ライフサイエンスコーパス: pulmonary neuroendocrine cell

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1 erentiation but had no effect on ciliated or pulmonary neuroendocrine cells.

2 sal cells, and activated UCHL1, a marker for pulmonary neuroendocrine cells.

3 olar septation and altered morphology of the pulmonary neuroendocrine cells.

4 elated peptide in embryonic and adult murine pulmonary neuroendocrine cells.

5 sruption of the ASH1 gene have no detectable pulmonary neuroendocrine cells.

6 s consistent with autonomic and C fibers and pulmonary neuroendocrine cells.

7 SCLC is thought to derive from pulmonary neuroendocrine cells.

8 ved with a lower incidence of hyperplasia of pulmonary neuroendocrine cells and aggressive liver, bil

9 Pulmonary neuroendocrine cells and clusters of these cel

10 is critically required for the formation of pulmonary neuroendocrine cells and is a marker for human

11 itrosamine-treated hamsters also demonstrate pulmonary neuroendocrine cell apoptosis in situ during t

12 H1) is selectively expressed in normal fetal pulmonary neuroendocrine cells, as well as in the divers

13 Increased pulmonary neuroendocrine cells containing bombesin-like

14 Increased numbers of pulmonary neuroendocrine cells containing bombesin-like

15 rucial role in hematopoietic, inner ear, and pulmonary neuroendocrine cell development and governs ce

16 Transient pulmonary neuroendocrine cell hyperplasia and non-neuroe

17 nomatous hyperplasia, and diffuse idiopathic pulmonary neuroendocrine cell hyperplasia.

18 8(+) macrophages 4 weeks after radiation and pulmonary neuroendocrine cells hyperplasia 6 weeks after

19 Idiopathic diffuse hyperplasia of pulmonary neuroendocrine cells (IDHPNC) is a clinicopath

20 in-releasing peptide (GRP) is synthesized by pulmonary neuroendocrine cells in inflammatory lung dise

21 lial bodies but a reduced number of solitary pulmonary neuroendocrine cells in the neonatal lung.

22 tion in GFI1 display abnormal development of pulmonary neuroendocrine cells, indicating that GFI1 is

23 on factor required for proper development of pulmonary neuroendocrine cells, is essential for the sur

24 Clara cells, together with ciliated and pulmonary neuroendocrine cells, make up the epithelium o

25 Gastrin-releasing peptide (GRP), secreted by pulmonary neuroendocrine cells, mediates oxidant-induced

26 ntiation in many organs, but its function in pulmonary neuroendocrine cell (PNEC) differentiation has

27 We initiated a transgenic model for primary pulmonary neuroendocrine cell (PNEC) hyperplasia/neoplas

28 ype II cells, free alveolar macrophages, and pulmonary neuroendocrine cells (PNEC).

29 r the distribution and abundance of solitary pulmonary neuroendocrine cells (PNECs) and neuroepitheli

30 Pulmonary neuroendocrine cells (PNECs) are proposed to b

31 Pulmonary neuroendocrine cells (PNECs) are the only inne

32 about receptor (Robo) genes are expressed in pulmonary neuroendocrine cells (PNECs), a rare, innervat

33 y reflexes - carotid body glomus cells, and 'pulmonary neuroendocrine cells' (PNECs) - are obscure.

34 ion requires augmented neural stimulation of pulmonary neuroendocrine cell secretion.

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