ライフサイエンスコーパス: pulmonary regurgitation

1 stent (BMS), although this treatment causes pulmonary regurgitation.

2 etralogy of Fallot) underwent PVR for severe pulmonary regurgitation.

3 ment, and all but 1 patient had mild or less pulmonary regurgitation.

4 er methods cannot reliably be used to assess pulmonary regurgitation.

5 n the management of conduit obstructions and pulmonary regurgitation.

6 te relief of the obstruction and significant pulmonary regurgitation.

7 he pulmonary valve in 6 young pigs to induce pulmonary regurgitation.

8 No patient had more than mild pulmonary regurgitation.

9 the late deleterious consequences of chronic pulmonary regurgitation.

10 ventricular to pulmonary artery stenosis or pulmonary regurgitation.

11 However, BMS resulted in free pulmonary regurgitation (21.3+/-10.7% versus 41.4+/-7.5%

12 n repaired tetralogy of Fallot (TOF) reduces pulmonary regurgitation and decreases right ventricular

13 y valve placement is an emerging therapy for pulmonary regurgitation and right ventricular outflow tr

14 Pulmonary regurgitation and RV volumes were assessed by

15 cy and childhood leads to reintervention for pulmonary regurgitation and stenosis in later life.

16 childhood had more than or equal to moderate pulmonary regurgitation, and fulfilled defined criteria

17 nt in MRI-defined ventricular parameters and pulmonary regurgitation, and results in subjective and o

18 ventricular systolic function and/or severe pulmonary regurgitation are at increased risk for advers

19 Nonsurvivors were more likely to have pulmonary regurgitation at any gestational age (61% vers

20 t of sustained ventricular tachycardia, with pulmonary regurgitation being the predominant haemodynam

21 er repair because it is associated with less pulmonary regurgitation, better exercise tolerance, and

22 Recent evidence suggests that pulmonary regurgitation causes significant morbidity, pr

23 ng of the RV to PVR in patients with chronic pulmonary regurgitation did not result in a measurable e

24 1% with data); no patient had more than mild pulmonary regurgitation early after implantation or duri

25 icuspid regurgitation (TR) and end-diastolic pulmonary regurgitation (EDPR) gradients in outpatients

26 Both HRV and BRS decreased with pulmonary regurgitation, elevated right ventricular end

27 n the past, residual defects such as chronic pulmonary regurgitation following repair of tetralogy of

28 ent of the tricuspid regurgitation gradient, pulmonary regurgitation gradient, pulmonary artery strok

29 inal synchrony improved significantly in the pulmonary regurgitation group (maximum wall delay P=0.03

30 MRI demonstrates the progression of pulmonary regurgitation in growing swine.

31 Fetuses with pulmonary regurgitation, indicating circular shunt physi

32 mm Hg on echocardiography without important pulmonary regurgitation (less than mild or regurgitant f

33 tients (n=31) and subgroups with predominant pulmonary regurgitation (n=13) or stenosis (n=18).

34 er surgical RV revalvulation for significant pulmonary regurgitation (n=21).

35 95% confidence interval, 1.1-1.5; P<0.001), pulmonary regurgitation (odds ratio, 2.9; 95% confidence

36 can treat this condition without consequent pulmonary regurgitation or cardiopulmonary bypass.

37 ventricular systolic function and/or severe pulmonary regurgitation (OR, 10.3) in a previously propo

38 ventricular ejection fraction and/or severe pulmonary regurgitation (OR, 9.0) and smoking history (O

39 ndocarditis (n=3, 2 with stenosis and 1 with pulmonary regurgitation), or right ventricular dysfuncti

40 he determinants of improvement after PVR for pulmonary regurgitation over a wide range of patient age

41 e whole cohort (P<0.001) and both subgroups (pulmonary regurgitation P=0.01; pulmonary stenosis P=0.0

42 RV) volume overload in patients with chronic pulmonary regurgitation, persistent RV dysfunction and s

43 ent (33+/-24.6 to 19.5+/-15.3, P<0.001), and pulmonary regurgitation (PR) (grade 2 of greater before,

44 t ventricular (RV) function in patients with pulmonary regurgitation (PR) after tetralogy repair rema

45 paired tetralogy of Fallot are monitored for pulmonary regurgitation (PR) and right ventricular (RV)

46 Repair of TOF often results in chronic pulmonary regurgitation (PR) and right ventricular (RV)

47 to examine the prevalence and predictors of pulmonary regurgitation (PR) following balloon dilation

48 diac flow measurement method for quantifying pulmonary regurgitation (PR) in an in vitro and a chroni

49 Pulmonary regurgitation (PR) is an important determinant

50 to quantify biventricular size and function, pulmonary regurgitation (PR), and myocardial viability.

51 digital color Doppler method for quantifying pulmonary regurgitation (PR), using an animal model of c

52 tricular outflow tract (RVOT) obstruction or pulmonary regurgitation (PR).

53 , and 94% of patients had moderate or severe pulmonary regurgitation (PR).

54 mic right ventricle, pulmonary hypertension, pulmonary regurgitation, pulmonary atrioventricular valv

55 similar TOF group, late PVR for symptomatic pulmonary regurgitation/RV dilation did not reduce the i

56 increased for the whole cohort (P=0.02) and pulmonary regurgitation subgroup (P=0.05); circumferenti

57 Pulmonary regurgitation was </=2+ in 97% of patients.

58 Pulmonary regurgitation was graded as mild (RF<20%), mod

59 Before implantation, pulmonary regurgitation was moderate or severe in 92 pat

60 Pulmonary regurgitation was the main underlying haemodyn

61 At least moderate neoaortic and pulmonary regurgitation were present in 3.4% and 6.6%, r

62 aying surgical replacement, but it creates a pulmonary regurgitation when crossing the valve.

63 with repaired tetralogy of Fallot often have pulmonary regurgitation, which is frequently overlooked

64 Patients with pulmonary regurgitation with or without stenosis after r

65 position in patients with moderate to severe pulmonary regurgitation with or without stenosis.