 and prepared lpp gene deletion mutants of Y. pseudotuberculosis YPIII, Y. pestis KIM/D27 (pigmentatio

300 de generated by auto-proteolysis of Yersinia pseudotuberculosis YscU, is secreted by the T3SS when ba