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1 analysis may provide novel insights into the pulmonary circulation.
2 cetylcholine and sodium nitroprusside in the pulmonary circulation.
3 fetus that directs blood flow away from the pulmonary circulation.
4 rtant mediator of vascular resistance in the pulmonary circulation.
5 and can lead to unpredictable effects on the pulmonary circulation.
6 the molecular diagnosis of disorders of the pulmonary circulation.
7 ent on reactions with deoxyhemoglobin in the pulmonary circulation.
8 right ventricle supporting both systemic and pulmonary circulation.
9 of the iron chelator desferrioxamine on the pulmonary circulation.
10 particles (2 x 10(8)) were infused into the pulmonary circulation.
11 eling and persistent vasoconstriction of the pulmonary circulation.
12 a-alveolar to alveolar vessels in the intact pulmonary circulation.
13 ors that escape degradation in bypassing the pulmonary circulation.
14 (P=0.03) as hemoglobin is oxygenated in the pulmonary circulation.
15 nce of normal hepatic venous drainage to the pulmonary circulation.
16 tion of sickled erythrocytes in the inflamed pulmonary circulation.
17 fetus that directs blood flow away from the pulmonary circulation.
18 5 mg/kg) produced selective dilation of the pulmonary circulation.
19 mouse by permanently obstructing 40% of the pulmonary circulation.
20 olerance by impairing blood flow through the pulmonary circulation.
21 rtension (PHTN) are unique properties of the pulmonary circulation.
22 /kg/min), the dilation was selective for the pulmonary circulation.
23 tes the membrane step in PG clearance by the pulmonary circulation.
24 and -independent dilators in the ovine fetal pulmonary circulation.
25 or absent in some SMC tissues, including the pulmonary circulation.
26 dure while preventing adverse effects on the pulmonary circulation.
27 ically like the native valve leaflets in the pulmonary circulation.
28 unting of blood between the systemic and the pulmonary circulations.
29 ve vascular disease in both the systemic and pulmonary circulations.
30 an resistance arteries from the systemic and pulmonary circulations.
31 1 (31 microgram/min) selectively dilated the pulmonary circulation, an effect again blocked after inh
32 resulted from (129)Xe transport through the pulmonary circulation and diffusion across the blood-gas
35 zing blood-O2 affinity for O2 loading in the pulmonary circulation and O2 unloading in the systemic c
37 hanisms underlying development of the normal pulmonary circulation and the essential relationship of
38 the fetus which directs blood away from the pulmonary circulation and towards the placenta where oxy
39 aternal hyperoxygenation affects human fetal pulmonary circulation and whether there is a gestational
40 ways and lung parenchyma are supplied by the pulmonary circulation, and changes in the pulmonary circ
41 low changes on eNOS expression in the normal pulmonary circulation, and to determine whether the incr
42 lar function and, hopefully, its coupling to pulmonary circulation; and 4) searching for effective th
45 sess the osmotic effectiveness of Het in the pulmonary circulation as judged by its exclusion from lu
46 t to provide adequate blood flow through the pulmonary circulation at a normal central venous pressur
47 d in some part to lack of pulsatility in the pulmonary circulation because of altered flow characteri
48 dulating vascular tone and remodeling in the pulmonary circulation, but its role in the pathogenesis
49 n (PAH), a proliferative vasculopathy of the pulmonary circulation, but the origin of vascular injury
51 sults demonstrate that although the existing pulmonary circulation can supply the metabolic needs for
52 effects on lung hyperinflation (and possibly pulmonary circulation) can explain the effects on exacer
54 majority of which affect the right-sided or pulmonary circulation, contribute significantly to morta
55 he pulmonary circulation, and changes in the pulmonary circulation could alter airway resistance or t
56 In arterial sections from the systemic and pulmonary circulation, CREB content was high in prolifer
58 se (OR = 4.8), metastatic cancer (OR = 4.6), pulmonary circulation disorders (OR = 2.9), congestive h
59 al disorders, peripheral vascular disorders, pulmonary circulation disorders, renal failure, solid tu
60 The patients with nonpulsatile group in the pulmonary circulation dropped the PVRI from 2.18+/-0.34
61 on to allow shunting from either systemic-to-pulmonary circulation (eg, in the first day of life) or
62 lation [FMD] in the brachial artery) and the pulmonary circulation (exhaled nitric oxide [NO] product
63 ss to constrictor agents, at a time when the pulmonary circulation exhibits varying degrees of vasoco
67 o increased leukocyte recruitment within the pulmonary circulation in a mouse acute endotoxemia model
68 ecome the first-line modality for imaging of pulmonary circulation in patients suspected of having pu
72 Thus the hypoxia-induced remodeling of the pulmonary circulation is a highly complex process where
73 atients with chronic heart failure (HF), the pulmonary circulation is a major source of endothelin-1
74 pacted by living at altitude, as the passive pulmonary circulation is dependent on the resistance of
75 dvancing gestation; this suggests that fetal pulmonary circulation is under acquired vasoconstriction
76 y C fibre endings, primarily supplied by the pulmonary circulation, is transmitted to this commissura
78 or other processes affecting the developing pulmonary circulation may represent a return to an earli
79 ctively investigate the role of NOS 3 in the pulmonary circulation, mice with targeted disruption of
81 ution from venous splanchnic beds to central pulmonary circulation need to be taken into account in s
82 s an important role in the remodeling of the pulmonary circulation, notably during exposure to hypoxi
83 ich breast cancer cells are infused into the pulmonary circulation of artificially ventilated explant
84 These TE structures have functioned in the pulmonary circulation of growing lambs for up to 4 month
85 forces maintain eNOS content in the normoxic pulmonary circulation of the adult rat, and suggest that
87 flation results in structural changes in the pulmonary circulation, potentially affecting pulmonary p
88 death, hypertension, ischemic heart disease, pulmonary circulation problems, stroke, and type 2 diabe
89 ardiography with detailed examination of the pulmonary circulation, pulse oximetry, complete blood co
90 pc-PH is amenable to therapies targeting the pulmonary circulation remains to be tested by properly d
91 presystemic pump may limit flow through the pulmonary circulation, restricting ventricular filling a
92 by abnormally elevated blood pressure of the pulmonary circulation that results, over time, from exte
94 ortant modulator of tone in the hypertensive pulmonary circulation, the roles of cyclic 3'-5'-guanosi
96 set latency of the excitation was within the pulmonary circulation time, consistent with being activa
97 The typical response of the adult mammalian pulmonary circulation to a low oxygen environment is vas
98 l PVR and limits the capability of the fetal pulmonary circulation to dilate or sustain vasodilation
99 account for the unique vulnerability of the pulmonary circulation to heterozygous mutations of BMP t
100 monary blood flow alters the response of the pulmonary circulation to hypothermic CPB; the increase i
101 the important contribution of the developing pulmonary circulation to lung growth in the setting of p
102 estational age-related response in the fetal pulmonary circulation to maternal hyperoxygenation durin
104 d contrast agents can now safely transit the pulmonary circulation to provide opacification of the le
105 e redistribution of EC-SOD from the lung and pulmonary circulation to the extracellular fluids is ben
106 onal contrast-enhanced MR angiography of the pulmonary circulation was feasible at 3.0 T and provided
107 onal contrast-enhanced MR angiography of the pulmonary circulation was performed with a 3.0-T MR syst
108 On the basis of the unique properties of the pulmonary circulation, we show how all relevant physiolo
111 ve potassium channels in the fetal and adult pulmonary circulation which regulate vascular tone in re
112 ion, including hemodynamic parameters in the pulmonary circulation, which are superior in their ident
113 of delivered cells exited the heart into the pulmonary circulation, with 26+/-3% (IM), 47+/-1% (IC),
114 promising agent for molecular imaging of the pulmonary circulation, with abundant specific binding si
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