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1 d A. fumigatus avirulent in a mouse model of pulmonary aspergillosis.
2 idia either in vitro or in a murine model of pulmonary aspergillosis.
3 als for comprehension of the pathogenesis of pulmonary aspergillosis.
4 s is the causative agent of allergic broncho-pulmonary aspergillosis.
5 , and the remaining 165 patients no invasive pulmonary aspergillosis.
6 d is avirulent in a murine model of invasive pulmonary aspergillosis.
7 tic cell transplant recipients with invasive pulmonary aspergillosis.
8 osuppressed mice from experimentally induced pulmonary aspergillosis.
9 nt in two distinct murine models of invasive pulmonary aspergillosis.
10 of haematogenously disseminated and invasive pulmonary aspergillosis.
11 ion in vitro and in mouse models of invasive pulmonary aspergillosis.
12 be antagonistic in the treatment of invasive pulmonary aspergillosis.
13 ung tissue recovered from mice with invasive pulmonary aspergillosis.
14 -mediated pH response in the pathogenesis of pulmonary aspergillosis.
15 f A. terreus to AmB in experimental invasive pulmonary aspergillosis.
16 ent a new strategy for treatment of invasive pulmonary aspergillosis.
17 d animals against subsequent lethal invasive pulmonary aspergillosis.
18 ay key roles in host defense against primary pulmonary aspergillosis.
19 important proximal signal in murine invasive pulmonary aspergillosis.
20  There was one transplant-related death from pulmonary aspergillosis.
21 t (n = 524), 79 patients had proven invasive pulmonary aspergillosis (15.1%).
22 2(-/-) mice displayed high susceptibility to pulmonary aspergillosis, a phenotype associated with a p
23 etermining BAL GM levels in the diagnosis of pulmonary aspergillosis among nonimmunocompromised hosts
24 .9%, likely reflecting the low prevalence of pulmonary aspergillosis among nonimmunosuppressed patien
25                       Incidences of invasive pulmonary aspergillosis, an infection caused predominant
26 ions include invasive aspergillosis, chronic pulmonary aspergillosis and bronchitis.
27 covered as the etiological agent of invasive pulmonary aspergillosis and had reduced in vitro suscept
28 plored targets for the treatment of invasive pulmonary aspergillosis and may potentiate both innate i
29  is known about the pathogenesis of invasive pulmonary aspergillosis and the relationship between the
30 ally successful if initiated early, although pulmonary aspergillosis and zygomycosis are portentous a
31 pulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, and aspergilloma.
32 at the inflammatory response during invasive pulmonary aspergillosis, and in particular the IL-1 axis
33       This is particularly true for invasive pulmonary aspergillosis, as so far, sources of (macro)el
34 n of neutrophils, animals developed invasive pulmonary aspergillosis, associated with delayed influx
35 c fibrosis patients without allergic broncho-pulmonary aspergillosis but sensitized to A. fumigatus a
36 spergillus fumigatus causes chronic cavitary pulmonary aspergillosis (CCPA) and allergic bronchopulmo
37  aspergillosis, two each with acute invasive pulmonary aspergillosis, chronic necrotizing pulmonary a
38 vitro, could account for chronic necrotizing pulmonary aspergillosis (CNPA), which is seen most commo
39                                      Chronic pulmonary aspergillosis (CPA) is an infectious disease t
40 body improves survival of mice with invasive pulmonary aspergillosis, demonstrating the potential of
41                                  In invasive pulmonary aspergillosis, direct invasion and occlusion o
42  Asp f 6 helped distinguish allergic broncho-pulmonary aspergillosis from A. fumigatus sensitization
43               Blood from three patients with pulmonary aspergillosis had positive PCR results: one pa
44 ecruitment in neutropenic mice with invasive pulmonary aspergillosis (IA).
45  detecting galactomannan (GM) for diagnosing pulmonary aspergillosis in 73 nonimmunocompromised patie
46                By use of a model of invasive pulmonary aspergillosis in corticosteroid-treated CF-1 m
47 respiratory tract colonization from invasive pulmonary aspergillosis in critically ill patients.
48    Therefore, we evaluated susceptibility to pulmonary aspergillosis in globally NADPH oxidase-defici
49 was assessed using a murine model of primary pulmonary aspergillosis in immunocompetent Crl:CF-1 mice
50 us is an important pathogen causing invasive pulmonary aspergillosis in immunocompromised patients.
51 ungal clearance during experimental invasive pulmonary aspergillosis in neutropenic mice.
52                                              Pulmonary aspergillosis in nonimmunocompromised hosts, a
53 ultaneous treatment of experimental invasive pulmonary aspergillosis in persistently neutropenic rabb
54 tion triazole, against experimental invasive pulmonary aspergillosis in persistently neutropenic rabb
55 undamental insights into the pathogenesis of pulmonary aspergillosis in the immunocompromised host.
56 ergillus colonization from putative invasive pulmonary aspergillosis in this patient group.
57                    Risk factors for invasive pulmonary aspergillosis (IPA) after kidney transplantati
58 tic factors and optimal therapy for invasive pulmonary aspergillosis (IPA) after kidney transplantati
59 age (BAL) fluid in the diagnosis of invasive pulmonary aspergillosis (IPA) among solid-organ transpla
60 gal therapy on an LFD developed for invasive pulmonary aspergillosis (IPA) detection.
61                                     Invasive pulmonary aspergillosis (IPA) is a frequently fatal infe
62                                     Invasive pulmonary aspergillosis (IPA) is a life-threatening lung
63                                     Invasive pulmonary aspergillosis (IPA) is a significant complicat
64 e to unresolved inflammation during invasive pulmonary aspergillosis (IPA) is associated with a poor
65                                     Invasive pulmonary aspergillosis (IPA) is frequent and often fata
66                                     Invasive pulmonary aspergillosis (IPA) is often a lethal entity i
67                                     Invasive pulmonary aspergillosis (IPA) is one of the major compli
68 cation of the causative organism in invasive pulmonary aspergillosis (IPA) is recommended.
69                                     Invasive pulmonary aspergillosis (IPA) is the most common manifes
70                        Diagnosis of invasive pulmonary aspergillosis (IPA) remains a major challenge
71                                     Invasive pulmonary aspergillosis (IPA) remains an important cause
72 ontribute to therapeutic failure in invasive pulmonary aspergillosis (IPA).
73 e gold standard test for diagnosing invasive pulmonary aspergillosis (IPA).
74 n antifungal immunity in a model of invasive pulmonary aspergillosis (IPA).
75 rst-line agent for the treatment of invasive pulmonary aspergillosis (IPA).
76 y facilitate the early diagnosis of invasive pulmonary aspergillosis (IPA).
77 aluation of patients with suspected invasive pulmonary aspergillosis (IPA).
78 ynamics of caspofungin (CAS) during invasive pulmonary aspergillosis (IPA).
79                                     Invasive pulmonary aspergillosis is a common and devastating comp
80                                     Invasive pulmonary aspergillosis is a devastating complication of
81                                     Invasive pulmonary aspergillosis is a devastating complication of
82                                     Invasive pulmonary aspergillosis is a leading cause of infection-
83                                              Pulmonary aspergillosis is a lethal mold infection in th
84                                     Invasive pulmonary aspergillosis is acquired through inhalation o
85                                     Invasive pulmonary aspergillosis is an emerging devastating infec
86                                     Invasive pulmonary aspergillosis is an important cause of morbidi
87                             The diagnosis of pulmonary aspergillosis is difficult, since the recovery
88  mononuclear cells into the lung in invasive pulmonary aspergillosis is in part mediated by MIP-1 alp
89 rs, and antifungal drug therapy for invasive pulmonary aspergillosis may be further understood.
90 nd in situ angiogenesis in a murine invasive pulmonary aspergillosis model.
91 ced mutant was hypervirulent in the invasive pulmonary aspergillosis murine model system and showed i
92 pulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergillosis (IA),
93 ngal lung burdens in a rat model of invasive pulmonary aspergillosis (p<0.05) compared to treatment w
94 kely to serve as an S source during invasive pulmonary aspergillosis since a sulfate transporter muta
95 s and in nine patients with allergic broncho-pulmonary aspergillosis (two with cystic fibrosis and se
96                             Six patients had pulmonary aspergillosis, two each with acute invasive pu
97                                     Invasive pulmonary aspergillosis was an independent risk factor f
98                                     Invasive pulmonary aspergillosis was defined according to Europea
99                  Probable or proven invasive pulmonary aspergillosis was diagnosed in 14% of our stud
100 l alkalinization in the host defense against pulmonary aspergillosis, we observed high morbidity of p

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