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1 SVR, although only 3 patients had increased pulmonary resistance.
2 al pressure, pulmonary arterial pressure, or pulmonary resistance.
3 the PDA was too small or because of elevated pulmonary resistance.
4 allergen, but significantly altered baseline pulmonary resistance.
5 sentan caused a dose-dependent fall in total pulmonary resistance (-20.0+/-11.0%, P=0.01) and mean pu
6 P < .05) that was due to an increased total pulmonary resistance (62 +/- 6 versus 33 +/- 2 mm Hg.min
7 y pressure (-4.1 mm Hg; P = 0.02), and total pulmonary resistance (-93.0 dynes x seconds/cm(5) ; P =
8 rk, we studied the roles of CG and NE in the pulmonary resistance against Mycobacterium bovis bacillu
10 e associated with virus-induced increases in pulmonary resistance and hyperresponsiveness to methacho
11 n-tbm (brevetoxin-2 minus the side chain) on pulmonary resistance and tracheal mucus velocity, a mark
13 y artery pressure by 12.6 +/- 3.7% and total pulmonary resistance by 13.1 +/- 6.4%, with no significa
14 here was a trend toward improvement in total pulmonary resistance during the 3-day delivery period.
18 up) correlated with both postoperative total pulmonary resistance index (R2=0.79, P<0.001) and postop
19 dex during exercise with a decrease in total pulmonary resistance index and an increase in stroke vol
20 (P<0.0001-interaction P=0.1), whereas total pulmonary resistance index was reduced at rest and reduc
21 n healthy conscious adults, (1) normal basal pulmonary resistance is maintained in part by continuous
22 sis (p = 0.01), and lacking abnormalities in pulmonary resistance (p = 0.028) and dynamic compliance
23 ized and instrumented for the measurement of pulmonary resistance (R(L)), dynamic compliance (Cdyn),
24 vasive technique also demonstrated increased pulmonary resistance responses to aerosolized methacholi
26 ffects of substance P on RAR activity, Cdyn, pulmonary resistance (RL), and arterial blood pressure w
27 t an increase in lung volume decreased lower pulmonary resistance (Rlp); however, within 30 min of sl
29 ng the ewe with 100% O2, causing fetal total pulmonary resistance to decrease from 1.18 +/- 0.14 to 0
30 erentially dilating systemic capacitance and pulmonary resistance vessels although only marginally di
31 n, (2) intrinsic hypoxic vasoconstriction of pulmonary resistance vessels, and (3) potential local an
32 mass, increased smooth muscle medial area in pulmonary resistance vessels, and significantly higher r
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