ライフサイエンスコーパス: psychomotor

1 tested in the MES (mice, ip), MES (rat, po), psychomotor 6 Hz (32 mA) (mice, ip), and hippocampal kin

2 the maximal electroshock (MES) test and the psychomotor 6 Hz (32 mA) seizure models.

3 en surgical residents (PGY 1-4) had baseline psychomotor abilities assessed, then were randomized to

4 cuits, representing emotional, cognitive and psychomotor abnormalities, in the pathophysiology of ear

5 equence, Hipk2(-/-) mutants show an array of psychomotor abnormalities.

6 ctures known to be involved in mediating the psychomotor-activating and incentive motivational effect

7 t engagement of the STN serves to dampen the psychomotor-activating and incentive motivational effect

8 ns produced a dose-dependent increase in the psychomotor-activating effects of cocaine, the rate at w

9 of Cdk5 in the adult forebrain increased the psychomotor-activating effects of cocaine.

10 Psychomotor activation encoding was expressed predominan

11 Among individual moderators, psychomotor activation had the largest moderator effect

12 e propose that endocannabinoid modulation of psychomotor activation is preferentially driven by CB1 r

13 droxydopamine lesion was used as an index of psychomotor activation, and in a second experiment, loco

14 ergic neurons does not alter cocaine-induced psychomotor activation, behavioral sensitization, or con

15 g-associated environment elicits conditioned psychomotor activation, which may be weakened following

16 abinoid signaling in methamphetamine-induced psychomotor activation.

17 eceptor signaling, thus leading to increased psychomotor activation.

18 rikingly, cocaine- and phencyclidine-induced psychomotor activities were enhanced in st-A(2A)R KO but

19 role of extrastriatal A(2A)Rs in modulating psychomotor activity is largely unexplored because of th

20 ine conditioned place preference and cocaine psychomotor activity while inducing depressive-like beha

21 n providing a prominent excitatory effect on psychomotor activity.

22 nifestations of this circuit abnormality are psychomotor agitation and stereotypical behaviors, which

23 ity, flight of ideas or racing thoughts, and psychomotor agitation.

24 lockade of GluR1/2 endocytosis prevented the psychomotor and cognitive phenotypes in Gfa2-A2AR KO mic

25 naling and thereby may influence some of the psychomotor and cognitive processes associated with schi

26 ., anxiety, psychosis, impulsivity, elevated psychomotor and cognitive processing speed, rather than

27 from those without and correlated best with psychomotor and executive function tests.

28 and cognitive performance measures included psychomotor and memory tasks and a standing balance task

29 Psychomotor and mental development indexes of the Bayley

30 There was improvement in growth and psychomotor and mental developmental scores after transp

31 drugs produces persistent increases in their psychomotor and physiological effects (sensitization), a

32 in Ras activity, thereby sensitizing mice to psychomotor and rewarding effects of morphine.

33 ually all drugs of abuse and regulates their psychomotor and rewarding effects.

34 leads to a marked increase in sensitivity to psychomotor and rewarding properties of amphetamine.

35 ttention/executive, visuospatial, motor, and psychomotor, and adjusted to each individual's best leve

36 maintenance on the reinforcing, subjective, psychomotor, and cardiovascular effects of active and in

37 The distinct neurocognitive, psychomotor, and mood disturbances observed in melanchol

38 Among children, iron may improve cognitive, psychomotor, and physical development, but the evidence

39 nds with an evolution of distinct cognitive, psychomotor, and social disturbances as the mice age.

40 ing dopamine concentrations in vivo and with psychomotor behavior in freely moving rats following the

41 2A)Rs in extrastriatal neurons in modulating psychomotor behavior using newly developed striatum-spec

42 nge and alterations in cognitive content and psychomotor behavior.

43 ay play important roles in the modulation of psychomotor behaviors, anxiety, depression, and pain sen

44 ffects of cannabinoids include disruption of psychomotor behaviour, short-term memory impairment, int

45 mental (beta = 0.1, 95% CI: -0.68, 0.88) or psychomotor (beta = -0.05, 95% CI: -0.79, 0.68) developm

46 greater emphasis to cognitive, physical, and psychomotor changes, and less to neurovegetative symptom

47 he rostral striatum and that is critical for psychomotor control.

48 refractory febrile and afebrile seizures and psychomotor decline.

49 -IIc, a rare human disorder characterized by psychomotor defects, developmental abnormalities, and le

50 f neurodevelopmental milestones and acquired psychomotor deficits.

51 llum, affected individuals had microcephaly, psychomotor delay, and ataxia.

52 d developmental delay, severe ID, no speech, psychomotor delay, and postnatal microcephaly.

53 rder of the CNS, characterized by nystagmus, psychomotor delay, progressive spasticity and cerebellar

54 ting with early-onset generalized hypotonia, psychomotor delay, refractory epilepsy, and elevated lac

55 had exercise intolerance, weakness, and mild psychomotor delay.

56 the 7 patients had learning difficulties and psychomotor delay.

57 th cerebellar vermis hypoplasia, ataxia, and psychomotor delay.

58 ngenital hypotonia early-onset glaucoma, and psychomotor delays.

59 racterized by high loadings on cognitive and psychomotor depressive symptoms.

60 [95% CI, -1.23 to -.22]; P = .01) and lower psychomotor development (Bayley test score difference: -

61 An increased risk of delayed psychomotor development (psychomotor scale score <85) wa

62 osages of FA supplements during pregnancy on psychomotor development after the first year of life has

63 tween prenatal exposure to total mercury and psychomotor development among female infants (beta = -1.

64 To measure children's mental and psychomotor development at 8 months of age, the authors

65 ay have a negative effect on both mental and psychomotor development in a maternal-birth cohort from

66 eatment prevents mortality and allows normal psychomotor development in patients with severe MTHFR de

67 In addition, psychomotor development in surviving patients treated wi

68 I were more likely than others to have a low Psychomotor Development Index (adjusted odds ratio = 1.7

69 mental development index (ORs, 1.3-1.6) and psychomotor development index (ORs, 1.5-2.4), and vision

70 lopmental outcome was assessed by use of the Psychomotor Development Index (PDI) and Mental Developme

71 age, the Mental Development Index (MDI) and Psychomotor Development Index (PDI) were measured using

72 icated by rSo(2), are associated with 1-year Psychomotor Development Index and brain magnetic resonan

73 s during supplementation and a higher Bayley Psychomotor Development Index at 30 mo of age but result

74 analyses that adjusted for age <or=30 days, Psychomotor Development Index score (P=0.02) and brain h

75 Lower Psychomotor Development Index scores were modestly assoc

76 es were motor development (assessed with the Psychomotor Development Index), cerebral palsy, hearing

77 In contrast, the Bayley Psychomotor Development Index, but not the Mental Develo

78 5 years, respectively, producing mental and psychomotor development indexes and verbal and performan

79 s in maternal prenatal urine with mental and psychomotor development indices (MDI/PDI) and evaluated

80 gnificantly associated with Bayley Mental or Psychomotor Development Indices at 1-3 years, but a 10-f

81 ation between iodine supplementation and the psychomotor development of infants in a birth cohort fro

82 95% confidence interval (CI): -2.18, -0.37]; psychomotor development score, beta = -0.92 (95% CI: -1.

83 mental development and 24- through 36-month psychomotor development scores were steeper for children

84 ties, subtle neurologic symptoms, or delayed psychomotor development were assessed for cobalamin stat

85 as the effect of treatment (survival and/or psychomotor development).

86 ve microcephaly, visual impairment, stagnant psychomotor development, abnormal extrapyramidal movemen

87 improvement with remarkable achievements in psychomotor development, along with dramatic reversion o

88 yielded inconsistent findings with regard to psychomotor development, negative findings with regard t

89 astic paraplegia, with normal or near-normal psychomotor development, preserved walking capability th

90 reduction in seizure frequency and improved psychomotor development.

91 etween 2,4,4'-trichlorobiphenyl exposure and psychomotor development.

92 gnancy is associated with adverse effects on psychomotor development.

93 -month-old-girl with microcephaly and proper psychomotor development.

94 features, including facial dysmorphologies, psychomotor developmental delays recognized since early

95 ons were found between MeHg and PUFAs on the Psychomotor Developmental Index (PDI) of the BSID-II.

96 scores on the Mental Developmental Index and Psychomotor Developmental Index of the Bayley Scales of

97 scores of the Mental Developmental Index and Psychomotor Developmental Index of the Bayley Scales of

98 nfant Development, Second Edition-Mental and Psychomotor Developmental indices were administered at a

99 ofacial anomalies, variable intellectual and psychomotor disability, and variable cardiac defects wit

100 phaly, craniofacial dysmorphism, progressive psychomotor disability, hyperkinetic movements, and axia

101 activity has been implicated in a number of psychomotor disorders, including Parkinson's disease (PD

102 rcuits implicated in an astonishing array of psychomotor disorders, including Parkinson's disease, sc

103 odevelopmental disorder featured by striking psychomotor dysfunction.

104 d mediate, at least in part, cocaine-induced psychomotor effects in vivo.

105 ypic counts, and exaggerated response to the psychomotor effects of amphetamine (AMPH).

106 The psychomotor effects of cocaine are mediated by dopamine

107 hyposensitivity, or hypersensitivity to the psychomotor effects of cocaine, altered striatal signali

108 The reinforcing and psychomotor effects of morphine involve opiate stimulati

109 Amphetamine sped task performance, but its psychomotor effects were not strongly related to its eff

110 ore treatment using 13 computerized tests of psychomotor, executive, memory-attention, processing spe

111 lirium, 88% of whom had hypoactive or normal psychomotor features.

112 Psychomotor findings are imperative in examination of cr

113 (P < .001), and diminished improvement in NP psychomotor function (P = .05).

114 CaMKIIalpha modulates dopamine signaling and psychomotor function in an activity-dependent manner.

115 the striatum preserved dopamine content and psychomotor function in both males and females.

116 Psychomotor function is most affected during acute intox

117 of common objects presented at baseline, and psychomotor function via the perceptual speed test (PST)

118 onic active cannabis use in humans may alter psychomotor function, brain activation, and hypothalamic

119 is associated with slower and less efficient psychomotor function, especially in male users, as indic

120 ive battery including tasks of attention and psychomotor function, verbal episodic memory, and spatia

121 rea and from hospital as well as recovery of psychomotor function.

122 modulation of striatal dopamine content and psychomotor function.

123 in the domains of verbal memory (P <.01) and psychomotor functioning (P <.03).

124 ry, hematology, coagulation, and urinalysis; psychomotor functioning (using the Wayne Saccadic Fixato

125 Other reports suggest negative effects on psychomotor functions such as driving, but no clear evid

126 Dopamine shapes a wide variety of psychomotor functions.

127 inical AEs (1 subject with drug-related [DR] psychomotor hyperactivity and insomnia); 16 subjects wit

128 , or monogenic syndromes with involvement of psychomotor impairment (PEX14, LRPPRC and KANSL1).

129 ant that can cause psychotic, cognitive, and psychomotor impairment in humans.

130 No evidence of opioid intoxication or psychomotor impairment was observed.

131 provided significant protection in the 6-Hz psychomotor model of pharmacoresistant epilepsy in mice.

132 nsions that index genetic risk for cognitive/psychomotor, mood, and neurovegetative symptoms.

133 r chronic dosing and was not associated with psychomotor or cognitive effects.

134 omarkers for folate and cobalamin status and psychomotor or mental development scores on the day of b

135 tions between manganese and mental (MDI) and psychomotor (PDI) development indices were estimated in

136 ill performance using a previously validated psychomotor performance test (maximum voluntary contract

137 For psychomotor performance testing, subjects with carpal tu

138 Clinical psychomotor performance was assessed using the Minimally

139 no improvement was seen in tests of clinical psychomotor performance.

140 Fatigue and MSDs impact psychomotor performance; therefore, these results warran

141 deling of excitatory synapses and persistent psychomotor plasticity in response to AMPH.

142 ological tests, particularly those measuring psychomotor processing speed and executive functioning.

143 In contrast, visual memory and psychomotor processing speed were between the borderline

144 desensitization affects the reinforcing and psychomotor properties of morphine has remained unexplor

145 -MGA-uria characterized by cataracts, severe psychomotor regression during febrile episodes, epilepsy

146 el displays high face validity for modelling psychomotor regression of a learned skill, a deficit tha

147 mild inflammation associated with changes in psychomotor responding, and suggest that inflammation-in

148 res of schizophrenia, namely enhanced MK-801 psychomotor response (positive symptoms) and decreased w

149 Gfa2-A2AR KO mice exhibited enhanced MK-801 psychomotor response and decreased working memory; this

150 changes in mesofrontal circuit activity and psychomotor response in adolescent mice than in adult mi

151 ion speed, information processing speed, and psychomotor response speed.

152 nosine A2A receptor links adenosine tone and psychomotor response to amphetamine, an indicator of dop

153 he ulnar nerve under HT7 acupoint suppressed psychomotor response to cocaine, which was abolished by

154 ansmitted via the DC pathway can inhibit the psychomotor response to cocaine.

155 ng periodontal procedures on patient recall, psychomotor response, oxygen saturation, and hemodynamic

156 was independent of actions on motivation or psychomotor response.

157 In experiment 1, the thermoregulatory and psychomotor responses produced by METH at 27 degrees C w

158 s associated with faster and more consistent psychomotor responses to visual stimulation.

159 ions of NALCN cause infantile hypotonia with psychomotor retardation and characteristic facies (IHPRF

160 ures, acquired microcephaly, hypertonia, and psychomotor retardation and died at age 7 mo despite sup

161 The rate of both psychomotor retardation and hearing deficit decreased wi

162 e lysosomal storage disease characterized by psychomotor retardation and ophthalmological abnormaliti

163 years who had either hearing deficit and/or psychomotor retardation and whose mothers had a confirme

164 They also present psychomotor retardation as well as increased emotional r

165 ressive disorder, and increased frequency of psychomotor retardation as well as other melancholic sym

166 n-Herndon-Dudley syndrome (AHDS) is a severe psychomotor retardation characterized by neurological im

167 significantly more fatigue, hypersomnia, and psychomotor retardation during the most severe major dep

168 l or early-infantile seizures and associated psychomotor retardation for KCNQ2 and KCNQ3 mutations.

169 arboxylate transporter 8 (MCT8) cause severe psychomotor retardation in children.

170 ects of MCOLN1 function result in mental and psychomotor retardation remain largely unknown.

171 domains of depression, contains measures of psychomotor retardation that could easily reflect fitnes

172 was referred to investigate the etiology of psychomotor retardation was later diagnosed to have fuma

173 don-Dudley syndrome (AHDS), a severe form of psychomotor retardation with abnormal thyroid hormone (T

174 omal storage disease characterized by severe psychomotor retardation, achlorhydria, and ophthalmologi

175 lated motivational symptoms such as anergia, psychomotor retardation, and fatigue.

176 ated motivational symptoms, such as anergia, psychomotor retardation, and fatigue.

177 kull, severe seizures, short limbs, profound psychomotor retardation, and hearing loss.

178 antihistamines are associated with sedation, psychomotor retardation, and reduced academic performanc

179 ired microcephaly, infantile-onset seizures, psychomotor retardation, choreoathetosis, dystonia, and

180 d other psychopathological symptoms (such as psychomotor retardation, lack of insight, poor attention

181 The patient has severe psychomotor retardation, seizures, failure to thrive and

182 toms such as anergia, fatigue, lassitude and psychomotor retardation, which cross multiple pathologie

183 time, including impaired motor function and psychomotor retardation.

184 those surviving beyond childhood have severe psychomotor retardation.

185 in patients with developmental dyslexia and psychomotor retardation.

186 t intractable multifocal seizures and severe psychomotor retardation.

187 tient who presented with epilepsy and severe psychomotor retardation.

188 as hospitalized for generalized seizures and psychomotor retardation.

189 an-Herndon-Dudley syndrome, characterized by psychomotor retardation.

190 tients had developmental delay, and many had psychomotor retardation.

191 sed risk of delayed psychomotor development (psychomotor scale score <85) was also evident among chil

192 had a statistically significantly lower mean psychomotor scale score (difference, -4.35 points; 95% C

193 We calculated mental scale and psychomotor scale scores.

194 ated to a 1.5-fold increase in the odds of a psychomotor score less than 85 (95% confidence interval:

195 The PCB-psychomotor score relation varied by study center (p < 0

196 enatal PCB exposure and children's mental or psychomotor scores (n = 1,207; multivariate adjusted bet

197 None of these markers was associated with psychomotor scores in the multiple regression models.

198 was found for any functional, cognitive, or psychomotor secondary outcome measure at an unadjusted 0

199 pocampus-dependent learning, and exaggerated psychomotor sensitivity to cocaine in mice.

200 d temporally to the emergence of exaggerated psychomotor sensitivity to cocaine.

201 These mice presented an exacerbated psychomotor sensitization and conditioned place preferen

202 , providing evidence that Arg regulates both psychomotor sensitization and decision-making processes

203 Both IEG induction and psychomotor sensitization are dependent upon dopamine an

204 neurons in context-specific cocaine-induced psychomotor sensitization in rats.

205 sion of DeltaFosB-T149A does not produce the psychomotor sensitization to chronic low-dose cocaine se

206 that mice lacking CRF1 receptors do not show psychomotor sensitization to EtOH, a phenomenon that was

207 t underlie the development and expression of psychomotor sensitization to EtOH.

208 reduced locomotor effect of morphine and the psychomotor sensitization to repeated morphine administr

209 greater ethanol-induced place preference and psychomotor sensitization, and greater ethanol consumpti

210 Utilizing cocaine psychomotor sensitization, we have examined phosphorylat

211 ns neurons are necessary for cocaine-induced psychomotor sensitization, whereas the majority of accum

212 ht to mediate drug-related behaviors such as psychomotor sensitization.

213 gly activated accumbens neurons that mediate psychomotor sensitization.

214 and prevented the expression of EtOH-induced psychomotor sensitization.

215 ession (IEG) in the striatum, and to produce psychomotor sensitization.

216 in mesocorticolimbic regions, and to produce psychomotor sensitization.

217 ediate early gene expression, and to produce psychomotor sensitization.

218 effects on the ability of cocaine to induce psychomotor sensitization.

219 us cocaine delivery on its ability to induce psychomotor sensitization.

220 Presently their usage in teaching psychomotor skills has scientific validity in specific t

221 rticle its efficacy in teaching the specific psychomotor skills of bronchoscopy were validated but it

222 evelopment followed by loss of cognitive and psychomotor skills.

223 speed (R=0.31 to 0.45, P<0.05) and increased psychomotor slowing (R=-0.35, P=0.015).

224 The authors hypothesized that since psychomotor slowing in depressed patients has been linke

225 tivational aspects of motivation, anergia or psychomotor slowing in depression, the impact of conditi

226 Psychomotor slowing may identify a subgroup of depressed

227 glutamate was associated with anhedonia and psychomotor slowing measured by the finger-tapping test,

228 pathological features of motivation, such as psychomotor slowing, anergia, and fatigue in depression.

229 illingness to expend effort for rewards; and psychomotor slowing.

230 aw score, -1.53 and -1.53, respectively), or psychomotor speed (change in raw score, 5.2 msec and 0.9

231 scores of 0-48), attention (Digit Span), and psychomotor speed (Digit Symbol Substitution).

232 ning, verbal memory, executive function, and psychomotor speed (P < .05 for all comparisons).

233 .001), executive functioning (P = .013), and psychomotor speed (P = .001).

234 al fasciculus was positively associated with psychomotor speed (P = .04, d = 0.16) in nonimpaired fig

235 h lower scores indicating fewer errors), and psychomotor speed (scores range from 100 to 5100 msec, w

236 Slow visual scanning and psychomotor speed (Trail Making Test, Part A: OR, 1.02;

237 n between duration of PPI use and scores for psychomotor speed and attention (mean score difference f

238 tween medication use and composite scores of psychomotor speed and attention, learning and working me

239 ficits in executive function, attention, and psychomotor speed are also seen in more chronic disease

240 ferential treatment response was specific to psychomotor speed because responders and nonresponders d

241 52, 3.65; P-trend = 0.02) resulted in better psychomotor speed measured by DSST scores.Higher intake

242 No significant findings were shown for psychomotor speed or delayed word recall.

243 sers had significantly slower performance on psychomotor speed tests.

244 he Digit Symbol Substitution Test (DSST) for psychomotor speed, and a modified Stroop interference te

245 learning, verbal memory, executive function, psychomotor speed, and fine motor skills (P < .05 for al

246 ted with overall performance, verbal memory, psychomotor speed, and fine motor skills, and sCD164 rem

247 rmed significantly worse on attention tests, psychomotor speed, and memory at t2 compared with t1 (P

248 are associated with fatigue severity, pain, psychomotor speed, and physical activity, while controll

249 mmon CFS symptoms (e.g., chronic pain, lower psychomotor speed, and reduced physical activity).

250 tests in the domains of delayed word recall, psychomotor speed, and verbal fluency were administered.

251 ance to middle age is associated with better psychomotor speed, executive function, and verbal memory

252 al changes in NABT predict faster decline in psychomotor speed, executive functions, and working memo

253 This study examined the utility of baseline psychomotor speed, measured with neuropsychological test

254 t majority of measures, including cognitive (psychomotor speed, timing, inhibitory control, cognitive

255 y diffuse but occurred primarily in areas of psychomotor speed, visual and working memory, verbal flu

256 depressive symptoms, physical activity, and psychomotor speed.

257 rkers were assessed along with anhedonia and psychomotor speed.

258 Deficits in verbal memory and psychomotor speed/executive function abilities strongly

259 bition via STI-571 infusion also enhance the psychomotor stimulant actions of cocaine.

260 ect that was enhanced by an injection of the psychomotor stimulant cocaine (10 mg/kg, i.p.).

261 as designed to investigate the rewarding and psychomotor stimulant effects of EM-1 in specific brain

262 mice treated with d-amphetamine (5 mg/kg), a psychomotor stimulant known to release AA from corticost

263 tereotypic motor responses characteristic of psychomotor stimulant sensitization correlate with an en

264 Methamphetamine (Meth) is a psychomotor stimulant strongly associated with increases

265 Methamphetamine is a highly addictive psychomotor stimulant yet the neurobiological consequenc

266 ess is not characterized for the most potent psychomotor stimulant, methamphetamine.

267 addition to the list of popular recreational psychomotor-stimulant compounds.

268 models that incorporate inhaled exposure to psychomotor stimulants are not commonly available.

269 accumbens as well as behavioral responses to psychomotor stimulants in female rats but not males.

270 Despite the widespread use of psychomotor stimulants in younger age groups, little is

271 Whereas the effect of psychomotor stimulants on ICSS has long been attributed

272 plore mechanisms by which dopamine-releasing psychomotor stimulants such as cocaine and amphetamine i

273 Adenosine A(2A) receptor antagonists are psychomotor stimulants that also hold therapeutic promis

274 cotine, like other addictive drugs including psychomotor stimulants, promotes dopamine release in the

275 been implicated in the rewarding effects of psychomotor stimulants, whereas recent work suggests tha

276 ty and elevates extracellular DA, leading to psychomotor stimulation and addiction, but the mechanism

277 tary ethanol consumption and ethanol-induced psychomotor stimulation.

278 d to improved treatment for motivational and psychomotor symptoms in psychiatry and neurology.

279 cal alterations in MSN activity patterns and psychomotor symptoms.

280 lular mechanisms of catatonia, an executive "psychomotor" syndrome that is observed across neuropsych

281 Nabilone (8 mg/day) modestly worsened psychomotor task performance.

282 atial navigation, but effects on attentional/psychomotor tasks were more variable.

283 he curriculum consisted of proficiency-based psychomotor training on a virtual reality simulator, cog

284 being a key mechanism for the regulation of psychomotor vigilance as a function of sleep loss.

285 come measure was median reaction time on the psychomotor vigilance task (PVT) at week 2 in each condi

286 nts performing a face/nonface categorization psychomotor vigilance task (PVT) over multiple experimen

287 of the dose-dependent effects of caffeine on psychomotor vigilance task (PVT) performance of sleep-de

288 resonance (MR) imaging was performed with a psychomotor vigilance task (sustained attention) and del

289 nse in the bilateral insular cortex during a psychomotor vigilance task (Z = 2.9-3.4, P = .01-.008) a

290 e validated the proposed unified model using psychomotor vigilance task data from three prior studies

291 ighttime testing of their performance on the Psychomotor Vigilance Test (change from baseline, a redu

292 ory observations of lapses of attention on a psychomotor vigilance test (PVT), in experiments on the

293 al data and reaction time performance on the psychomotor vigilance test (PVT).

294 We validated the model on psychomotor vigilance test data from two studies involvi

295 he Clinical Global Impression of Change, the Psychomotor Vigilance Test, diaries of patients, and day

296 For the psychomotor vigilance test, it accounts for lapses in pe

297 detection task (a "simple RT task") and on a psychomotor vigilance test.

298 Subjective sleepiness, but not psychomotor vigilance, improved during a 2-week course o

299 references was not correlated with change in psychomotor vigilance.

300 a night of REM disruption without changes in psychomotor vigilance.