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1 n a child with sickle cell disease and a new pulmonary infiltrate.
2 lanoma presented with the chief complaint of pulmonary infiltrates.
3 noninfectious complications that manifest as pulmonary infiltrates.
4 and an increase in macrophage percentage in pulmonary infiltrates.
5 y-function tests or resolution or absence of pulmonary infiltrates.
6 ICU over a 3-year period, 44% (40) developed pulmonary infiltrates.
7 tors of pneumonia versus other etiologies of pulmonary infiltrates.
8 alter CTL generation or to affect Ad-induced pulmonary infiltrates.
9 but, of 6 patients at level III, 1 developed pulmonary infiltrates, 1 developed hypotension (both res
12 ing a mortality rate of 50% in subjects with pulmonary infiltrates and an overall mortality of 33.3%.
13 but reversible early graft dysfunction with pulmonary infiltrates and hypoxemia, attributed to ische
15 r infusion, they became critically ill, with pulmonary infiltrates and lung injury, renal failure, an
16 ce of severe graft dysfunction manifested as pulmonary infiltrates and severe hypoxemia with onset in
17 pulmonary tuberculosis, one having fever and pulmonary infiltrates and the other having subclinical d
18 ction in lung with LRTD symptoms without new pulmonary infiltrates), and proven (PIV detection in lun
20 rent episodes of wheezing, mucus production, pulmonary infiltrates, and elevated levels of serum IgE.
22 I, and they presented more often with shock, pulmonary infiltrates, and renal dysfunction (p < 0.0001
25 olonization, IgE and IgG anti-Af antibodies, pulmonary infiltrates, bronchiectasis, and pulmonary fib
26 dentifying pneumonia as a potential cause of pulmonary infiltrates, but for the likely etiology of th
29 Clinical presentations included asymptomatic pulmonary infiltrates, chronic cough, and shortness of b
30 primed for a strong cytolytic response and a pulmonary infiltrate consisting primarily of mononuclear
31 .v. yeast wall glucan developed consolidated pulmonary infiltrates consisting predominantly of macrop
32 of organism-mediated pulmonary injury and of pulmonary infiltrates detected by thoracic computed tomo
34 eumonia, accurately identified patients with pulmonary infiltrates for whom monotherapy with a short
35 sis in 73 nonimmunocompromised patients with pulmonary infiltrates for whom the test was ordered.
36 hest syndrome (ACS) is the presence of a new pulmonary infiltrate in combination with fever or respir
40 therapy in liver transplant recipients with pulmonary infiltrates in the intensive care unit (ICU).
41 ious pulmonary complications that present as pulmonary infiltrates include idiopathic pneumonia syndr
42 ficiency virus-specific cytotoxic T cells in pulmonary infiltrates, increased survival time, and a re
44 had evidence of mediastinitis and extensive pulmonary infiltrates late in their course of illness.
45 esent with hypoxemic respiratory failure and pulmonary infiltrates, meeting criteria for acute respir
46 -) animals had eosinophilic and neutrophilic pulmonary infiltrates not present in wild-type or IFN-ga
47 lized for CAP; 5.4% had PO-CAP, defined as a pulmonary infiltrate occurring distal to an obstructed b
48 lvement (RD, 25.5%; 95% CI, 13.9% to 37.0%), pulmonary infiltrates (odds ratio, 4.9; 95% CI, 1.5-16.2
49 I, 1.2-55.2]; p = 0.032), and more extensive pulmonary infiltrates (odds ratio, 9.7 [95% CI, 3.6-25.9
52 s syndrome presents as hypoxia and bilateral pulmonary infiltrates on chest imaging in the absence of
53 of > 0.5 while intubated, bilateral diffuse pulmonary infiltrates on chest radiograph, and exclusion
54 CI, 1.6 to 3.7), and multilobar radiographic pulmonary infiltrate (OR = 3.1; 95% CI, 1.9 to 5.1).
55 unexplained fever, weight gain, dyspnea with pulmonary infiltrates, pleuropericardial effusion, hypot
56 ack male with sickle cell disease, bilateral pulmonary infiltrates, refractory hypoxemia, and unstabl
59 hypoxemic respiratory failure with bilateral pulmonary infiltrates that are not attributed to left at
60 considered in the differential diagnosis of pulmonary infiltrates that occur acutely after bone marr
61 who developed fever, hypoxia, and bilateral pulmonary infiltrates two and a half years after orthoto
65 etection in upper respiratory tract with new pulmonary infiltrates with/without LRTD symptoms), proba
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