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1 tion of inspired oxygen, shunt fraction, and pulmonary arterial pressure.
2 xygenation without a significant decrease in pulmonary arterial pressure.
3 pulmonary capillary wedge pressure and mean pulmonary arterial pressure.
4 Mean pulmonary arterial pressure.
5 ally in the presence of chronic elevation of pulmonary arterial pressures.
6 ks, significant decreases were noted in mean pulmonary arterial pressure (-10%) and in pulmonary vasc
8 at pre-stage II evaluation, including higher pulmonary arterial pressures (15.4+/-3.0 versus 14.5+/-3
9 ressure (10+/-4 versus 6+/-3 mm Hg; P=0.02), pulmonary arterial pressure (22+/-8 versus 11+/-4 mm Hg;
10 received simvastatin from Day 5 to 35 (mean pulmonary arterial pressure = 27 +/- 3 mm Hg, RV/LV+S =
11 s and right ventricular workload by lowering pulmonary arterial pressure (29.6 +/- 1.3 vs. 24.6 +/- 1
12 ations included 79+/-9% (mean+/-SEM) rise in pulmonary arterial pressure, 30+/-7% decline in cardiac
13 improvements in hemodynamic variables (mean pulmonary arterial pressure: 40 +/- 12 mm Hg vs. 33 +/-
14 an-European study, 91 patients with PH (mean pulmonary arterial pressure 46+/-15 mm Hg) underwent cli
15 , rats that received vehicle had higher mean pulmonary arterial pressures (53 +/- 2 mm Hg) and right
17 ve pulmonary vasodilator, leading to reduced pulmonary arterial pressure and improved ventilation/per
18 via the suppression of the acute increase in pulmonary arterial pressure and improvement of right ven
19 nstrate that SB525334 significantly reverses pulmonary arterial pressure and inhibits right ventricul
22 sed, and there was a small reduction in mean pulmonary arterial pressure and pulmonary vascular resis
24 hypoxemia with a pronounced increase in mean pulmonary arterial pressure and pulmonary vascular resis
25 mia but caused significant increases in mean pulmonary arterial pressure and pulmonary vascular resis
26 d results in an additive favorable effect on pulmonary arterial pressure and pulmonary vascular resis
27 1, and 0.3 mg x kg(-1) x h(-1)) reduced mean pulmonary arterial pressure and pulmonary vascular resis
28 mice was associated with a reduction in mean pulmonary arterial pressure and pulmonary vascular resis
29 ivity, and cGMP levels and partially restore pulmonary arterial pressure and pulmonary vascular resis
30 nitric-oxide synthase (eNOS) have increased pulmonary arterial pressure and pulmonary vascular resis
31 vector encoding the eNOS gene (AdCMVeNOS) on pulmonary arterial pressure and pulmonary vascular resis
32 We conclude that iron availability modifies pulmonary arterial pressure and pulmonary vascular respo
33 lumen and contribute to the elevation of the pulmonary arterial pressure and reduce local lung tissue
34 , and angio-obliteration leading to elevated pulmonary arterial pressure and resistance, right ventri
36 ne whether varying iron availability affects pulmonary arterial pressure and the pulmonary vascular r
38 rtension (PPH) is characterized by increased pulmonary arterial pressure and vascular resistance.
39 lth and disease, including associations with pulmonary arterial pressure, and adverse neurological se
40 ean arterial pressure (MAP), cardiac output, pulmonary arterial pressure, and calculated pulmonary an
42 nts of Ex-PHT are male sex, resting systolic pulmonary arterial pressure, and exercise parameters of
43 ent for recipient age, body mass index, mean pulmonary arterial pressure, and pretransplant diagnosis
45 In controls subjects, right atrial pressure, pulmonary arterial pressure, and pulmonary capillary wed
47 eases (<10 percent) in systemic arterial and pulmonary arterial pressures, and it had no effect on pu
48 t, oxygen delivery [DO2]), mean systemic and pulmonary arterial pressures, and the oxygenation index
49 lling pressure and the resultant increase in pulmonary arterial pressure are associated with disrupti
50 hypertrophy, restoring right ventricular and pulmonary arterial pressures, as well as the pulmonary v
51 ide animals showed a significant increase in pulmonary arterial pressure at 30 mins (47.5 +/- 2.4 and
52 ndary end points comprised cardiac index and pulmonary arterial pressure at rest and during exercise
53 oppler estimates and invasive measurement of pulmonary arterial pressure at rest and peak exercise we
54 ventilation, ventilation-perfusion matching, pulmonary arterial pressure, basal airway tone, and resp
55 id regurgitation velocity-time integral, and pulmonary arterial pressure between patients with and wi
57 ean right atrial pressure by 12 +/- 3%, mean pulmonary arterial pressure by 13 +/- 2%, and pulmonary
58 g vasculature, dose-dependently reducing the pulmonary arterial pressure by as much as 9 mmHg with no
59 used significant decreases in total Hb, mean pulmonary arterial pressure, cardiac index and systemic
60 rch infusion was accompanied by increases of pulmonary arterial pressure, cardiac index, and blood ox
62 chi(2), 28.8; P=0.003) and exercise systolic pulmonary arterial pressure (chi(2), 40.1; P=0.002) and
63 nals, echocardiographic measures of systolic pulmonary arterial pressure correlated reasonably well w
64 pressure of at least 10 mm Hg with the mean pulmonary arterial pressure decreasing to 40 mm Hg or be
67 p had significantly higher mean systemic and pulmonary arterial pressures during resuscitation in com
69 sociated with HSA, which raised systemic and pulmonary arterial pressures from baseline values of 86
71 zation to confirm the diagnosis of PAH (mean pulmonary arterial pressure >/=25 mm Hg and pulmonary ca
72 namics consistent with POPH (defined as mean pulmonary arterial pressure >25 mm Hg and pulmonary vasc
74 ng PHT and Ex-PHT were defined as a systolic pulmonary arterial pressure >50 and >60 mm Hg, respectiv
76 on and, with the exception of an increase in pulmonary arterial pressure, had no adverse effects on h
77 ard ratio, 1.09; P=0.027), exercise systolic pulmonary arterial pressure (hazard ratio, 1.03; P<0.001
78 , 5.2; 95% CI, 1.8-14.8; P = 0.002) and mean pulmonary arterial pressure (hazard ratio, 1.04; 95% CI,
82 tion of AdRSVeNOS attenuated the increase in pulmonary arterial pressure in mice exposed to the fibro
87 Acute increases in PaO2, decreases in mean pulmonary arterial pressure, intensity of mechanical ven
88 O(2) (pO(2))] elicits signaling to regulate pulmonary arterial pressure is incompletely understood.
89 tested the hypotheses that (1) elevated mean pulmonary arterial pressure is the most important hemody
90 s model, we found that the acute increase in pulmonary arterial pressure leading to right ventricle f
91 e and noninvasive), central venous pressure, pulmonary arterial pressure, left and right atrial press
92 arge body mass index, preoperative increased pulmonary arterial pressure, low stroke volume index, hy
94 ry arteries, leads to sustained elevation of pulmonary arterial pressure (mean >25 mm Hg at rest or >
97 death, mean systemic pressure (mSP) and mean pulmonary arterial pressure (mPA) emerged as the most im
98 ons between biventricular MPRI and both mean pulmonary arterial pressure (mPAP) (RV MPRI: rho = -0.59
99 ptolide-treated rats demonstrated lower mean pulmonary arterial pressure (mPAP) than vehicle-treated
100 ary hypertension (PH) is diagnosed by a mean pulmonary arterial pressure (mPAP) value of at least 25
101 heal instillation of liposomal fasudil, mean pulmonary arterial pressure (MPAP) was reduced by 37.6+/
102 L-NAME resulted in a larger increase in mean pulmonary arterial pressure (MPAP) when compared with sa
103 used a marked and sustained decrease in mean pulmonary arterial pressure (MPAP), it also decreased me
104 e, pulmonary artery occlusion pressure, mean pulmonary arterial pressure (MPAP), systemic vascular re
106 (odds ratio, 4.3; P=0.002), resting systolic pulmonary arterial pressure (odds ratio, 1.16; P=0.002),
107 (odds ratio 1.60, 95% CI 1.09 to 2.32), mean pulmonary arterial pressure of > or = 20 mm Hg (odds rat
108 progressively to 50 mL/kg/min, maintaining a pulmonary arterial pressure of < 25 mm Hg and a left atr
109 e is generally defined as a decrease in mean pulmonary arterial pressure of at least 10 mm Hg with th
110 acterized by a progressive elevation in mean pulmonary arterial pressure, often leading to right vent
112 ), with no changes in right atrial pressure, pulmonary arterial pressure, or pulmonary resistance.
114 is found ventricle classification (P=0.001), pulmonary arterial pressure (P</=0.001) annulus area (P=
117 ransposase showed a significant reduction in pulmonary arterial pressure (PABP, 31.67+/-6.03 mmHg, P<
118 variables were mean arterial pressure, mean pulmonary arterial pressure, PAOP, pulmonary capillary p
121 RBCs from hypoxemic patients with elevated pulmonary arterial pressure (PAP) exhibit a similar FeNO
123 luded mean right atrial pressure (RAP), mean pulmonary arterial pressure (PAP), cardiac index, transp
126 nous flow can occur when PA exceeds both the pulmonary arterial pressure (Ppa) and pulmonary venous p
127 , rats that received vehicle had higher mean pulmonary arterial pressures (Ppa = 41 +/- 3 mm Hg) (p <
128 ements (mean+/-SD, 8.3+/-2.8 per subject) of pulmonary arterial pressure, pulmonary arterial wedge pr
131 amic variables (cardiac output, systemic and pulmonary arterial pressures, pulmonary artery occlusion
134 groups 2 to 4, hemodynamic functions (except pulmonary arterial pressure) recovered, yet neither tota
135 usion resulted in a greater increase in mean pulmonary arterial pressure relative to cardiac output i
137 f statin therapy in the improvement of 6MWD, pulmonary arterial pressure, right atrial pressure, card
141 ood was allowed to drain into a reservoir as pulmonary arterial pressure started to rise after veratr
146 pulmonary capillary wedge pressure and mean pulmonary arterial pressure to volume loading with rapid
147 In patients with grade A TR signals, mean pulmonary arterial pressure-to-workload ratio at a thres
148 H based on QRS-gated DPD demonstrated higher pulmonary arterial pressures versus isolated postcapilla
155 ate, mean transmitral gradient, and systolic pulmonary arterial pressure were assessed at different s
157 nnular plane systolic excursion and systolic pulmonary arterial pressure were measured at rest and du
158 for 10 minutes), and LAP, systemic flow, and pulmonary arterial pressure were measured in both fixed
161 , in turn, may play a key role in increasing pulmonary arterial pressure, which is involved in the de
162 ikewise, none of the other indices including pulmonary arterial pressure (WMD: -0.97 mmHg, 95%CI: -4.
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