ライフサイエンスコーパス: pulmonary hemorrhage

1 osition, destruction of lung parenchyma, and pulmonary hemorrhage).

2 ratory failure caused by lung hypoplasia and pulmonary hemorrhage.

3 icities, including venoocclusive disease and pulmonary hemorrhage.

4 Only one patient had grade 3 pulmonary hemorrhage.

5 ed variables were evaluated as predictors of pulmonary hemorrhage.

6 apy-plus-bevacizumab group, including 5 from pulmonary hemorrhage.

7 e were used to develop a guinea pig model of pulmonary hemorrhage.

8 been isolated from the lung of a child with pulmonary hemorrhage.

9 There were six deaths from pulmonary hemorrhage.

10 , causing progressive glomerulonephritis and pulmonary hemorrhage.

11 severity of viral pneumonitis, and prevented pulmonary hemorrhage.

12 stinal hemorrhage, hemorrhagic cystitis, and pulmonary hemorrhage.

13 atient died shortly after BPA as a result of pulmonary hemorrhage.

14 .3 [95% CI, 0.3 to 8.3]) and a lower rate of pulmonary hemorrhage (5.6% vs 8.9%; OR, 0.60 [95% CI, 0.

15 ized woman with known SLE who presented with pulmonary hemorrhage, acute renal failure, change in men

16 newborn who developed glomerulonephritis and pulmonary hemorrhage after transplacental transfer of an

17 Pulmonary hemorrhage after TTLB is common but rarely req

18 uction in the level of Foxf1 caused neonatal pulmonary hemorrhage and abnormalities in alveologenesis

19 itive microscopic polyangiitis, resulting in pulmonary hemorrhage and acute renal failure.

20 amination of patient 1 showed focal areas of pulmonary hemorrhage and diffuse alveolar damage, spleni

21 The recent increase in idiopathic pulmonary hemorrhage and hemosiderosis in young infants,

22 ciated with human health concerns, including pulmonary hemorrhage and hemosiderosis.

23 inflammatory vasculitides, which often cause pulmonary hemorrhage and infarction, result in florid bu

24 h severe respiratory failure with associated pulmonary hemorrhage and may be a life-sustaining suppor

25 condition of infants who present with acute pulmonary hemorrhage and respiratory failure.

26 e died immediately after birth due to severe pulmonary hemorrhage and structural defects in arterial

27 ate or severe hemorrhagic cystitis, 2.8% had pulmonary hemorrhage, and 2% had intracranial hemorrhage

28 times greater in patients who presented with pulmonary hemorrhage, and 3.78 (1.22, 11.70) times great

29 nical findings included respiratory failure, pulmonary hemorrhage, and culture-negative septic shock.

30 ematurity, duration of parenteral nutrition, pulmonary hemorrhage, and white matter injury (p<0.01 fo

31 m, also induced modest (25%) to severe (25%) pulmonary hemorrhage, another important feature of anti-

32 y and immune-related disorders causing acute pulmonary hemorrhage are reviewed.

33 prognostic indicators: (1) ANCA pattern; (2) pulmonary hemorrhage at onset; (3) presence of extrarena

34 ower in-hospital mortality and likelihood of pulmonary hemorrhage but not with differences in necroti

35 y copathogens, radiographic presentation, or pulmonary hemorrhage did not alter predictive values.

36 Primary outcomes were pulmonary hemorrhage, documented hemoptysis, and bleedin

37 events occurred in 4 cases (15%), including pulmonary hemorrhage, hypotension requiring vasoactive s

38 is, has previously been shown to cause fatal pulmonary hemorrhage in guinea pigs and asymptomatic chr

39 cause acute lethal infection with prominent pulmonary hemorrhage in guinea pigs.

40 ion of important clinical concepts regarding pulmonary hemorrhage in infants and children, recent rep

41 as demonstrated an association between acute pulmonary hemorrhage in infants and exposure to Stachybo

42 oimmune process may be the etiology of fatal pulmonary hemorrhage in leptospirosis.

43 The only complication was a self-limited pulmonary hemorrhage in one patient.

44 a cause of sudden death, despite evidence of pulmonary hemorrhage in one.

45 Acute idiopathic pulmonary hemorrhage is a unique condition of infants wh

46 The hypothesis that acute idiopathic pulmonary hemorrhage is caused by toxigenic mold has not

47 ach to reducing the exposure of infants with pulmonary hemorrhage is suggested.

48 The etiology of pulmonary hemorrhage is unclear.

49 urther intraprocedure complications included pulmonary hemorrhage (n = 2), ventricular fibrillation (

50 Pulmonary hemorrhage occurred in 483 of the 1175 TTLBs (

51 Both were pulmonary hemorrhages, one occurring during treatment an

52 de therapy did not increase the incidence of pulmonary hemorrhage or other adverse events.

53 dysfunction (OR 1.89, 95% CI 1.17 to 3.03), pulmonary hemorrhage (OR 2.23, 95% CI 1.11 to 4.50), neu

54 Severe (grade >or= 3) pulmonary hemorrhage (PH) in advanced non-small-cell lun

55 nd 1 patient with cholangiocarcinoma died of pulmonary hemorrhage secondary to a cavitating pulmonary

56 ry outcomes were major neonatal morbidities (pulmonary hemorrhage, severe bronchopulmonary dysplasia,

57 use strains was associated with jaundice and pulmonary hemorrhage, similar to the patient from whom t

58 nuing sorafenib, and one patient experienced pulmonary hemorrhage that was considered drug related.

59 The rate of fatal pulmonary hemorrhage was consistent with previous bevaci

60 Pulmonary hemorrhage was graded as follows: 0, none; 1,

61 The relative risk of pulmonary hemorrhage was no different by ANCA pattern.

62 Gross pulmonary hemorrhage was noted in most Abeta-infused ani

63 Areas of pulmonary hemorrhage were observed in bleomycin-treated

64 ospira acquired from patients suffering from pulmonary hemorrhage were used to develop a guinea pig m

65 n between squamous cell histology and severe pulmonary hemorrhage, whereas for pemetrexed, superior t

66 All patients had resolution of their pulmonary hemorrhage within 24 hrs.