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1 tress disorder [PTSD], eating disorders, and psychoses).
2 re and function in schizophrenia and related psychoses.
3 lopment of schizophrenia as opposed to other psychoses.
4 ed with 14% of patients with other remitting psychoses.
5 6 patients with other nonaffective remitting psychoses.
6 ferential and common manifestations of the 2 psychoses.
7 sociated with a 2-fold risk for nonaffective psychoses.
8 fewer negative symptoms than other remitting psychoses.
9 rate was less marked in women with affective psychoses.
10 e fertility rate in women with non-affective psychoses.
11 ial vulnerability to schizophrenia and other psychoses.
12 ntification of candidate genes for the major psychoses.
13 schizophrenia; and 5013, other nonaffective psychoses.
14 aving schizophrenia and related nonaffective psychoses.
15 turbances of GABAergic function in the major psychoses.
16 schizophrenia with those of nonschizophrenic psychoses.
17 R schizophrenia and 42 with nonschizophrenic psychoses.
18 much lower for schizoaffective and atypical psychoses.
19 gher in patients with schizophrenia spectrum psychoses.
20 or sibling pairs concordant for nonaffective psychoses.
21 s for the treatment of schizophrenia-related psychoses.
22 t significantly influenced DMN modulation in psychoses.
23 es with other mental disorders, particularly psychoses.
24 present for both affective and nonaffective psychoses.
25 ests overlapping genetic determinants across psychoses.
26 adult, and 0.8% (0.3-1.3) for non-affective psychoses.
27 s primarily based on hospital admissions for psychoses.
28 examine the risk for developing nonaffective psychoses.
29 substance-induced psychosis, and 27.9% other psychoses.
30 lial transmission of schizophrenia and other psychoses.
31 distinction between schizophrenia and other psychoses.
32 rity ethnic groups are at increased risk for psychoses.
33 velopmental delay, schizophrenia and related psychoses.
34 tterns were broadly upheld for the affective psychoses.
35 alcohol-use and substance-use disorders, and psychoses.
36 ug abuse as causative agents in the onset of psychoses.
37 ticolimbic subregions of subjects with major psychoses.
38 to changes in the current nosology of major psychoses.
39 and subjective effects resembling endogenous psychoses.
40 disturbed in schizo-affective and affective psychoses.
41 T1 variant in schizophrenia and drug-induced psychoses.
42 a, severe depression, and other nonaffective psychoses.
43 ith COS but not in adolescents with atypical psychoses.
44 igher for the patients with nonschizophrenia psychoses.
45 , of the subjects with schizophrenia-related psychoses; 50% were identified by all three variables co
46 nia and for other nonaffective and affective psychoses (adjusted hazard ratio for schizophrenia for a
49 lence for schizophrenia and other functional psychoses among communal Hutterites as well as among the
50 ated with an increased risk for nonaffective psychoses among offspring in adjusted models (HR, 1.32;
54 between schizophrenia and other nonaffective psychoses and a theoretically derived measure of hypoxic
56 here was also lower prevalence for affective psychoses and adjustment reaction disorders among the co
59 n important source of income for people with psychoses and confer eligibility for health insurance.
62 for investigating the role of monoamines in psychoses and neurodegenerative and stress-related disor
64 seline diagnosis of schizophrenia or related psychoses and with whom nonusers were matched according
66 patients with first-episode nonschizophrenia psychoses, and 77 healthy comparison subjects were asses
67 sis (e.g., schizophrenia, other nonaffective psychoses, and affective psychoses) have distinct forms
68 ially useful in the assessment of idiopathic psychoses, and cognitive/perceptual neurological signs m
69 assess the prevalence of seizure disorders, psychoses, and mental retardation in urban and rural Moz
73 ssociated with schizophrenia and other major psychoses, and understanding their normal functions will
74 hosis spectrum (SPS) disorders and affective psychoses (APs) or observed high-risk offspring into mid
75 the 1960s notes that episodes with paranoid psychoses are more prevalent in temporal lobe epilepsy (
76 hold model incorporating affective and other psychoses as a phenotype intermediate between schizophre
77 are observed, similar to those seen in major psychoses, as well as a marked reduction in GABA-recepto
78 ibling pairs concordant for all nonaffective psychoses ascertained in the Irish Study of High-Density
79 risk of schizophrenia and other nonaffective psychoses associated with hypoxic-ischemia-related fetal
81 risk of schizophrenia and other nonaffective psychoses associated with this classification of anteced
82 ical and family study of patients with major psychoses at a VA medical center and evaluated with the
83 ars) included 2910 persons with nonaffective psychoses at the end of follow-up, of whom 704 had narro
85 agnoses of schizophrenia, other nonaffective psychoses, bipolar or depressive disorders, neurotic and
87 disorders are the most prevalent idiopathic psychoses, but their outcome from onset has rarely been
89 search should focus on differential rates of psychoses by ethnicity rather than between generations.
94 armacology; some disorders such as epilepsy, psychoses, depression and anxiety were regarded as super
95 gy remains the primary means for classifying psychoses despite considerable evidence that this method
97 creases the risk of later schizophrenia-like psychoses, especially in genetically vulnerable individu
100 th severe mental illnesses, 75% with chronic psychoses, from an inner-city catchment area were random
103 Patients with schizophrenia and related psychoses have an excess of minor neurological abnormali
104 other nonaffective psychoses, and affective psychoses) have distinct forms of behavioral problems (i
105 primary and secondary discharge diagnosis of psychoses (ICD-9 codes 290.x-299.x) after RT and mortali
110 l status and with schizophrenia versus other psychoses in European and African ancestry FEP patients
111 at given the same age structure, the risk of psychoses in first and second generations of the same et
112 locales and cultures finds increased risk of psychoses in first- and second-generation immigrant popu
115 racterized the epidemiology of first-episode psychoses in rural or urban settings since the introduct
116 tion of incidence of schizophrenia and other psychoses in terms of sex, age, ethnicity, and place.
119 between creative individuals and those with psychoses, indicating that creativity and psychosis shar
120 tios (HRs) for the diagnosis of nonaffective psychoses (International Statistical Classification of D
122 ence of schizophrenia and other nonaffective psychoses is greater in urban than rural areas, but the
123 of whether the present classification of the psychoses is in urgent need of reconceptualization.
125 strated that family history of schizophrenia/psychoses is partly mediated through the individual's ge
126 ied register-based diagnoses of nonaffective psychoses made between 1987 and 2003 and comparison subj
127 ive disorder (n = 178); and all nonaffective psychoses (n = 216), and their nonpsychotic relatives (n
129 ), 23.2 (95%CI: 18.3-29.5) for non-affective psychoses (N = 8), 15.2 (95%CI: 11.9-19.5) for schizophr
131 hizophrenia (N=90) and with nonschizophrenia psychoses (N=39) and carefully matched healthy subjects
132 ith ICD diagnoses of schizophrenia and other psychoses (N=98,082) were linked to the crime register t
133 atric disorders, including epilepsy, stroke, psychoses, obsessive compulsive disorder, phobias, psych
136 management of methamphetamine (METH)-induced psychoses often involves treatment with the typical anti
138 p rates of less than 80%, included affective psychoses, or did not use a standardized assessment of D
139 positive predictive value=80%) and affective psychoses (positive predictive value=83%) but much lower
141 ts who did not develop schizophrenia-related psychoses ranged from 18% for those with deficits in att
142 ith 77% of the patients with other remitting psychoses, received a research diagnosis of schizophreni
144 r wholly right nor wholly wrong: the 2 major psychoses show both distinctive and similar patterns of
145 -NDI captures brain differences in the major psychoses that are not accessible with other structural
147 izophrenia researchers to ascribe late-onset psychoses to organic factors, have led to such cases occ
148 s, key informants most frequently attributed psychoses to supernatural causes, followed by seizure di
149 mity of incidence of schizophrenia and other psychoses; variation would have implications for their c
152 age-sex standardized incidence rate for all psychoses was higher in Southeast London (IRR, 49.4 [95%
153 ociated with family history of schizophrenia/psychoses was mediated through the polygenic risk score.
155 or skills to adulthood schizophrenia-related psychoses were examined in separate path analyses by usi
156 incidence of both nonaffective and affective psychoses were found for all of the black and minority e
157 DSM-IV schizophrenia and other nonaffective psychoses were identified using the Diagnostic Interview
161 ed with 12% of patients with other remitting psychoses, were given the residual diagnosis of psychoti
162 of substance abuse or dependence and primary psychoses, while older age predicted major mood disorder
163 rrent therapeutic management of METH-induced psychoses with haloperidol may be contraindicated becaus
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