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1 d A. fumigatus avirulent in a mouse model of pulmonary aspergillosis.
2 idia either in vitro or in a murine model of pulmonary aspergillosis.
3 als for comprehension of the pathogenesis of pulmonary aspergillosis.
4 s is the causative agent of allergic broncho-pulmonary aspergillosis.
5 , and the remaining 165 patients no invasive pulmonary aspergillosis.
6 d is avirulent in a murine model of invasive pulmonary aspergillosis.
7 tic cell transplant recipients with invasive pulmonary aspergillosis.
8 osuppressed mice from experimentally induced pulmonary aspergillosis.
9 nt in two distinct murine models of invasive pulmonary aspergillosis.
10 of haematogenously disseminated and invasive pulmonary aspergillosis.
11 ion in vitro and in mouse models of invasive pulmonary aspergillosis.
12 be antagonistic in the treatment of invasive pulmonary aspergillosis.
13 ung tissue recovered from mice with invasive pulmonary aspergillosis.
14 -mediated pH response in the pathogenesis of pulmonary aspergillosis.
15 f A. terreus to AmB in experimental invasive pulmonary aspergillosis.
16 ent a new strategy for treatment of invasive pulmonary aspergillosis.
17 d animals against subsequent lethal invasive pulmonary aspergillosis.
18 ay key roles in host defense against primary pulmonary aspergillosis.
19 important proximal signal in murine invasive pulmonary aspergillosis.
20 There was one transplant-related death from pulmonary aspergillosis.
22 2(-/-) mice displayed high susceptibility to pulmonary aspergillosis, a phenotype associated with a p
23 etermining BAL GM levels in the diagnosis of pulmonary aspergillosis among nonimmunocompromised hosts
24 .9%, likely reflecting the low prevalence of pulmonary aspergillosis among nonimmunosuppressed patien
27 covered as the etiological agent of invasive pulmonary aspergillosis and had reduced in vitro suscept
28 plored targets for the treatment of invasive pulmonary aspergillosis and may potentiate both innate i
29 is known about the pathogenesis of invasive pulmonary aspergillosis and the relationship between the
30 ally successful if initiated early, although pulmonary aspergillosis and zygomycosis are portentous a
32 at the inflammatory response during invasive pulmonary aspergillosis, and in particular the IL-1 axis
34 n of neutrophils, animals developed invasive pulmonary aspergillosis, associated with delayed influx
35 c fibrosis patients without allergic broncho-pulmonary aspergillosis but sensitized to A. fumigatus a
36 spergillus fumigatus causes chronic cavitary pulmonary aspergillosis (CCPA) and allergic bronchopulmo
37 aspergillosis, two each with acute invasive pulmonary aspergillosis, chronic necrotizing pulmonary a
38 vitro, could account for chronic necrotizing pulmonary aspergillosis (CNPA), which is seen most commo
40 body improves survival of mice with invasive pulmonary aspergillosis, demonstrating the potential of
42 Asp f 6 helped distinguish allergic broncho-pulmonary aspergillosis from A. fumigatus sensitization
45 detecting galactomannan (GM) for diagnosing pulmonary aspergillosis in 73 nonimmunocompromised patie
48 Therefore, we evaluated susceptibility to pulmonary aspergillosis in globally NADPH oxidase-defici
49 was assessed using a murine model of primary pulmonary aspergillosis in immunocompetent Crl:CF-1 mice
50 us is an important pathogen causing invasive pulmonary aspergillosis in immunocompromised patients.
53 ultaneous treatment of experimental invasive pulmonary aspergillosis in persistently neutropenic rabb
54 tion triazole, against experimental invasive pulmonary aspergillosis in persistently neutropenic rabb
55 undamental insights into the pathogenesis of pulmonary aspergillosis in the immunocompromised host.
58 tic factors and optimal therapy for invasive pulmonary aspergillosis (IPA) after kidney transplantati
59 age (BAL) fluid in the diagnosis of invasive pulmonary aspergillosis (IPA) among solid-organ transpla
64 e to unresolved inflammation during invasive pulmonary aspergillosis (IPA) is associated with a poor
88 mononuclear cells into the lung in invasive pulmonary aspergillosis is in part mediated by MIP-1 alp
91 ced mutant was hypervirulent in the invasive pulmonary aspergillosis murine model system and showed i
92 pulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergillosis (IA),
93 ngal lung burdens in a rat model of invasive pulmonary aspergillosis (p<0.05) compared to treatment w
94 kely to serve as an S source during invasive pulmonary aspergillosis since a sulfate transporter muta
95 s and in nine patients with allergic broncho-pulmonary aspergillosis (two with cystic fibrosis and se
100 l alkalinization in the host defense against pulmonary aspergillosis, we observed high morbidity of p
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