ライフサイエンスコーパス: puff

1 icipation of drug-related rewards (cigarette puff).

2 ard (mealworms) and one with punishment (air puff).

3 placebo or active cannabis 'puffs' ($0.25-$5/puff).

4 or by focal application of glutamate (20 mM puffs).

5 al responses into discrete localized events (puffs).

6 ictive relationship between the tone and the puff.

7 arousal responses to hypoxia, sound, and air puff.

8 ist methysergide and mimicked by a serotonin puff.

9 d, a small liquid reward, or an aversive air-puff.

10 IP(3)Rs may simultaneously be open during a puff.

11 19 ms, representing the rising phase of the puff.

12 s' reside within, not at, the borders of the puff.

13 to light mechanical stimulation from an air puff.

14 ing the startle response with unexpected air puffs.

15 amplitudes, kinetics, and spatial spread of puffs.

16 e evident within the durations of individual puffs.

17 ceptor (IP3R) channels underlying local Ca2+ puffs.

18 smic reticulum to generate repetitive Ca(2+) puffs.

19 and rewards than after punished CSs and air puffs.

20 e activation can be visualized as chromosome puffs.

21 hich to calibrate the Ca(2+) flux underlying puffs.

22 nd puffs with sixty-second intervals between puffs.

23 Neither subunit appears to be recruited to puffs.

24 rint, yet are short lived compared to oocyte puffs.

25 wave-like releases interspersed with several puffs.

26 constitute elementary signals called calcium puffs.

27 eavy smokers due to more frequent, intensive puffing.

28 reliable indices of heat load applied during puffing.

29 and pyrolysis regions of a cigarette during puffing.

30 0.15 to 0.43]), reduced mean daily albuterol puffs (-0.27 puff/d [CI, -0.49 to -0.04 puff/d]), and de

31 or decline 1-12 placebo or active cannabis 'puffs' ($0.25-$5/puff).

32 e-liquid consumed doubled from 3.7 to 7.5 mg puff(-1) and the total aldehyde emission rates tripled f

33 de emission rates tripled from 53 to 165 mug puff(-1), with acrolein rates growing by a factor of 10.

34 jects smoked 1 of 5 amounts (none, 1 puff, 3 puffs, 1 full cigarette, or to satiety [2(1/2) to 3 ciga

35 2000 ms before and coterminated with an air puff (100 ms) directed at the cornea as the US.

36 Puffing 25 microM of SP onto SGNs suppressed IK+ by 43 +

37 ons, subjects smoked 1 of 5 amounts (none, 1 puff, 3 puffs, 1 full cigarette, or to satiety [2(1/2) t

38 In both species, hyperpolarizing HCs by puffing a glutamate antagonist, 6,7-dinitro-quinoxaline-

39 ng in collision induced desorption of water (puffing), a process that occurs with significant probabi

40 the venom gland transcriptome of the African puff adder Bitis arietans using paired-end reads sequenc

41 , Cerastes cerastes cerastes (Egypt) and the puff adder, Bitis arietans (Nigeria).

42 y Echis saw scaled vipers and Bitis arietans puff adders is the leading cause of death and morbidity

43 mug) of once-daily tiotropium or placebo (2 puffs) administered through the Respimat device every ev

44 2.5 mug (2 puffs of 1.25 mug), or placebo (2 puffs), administered through the Respimat device as add-

45 ion time of puffs lengthened with increasing puff amplitude size, consistent with independent closing

46 nation time with cluster size independent of puff amplitude.

47 Puff amplitudes (fluorescence ratio change) are on avera

48 Mean puff amplitudes scale with cluster size, but individual

49 correlations between interpuff intervals and puff amplitudes.

50 hannel recruitment by CICR further determine puff amplitudes.

51 Several parameters, including puff and blip durations, puff latency and frequency, and

52 ture geometries on performance of CO2 Huff-n-Puff and CO2 continuous injection.

53 mall Ca(2+) signals that immediately precede puffs and are interpreted to arise through opening of si

54 while varying the polar angle of facial air puffs and close-up visual stimuli.

55 erent ocean regions and colony morphologies (puffs and rafts).

56 Puffs and sparks are localized intracellular Ca(2+) elev

57 onstant of Ca(2+) inactivation and find that puffs and sparks are often less sensitive to variations

58 zed intracellular Ca(2+) elevations known as puffs and sparks arise from the cooperative activity of

59 We study how the statistics of simulated puffs and sparks depend on the kinetics and dissociation

60 raction of inactivated channels that exhibit puffs and sparks that are nearly time-reversible and ter

61 c Ca(2+) excitability" reminiscent of Ca(2+) puffs and sparks where channels open and close in a conc

62 nactivation often leads to time-irreversible puffs and sparks whose termination is facilitated by the

63 sively fast Ca(2+) inactivation can preclude puffs and sparks, moderately fast Ca(2+) inactivation of

64 ation of IP(3)Rs and RyRs in the dynamics of puffs and sparks, we formulate and analyze Markov chain

65 py production rate of Markov chain models of puffs and sparks.

66 onstant is much greater than the duration of puffs and sparks.

67 a deterministic numerical model to simulate puffs and trigger events.

68 s and global Ca(2+) waves, resembling Ca(2+) puffs and waves mediated by inositol trisphosphate (IP(3

69 To elucidate the relation of puffs and waves, we here model a cluster of IP3R channel

70 d InsP3Rs open together giving a local 'Ca2+ puff', and as puffs become more frequent they ignite reg

71 ms macroscopic, fusiform (tufts), spherical (puffs) and raft-like colonies that provide a pseudobenth

72 compared to 19 ms for the mean rise time of puffs, and their spatial extent is approximately 3 times

73 At P14, puff- and light-evoked NMDAR-mediated responses in both

74 elicited by either electrical stimulation or puff application in labelled PVN neurons was significant

75 Calcium puffs are local Ca(2+) release events that arise from a

76 Puffs are local Ca(2+) signals that arise by Ca(2+) libe

77 Puffs are local Ca(2+) signals that arise by Ca(2+) libe

78 Calcium puffs are local transient Ca(2+) releases from internal

79 Puffs are localized, transient elevations in cytosolic C

80 stic and deterministic simulations show that puffs are not stereotyped events of constant duration bu

81 indicator dye (fluo-4-dextran), we show that puffs are often preceded by small, transient Ca(2+) elev

82 ), i.e., the waiting time between successive puffs, are found to be well characterized by a probabili

83 r amplitude ratio was obtained assuming that puffs arise from the synchronous opening of 25-35 IP(3)R

84 g studies have revealed local Ca2+ signals ("puffs") arising from clusters of IP3R, and patch-clamp s

85 s (CS) followed 250 ms later by a 100 ms air puff as an unconditioned stimulus (US) coterminating wit

86 conditioned stimulus (CS) followed by an air puff as an unconditioned stimulus (US) that coterminated

87 conditioned stimulus (CS) and a corneal air puff as the unconditioned stimulus (US).

88 Despite the presence of a puff-associated oscillation in the rate of nicotine accu

89 ulation during cigarette smoking and/or (ii) puff-associated spikes in brain nicotine concentration.

90 10 nondependent smokers (NDS), suggest that puff-associated spikes in the brain nicotine concentrati

91 ected ecdysone-responsive genes reveals that puffing at Eip74EF ceases within an hour or two of cohes

92 to the variability of amplitudes of repeated puffs at a site but the amplitudes of successive puffs w

93 opipette stimulates local, repetitive Ca(2+) puffs at the region of cell contact.

94 al entry and infection, we mutated three VP2 puff B amino acids proposed to make contact with sialic

95 together giving a local 'Ca2+ puff', and as puffs become more frequent they ignite regenerative Ca2+

96 from single-channel openings to local Ca(2+) puffs believed to arise from several to tens of clustere

97 owing a HGRAP choice that resulted in an air-puff, but this did not deter chickens from subsequently

98 r/channel, and we propose that they initiate puffs by recruiting neighboring IP(3)Rs within the clust

99 products (AGEs) and antioxidant capacity of puffed cereals was studied.

100 MF looked like the most appropriate index in puffed cereals.

101 Most interestingly, Ca(2+) puffs cluster during oocyte maturation resulting in a co

102 rder; control (C); control with noise of air puff (CN); air puff to conspecific hen (APC); air puff t

103 rom vaporizing PG and GLY under mild, single puff conditions.

104 Because Ca(2+) puffs constitute the building blocks from which cellular

105 ), reduced mean daily albuterol puffs (-0.27 puff/d [CI, -0.49 to -0.04 puff/d]), and decreased mean

106 erol puffs (-0.27 puff/d [CI, -0.49 to -0.04 puff/d]), and decreased mean asthma symptom score (-0.26

107 4), and increased albuterol rescue use (0.95 puffs/d, p = 0.004) compared with B16Gly/Gly subjects.

108 ypic analyses confirm that sco is allelic to puff daddy(gw1) (pfd(gw1)), a null mutant in fbn2b, and

109 For a high rate of 250 puff day(-1) using a typical vaping regime and popular t

110 imilarly rapid inhibitory process terminates puffs despite local [Ca(2+)] elevation that would otherw

111 GABA puffs diluted in SM/CSF solution or SM/CSF solution alon

112 responded both to visual stimuli and to air puffs directed at the head and abdomen.

113 of LPA2 disorganizes the gradient of Ca(2+) puffs, disrupts gradient sensing, and reduces the direct

114 Vapers' mean puff durations (2.0 s and 2.2 s) were similar with both

115 Among cigarette experimenters (1 puff), ever e-cigarette use was associated with higher o

116 IP(3), and increased numbers of local Ca(2+) puffs evoked by weaker photorelease.

117 Initial puffs evoked following photorelease of IP3-which would n

118 PFC produced a significant inhibition of air-puff-evoked blinks and reduced the generation of CRs com

119 Oocytes loaded with CR or PV showed Ca(2+) puffs following photolysis flashes that were subthreshol

120 We find that the initial triggering rate of puffs following photorelease of IP(3), and the average f

121 f an altered chromatin state associated with puff formation.

122 These properties are similar to Ca(2+) "puffs" found in oocytes.

123 nt, including refractoriness and increase of puff frequency during the inter-wave interval.

124 average amount of Ca(2+) released per puff x puff frequency) varies about the square of cluster size,

125 lular signaling and, because of their higher puff frequency, preferentially act as pacemakers to init

126 lution of locally disordered patches (active puffs) from an ordered vortex-lattice flow state.

127 Electrical stimulation or puffing GABA (gamma-amino butyric acid) receptor blocker

128 da and xi, which represent the basal rate of puff generation and the recovery rate from refractorines

129 ated somata/dendrites; this was confirmed by puffing glycine onto these two regions in retinal slices

130 molecular complexes of LPA2 trigger a Ca(2+) puff gradient that governs gradient sensing and directio

131 ficant positive effect on the pliability and puffing height of chapatti.

132 nt study, we developed a novel compressional-puffing-hydrothermal extraction (CPHE) process which pri

133 We have focused on DNA puff II/9A, which amplifies approximately 17-fold over t

134 ene to examine the structure of a heat shock puff in the presence and absence of insulators.

135 Furthermore, Ca(2+) puffs in eggs have a larger spatial fingerprint, yet are

136 we provide evidence that heat shock-induced puffs in JIL-1 null mutant backgrounds are strongly labe

137 ing high-amplitude local Ca signals known as puffs in neurons and sparks in muscle cells.

138 n ASMC, as well as the statistics of calcium puffs in other cell types, can be quantitatively reprodu

139 We imaged puffs in SH-SY5Y cells using total internal fluorescence

140 phosphorylation at transcriptionally active puffs in such polytene squash preparations after heat sh

141 smooth muscle and from IP(3)-mediated Ca(2+) puffs in Xenopus oocytes.

142 zed elementary Ca(2+) release events (Ca(2+) puffs) in oocytes and eggs and show that the sensitivity

143 The spatial extent of puffs increased with their amplitude, and puffs of simil

144 Puffing influenced starch in vitro digestibility, being

145 of IP3Rs increases, the average duration of puffs initially increases but then becomes saturated, wh

146 volumes (52.2 mL and 83.0 mL) and mean inter-puff intervals (23.2 s and 29.3 s) differed significantl

147 ich was restored by rhythmic delivery of air puffs into the nasal cavity.

148 A point-source model of Ca(2+) puffs is then constructed based on the new IP3R model an

149 Information in the ion "puffs" is received and processed by the cell through res

150 Conversely, depolarizing HCs by puffing kainic acid (KA) into the outer plexiform layer

151 nt antioxidant capacity revealed by digested puffed kernels can be ascribed to both the new formed Ma

152 s CS extracts (CSEs) plus IL-1beta [CSE (6.4 puffs/L)/IL-1beta (2 mug/L)].

153 mal correspondence with experimental data of puff latencies after photorelease of IP3 was obtained wh

154 responses, we then simulated measurements of puff latency after step increases of [IP3].

155 rameters, including puff and blip durations, puff latency and frequency, and frequency of repetitive

156 The mean termination time of puffs lengthened with increasing puff amplitude size, co

157 in (small food quantity), no-risk (of an air-puff) (LGNAP) option.

158 (a P2Y(1) antagonist, 1 muM), ongoing spark/puff like activity and rhythmic intracellular Ca(2+) wav

159 esemble the elementary events ("sparks" and "puffs") mediated by IP(3) receptors and ryanodine recept

160 In this article, using a stochastic puff model and a single-channel data-based IP3R model, w

161 imuli (USs) (liquid rewards and aversive air puffs), newly learned reinforcement-predictive visual st

162 The amplitude of NMDAR currents elicited by puff NMDA application to dorsal horn neurons was also si

163 currents of dorsal horn neurons elicited by puff NMDA application.

164 d the increased amplitude of NMDAR-EPSCs and puff NMDA currents in labeled PVN neurons in SHRs but ha

165 y increased the amplitude of NMDAR-EPSCs and puff NMDA currents in PVN neurons in WKY rats but not in

166 he basal amplitude of evoked NMDAR-EPSCs and puff NMDA currents in retrogradely labeled PVN neurons w

167 e basal amplitudes of evoked NMDAR-EPSCs and puff NMDA currents were significantly higher in SHRs tha

168 a larger reduction in evoked NMDAR-EPSCs and puff NMDA-elicited currents of PVN neurons in SHRs than

169 NMDAR-mediated EPSCs and puff NMDA-elicited currents were recorded in spinally pr

170 excitatory postsynaptic currents (EPSCs) and puff NMDA-elicited currents were recorded in spinally pr

171 Both dsGC types responded to puffed NMDA between P7 and P28; and both types exhibited

172 contrast, neither CPD, total daily number of puffs, nor TDPV predicted nicotine metabolite levels abo

173 ster sizes from the magnitude of the largest puff observed at each site relative to the signal from a

174 , and we investigated how the probability of puff occurrence varies with cluster size.

175 exposed to nicotine (i.e., smoked at least a puff of a cigarette) (N=292).

176 ast some nicotine exposure (i.e., at least a puff of a cigarette).

177 procedure in which a tone was followed by a puff of air to the eye.

178 ng this technique, we found that every 70 mL puff of an e-cigarette deposited 0.019% e-liquid (v/v) i

179 Twice daily beclomethasone treatment was one puff of beclomethasone (40 mug per puff) or placebo give

180 like food body that, when bitten, releases a puff of pollen allowing transfer to stigmas by wind or t

181 ptors in terminals with a spatially confined puff of ryanodine caused a decline in terminal ER [Ca(2+

182 ympathetic nerve activity induced by a 50-mL puff of smoke directed to the external nares of the rabb

183 was scanned following inhalation of a single puff of smoke from a cigarette containing (11)C-nicotine

184 chemicals were delivered by the conventional puffing of stimulus on the antennae, the receptor respon

185 ive 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug) of once-daily tiotropium or placebo (

186 ium 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug), or placebo (2 puffs), administered t

187 17 years were randomized to receive 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug) of on

188 ed to receive once-daily tiotropium 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug), or p

189 fs of beclomethasone or placebo for each two puffs of albuterol (180 mug) needed for symptom relief.

190 baseline to week 48 in mean daily number of puffs of albuterol, mean total asthma symptom score, and

191 Rescue beclomethasone treatment was two puffs of beclomethasone or placebo for each two puffs of

192 he rate of RBC depolarization in response to puffs of CPPG and consequently potentiated the transient

193 rs were recorded in response to 6 successive puffs of GABA diluted in the survival medium (SM), showi

194 ns, and they also responded more strongly to puffs of nicotine.

195 of puffs increased with their amplitude, and puffs of similar size were of similar width, independent

196 creased postsynaptic responsiveness to local puffs of the GABAA receptor agonist isoguvacine.

197 sophila CHD1 localizes to the interbands and puffs of the polytene chromosomes, which are classic sit

198 However, DNA puffs of the salivary gland giant polytene chromosomes o

199 c agonist isoproterenol, we observed bright "puffs" of locally increased surface fluorescence intensi

200 Smoking 0.13 (1 to 2 puffs) of a cigarette resulted in 50% occupancy of alpha

201 Likewise, repetitive iontophoresis (or puffs) of glutamate (or AMPA) onto the dendrites of amac

202 rats learned the location of an aversive air puff on a linear track, as well as during sleep before a

203 to react in an exaggerated fashion to an air puff on the face and to objects approaching the face, wh

204 be detected in slices of the rodent MS/DB by puffing on acetylcholine (ACh).

205 pe II fibers was constructed by successively puffing onto OHCs along the cochlear spiral, up to 180 m

206 Focal DHPG puffs onto granule cells or bath application after glome

207 ing GABA(C) receptors to two rapid glutamate puffs onto the bipolar cell terminal.

208 y calcium-induced calcium release to evoke a puff, optimal correspondence with experimental data of p

209 risk (1 in 4 probability of receiving an air-puff) option (HGRAP) or a low-gain (small food quantity)

210 in mean arterial pressure in response to air puff or noise startle.

211 lgi cell responses could be evoked using air puff or tactile stimuli and under four different anaesth

212 t was one puff of beclomethasone (40 mug per puff) or placebo given in the morning and evening.

213 in mL) (P < 0.05) and total daily number of puffs (P < 0.05), but not other topographical measures,

214 respiratory tract infection and less than 2 puffs per day of short-acting beta-agonist; they also ex

215 average consumption of 517 +/- 465 estimated puffs per month, indicating mild to moderate cannabis de

216 ngle whiskers were deflected with 0.5-18 air puffs per second (apps), a range that includes the whisk

217 Similar results were found for the number of puffs per vaping episode for low (adjusted RR, 2.05; 95%

218 er of vaping episodes per day, and number of puffs per vaping episode) at the 6-month follow-up.

219 y an appearance of large vesicles around the puffing pipette.

220 y responses to aversive stimuli, such as air puff-predictive cues or air puff, remained unimpaired.

221 pattern, observed in raw rice was altered by puffing process and led to the formation of B- and V-typ

222 the antioxidant properties was observed upon puffing process as compared to raw rice samples.

223 ence the present study demonstrates that the puffing process leads to the significant changes in the

224 this study was to investigate the effect of puffing process on the physical, antioxidant properties

225 sity of those peaks was decreased during the puffing process.

226 main in the polytene chromosomes, known as a puff, produced by transcription of the 87A heat shock pr

227 tivated by spontaneous Ca(2+) events (Ca(2+) puffs/pulsars).

228 We found that motivation to smoke (puff rate) predicted magnitude of DA release in limbic s

229 In contrast, GL DHPG puffs readily increased mIPSCs.

230 uli, such as air puff-predictive cues or air puff, remained unimpaired.

231 of release channels involved during a Ca(2+) puff renders the puffs stochastic, with distributed ampl

232 extent is approximately 3 times smaller than puffs (respective widths at half peak amplitude 0.6 and

233 Cortical postsynaptic air-puff responses in the trident representation show much l

234 ulated a model to explain the control of the puffing sequence on Drosophila polytene chromosomes init

235 This gives rise to the observed asymmetric puff shape.

236 om single IP3R (blips) and clusters of IP3R (puffs) showed little temperature dependence, whereas the

237 3) concentration, although the proportion of puffs showing resolved triggers was greatest (approximat

238 consequence is that the signaling power of a puff site (average amount of Ca(2+) released per puff x

239 nt for targeting to transcriptionally active puff sites in chromatin, supporting a role for this doma

240 ed with some of the transcriptionally active puff sites in Drosophila.

241 The locations of puff sites observed by Ca(2+) imaging remain static over

242 ion coefficient<0.003 microm2 s(-1)), as are puff sites over prolonged periods, suggesting that the a

243 loA associates with transcriptionally active puff sites within Drosophila polytene chromosomes and ex

244 lusters differ appreciably between different puff sites, and we investigated how the probability of p

245 for the IP3R clusters underlying functional puff sites.

246 ated channels throughout the duration of the puff/spark event.

247 luminal depletion in the dynamics of Ca(2+) puff/spark termination in release sites composed of Ca(2

248 In the second case, puff/spark termination is promoted by the local depletio

249 nimal Ca(2+) release site models may exhibit puff/spark-like dynamics in either of two distinct param

250 In one case, puffs/spark termination is due to the process of stochas

251 espondence with experimental measurements of puff spatial width and puff/trigger amplitude ratio was

252 sity significantly varied (P<0.05) among the puffing stages and was lowest in expanded rice.

253 ses of six trigeminal sensory neurons to air puff stimulation of facial hair mechanoreceptors were si

254 agnetoencephalographic (MEG) elicited by air puff stimulation of right index finger and recorded usin

255 Tactile experiments using full-body air-puff stimulation suits revealed somatotopic areas of the

256 rigeminal reflex blinks were evoked with air puff stimuli directed at the cornea in darkness and at t

257 ls involved during a Ca(2+) puff renders the puffs stochastic, with distributed amplitudes, durations

258 the effect of PSEs (minus PNEs) on number of puffs taken from a cigarette (r=0.6, p=0.001).

259 ting the run direction that involved the air puff than for the 'safe' direction.

260 ing more strongly after punished CSs and air puffs than after rewarded CSs and rewards.

261 familiar equilibrium (or longtime transient) puffs that are spatially localized and keep their size i

262 es a quantal dissection of the local calcium puffs that constitute building blocks of cellular Ca(2+)

263 to the number of IP(3)Rs that open during a puff, their spatial distribution within a cluster, and h

264 C); control with noise of air puff (CN); air puff to conspecific hen (APC); air puff to observer hen

265 (CN); air puff to conspecific hen (APC); air puff to observer hen (APH).

266 trigeminal blink reflex by delivering an air puff to one eye as saccades were evoked by sub-optimal s

267 ent of this controversy by delivering an air puff to one eye to invoke the trigeminal blink reflex as

268 r visual stimuli paired with an aversive air-puff to the eye in a trace-conditioning paradigm.

269 ally salient unconditioned stimulus (US; air puff to the eye).

270 imilar set of movements was evoked by an air puff to the monkey's cheek.

271 ired with painful esophageal distention, air puff to the wrist, or nothing, which acted as unconditio

272 93129's inhibitory effect was strongest when puffed to STN-->SNr axon terminals in SNr, indicating a

273 show that product characteristics influence puffing topography and, therefore, the results obtained

274 We adapted a cigarette puffing topography device for use with e-cigarettes.

275 We used puffing topography to measure directly product use.

276 Consumer puffing topography was measured for 60 vapers provided w

277 Understanding the factors that affect puffing topography will be important for standardising t

278 ental measurements of puff spatial width and puff/trigger amplitude ratio was obtained assuming that

279 al "trigger" channel, and the probability of puff triggering thus increases steeply with increasing n

280 ays/40 IU twice daily) or placebo (8 days/10 puffs twice daily), initiated within 12 days posttrauma.

281 ne inhaled corticosteroids (QVAR 80 mug, two puffs twice per day, equivalent to 800 mug per day beclo

282 e average frequency of subsequent repetitive puffs, vary about linearly with cluster size.

283 two times more likely to work for cigarette puffs versus money in a progressive ratio, choice task (

284 l IP(3)R channel within a cluster triggers a puff via Ca(2+)-induced Ca(2+) release.

285 Total daily puff volume (TDPV; in mL) (P < 0.05) and total daily num

286 g the expired carbon monoxide level or total puff volume, suggesting minimal compensation.

287 lar with both types of e-cigarette, but mean puff volumes (52.2 mL and 83.0 mL) and mean inter-puff i

288 Under all regimes, air puff volumes were within 1 mL of the target and aerosol

289 a 500-msec tone; in the trace paradigm, the puff was delivered after a 700-msec empty "trace" interv

290 In the delay paradigm, a mild puff was delivered to the eye at the end of a 500-msec t

291 bility to blink in response to a corneal air puff was monitored weekly for 9 weeks.

292 Formaldehyde intake from 100 daily puffs was higher than the amount inhaled by a smoker con

293 Most notably, puffs were never observed after loading BAPTA, and this

294 s at a site but the amplitudes of successive puffs were uncorrelated, even though we observed statist

295 eptor-mediated Ca(2+) release events (Ca(2+) puffs) were more frequent in HF atrial myoctes and were

296 new design incorporates venting to prevent "puffing" when the individual flow channels are opened.

297 Elementary Ca(2+) signals, such as "Ca(2+) puffs", which arise from the release of Ca(2+) from endo

298 eover, the appearance of a new population of puffs with longer latencies, prolonged durations, and at

299 nnected to a pump drawing air for two second puffs with sixty-second intervals between puffs.

300 site (average amount of Ca(2+) released per puff x puff frequency) varies about the square of cluste