ライフサイエンスコーパス: psychogenic

1 categorized as 'atypical', 'idiopathic' or 'psychogenic'.

2 epticus such as nonconvulsive, autonomic and psychogenic.

3 ement disorders may still be misdiagnosed as psychogenic.

4 tantaneous frequencies is evidence against a psychogenic aetiology of tremor.

5 ecords and neuropsychological findings in 53 psychogenic amnesia cases (ratio of 3:1, males:females),

6 (vi) Does psychogenic amnesia involve the same mechanisms as organ

7 Consequently, the literature on psychogenic amnesia is somewhat fragmented and offers li

8 ms of Markowitsch's and Kopelman's models of psychogenic amnesia, and with respect to Anderson's neur

9 In conclusion, the outcome in psychogenic amnesia, particularly those characterized by

10 iates the endocrine and anxiety responses to psychogenic and interoceptive stress.

11 This suggests that psychogenic and organic dystonia have different cortical

12 eal-life ambulatory setting in patients with psychogenic and organic tremor by actigraphy, and compar

13 of men with erectile dysfunction of organic, psychogenic, and mixed causes.

14 ile dysfunction of vasculogenic, neurogenic, psychogenic, and mixed causes.

15 tors, such as palatal tremor, may occur on a psychogenic basis.

16 head injury was actually more common in our psychogenic cases (P = 0.012), perhaps reflecting a 'lea

17 Biological and psychogenic causes contribute to a multifactorial model

18 it remained unknown how chronic exposure to psychogenic challenges affects excitatory and inhibitory

19 ful for distinguishing between epileptic and psychogenic convulsive seizures.

20 onclusive features of somatoform disorder or psychogenic disorder can be found and, in these patients

21 isorders and, if unrecognised as a sign of a psychogenic disorder, it may lead to unnecessary and occ

22 graine aura, post-hallucinogen flashback, or psychogenic disorder.

23 %) controls (p=0.049 when psychogenic vs non-psychogenic disorders or controls were compared).

24 rganic damage but associated with emotional "psychogenic" disturbances.

25 ss and may be misdiagnosed or malingering or psychogenic dizziness because they often present with co

26 zziness; a refinement of earlier concepts of psychogenic dizziness, phobic postural vertigo, and spac

27 s, and medical illnesses that masquerade as 'psychogenic' dizziness are examined.

28 e conceptualization to the common notion of 'psychogenic' dizziness is presented.

29 h organic dystonia to that in a patient with psychogenic dystonia (Psyd) and in healthy waking monkey

30 in functional neuroimaging of patients with psychogenic dystonia and genetically determined dystonia

31 In contrast, the psychogenic dystonia group showed the opposite pattern,

32 l cortex that was common to both organic and psychogenic dystonia groups (compared with control subje

33 cortical differentiation between organic and psychogenic dystonia in terms of regional blood flow, bo

34 Comparing organic dystonia with psychogenic dystonia revealed significantly greater regi

35 od flow in the primary motor cortex, whereas psychogenic dystonia was associated with significantly g

36 efrontal cortex, but there are no studies on psychogenic dystonia, and comparisons with findings in o

37 tudied patients did not have any evidence of psychogenic dystonia, and detailed investigation failed

38 il strict criteria for a somatoform disorder/psychogenic dystonia.

39 n was confirmed in, but was not specific to, psychogenic dystonia.

40 ria for documented or clinically established psychogenic dystonia; 29% fulfilled DSM-IV (Diagnostic a

41 ii) fugue-to-focal retrograde amnesia; (iii) psychogenic focal retrograde amnesia following a minor n

42 owever, some tests reported to differentiate psychogenic from neurological movement disorders may hav

43 nse of personal identity was confined to the psychogenic group.

44 iographical memory loss differed between the psychogenic groups: fugue cases showed a severe and unif

45 In this context, the term 'psychogenic' has been replaced by 'functional' movement

46 buted to toxic exposure had features of mass psychogenic illness - notably, widespread subjective sym

47 Mass psychogenic illness may be difficult to differentiate fr

48 f the anxiety surrounding an episode of mass psychogenic illness requires prompt recognition and a de

49 this central "itch matrix" may give rise to psychogenic itch disorders.

50 ght in to a genuinely experienced functional/psychogenic leg paralysis following injury.

51 very few case series of patients with acute psychogenic memory loss (also known as dissociative/func

52 We found that our patients with psychogenic memory loss fell into four distinct groups,

53 The prognosis of functional (or psychogenic) motor symptoms (weakness and movement disor

54 Functional (psychogenic) motor symptoms are commonly encountered in

55 Psychogenic movement disorder continues to be a difficul

56 nt disorders continues, but a diagnosis of a psychogenic movement disorder should be actively sought

57 reatment-induced dyskinesias, can be seen in psychogenic movement disorder, and abnormal movements th

58 uld always be screened for the presence of a psychogenic movement disorder.

59 Psychogenic movement disorders (PMDs) may be difficult t

60 Diagnosis and treatment of psychogenic movement disorders are challenging for both

61 Probably more commonly, however, psychogenic movement disorders are underdiagnosed.

62 nic movement disorders cases, 4/11 (36%) non-psychogenic movement disorders cases and 4/12 (33%) cont

63 Convergence spasm was present in 9/13 (69%) psychogenic movement disorders cases, 4/11 (36%) non-psy

64 The few clinical trials specific to psychogenic movement disorders focus on antidepressants

65 ovide benefit in the clinical examination of psychogenic movement disorders patients.

66 The diagnostic criteria for psychogenic movement disorders provide a degree of diagn

67 The neurobiological basis of psychogenic movement disorders remains poorly understood

68 ogic sign, 36 subjects were studied, 13 with psychogenic movement disorders, 11 with organic movement

69 d laboratory findings that may help diagnose psychogenic movement disorders, and the ongoing controve

70 s can demonstrate characteristic features of psychogenic movement disorders, such as entrainment or s

71 Functional (psychogenic) movement disorders (FMD) are part of the wi

72 , neuroimaging and management of functional (psychogenic) movement disorders (FMD).

73 the neurobiological mechanism of functional (psychogenic) movement disorders (FMDs).

74 Patients with psychogenic (n = 6) or organic (n = 5, DYT1 gene mutatio

75 Functional (psychogenic) neurological symptoms are frequently encoun

76 t work on the diagnosis and investigation of psychogenic non-epileptic attacks, drop attacks, falls,

77 Some evidence suggests that psychogenic non-epileptic seizures (PNES) are associated

78 Psychogenic non-epileptic seizures (PNES) are classified

79 ily doctors of 260 consecutive patients with psychogenic non-epileptic seizures (PNES), 5-10 years af

80 ave been few studies of long-term outcome in psychogenic non-epileptic seizures (PNES), and none of l

81 s involved in the treatment of patients with psychogenic non-epileptic seizures and generates ideas f

82 While the diagnostic features of psychogenic non-epileptic seizures have been better char

83 nd economic burdens from diagnostic delay of psychogenic nonepileptic seizures (PNES) requires prompt

84 rsion disorder, seizures type, also known as psychogenic nonepileptic seizures (PNES).

85 Our knowledge of longer term outcome in psychogenic nonepileptic seizures (PNESs) patients is li

86 erentiation between convulsive epileptic and psychogenic nonepileptic seizures (PNESs).

87 s the recent studies assessing patients with psychogenic nonepileptic seizures and developments in tr

88 Psychogenic nonepileptic seizures, the most common conve

89 ncrease in the number of publications about (psychogenic) nonepileptic seizures (NES) over the past t

90 Other names include psychogenic or 'non-organic' paralysis.

91 clues as to whether the underlying cause is psychogenic or organic.

92 ements that would not normally be considered psychogenic or produced by psychological factors, such a

93 t that confirms the diagnosis and excludes a psychogenic or voluntary torticollis in individual patie

94 ymptoms that have been called 'hysterical', 'psychogenic' or 'medically unexplained', which we will c

95 ile dysfunction of neurogenic, vasculogenic, psychogenic, or mixed causes, respectively.

96 ary skin conditions, as well as neuropathic, psychogenic, or systemic disorders like chronic liver di

97 .03-1.24]), migraine (1.34 [1.02-1.77]), and psychogenic pain (1.58 [1.11-2.26]).

98 dache or tension headache, fibromyalgia, and psychogenic pain) and subsequent suicide death (assessed

99 -1.45] for back pain to 2.61 [1.82-3.74] for psychogenic pain).

100 oses, in particular back pain, migraine, and psychogenic pain.

101 ), dissociative motor disorder (ICD-10) and 'psychogenic' paralysis.

102 arkinson's disease', 'Paradoxical kinesia', 'Psychogenic parkinsonism', 'Functional somatic syndromes

103 Psychogenic patients reported significantly more of the

104 ounter the duality of the somatogenic versus psychogenic perspective and suggest a more integrated as

105 younger than 18 years, anoxic/myoclonic SE, psychogenic SE, simple partial SE, and absence SE.

106 pain (OR, 2.20; 95% CI, 1.54-3.15; 1 study), psychogenic seizures (OR, 2.96; 95% CI, 1.12-4.69, I(2)

107 We show in rodents that repeated psychogenic stress (predator) induces long-lasting sensi

108 Psychogenic stress may be associated with the developmen

109 l restraint, a commonly used rodent model of psychogenic stress.

110 scular, and behavioral responses to an acute psychogenic stress.

111 ough c-Fos was increased after exposure to a psychogenic stressor (i.e., open field).

112 of exposure to predator odor stress (POS), a psychogenic stressor, on the concurrent efflux of four m

113 the nervous system to decrease reactivity to psychogenic stressors and facilitate social behavior, wh

114 lesions enhance corticosterone responses to psychogenic stressors via trans-synaptic influences on p

115 tary-adrenal (HPA) axis is hyporesponsive to psychogenic stressors.

116 Two patients developed psychogenic symptoms during the head-up tilt test.

117 We studied 9 patients (five females) with psychogenic tics representing 4.9% of all 184 patients f

118 pecific feature distinguishing dystonic from psychogenic torticollis.

119 sease (n = 20), essential tremor (n = 8) and psychogenic tremor (n = 7).

120 Eight patients with psychogenic tremor and all patients with organic tremor

121 Ten participants with psychogenic tremor and eight with organic tremor, diagno

122 These data demonstrate that patients with psychogenic tremor fail to accurately perceive that they

123 Psychogenic tremor involved fewer limbs and fewer limb s

124 st that maintenance of phasic contraction in psychogenic tremor is not due to intrinsic instability o

125 ay have incomplete specificity; for example, psychogenic tremor may not always be associated with com

126 actigraphy recordings, whereas patients with psychogenic tremor reported 65% more tremor than actigra

127 essential tremor subjects but in none of the psychogenic tremor subjects.

128 Psychogenic tremor was not maintained while tapping with

129 rders, such as entrainment or suppression of psychogenic tremor with contralateral hand movements.

130 tial tremor and in none of the subjects with psychogenic tremor.

131 framework for understanding the mechanism of psychogenic tremor.

132 t report describes seven elite athletes with psychogenic vocal cord dysfunction who presented with ac

133 cases and 4/12 (33%) controls (p=0.049 when psychogenic vs non-psychogenic disorders or controls wer