ライフサイエンスコーパス: pulmonary tumor

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1 lls also inhibits local growth of metastatic pulmonary tumor.

2 eatment of s.c. tumors as well as metastatic pulmonary tumors.

3 ffects were observed in established s.c. and pulmonary tumors.

4 gression that enhanced survival in mice with pulmonary tumors.

5 onged survival times in mice with metastatic pulmonary tumors.

6 of peptide hormones in the growth biology of pulmonary tumors.

7 as a methodology for the detection of small pulmonary tumors.

8 y for the detection and serial monitoring of pulmonary tumors.

9 of the lung is a subtype of highly invasive pulmonary tumors and is associated with decreased or abs

10 However, detection of mouse pulmonary tumors and their subsequent response to therap

11 used to detect submillimeter CEA-expressing pulmonary tumors before they become visible to the naked

12 tion algorithms described by our laboratory, pulmonary tumor burden can be quantitatively measured in

13 Pulmonary tumor burden was significantly (P < .01) lower

14 d die at 4 months as a result of progressive pulmonary tumor burden.

15 CD4+ cells were prevented from infiltrating pulmonary tumors by pretreatment with pertussis toxin.

16 that the appearance and regression of these pulmonary tumors can be readily monitored in anesthetize

17 s virus type 11 (HPV-11) genome, whereas the pulmonary tumor cells contained a 10.4-kb genome.

18 Submillimeter pulmonary tumor colonies could be visualized with both s

19 nide resulted in more than 80% inhibition of pulmonary tumor formation compared to the aerosol contro

20 ical abnormalities found in the proximity of pulmonary tumors from a series of 59 patients.

21 Pulmonary tumors from Arf-knockout mice exhibited increa

22 re hyperinvasive to EGF and showed increased pulmonary tumor growth upon tail vein injection.

23 rchitecture, or primary (s.c.) or secondary (pulmonary) tumor growth.

24 , but not with oncogenic EGFR(L858R), caused pulmonary tumors in transgenic mice that were phenotypic

25 The pathogenesis of pulmonary tumors induced by a tobacco carcinogen, 4-(met

26 COOH-terminal ADAMTS-1 fragment can inhibit pulmonary tumor metastasis.

27 enic organizing pneumonia ([COP] n = 4); and pulmonary tumor (n = 4).

28 Because pulmonary tumor necrosis factor-alpha expression is know

29 oarrestin in HT1080 tumor cells and measured pulmonary tumor nodule formation in nude mice.

30 2- and IL-18-cultured TDLN cells infiltrated pulmonary tumor nodules and eradicated established tumor

31 (n = 5) within the lungs with BLI-detectable pulmonary tumor nodules as compared with controls (n = 4

32 xpressing exogenous CYLD were unable to form pulmonary tumor nodules following tail-vein injection.

33 y invasive melanoma cell line, B16F10, forms pulmonary tumor nodules in normal C57BL6 mice.

34 ith influenza virus, increased the number of pulmonary tumor nodules.

35 , ROC area = 0.75; CT, ROC area = 0.75), and pulmonary tumor (radiography, ROC area = 0.73; CT, ROC a

36 orthotopic RENCA cell renal transplantation, pulmonary tumor spread was inhibited by pharmacologic bl

37 alectomy significantly reduced the extent of pulmonary tumor spread, whereas aldosterone infusion rec

38 infiltrated and proliferated extensively in pulmonary tumors, while also stimulating tumor antigen-s

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