ライフサイエンスコーパス: psychotic disorder

1 q11DS, 55 (13.4%) were diagnosed as having a psychotic disorder.

2 ith later hospitalization for a nonaffective psychotic disorder.

3 unded yet tenacious beliefs and a symptom of psychotic disorder.

4 spectively identify patients at high risk of psychotic disorder.

5 se who do and do not convert to a full-blown psychotic disorder.

6 Schizophrenia is currently classified as a psychotic disorder.

7 participants were known to have developed a psychotic disorder.

8 ted with a later diagnosis of a nonaffective psychotic disorder.

9 with at least two siblings suffering from a psychotic disorder.

10 Schizophrenia is a highly heritable psychotic disorder.

11 the criteria for schizophrenia or a related psychotic disorder.

12 es not reflect the presence of an underlying psychotic disorder.

13 cting later hospitalization for nonaffective psychotic disorder.

14 lly defined suspected PEs, definite PEs, and psychotic disorder.

15 subjects clinically at risk of developing a psychotic disorder.

16 escence is associated with increased risk of psychotic disorders.

17 ls of neural noise have been demonstrated in psychotic disorders.

18 number of autoimmune diseases in people with psychotic disorders.

19 etting the stage for vulnerability for later psychotic disorders.

20 ajor psychiatric disorders, particularly for psychotic disorders.

21 tions in the hippocampus were evident across psychotic disorders.

22 e underlying mechanism and for prevention of psychotic disorders.

23 help elucidate etiological underpinnings of psychotic disorders.

24 but remain less well characterized in other psychotic disorders.

25 ith the lack of friendships in patients with psychotic disorders.

26 pression, posttraumatic stress disorder, and psychotic disorders.

27 ball responses among patients with different psychotic disorders.

28 ciated with increased risk for affective and psychotic disorders.

29 ture of schizophrenia and is common in other psychotic disorders.

30 ed to be a pathophysiological contributor to psychotic disorders.

31 ctive study of first-admission patients with psychotic disorders.

32 ed dopamine synthesis capacity to particular psychotic disorders.

33 misdiagnosed in the United States as primary psychotic disorders.

34 delusions are a common symptom of a range of psychotic disorders.

35 es predisposing to schizophrenia and related psychotic disorders.

36 tially targeted by schizophrenia and related psychotic disorders.

37 mprove functional outcome among persons with psychotic disorders.

38 Among the men, 2.2% were hospitalized with psychotic disorders.

39 brain structural abnormality common to both psychotic disorders.

40 from that of patients with nonschizophrenic psychotic disorders.

41 ate may play a role in neurodegenerative and psychotic disorders.

42 ion in first-episode schizophrenia and other psychotic disorders.

43 represents a novel treatment development for psychotic disorders.

44 tical connectivity that putatively underlies psychotic disorders.

45 90s but persisted for African Americans with psychotic disorders.

46 and Related Health Problems, Tenth Revision psychotic disorders.

47 y living situation of older outpatients with psychotic disorders.

48 age-specific fertility rates for women with psychotic disorders.

49 al of maintenance treatment in patients with psychotic disorders.

50 women of childbearing age (15-44 years) with psychotic disorders.

51 l exposures might increase the later risk of psychotic disorders.

52 atients (ages 18 to 55 years) with DSM-III-R psychotic disorders.

53 treatment, particularly for individuals with psychotic disorders.

54 treated a higher proportion of patients with psychotic disorders.

55 e that included the full range of functional psychotic disorders.

56 ve sample of persons newly hospitalized with psychotic disorders.

57 ers, then those with schizophrenia and other psychotic disorders.

58 than half of that for patients with mood or psychotic disorders.

59 tion among young adults with newly diagnosed psychotic disorders.

60 ant effect on the etiology and the course of psychotic disorders.

61 hat may contribute to the pathophysiology of psychotic disorders.

62 eptor hypofunction in the pathophysiology of psychotic disorders.

63 mechanism underlies cognitive impairment in psychotic disorders.

64 tervention and the personalized treatment of psychotic disorders.

65 ted to the underlying familial diathesis for psychotic disorders.

66 le evidence that the thalamus is abnormal in psychotic disorders.

67 l categorization schemes for differentiating psychotic disorders.

68 al characteristic of schizophrenia and other psychotic disorders.

69 e that cannabis use can increase the risk of psychotic disorders.

70 nditions: post-traumatic stress disorder and psychotic disorders.

71 escence, is important in the pathogenesis of psychotic disorders.

72 ssessment and management in individuals with psychotic disorders.

73 on to baseline cannabis use and diagnosis of psychotic disorders.

74 e have increased prevalence in patients with psychotic disorders.

75 ce of persecutory delusions in patients with psychotic disorders.

76 psychophysiological alterations observed in psychotic disorders.

77 ups of disorders, the neurodevelopmental and psychotic disorders.

78 se, and type of cannabis used on the risk of psychotic disorders.

79 ffected the association between cannabis and psychotic disorders.

80 tanding of common mechanisms between FTD and psychotic disorders.

81 e of 5-HT2B receptors in the neurobiology of psychotic disorders.

82 e pathophysiology of schizophrenia and other psychotic disorders.

83 malities reported in adults with established psychotic disorders.

84 (15 subjects), depression (11 subjects), and psychotic disorders (10 subjects).

85 disorders or mental retardation (17.3%), and psychotic disorders (14.2%).

86 as mood disorders, followed by schizophrenia/psychotic disorders (14.6%), anxiety disorders (9.3%), a

87 is study of 304 adults at ultrahigh risk for psychotic disorders, 153 (50.3%) received omega-3 PUFAs

88 continuum model of cognitive dysfunction in psychotic disorders, 3) reports familiality of cognitive

89 with first-episode schizophrenia and related psychotic disorders, 34 patients with first-episode nons

90 ssment was lower in patients who developed a psychotic disorder (65.5 [12.0]) compared with those wit

91 (65.5 [12.0]) compared with those without a psychotic disorder (74.0 [14.0]).

92 In both cohorts, non-affective psychotic disorders (adjusted hazard ratio [HR] 1.25, 95

93 ct, a study of first-admission patients with psychotic disorders admitted to psychiatric facilities b

94 n, and deprivation on risk of developing any psychotic disorder, all with qualitative patterns of int

95 en thought to be critical in the etiology of psychotic disorders, although the differential roles pla

96 dysfunction has often been reported in other psychotic disorders, although there are inconsistencies

97 y marker in assessing the risk of developing psychotic disorders among ultra-high-risk individuals.

98 ease in the risk of having a broadly defined psychotic disorder, an almost 8.5-fold increase in the r

99 Most were African American and had a chronic psychotic disorder and a comorbid substance use disorder

100 ical psychotic symptoms), 50 patients with a psychotic disorder and auditory verbal hallucinations, a

101 The presence of a psychotic disorder and cognitive deficits were also eval

102 alth services among patients with a lifetime psychotic disorder and current chronic PTSD.

103 pocampal hypermetabolism leads to atrophy in psychotic disorder and suggest glutamate as a pathogenic

104 s between those who subsequently developed a psychotic disorder and those who did not was distinguish

105 ifty outpatients with schizophrenia or other psychotic disorders and 19 normal comparison subjects ag

106 e role of oxytocin in the pathophysiology of psychotic disorders and addictions and to extend clinica

107 ould be responsible for its association with psychotic disorders and delusions.

108 Patients with other psychotic disorders and depressive psychosis also demons

109 ven lower, even for severe disorders such as psychotic disorders and epilepsy.

110 plications for understanding the etiology of psychotic disorders and for informing social policy.

111 maller than that of patients with delusional psychotic disorders and healthy subjects.

112 ing study in a large series of probands with psychotic disorders and healthy volunteers as part of th

113 Implications for treatment of parents with psychotic disorders and high-risk children are discussed

114 are utilized extensively in the treatment of psychotic disorders and other psychiatric conditions, bu

115 cal displacement of the postcentral gyrus in psychotic disorders and support the hypothesis that anom

116 s mortality; 2) mortality among persons with psychotic disorders and the extent to which cannabis use

117 ations for understanding the pathogenesis of psychotic disorders and the specific deficits that seem

118 al to shed light on the development of major psychotic disorders and to alter their course.

119 road array of intermediate phenotypes across psychotic disorders and to test the hypothesis that inte

120 ally examined the aetiological links between psychotic disorders and violence.

121 organic mental disorders, mood disorders, or psychotic disorders and who were discharged after a comp

122 Of these, 79 (1.7%) met criteria for a psychotic disorder, and of those, only 50% sought profes

123 , delusional disorder, or affective-spectrum psychotic disorder, and psychotic symptoms scoring at le

124 ith schizophrenia, 45 individuals with other psychotic disorders, and 33 healthy control subjects.

125 familiality of cognitive impairments across psychotic disorders, and 4) evaluates cognitive impairme

126 ble death, especially among individuals with psychotic disorders, and may also be common among noncli

127 Psychotic disorders appear to be characterized by signif

128 Psychotic disorders are associated with an increased ris

129 mic-pituitary-adrenal activity indicate that psychotic disorders are associated with elevated cortiso

130 Psychotic disorders are characterized by aberrant neural

131 Psychotic disorders are characterized by attenuated acti

132 al manifestations of schizophrenia and other psychotic disorders are correlated in affected sibling p

133 Psychotic disorders are currently grouped under broad ph

134 The psychotic disorders are one example of this expanding sp

135 y, but the specific network abnormalities in psychotic disorders are poorly understood.

136 omains confirm substance-induced and primary psychotic disorders as distinct entities.

137 Transition to psychotic disorder, as measured using the Comprehensive

138 nding the basis for deficient PPI and MMN in psychotic disorders, as reduced NMDA activity is implica

139 nd is thought to be dysregulated in mood and psychotic disorders, as well as in various phases of opi

140 ups: a schizophrenia group and those with no psychotic disorder at age >/=25 years.

141 Risks of PEs and of psychotic disorder at age 18 years were also increased w

142 092 individuals (0.95%) not diagnosed with a psychotic disorder at the time of examination were hospi

143 requency of experiences at age 12 predicting psychotic disorders at age 18 ranged from 5.5% to 22.8%.

144 The risk of psychotic disorders at age 18 was greater in those with

145 experiences (PE; n=20) at increased risk for psychotic disorders; (b) people with extremely elevated

146 as not associated with an excess of familial psychotic disorder but was associated with a higher morb

147 equently seen in patients with first-episode psychotic disorders but are generally considered to be d

148 ourse, cognitive deficits are present in all psychotic disorders but are most severe and pervasive in

149 At the time of investigation, none had any psychotic disorder, but on detailed interview some of th

150 nt dopamine antagonist that is used to treat psychotic disorders, but 50% of treated patients develop

151 etic contribution to the major categories of psychotic disorders, but few of these have employed oper

152 s of violence are increased in patients with psychotic disorders, but the contribution of triggers fo

153 ound an excess mortality among subjects with psychotic disorders, but the level did not differ betwee

154 pothesis that cannabis increases the risk of psychotic disorders by inducing the same dopaminergic al

155 le participants were patients with diagnosed psychotic disorders (clinical group) and adults in the g

156 Schizophrenia is a psychotic disorder commonly attributed to the interactio

157 Psychotic disorders comorbid with personality disorders

158 abis every day conferred the highest risk of psychotic disorders compared with no use of cannabis (ad

159 with a doubling of the risk of developing a psychotic disorder, compared with no relevant complicati

160 ates of smoking were reported in people with psychotic disorders, compared with controls.

161 Patients with chronic psychotic disorders (CPD) exhibit deficient sensorimotor

162 Psychotic disorders defined by the DSM-IV.

163 1957 in Finland were screened for nonorganic psychotic disorder diagnoses as recorded on an inpatient

164 ata for age, sex, ethnicity, marital status, psychotic disorder diagnosis, subsequent hospital admiss

165 tor for psychosis rather than for a specific psychotic disorder diagnosis.

166 Reliability for psychotic disorders, eating disorders, antisocial person

167 Age-related patterns of addictive and psychotic disorders echo their prevalence in the general

168 Individuals with psychotic disorders experience substantial health dispar

169 efficiency was also significantly reduced in psychotic disorders (F3,587 = 4.01, P = .008) and positi

170 in an epidemiologically defined cohort with psychotic disorders followed for 20 years after first ho

171 Patients were drawn from a cohort with psychotic disorders followed since first hospitalization

172 Research in psychotic disorders following traumatic brain injury mus

173 Patients diagnosed with psychotic disorders (for example, schizophrenia and bipo

174 y control participants and 375 patients with psychotic disorders from the Bipolar-Schizophrenia Netwo

175 anomalous cerebral asymmetry is a feature of psychotic disorders generally, including psychotic mood

176 All patients with psychotic disorders had significantly reduced CON local

177 disorders and patients with nonschizophrenic psychotic disorders had smaller left entorhinal cortex v

178 sorders, schizophrenia, and nonschizophrenic psychotic disorders) had substantially increased risks o

179 elevated dopamine synthesis capacity once a psychotic disorder has developed, little is known about

180 tent methamphetamine psychosis and a primary psychotic disorder has grown increasingly important.

181 ansition in the clinical characterization of psychotic disorders has occurred.

182 Depression, anxiety, and psychotic disorders have been associated with an increas

183 Adults with psychotic disorders have dysconnectivity in critical bra

184 general population, individuals with severe psychotic disorders have increased risks for smoking (od

185 ng-term trajectories of social impairment in psychotic disorders have rarely been studied systematica

186 lator for the individualized prediction of a psychotic disorder in a 2-year period.

187 of AKT1 influences the risk of developing a psychotic disorder in cannabis users.

188 the development of psychotic experiences and psychotic disorders in a large population-based sample o

189 e calculated an overall relative risk of new psychotic disorders in daily smokers versus non-smokers

190 have been identified as raising the risk for psychotic disorders in longitudinal studies.

191 symptoms of patients later hospitalized for psychotic disorders in primary mental health outpatient

192 comorbidity between substance use and severe psychotic disorders in the Genomic Psychiatry Cohort.

193 Psychotic disorders in young adults are relatively uncom

194 Psychotic disorders (including schizophrenia, schizoaffe

195 nd feasible in patients with PTSD and severe psychotic disorders, including current symptoms.

196 Diabetes is highly prevalent in people with psychotic disorders, including schizophrenia and schizoa

197 urenic acid have been found in patients with psychotic disorders, including schizophrenia.

198 uctural brain abnormalities are prominent in psychotic disorders, including schizophrenia.

199 The mechanism through which psychotic disorders influence violence also remains unce

200 Diagnosis of a psychotic disorder, initial IQ, longitudinal IQ trajecto

201 duals who may develop schizophrenia or other psychotic disorders is critical for predicting psychosis

202 etes pharmacotherapy and drug management for psychotic disorders is essential, irrespective of whethe

203 Knowledge of fertility rates in women with psychotic disorders is fundamental for clinicians and re

204 The onset of psychotic disorders is preceded by a high-risk phase cha

205 he general cognitive deficit observed across psychotic disorders is similarly associated with functio

206 th concern associated with schizophrenia and psychotic disorders is the long-term disability that inv

207 ffective acute treatment for severe mood and psychotic disorders, it remains controversial because of

208 resent with unusual symptoms associated with psychotic disorders, leading to misdiagnosis and inappro

209 prevent the onset of schizophrenia and other psychotic disorders, longitudinal high-risk research on

210 t histories of major depression, anxiety, or psychotic disorders (low-risk group) completed a functio

211 psychiatric disorders and that patients with psychotic disorders may develop dementia more often than

212 and psychotic depression suggest that these psychotic disorders may have common pathophysiological f

213 elevating dopamine antagonists used to treat psychotic disorders may initiate and promote breast canc

214 x volume in first-episode, neuroleptic-naive psychotic disorders may not be a confound of the effects

215 t is unique to schizophrenia or is common to psychotic disorders more broadly, and its associations w

216 sms may shed light on the pathophysiology of psychotic disorders more generally.

217 N=37), depressive psychosis (N=39), or other psychotic disorders (N=46) following index presentation,

218 The women with psychotic disorders (N=7,936) had a lower overall genera

219 (ASD) are at increased risk for nonaffective psychotic disorder (NAPD) or bipolar disorder (BD) is un

220 of neighbourhood variations of non-affective psychotic disorders (NAPD) have focused mainly on incide

221 Subjects with psychotic disorder not otherwise specified also had qual

222 abnormalities were seen in the subjects with psychotic disorder not otherwise specified except that t

223 izophrenia, of children and adolescents with psychotic disorder not otherwise specified to that of ch

224 th childhood-onset schizophrenia and 26 with psychotic disorder not otherwise specified) and their re

225 choses, were given the residual diagnosis of psychotic disorder not otherwise specified.

226 or state-related phenomenon in subjects with psychotic disorder not otherwise specified.

227 eater than twofold increased likelihood of a psychotic disorder (odds ratio = 2.18 [95% confidence in

228 ntly increased risk for the development of a psychotic disorder (odds ratio = 2.49; 95% CI, 1.24-5.00

229 Many people who struggle with psychotic disorder often refuse offers of help, includin

230 tion effect of cannabis use and diagnosis of psychotic disorders on mortality.

231 infarction (one patient on eliglustat), and psychotic disorder (one patient on imiglucerase).

232 However, for adults with psychotic disorder or bipolar disorder, the additional c

233 ued because of an adverse event (four due to psychotic disorder or hallucination within 10 days of st

234 erlie the propensity of stress to exacerbate psychotic disorders or predispose an individual to drug-

235 ultra-high risk for psychosis do not develop psychotic disorder over the medium term.

236 upport an overall change in the incidence of psychotic disorder over time, though diagnostic shifts (

237 l high risk (HR) of psychosis will develop a psychotic disorder over time.

238 eir patients with schizophrenia or a related psychotic disorder participated in a European multi-cent

239 Here, we examine gyrification in psychotic disorder patients and their first-degree relat

240 igh across a broad spectrum of patients with psychotic disorders; patients with a history of a curren

241 ntify individuals at high risk of developing psychotic disorders prior to disease onset.

242 e compared between 243 control subjects, 388 psychotic disorder probands, and 300 of their first-degr

243 Of the remaining patients, nine developed a psychotic disorder (psychotic transition group) and 15 d

244 nia diagnosis from those who developed other psychotic disorders (R(2) = 9.2%, p = .002).

245 en reported in a proportion of patients with psychotic disorders, raising hopes for more appropriate

246 For visits specified for psychotic disorders, receipt of atypical antipsychotics

247 effective for individuals with nonaffective psychotic disorders, reducing hospital readmissions appr

248 Present classifications of psychotic disorder remain based on the presence of speci

249 utoantibodies against glutamatergic NMDAR in psychotic disorders remains controversial, with detectio

250 First-admission patients with psychotic disorders run the risk of being misclassified

251 Patients with psychotic disorders (schizophrenia, n = 219; schizoaffec

252 sive-compulsive and panic symptoms and three psychotic disorders--schizophrenia/schizoaffective disor

253 ring a medical visit, patients completed the psychotic disorders section of the Mini International Ne

254 cts of organic-induced and substance-induced psychotic disorder show that our diagnostic classificati

255 The risk of individuals having a psychotic disorder showed a roughly three-times increase

256 asingly used in the treatment of a number of psychotic disorders since their introduction in 1988, wi

257 gnificantly mediated the association between psychotic disorder status and general cognition (beta =

258 increased risk of mental illnesses including psychotic disorders such as schizophrenia.

259 ences differed from that seen in adults with psychotic disorders, suggesting important differences be

260 markedly lower fertility rates in women with psychotic disorders than in matched normal comparison su

261 a substance-induced psychosis and a primary psychotic disorder that co-occurs with the use of alcoho

262 to dissecting the genetic basis of mood and psychotic disorders that can inform future large-scale c

263 clinicians to correctly classify early-phase psychotic disorders that co-occur with substance use.

264 immune response in schizophrenia and related psychotic disorders that, we think, will be of interest

265 en neuronal ensembles has been implicated in psychotic disorders, the neuronal activity of N-desmethy

266 previous diagnosis of schizophrenia or other psychotic disorder, those with glomerular disease, tubul

267 ioning, but few studies have linked risk for psychotic disorders to a neural measure evoked during a

268 ng clinical utility of antipsychotics beyond psychotic disorders to include depressive, bipolar, and

269 amination were hospitalized for nonaffective psychotic disorder up to 9 years after the index examina

270 D IMPLICATIONS OF KEY FINDINGS: Incidence of psychotic disorders varied markedly by age, sex, place a

271 Presence of ICD-10 F10-33 psychotic disorder was confirmed using OPCRIT [operation

272 or bipolar disorder, schizophrenia, or other psychotic disorder was recorded on at least one inpatien

273 Severity of cognitive impairments across psychotic disorders was consistent with a continuum mode

274 cortex volume of patients with nondelusional psychotic disorders was significantly smaller than that

275 cohort of 470 first-admission patients with psychotic disorders was systematically assessed at basel

276 replication; psychotic experiences (PEs) and psychotic disorder were measured by a semistructured int

277 le-aged and elderly outpatients with primary psychotic disorders were administered the UPSA, the Matt

278 and those diagnosed with major depression or psychotic disorders were excluded.

279 Psychotic disorders were present in 41% of adults over a

280 hirty-two hospitalized patients with chronic psychotic disorders were randomly assigned to community

281 icted to studies of more clinically relevant psychotic disorders were similar.

282 2, positive predictive values for predicting psychotic disorders were too low to offer real potential

283 represent candidates that may be causal for psychotic disorders when considered in the context of th

284 tern of cortical thinning as patients with a psychotic disorder, which is suggestive of a similar, bu

285 n a generalizability cohort of patients with psychotic disorders who were hospitalized for an acute p

286 ded a sample of patients with a pre-existing psychotic disorder with a follow-up duration of at least

287 ssion to compare persons hospitalized with a psychotic disorder with those who had never been hospita

288 Psychotic disorders with comorbid personality disorders

289 served between cannabis use and diagnosis of psychotic disorders with regard to mortality.

290 Fifteen individuals with VCFS (30%) had a psychotic disorder, with 24% (n = 12) fulfilling DSM-IV

291 s not associated with an increased risk of a psychotic disorder, with lifetime cannabis use, or with

292 The UHR patients are at long-term risk for psychotic disorder, with the highest risk in the first 2

293 the hippocampal subfields were seen in all 3 psychotic disorders, with the most prominent differences

294 ed risk for hospitalization for nonaffective psychotic disorder within 14 days after examination (haz

295 als presenting with these features develop a psychotic disorder within 3 years.

296 ed on current clinical criteria convert to a psychotic disorder within a 2.5-year follow-up period.

297 en who had never been hospitalized, men with psychotic disorders without a personality disorder or a