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1 ill UK patients with COVID-19 and suspected pulmonary aspergillosis.
2 f A. terreus to AmB in experimental invasive pulmonary aspergillosis.
3 ent a new strategy for treatment of invasive pulmonary aspergillosis.
4 d animals against subsequent lethal invasive pulmonary aspergillosis.
5 ay key roles in host defense against primary pulmonary aspergillosis.
6 important proximal signal in murine invasive pulmonary aspergillosis.
7 ses susceptibility to postinfluenza invasive pulmonary aspergillosis.
8 There was one transplant-related death from pulmonary aspergillosis.
9 th was the predominant histologic pattern of pulmonary aspergillosis.
10 an immunosuppressed murine model of invasive pulmonary aspergillosis.
11 influenza virus-infected mice from invasive pulmonary aspergillosis.
12 s associated with susceptibility to invasive pulmonary aspergillosis.
13 as associated with the incidence of invasive pulmonary aspergillosis.
14 iation between severe influenza and invasive pulmonary aspergillosis.
15 and in a neutropenic mouse model of invasive pulmonary aspergillosis.
16 d A. fumigatus avirulent in a mouse model of pulmonary aspergillosis.
17 idia either in vitro or in a murine model of pulmonary aspergillosis.
18 als for comprehension of the pathogenesis of pulmonary aspergillosis.
19 s is the causative agent of allergic broncho-pulmonary aspergillosis.
20 , and the remaining 165 patients no invasive pulmonary aspergillosis.
21 d is avirulent in a murine model of invasive pulmonary aspergillosis.
22 tic cell transplant recipients with invasive pulmonary aspergillosis.
23 osuppressed mice from experimentally induced pulmonary aspergillosis.
24 nt in two distinct murine models of invasive pulmonary aspergillosis.
25 of haematogenously disseminated and invasive pulmonary aspergillosis.
26 ion in vitro and in mouse models of invasive pulmonary aspergillosis.
27 be antagonistic in the treatment of invasive pulmonary aspergillosis.
28 ung tissue recovered from mice with invasive pulmonary aspergillosis.
29 -mediated pH response in the pathogenesis of pulmonary aspergillosis.
31 2(-/-) mice displayed high susceptibility to pulmonary aspergillosis, a phenotype associated with a p
32 f critically ill patients, putative invasive pulmonary aspergillosis according to AspICU algorithm wa
35 study we evaluated the incidence of invasive pulmonary aspergillosis among intubated patients with cr
36 etermining BAL GM levels in the diagnosis of pulmonary aspergillosis among nonimmunocompromised hosts
37 .9%, likely reflecting the low prevalence of pulmonary aspergillosis among nonimmunosuppressed patien
40 covered as the etiological agent of invasive pulmonary aspergillosis and had reduced in vitro suscept
41 plored targets for the treatment of invasive pulmonary aspergillosis and may potentiate both innate i
43 Intensive Care Medicine influenza-associated pulmonary aspergillosis and the European Confederation o
44 is known about the pathogenesis of invasive pulmonary aspergillosis and the relationship between the
45 ally successful if initiated early, although pulmonary aspergillosis and zygomycosis are portentous a
47 at the inflammatory response during invasive pulmonary aspergillosis, and in particular the IL-1 axis
49 n of neutrophils, animals developed invasive pulmonary aspergillosis, associated with delayed influx
51 e diagnosed with putative or proven invasive pulmonary aspergillosis, based on the validated AspICU a
52 Reaching a consensual definition of invasive pulmonary aspergillosis becomes mandatory and confers fu
53 c fibrosis patients without allergic broncho-pulmonary aspergillosis but sensitized to A. fumigatus a
54 es were classified as coronavirus-associated pulmonary aspergillosis (CAPA) according to previous con
55 es were classified as coronavirus associated pulmonary aspergillosis (CAPA) according to previous con
56 The literature regarding COVID-19-associated pulmonary aspergillosis (CAPA) has shown conflicting obs
57 or coronavirus disease (COVID-19)-associated pulmonary aspergillosis (CAPA) has yet to be explored.
58 ronavirus disease 2019 (COVID-19)-associated pulmonary aspergillosis (CAPA) is a frequent superinfect
60 ronavirus disease 2019 (COVID-19)-associated pulmonary aspergillosis (CAPA) occurs in critically ill
61 pectively classified for COVID-19-associated pulmonary aspergillosis (CAPA) status following the 2020
63 9 (COVID-19) may develop COVID-19-associated pulmonary aspergillosis (CAPA), which impacts their chan
64 is, including those with COVID-19-associated pulmonary aspergillosis (CAPA), which in its early stage
67 spergillus fumigatus causes chronic cavitary pulmonary aspergillosis (CCPA) and allergic bronchopulmo
68 aspergillosis, two each with acute invasive pulmonary aspergillosis, chronic necrotizing pulmonary a
69 vitro, could account for chronic necrotizing pulmonary aspergillosis (CNPA), which is seen most commo
75 ) in assessing treatment response in chronic pulmonary aspergillosis (CPA) remains to be determined.
78 body improves survival of mice with invasive pulmonary aspergillosis, demonstrating the potential of
82 Asp f 6 helped distinguish allergic broncho-pulmonary aspergillosis from A. fumigatus sensitization
83 ill patients with influenza associated with pulmonary aspergillosis from three hospital ICUs between
90 ogens, in patients with influenza-associated pulmonary aspergillosis (IAPA) or coronavirus disease (C
91 detecting galactomannan (GM) for diagnosing pulmonary aspergillosis in 73 nonimmunocompromised patie
92 tunistic fungal pathogen and causes invasive pulmonary aspergillosis in conditions with compromised i
95 Therefore, we evaluated susceptibility to pulmonary aspergillosis in globally NADPH oxidase-defici
96 ives: We investigated the invasive nature of pulmonary aspergillosis in histology specimens of influe
97 was assessed using a murine model of primary pulmonary aspergillosis in immunocompetent Crl:CF-1 mice
98 us is an important pathogen causing invasive pulmonary aspergillosis in immunocompromised patients.
101 (LFA) with automated reader for diagnosis of pulmonary aspergillosis in patients with COVID-19-associ
102 tion triazole, against experimental invasive pulmonary aspergillosis in persistently neutropenic rabb
103 ultaneous treatment of experimental invasive pulmonary aspergillosis in persistently neutropenic rabb
104 undamental insights into the pathogenesis of pulmonary aspergillosis in the immunocompromised host.
106 ncommon but severe clinical form of invasive pulmonary aspergillosis in which the fungal infection is
107 literature related to diagnosis of invasive pulmonary aspergillosis, invasive candidiasis, and the c
109 tic factors and optimal therapy for invasive pulmonary aspergillosis (IPA) after kidney transplantati
110 age (BAL) fluid in the diagnosis of invasive pulmonary aspergillosis (IPA) among solid-organ transpla
111 al setting, is highly suggestive of invasive pulmonary aspergillosis (IPA) and associated with specif
112 kemia during the study period, with invasive pulmonary aspergillosis (IPA) complicating 6.8% of ISA,
122 e to unresolved inflammation during invasive pulmonary aspergillosis (IPA) is associated with a poor
130 stem cell transplant recipients to invasive pulmonary aspergillosis (IPA), as correct levels of pers
132 ncluding 20 with probable or proven invasive pulmonary aspergillosis (IPA), seven with mucormycosis,
152 mononuclear cells into the lung in invasive pulmonary aspergillosis is in part mediated by MIP-1 alp
156 ced mutant was hypervirulent in the invasive pulmonary aspergillosis murine model system and showed i
157 pulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergillosis (IA),
158 ngal lung burdens in a rat model of invasive pulmonary aspergillosis (p<0.05) compared to treatment w
159 , in a separate cohort of confirmed invasive pulmonary aspergillosis patients, polymorphisms in the I
161 in patients with proven or probable invasive pulmonary aspergillosis, should combination therapy with
162 in patients with proven or probable invasive pulmonary aspergillosis, should combination therapy with
163 kely to serve as an S source during invasive pulmonary aspergillosis since a sulfate transporter muta
164 the role of PCR in the diagnosis of invasive pulmonary aspergillosis, the role of beta-d-glucan assay
165 um and BAL and for the diagnosis of invasive pulmonary aspergillosis, the role of PCR in the diagnosi
166 is associated with poor outcome to invasive pulmonary aspergillosis, this suggested that IFNAR2 may
167 or patients with proven or probable invasive pulmonary aspergillosis to receive either initial combin
168 or patients with proven or probable invasive pulmonary aspergillosis to receive either initial combin
169 s and in nine patients with allergic broncho-pulmonary aspergillosis (two with cystic fibrosis and se
171 iagnosis of probable/proven viral-associated pulmonary aspergillosis (VAPA) was made based on the Int
175 l alkalinization in the host defense against pulmonary aspergillosis, we observed high morbidity of p
177 environmental fungus that can cause invasive pulmonary aspergillosis when spores are inhaled into the
178 piratory disease, and postinfluenza invasive pulmonary aspergillosis, which is becoming a well-recogn
179 ts underwent screening protocol for invasive pulmonary aspergillosis with bronchoalveolar lavage gala
180 underwent a screening protocol for invasive pulmonary aspergillosis with bronchoalveolar lavage gala
181 3-beta-d-glucan concentrations than invasive pulmonary aspergillosis without tracheobronchial lesions
182 s between tracheobronchial aspergillosis and pulmonary aspergillosis without tracheobronchial lesions