ライフサイエンスコーパス: pulmonary alveolar proteinosis

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1 nst GM-CSF from six patients with idiopathic pulmonary alveolar proteinosis.

2 nction disrupt surfactant clearance, causing pulmonary alveolar proteinosis.

3 F autoantibodies isolated from patients with pulmonary alveolar proteinosis.

4 possible contributor to the pathogenesis of pulmonary alveolar proteinosis.

5 haracteristic of atherosclerotic lesions and pulmonary alveolar proteinosis.

6 (diffusion 79% and ventilatory defects 63%, pulmonary alveolar proteinosis 18%, pulmonary arterial h

7 We studied 12 subjects with pulmonary alveolar proteinosis, 61 healthy control subje

8 L-5 exhibit a lung disorder similar to human pulmonary alveolar proteinosis, a rare disease with cong

9 phil functions are impaired in patients with pulmonary alveolar proteinosis and that GM-CSF autoantib

10 ory disease syndrome, cigarette smoking, and pulmonary alveolar proteinosis and the implications of o

11 h myelodysplasia, cytogenetic abnormalities, pulmonary alveolar proteinosis, and myeloid leukemias.

12 including atherosclerosis, type 2 diabetes, pulmonary alveolar proteinosis, and obesity, have a chro

13 xpressed human GM-CSF and IL-3 and developed pulmonary alveolar proteinosis because of elimination of

14 autoimmune polyendocrine syndrome type I and pulmonary alveolar proteinosis, detecting ACAA levels co

15 Neutrophils from subjects with pulmonary alveolar proteinosis had normal ultrastructure

16 Hereditary pulmonary alveolar proteinosis (hPAP) caused by granuloc

17 yeloid cell disorder identical to hereditary pulmonary alveolar proteinosis (hPAP) in children with C

18 Idiopathic pulmonary alveolar proteinosis is caused by autoantibodi

19 Mice lacking beta c show a pulmonary alveolar proteinosis-like disease and reduced

20 (n = 44), bronchiolitis obliterans (n = 21), pulmonary alveolar proteinosis (n = 12), pulmonary fibro

21 ed mortality from infection in patients with pulmonary alveolar proteinosis occurs in association wit

22 ents may be at risk for later development of pulmonary alveolar proteinosis or other opportunistic in

23 of neutrophils are impaired in patients with pulmonary alveolar proteinosis, owing to the presence of

24 e have observed that it is elevated in human pulmonary alveolar proteinosis (PAP) and in the GM-CSF k

25 Rbetac(-/-) mice, respectively) resulted in pulmonary alveolar proteinosis (PAP) but no hematologic

26 o characterize the frequency and features of pulmonary alveolar proteinosis (PAP) in patients with AD

27 Pulmonary alveolar proteinosis (PAP) is a rare disease c

28 Pulmonary alveolar proteinosis (PAP) is a rare disorder

29 Pulmonary alveolar proteinosis (PAP) is a rare lung dise

30 Pulmonary alveolar proteinosis (PAP) is a rare lung diso

31 Pulmonary alveolar proteinosis (PAP) is a rare lung synd

32 Pulmonary alveolar proteinosis (PAP) is a rare syndrome

33 Primary pulmonary alveolar proteinosis (PAP) is a rare syndrome

34 Pulmonary alveolar proteinosis (PAP) is an idiopathic lu

35 In patients with pulmonary alveolar proteinosis (PAP) syndrome, disruptio

36 (GM-CSF) autoantibodies are thought to cause pulmonary alveolar proteinosis (PAP), a rare syndrome ch

37 on computed tomography is characteristic for pulmonary alveolar proteinosis (PAP), it is not specific

38 nt homeostasis and how its disruption causes pulmonary alveolar proteinosis (PAP), we evaluated lipid

39 cryptococcal meningitis who later developed pulmonary alveolar proteinosis (PAP).

40 phagocyte lineage cells and severe secondary pulmonary alveolar proteinosis (PAP).

41 eolar macrophages that partially rescued the pulmonary alveolar proteinosis syndrome.

42 The neutrophil dysfunction characteristic of pulmonary alveolar proteinosis was reproduced in a dose-

43 Five patients developed pulmonary alveolar proteinosis without mutations in the

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