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1 e was mitral (44%), aortic (48%), both (2%), pulmonic (0.4%), or transcatheter aortic valve replaceme
2 neous therapies for the treatment of aortic, pulmonic, and mitral valve disease.
3 vere cardiovascular abnormalities, including pulmonic arterial stenosis and ventricular septal defect
4 , including variants with pulmonic stenosis, pulmonic atresia and absent pulmonary valve.
5  20%), Ebstein's anomaly (4 patients, 6.5%), pulmonic atresia or stenosis (7 patients, 11.5%), truncu
6  the cardiovascular system into systemic and pulmonic circulations is a complex morphogenetic process
7 the electrocardiographic wave complex to the pulmonic component of the second heart sound.
8                                              Pulmonic expression of IFN-gamma, IL-12, TNF-alpha, or N
9 on of persistent tuberculosis, modulates the pulmonic expression of specific immunologic factors, and
10 ow probes were used for obtaining aortic and pulmonic flows.
11                                  Analysis of pulmonic gene and protein expression of mice in the low-
12 fferences in the cellular composition of the pulmonic granuloma of the CXCR3(-/-) and wild-type mice
13 increased tissue bacillary burden and severe pulmonic histopathological deterioration that was associ
14 to become a compact ring at the level of the pulmonic infundibulum over several days of development (
15 further investigated and that atrial volume, pulmonic outflow and interventricular septal motion may
16 s were similar for tricuspid (P = 0.002) and pulmonic (P = 0.012) regurgitation.
17 or stenotic lesions with implantation in the pulmonic position in 2000, treatment for valvular heart
18 n, severe pulmonary hypertension, and severe pulmonic regurgitation with RV dysfunction.
19 ressive RV dilation in a patient with severe pulmonic regurgitation.
20 nction, and 13 had severe RV dilation due to pulmonic regurgitation.
21 ortic dextroposition and isolated peripheral pulmonic stenosis (PPS).
22 ort stature and heart disease (most commonly pulmonic stenosis and hypertrophic cardiomyopathy).
23  seen in patients with AGS (e.g., peripheral pulmonic stenosis and tetralogy of Fallot) in the absenc
24 ation is the treatment of choice in critical pulmonic stenosis but remains somewhat controversial for
25  septal defect, patent ductus arteriosus, or pulmonic stenosis have had infective endocarditis after
26 were pioneered with balloon valvuloplasty of pulmonic stenosis in infants.
27    Genotype-phenotype analysis revealed that pulmonic stenosis was more prevalent among the group of
28 lastic left heart syndrome, aortic stenosis, pulmonic stenosis, and tetralogy of Fallot.
29                      Some patients also have pulmonic stenosis, persistence of a left-sided superior
30 oF were represented, including variants with pulmonic stenosis, pulmonic atresia and absent pulmonary
31 res and cardiac abnormalities, with frequent pulmonic stenosis.
32 ome cases of isolated tetralogy of Fallot or pulmonic stenosis.
33 ghtward displacement of the aortic valve and pulmonic stenosis.
34 e premature, 3 had neonatal lupus, and 1 had pulmonic stenosis.
35 ptal defects, and valvular defects including pulmonic stenosis.
36 fects, atrioventricular cushion defects, and pulmonic stenosis.
37 r outflow tract (RVOT), presumably below the pulmonic valve (PV).
38 lities clearly characterized transaortic and pulmonic valve and pulmonary vein blood flow, including
39             In vitro experiments using human pulmonic valve endothelial cells demonstrate that miR-21
40 om the deformed aortic valve and the healthy pulmonic valve revealed the presence of B. clarridgeiae
41 ry to the right ventricular outflow tract or pulmonic valve.
42  perivalvular tricuspid valves (5 patients), pulmonic valves (6 patients), or both valves (10 patient
43  (VSD), abnormal formation of the aortic and pulmonic valves, leading to valvular stenosis, and abnor
44 diogram abnormalities, Ocular hypertelorism, Pulmonic valvular stenosis, Abnormalities of genitalia,

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