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1                               A subset of 25 pulmonary TB patients who had a positive skin reaction t
2 after intradermal injection of PPD among 364 pulmonary TB patients in Cambodia.
3 icts/counties (each with at least 300 active pulmonary TB patients registered in 2009) within the pro
4 nted with untreated HIV infection and active pulmonary TB.
5 s I ranking HRCT criteria to diagnose active pulmonary TB were 95%, 40% and 1.4, respectively.
6 tegies for evaluating outpatients for active pulmonary TB at the San Francisco Department of Public H
7 , 2 had TB lymphadenopathy, and 1 had active pulmonary TB.
8             Cases suspected of having active pulmonary TB whose smears are negative can benefit from
9 uberculosis test for the diagnosis of active pulmonary TB (PTB) with whole blood, plasma, and serum f
10 with symptoms and signs suggestive of active pulmonary TB that were systematically confirmed or ruled
11 atients with class III ranking showed active pulmonary TB.
12 lt South African cohort (n = 72) with active pulmonary TB (on treatment for 1-4 mo) or pulmonary TB t
13 th symptoms and signs consistent with active pulmonary TB and complete clinical diagnosis were strati
14  were collected from 20 subjects with active pulmonary TB and from 20 healthy controls.
15 curacy of this assay in patients with active pulmonary TB and in control patients with or without lat
16 ensure that contacts of patients with active pulmonary TB are identified and appropriately screened.
17 were identified for 349 patients with active pulmonary TB.
18  of sera from patients diagnosed with active pulmonary TB.
19             Data were collected on all adult pulmonary TB patients registered at 25 public health cli
20 Xpert Ultra cartridge for diagnosis of adult pulmonary TB may have different consequences in differen
21 s, and restriction of this analysis to adult pulmonary TB.
22                   We further find that after pulmonary TB infection, it still takes many days before
23 uced T cell responses and protection against pulmonary TB.
24 t implications in vaccine strategies against pulmonary TB and other intracellular infections in the l
25 y influences susceptibility to meningeal and pulmonary TB by different immune mechanisms.
26 sted case-control study on air pollution and pulmonary TB, we observed positive associations with amb
27 re, we present a detailed comparison between pulmonary TB and SARC, including whole-blood gene expres
28 mental findings showed a causal link between pulmonary TB and lung tumorigenesis and established a ge
29 randomly selected U.S.-born and foreign-born pulmonary TB patients from 1993.
30 iciency virus-negative patients with chronic pulmonary TB.
31 dized patients (SPs) presenting with classic pulmonary TB symptoms were deployed in 3 provinces of Ch
32 proportion of younger patients with clinical pulmonary TB due to NTM and co-infection with HIV and th
33 extrapulmonary TB cases but not in clustered pulmonary TB cases.
34                    We used culture-confirmed pulmonary TB as the gold standard, and compared accuracy
35 3/51 (65%) and 33/51 (65%) culture-confirmed pulmonary TB cases, respectively; Xpert MTB/RIF detected
36 nes in participants with previous or current pulmonary TB may have the potential for causing harmful
37                       All patients developed pulmonary TB, either alone or with extrapulmonary diseas
38 s associated with reduced risk of developing pulmonary TB but increased risk of rapid progression to
39 associated with increased risk of developing pulmonary TB.
40             41 subjects with newly-diagnosed pulmonary TB (cases) were compared to 82 healthy control
41 e of multi-detector HRCT chest in diagnosing pulmonary TB cases whose sputum smears are negative and
42 1 replication in alveolar macrophages during pulmonary TB.
43 sease-resistant and -susceptible mice during pulmonary TB.
44 ients presenting with a productive cough for pulmonary TB, Xpert blood offers no diagnostic advantage
45 m a genome-wide association study (GWAS) for pulmonary TB, we found that the response eQTL were more
46 approaches and experimental mouse models for pulmonary TB we characterized MDSCs as novel myeloid pop
47             We estimate risk ratios (RR) for pulmonary TB associated with BCG, IPT, and latent TB inf
48 rolled hospitalized adults suspected to have pulmonary TB in Kampala, Uganda.
49 3-Gal9 pathway plays a similar role in human pulmonary TB.
50 nd persistent in a subset of immunocompetent pulmonary TB patients and is characterized by antigen-sp
51  lung may affect presentation and outcome in pulmonary TB, and an understanding of the development of
52 rrow chimeras demonstrate that reductions in pulmonary TB immunopathology are dependent on hematopoie
53        Since the cellular immune response in pulmonary TB requires lymphocyte--macrophage interaction
54 ing might regulate monocyte MMP secretion in pulmonary TB during cell adhesion to the extracellular m
55 ity with both CCP and CAP frequently seen in pulmonary TB.
56 preventive therapy for persons with inactive pulmonary TB.
57 identified all adults (>15 yr) with incident pulmonary TB (index cases) diagnosed at 106 public healt
58  From 1 June 2011 to 7 March 2012, 4,292 new pulmonary TB patients were enrolled across the 36 cluste
59 SA in the induced sputum samples from 56% of pulmonary TB patients.
60 ection, all animals remained asymptomatic of pulmonary TB.
61 ever, from May to September 1997, 3 cases of pulmonary TB were reported among medical waste treatment
62 y was performed using records on contacts of pulmonary TB patients at the Public Health Service Amste
63                         Of 9,332 contacts of pulmonary TB patients, 4,774 were screened for latent TB
64  frequently are not used in the diagnosis of pulmonary TB cases, particularly TB cases with smear-neg
65  is a reliable method for rapid diagnosis of pulmonary TB, irrespective of the AFB smear result.
66 abinomannan (LAM) would improve diagnosis of pulmonary TB.
67 s or DNA accumulate on the oral epithelia of pulmonary TB patients, and can be collected and detected
68 ociety where TB was prevalent, evaluation of pulmonary TB before prescription of PPI or H2RA is warra
69                             The incidence of pulmonary TB was estimated in miners with and those with
70 ssify the images as having manifestations of pulmonary TB or as healthy.
71     In this study, we used a rabbit model of pulmonary TB to evaluate the impact of adjunctive immune
72 n the highly susceptible guinea pig model of pulmonary TB, a model noteworthy for its close resemblan
73         We enrolled adults with suspicion of pulmonary TB from health facilities in southwestern Ugan
74 mmune modulation to improve the treatment of pulmonary TB and reduce the risk of chronic respiratory
75 thesized that aerosol IFN-gamma treatment of pulmonary TB would increase expression of genes importan
76 ve pulmonary TB (on treatment for 1-4 mo) or pulmonary TB treated at least 12 months before study ent
77  were obtained for cases of culture-positive pulmonary TB (PTB; 91.3%) and extrapulmonary TB (EPTB; 9
78 pproximately 54 (74%) of 72 culture-positive pulmonary TB cases over a 1-year period while requiring
79    Comparison of serum from culture-positive pulmonary TB patients and TB suspects systematically rul
80             Consecutive adult smear-positive pulmonary TB patients presenting to an urban hospital in
81 um samples from patients with smear-positive pulmonary TB who were consecutively enrolled at 10 inter
82 n 4 of 16 subjects (25%) with smear-positive pulmonary TB.
83 ine to participants with current or previous pulmonary TB induced a robust immune response and is not
84 ens, in individuals with current or previous pulmonary TB.
85  the overwhelming majority of culture-proven pulmonary TB cases are diagnosed from the first or secon
86 monary TB compared with patients with purely pulmonary TB (p = 0.01) and was amplified 2.6-fold at di
87 om PPD-anergic as compared with PPD-reactive pulmonary TB patients.
88  organs/tissues in the progression of severe pulmonary TB.
89 c sputum evaluation with Xpert for suspected pulmonary TB, in each of 3 emblematic settings: an HIV c
90                           We identified that pulmonary TB patients have reduced expression of Tim3 on
91  conditional logistic regression models, the pulmonary TB odds ratios (95% confidence intervals) for
92 , that are promising susceptibility genes to pulmonary TB.
93 ophozoites and cysts to Balb/c mice leads to pulmonary TB.
94 to meningeal TB (OR, 3.02; P < .001) than to pulmonary TB (OR, 1.55; P = .22).
95 V)-infected and 416 HIV-negative adults with pulmonary TB at the time of initiating chemotherapy and
96 ed and forty six persons were diagnosed with pulmonary TB in the time period analyzed.
97 count in contacts of patients diagnosed with pulmonary TB.
98                                Patients with pulmonary TB (n = 49) and healthy volunteers with presum
99 -CCP and anti-CAP in sera from patients with pulmonary TB (n = 49), RA patients (n = 36), and control
100 ere evaluated in 358 Cambodian patients with pulmonary TB and 106 tuberculin-positive control subject
101                  We studied 21 patients with pulmonary TB and 7 healthy subjects.
102  weeks of TB treatment from 39 patients with pulmonary TB from Kampala, Uganda enrolled in the Center
103 veolar lavage (BAL) cells from patients with pulmonary TB would have increased spontaneous release of
104                   Moreover, in patients with pulmonary TB, lung damage correlated with increased seru
105 illion genetic variants in 5,530 people with pulmonary TB and in 5,607 healthy controls.

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